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PMHx: Left-Sided Heart Failure (HF)
timing
dyspnea→progress slowly/rapidly
provoking factors
exertion
lying flat
pallitative factors
rest
sitting up
may become persistent
associated sxs
often cough
orthorpnea
paroxysmal nocturnal dyspnea
sometimes wheezing
PMH/SH/FH
PMHx heart dx/predisposing RFs
PMHx: Spontaneous Pneumothorax
timing
dyspnea→sudden onset
provoking factors
none
pallitative factors
none
associated sxs
pleuritic chest pain
cough
PMHx/SHx/FHx
often:
previously healthy young adult
adult+emphysema
smoking hx
PMHx: Acute Pulmonary Embolism (PE)
timing
dyspnea+tachypnea→sudden onset
provoking factors
exertion
pallitative factors
rest
(although) may become persistent
associated sxs
(maybe) none
pleuritic chest pain
cough
syncope
hemoptysis
DVT→UL leg pain
PMHx/SHx/FHx
postpartum
recent surgery
prolonged bed rest
hip fracture
DVT
hypercoagulable states
PMHx: Chronic Bronchitis
timing
chronic productive cough→slowly preogrssive dyspnea
provoking factors
exertion
inhaled irritants
respiratory infxns
pallitative factors
expectoration
rest
dypnea may be persistent
associated sxs
chronic productive cough
recurrent respiratory infxns
wheezing may develop
clubbing
PMHx/SHx/FHx
smoking hx
air pollutants
recurrent respiratory infxns
PMHx: COPD
timing
slowly progressive dyspnea→relatively mild cough later
provoking factors
exertion
pallitative factors
rest
dyspnea maybe persistent
associated sxs
cough+scant mucoid production
PMHx/SHx/FHx
smoking hx
air pollutants
maybe familial
PMHx: Asthma
timing
acute episodes
separated by sx-free periods
nocturnal episodes→common
provoking factors
variable
pallitative factors
separation from aggravating factors
associated sxs
wheezing
cough
tightness
PMHx/SHx/FHx
environmental conditions
may be FHx
PMHx: Diffuse Interstitial Lung Disease
timing
dyspnea→progressive
varies in its rate of development with the cause
provoking factors
exertion
pallitative factors
rest
dyspnea may become persistent
associated sxs
often:
weakness
fatigue
cough
less common than other lung dxs
PMHx/SHx/FHx
varied
exposure to trigger substance
PMHx: Pneumonia (PNA)
timing
acute illness
timing→varies with the causative agent
provoking factors
exertion
smoking
pallitative factors
rest
dyspnea may become persistent
associated sxs
pleuritic chest pain
cough
sputum
fever
PMHx/SHx/FHx
varied
PMHx: Pulmonary Tuberculosis (TB)
timing
variable
latent+active dx
provoking factors
exertion
pallitative factors
rest
dyspnea may persist
associated sxs
early→no sxs
later:
anorexia
weight loss
fatigue
night sweats
fever
cough
purulent sputum
blood-tinged
PMHx/SHx/FHx
high-risk pts
immunocompromised
PE: Normal
PP
tracheobronchial tree+alveoli→open
pleurae→thin+close together
mobility of the chest wall→unimpaired
percussion→resonant
trachea→midline
breath sounds→vesicular
added sounds
none
tactile fremitus+voice sounds
“normal”
PE: Left-Sided Heart Failure (HF)
PP
increased pressure in pulmonary Vs→congestion+interstitial edema
-around the alveoli
bronchial mucosa may become edematous
percussion→resonant
trachea→midline
breath sounds→vesicular
added sounds
late inspiratory crackles→in dependent portions of the lungs
possibly wheezes
tactile fremitus+voice sounds
“normal”
PE: Chronic Bronchitis
PP
bronchi chronically inflamed+productive cough
airway obstruction may develop
percussion→resonant
trachea→midline
breath sounds→vesicular
added sounds
none
inspiration→scattered coarse crackles
possible wheezes
tactile fremitus+voice sounds
“normal”
PE: Lobar Pneumonia (PNA)
PP
alveoli filled with infxd/inflamed fluid
percussion→dull over area
trachea→midline
breath sounds→bronchial over area
added sounds
late inspiratory crackles over area
tactile fremitus+voice sounds
increased
egophany+bronchophony+whispered pectoriloquy→positive
PE: Atelectasis
PP
mucus plug obstructs bronchial air flow→affected alveoli colapse→airless
percussion→dull over area
trachea→may be shifted to involved side
breath sounds→bronchial plug persists→usually absent
added sounds
none
tactile fremitus+voice sounds
bronchial plug persists→usually absent
PE: Pleural Effusion
PP
fluid accumulates in pleural space→separates air-filled lung from chest wall→blocks the transmission of breath sounds
percussion→dull
trachea→shifted towards unaffected side
breath sounds
decreased
absent
bronchial
added sounds
none
crackles
pleural rub
tactile fremitus+voice sounds
decreased→absent
PE: Pneumothorax (PTX)
PP
percussion→hyperresonant
trachea→tension PTX→shifted towwards unaffected side
breath sounds
decreased
over pleural air→aobsent
added sounds
none
possibel pleural rub
tactile fremitus+voice sounds
decreased
PE: COPD
PP
distal air spaces enlarge→lungs become hyperinflated
-slowly progressive dx
-chronic bronchitis may precede/follwo COPD development
percussion→diffusely hyperresonant
trachea→midline
breath sounds
decreased
absent
delayed expiration
added sounds
none
crackles
wheeze
rhonchi
tactile fremitus+voice sounds
decreased
Asthma
PP
during attacks: bronchial hyperresponsiveness+underlying inflammation→airflow decreases+lungs hyperinflate→reversible airflow obstruction
widespread
percussion→resonant→diffusely hyperresonant
trachea→midline
breath sounds
vesicular
obscured
added sounds
wheezes
possible crackles
tactile fremitus+voice sounds
decreased