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hallucinogens
marijuana, LSD, psilocybin, mescalin, DMT
alter sensory perception, induce visual and auditory hallucinations as they separate user from reality and disrupt thought processes
stimulants
nicotine, caffeine, cocaine, amphetamines, methamphetamines, MDMA
make people feel more alert and energetic by boosting CNS activity
depressants
alcohol, barbiturates, opiates, benzodiazepines
dampen activity in CNS
agonists
drug attaches to a receptor and produces neural actions that mimic or enhance those of a neurotransmitter
antagonists
block action of neurotransmitter
re-uptake enhancers/inhibitors
alters neural transmission by enhancing or inhibiting the reuptake of neurotransmitters in the synapse
binge drinking
5+ drinks on one occasion
heavy drinking
5+ occasions of binge drinking in the past month
blood alcohol level
amount of alcohol in the blood
death occurs at 0.35% or more
moderate alcohol use
no more than 2 drinks a day for men and 1 a day for women
physical effects of excessive alcohol use
brain shrinkage, inhibition of neurogenesis, effects on hippocampus, weakens immune system, disrupts growth hormone, promotion of fat deposits on heart muscle, promotes formation of ulcers and liver disease
psychological effects of excessive alcohol use
behavioral disinhibition, alcohol myopia, conflicts in interpersonal relationships, challenges in school and work, heightened pain and frustration sensitivity
vapes
contain nicotine, carcinogens, and toxic metal nanoparticles released from heating
harder to gauge dosage
not enough scientific research about consequences
tobacco use and abuse
single most preventable cause of illness, disability, and premature death
responsible for 1 in 5 US deaths
reduced life expectancy
increased heart rate, bp, bronchial congestion, respiratory infections
initiation smoking
starting or experimenting with tobacco use
maintenance of smoking
maintaining use, nicotine-titration model, affect management model
cessation smoking
stopping tobacco use
relapse smoking
restarting tobacco use after cessation
marijuana
can help relieve negative symptoms of aids, cancer, epilepsy, multiple sclerosis, autoimmune diseases, inflammation, and pain
health consequences: increased risk for lung disease, heart attacks, accidents
illicit drugs
opioids=depressants
highly addictive
increased availability=increased use and overdose
dependance
a state in which the drug is required for a person to function normally
tolerance
symptom of dependence
progressively decreasing responsiveness to a frequently used drug
neuroadaptation
withdrawal
unpleasant physical and psychological symptoms that occur when a person abruptly ceases using certain drugs
biomedical addiction model
addiction as a disease
assumes a person inherits a biological vulnerability to physical dependance
genetic liability
evidence in twin and family studies, associations between neurotransmitter deficiencies and well being
reward addiction model
addiction as pleasure-seeking
dopamine hit
overstimulation of reward circuit, pleasure pathway nerves causes pleasurable “high” that leads to continuing drug usage
evidence: some drugs increase dopamine
social learning addiction model
addiction as a learned behavior
shaped by conditioning, learning, sociocultural, cognitive factors, motivations, intentions, attitudes, family, peers, media, neighborhoods, etc.
gateway drugs
people start with mild and easier substances and progress to more dangerous drugs in pursuit of more dopaminergic and pleasurable effects
prevention
preventing abuse from happening before it occurs (DARE)
treatment
stopping abuse after it has begun (AA)
harm reduction approaches
reduce certain health and safety issues associated with substance use (syringe services, naloxone distribution programs)
biological processes stress
physiological systems react
nervous system
psychological influences stress
how we appraise or think about events impact how stressed we feel, as well as how our health is impacted
personality traits
sociocultural factors stress
impact the types of stressor we are exposed to, how we appraise them, and ultimately how they affect us
social support
significant life event
big life disrupting events that cause great amounts of stress
Holes and Rah Life Change Units and the Social Readjustment Rating Scale
first systematic attempt to link stress to health
Daily Hassles
Most significant source of stress
persistent ones are a stronger predictor of health problems that either significant life events or the frequency of daily uplifts
features of stressors
controllability, predictability, persistence
features of stress appraisals
challenge versus threat appraisal
resources meet or fail to meet demands
adverse childhood experiences
exposure to early life stress and childhood trauma can have adverse consequences on health decades later
individuals with higher were at higher risk for substance use, cancer, heart disease, respiratory disease, poor mental health, and interpersonal and self-directed violence
stress of emerging adulthood
increase at this stage
frequently: move, change jobs, lack of career progress, move back home, living with romantic partner
body response to stressors
increased heart rate, dilated pupils, stress hormones released, digestion slows, increased blood flow to the muscles, stored energy to a form that is directly unusable by muscles
sympatho-adreno-medullary (SAM) axis
fast-acting endocrine response
sympathetic NS activates adrenal stress hormones
fight or flight
hypothalamic-pituitary-adrenocortical (HPA) axis
slower acting endocrine response
body’s delayed response to stress, involving the secretion of corticosteroid hormones from adrenal cortex
returns to homeostasis
what can short term stress trigger in immune function
up regulation
what can long term stress trigger in immune function
suppressing immunity and increasing inflammation
reduced immune function follows
divorce, bereavement, unemployment, and exam periods
how does stress make you sick
more infections from flue, chickenpox, mono, and other viruses
autoimmune disorders
coronary artery disease
delayed wound healing
direct effect hypothesis stress to disease
immunosuppressant part of the body’s natural response to stress
HPA and SAM axes activate release of stress hormones
stress hormones bind to receptors on T and B cells and lymphocytes
indirect effect hypothesis stress to disease
stress may encourage maladaptive behaviors that disrupt immune functioning
substance use, poor sleep, exercise, and nutrition
allostatic load
cumulative long term effects of the body’s physiological response to stress
decreased immunity and hippocampal size and function, increased abdominal fat, overproduction of pro-inflammatory cytokines
sex
assigned at birth, chromosomes,
intersex
XXY, XYY
gender
social and cultural construct
gender roles
traits, behaviors, and responsibilities shaped by sociocultural expectations based on an individuals gender Rosenthal
identity
how in your head experience and define your gender, based on how much you align with what you understand the options for gender to be
attraction
how you find yourself feeling drawn to some other people in sexual or romantic ways
expression
how you present gender
sex (genderbread)
physical traits you’re born with or develop that we think of as sex characteristics as well as that assigned of birth
social role theory
suggests that the division of labor creates norms about roles women and men fulfill - Eagly
gender based stigma’s influence
health disparities
caregiving
women primarily fulfil, men seek outside help
men 7x more likely to divorce wives following cancer diagnosis than women
consequences of female gender role for womens health
breast cancer → lack of femineity
young women who highly endorse traditional conceptions of femininity → lower conception and condom use + lower self esteem and greater depressive symptoms
consequence of male gender role for mens health
greater value on dominant male gender role → less wiling to seek counseling
undergrad men who feel masculinity threatened → higher rates of cardiovascular and metabolic disease
prostate cancer → threat to masculinity
women and sexism
greater adverse mental health outcomes and negative health habits
prejudice in health care → less likely to go to cancer screening and use health care when sick
women exposed to hostile sexism in lab setting → poor physiological reactions
minority stress model
stigmatized identities experience health disparities due to additive general life stressors and can occur at individual, interpersonal, and structural level
intersection in race/ethnicity and sexual minorities - sleep
black and Latino sexual minority men → shorter sleep