student made questions wednesday section

Which event occurs during the cellular phase rather than the vascular phase?

A. Initial vasoconstriction of blood vessels

B. Increased vessel permeability leading to fluid leakage

C. Neutrophils migrating out of the vessel and toward the injury site

D. Vasodilation causing redness and warmth

C. Neutrophils migrating out of the vessel and toward the injury site

Which of the following is incorrect in regards to vascular permeability?

A. Caused by release of histamine and bradykinin 

B. Results in fluid extravasation

C. Aids in the transport of WBCs to the injury site 

D. Causes vasoconstriction to reduce blood flow to the injured area

D. Causes vasoconstriction to reduce blood flow to the injured area

Chemotaxis is an important part of the initial white blood cell (WBC) response to inflammation. Which of the following best defines chemotaxis?

A. When WBCs attach to or move along the endothelium.

B. When the number of WBCs increase

C. When WBCs are attracted to chemical signals released at the site of injury.

 D. When WBCs release inflammatory mediators such as histamine

C. When WBCs are attracted to chemical signals released at the site of injury.

Which pro-inflammatory mediator is associated with cachexia?

A. Leukotrienes

B. Histamine

C. TNF Alpha

D. Interleukins 

E. Prostaglandins 

C. TNF Alpha

Which of the following proteins is associated with the acute phase of inflammation?

A. Collagen 

B. C-reactive protein (CRP)

C. Leukocytes 

D. Cytokines

B. CRP

A patient undergoes a clean surgical incision that is closed with sutures. Approximately 5 days after the procedure, granulation tissue fills the incision space and there is progressive new blood vessel growth. Is this primary, secondary, or tertiary intention?

A) Primary

B) Secondary

C) Tertiary

D) None of the above

A. Primary Intention

Which of the following isn’t a systemic response due to inflammation?

A. Weight loss

B. Fever 

C. Anemia

D. Depression

D. Depression

How do NSAIDs interact with prostaglandins to relieve the side effects of inflammation?

A. They block pain receptors and cause prostaglandins to degenerate within the cell

B. They block the formation of prostaglandins via the cyclooxygenase enzyme pathways

C. They only stimulate the Cox-1 pathway 

D. They only block the Cox-2 pathway 

B. They block the formation of prostaglandins via the cyclooxygenase enzyme pathways