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Cerebral Palsy
complex, heterogenous condition that primarily impacts motor function
can occur at different times during development
prenatal (before birth)
perinatal (around birth)
postnatal
brain-based, non progressive, permanent condition
variability in severity
Features of CP
motor impairment with accompanying disorders of sensory function, cognition, speech, and sometimes seizures
disorder in sensorimotor development that is manifested by
abnormal muscle tone
stereotypical patterns of movement
Causes of CP
the primary cause is NOT BIRTH COMPLICATIONS
most are congenital
risk factors
low birth weight
premature birth
postpartum impact of prematurity/low birth weight
maternal infection or infant infection
fetal stroke
gene mutations
TBI
Hypertonicity
muscles are more resistive to sudden, passive movement
80% of CP cases have this
Contractures
permanent shortening of a muscle or joint and deformities
Spastic Hemiplegia
involves one entire side of the body, including the head, neck, and trunk
Spastic Diplegia
involves both lower extremities, with mild incoordination, tremors, or less severe spasticity in the upper extremities
Spastic quadriplegia
impacts all limbs symmetricaly
typically flexor pattern in UE’s and extensor pattern in LE’s
still may be greater in one area vs. another
Athetosis
slow, writhing, involuntary movements of the face and extremities or the proximal parts of the limbs and trunk
increases with emotional tension and are not present during sleep
Ataxia
unsteadiness and difficulties with balance, particularly with ambulating
walk faster to compensate for lack of stability and control
controlled movements clumsy
Hypotonicity
low tone, floppy
Mixed
both low and high tone
Diagnosis
no definitive test
usually occurs when motor milestones are not met in infancy and preschool years
rarely diagnosed at birth-usually takes time to notice symptoms
Differential diagnosis
types of movement patterns observed in child
atypical or stereotypical movements
rules out other diagnoses such as MD
genetic testing
MRI, CT: evidence of lesions in the brain
Course and Prognosis
varies depending on type, severity, and presence of associated problems
secondary problems occur with severe form
contractures
musculoskeletal deformities
arthritis
survival is deemed to be good, but lower than general population
adults with CP are more likely to die from respiratory complications
Motor symptoms
reflex abnormalities
hyperreflexia: overactive reflex response to stimulus
clonus: involuntary rhythmic muscle contractions
enhanced stretch reflex
overflow: extraneous movements
Delayed motor development
failure to achieve motor milestones
Atypical Motor performance
asymmetrical hand use, unusual crawling or gait, uncoordinated reach, tremors, rigid muscles, oral-motor difficulties
Medical/Surgical Management
reduce spasticity
botox
baclofen
intrathecal baclofen pump
surgical tendon lengthening and tendon transfers
prevent contractures/increase ROM
splinting
orthotics
positioning
neurology to address seizures
ophthalmologist to address visual challenges
ENT & audiology: hearing