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Right Hemisphere Dysfuncation
Damage tends to be focal, eg from a stroke
Traumatic Brain Injury (TBI)
damges tends to be diffuse
Right Hemisphere Dysfunction
Result from neurological damage to the right cerebral hemisphere, typically caused by a stoke
Symptoms are quite different to what is typically seen in a left hemisphere stroke. Why?
Right hemisphere stroke can still cause dysarthria due to damage to the motor cortex
Right hemisphere stroke are slightly less common than left hemisphere stokes
Right hemisphere typically processes non-linear spatial and visual information
Left Neglect
impairment of response to stimuli on the patient left
May co exist w/ visual field cut
Think of at least two safety implications of this
Perceptual Impairments
Anosgnosia: denial of illness (poor insight)
More difficulty with spatial than temporal orientation
Recognition and expression of emotion
Typical presentation is a flat affect
Can fail to appreciate change in emotional tone
Attentional Impairments
Difficulty focusing, maintaining attention, and shifting attention
Difficulty determining what is and what is not important
Communication Symptoms:Diminished speech prosody
Slower than normal speech rate, robot-like quality
Reduced emphatic stress in phrases and sentences
Communication Symptoms: Anomalous content and organization of connected speech
Speech is often excessive, confabulatory, and inappropriate
Narratives are fragmented, lack cohesion, don’t seem to have a point, focus on incidental details, fail to link events, insert tangential comments, and permit personal experiences and opinions to intrude
Communication Symptoms:Impaired comprehension of narratives
Difficulty with implied meanings, often concrete interpretations only
Interpret metaphors and idioms literally
Communication Symptoms:Pragmatic impairments
Begin and end conversations abruptly
Talk excessively and without regard for their listener
TBI
neurological damage to the brain resulting from the impact of external forces
Can be caused by direct or indirect trauma, injury may be penetrating or non-penetrating (closed head), damage is typically diffuse
TBI: Primary Injury
skull fracture
contusions/hemorrhage
concussions
lacerations
diffuse axonal injury
TBI: Secondary Injury
Ischemia
Hypoxia
Hypo/Hypertension
Cerebral Edema
Elevated Intracranial Pressure
Hypercapnia
Meningities
Seizures
Neuro-Cognitive Consequences of TBI
variety of behavioral, physical, sensory, language, and swallowing deficits
Orientation
person, place, time, purpose, confusion
Memory
Short-term/working memory, post-traumatic amnesia
TBI Treatment: External aids
computer, smartphones, calendars, timer, checklist
TBI Treatment: Internal Aids
mnemonics, visual imagery, encoding, association, chucking
Best Prognostic Indicator
rapidity of emergence from post-traumatic amnesia: this indicates the severity of the ongoing injury
Rancho Level: Early
I-II
Rancho Level: Middle
IV-VI
Rancho Level: Late
VII-X
GOAT
Orientation
SCATBI
Standardized Test
Compensatory Treatment
Accommodations and/or Modifications may be needed
Restoration Treatment
Focuses on the function to achieve the highest level of independent functioning