Edema, lung volume and lymphedema

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46 Terms

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edema pitting scale 1+

indentation is barely detectable

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edema pitting scale 2+

slight indentation visible when skin is depressed, returns to normal in 15s

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edema pitting scale 3+

deeper indentation occurs when pressed and returns to normal within 30s

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edema pitting scale 4+

indentation lasts for more than 30s

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Diaphragm resting position with SCI

lower resting position → decreased inspiratory reserve

  • when upright → lower → decreased IR

  • when supine → better position of diaphragm


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What may be beneficial for abdominal support for a SCI pt who lacks abdominal muscle control?

Abdominal binder, provides support

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FEV1

forced expiratory volume in 1s of FVC

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FEV1 normal value

>70% of FVC exhaled within 1s

  • FEV1/FVC x 100 > 70%


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FEF

Forced expiratory flow rate 25—75% of exhaled volume on FVC exhalation curve

  • more specific to smaller airways, dramatic changes with disease compared to FEV1


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<p>What is seen here?</p>

What is seen here?

Obstructive disease LVs

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<p>What is seen here?</p>

What is seen here?

Normal LVs

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ventilation

movement of air in and out of the lungs

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respiration

gas exchange within the lungs

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Normal PaO2

90—100 mmHg; 98—100% SpO2

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Abnormal PaO2 warranting supplemental O2 consideration

55 mmHg; 88% SpO2

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Hypoxemia

PaO2 < 80mmHg

Low level of O2 in arterial blood

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Hypoxia

Low level of O2 in tissue

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Normal pH

7.35—7.45

If normal → compensated

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Normal PaCO2

35—45mmHg

if normal → metabolic

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Normal HCO3-

22—26 mEq/L

if normal → respiratory

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What is it called is all 3 ABG measurements are abnormal?

Partially compensated

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Decreased pH, increased PaCO2, normal HCO3-

Respiratory acidosis

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Increased pH, decreased PaCO2, normal HCO3-

Respiratory alkalosis

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Decreased pH, normal PaCO2, decreased HCO3-

Metabolic acidosis

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Increased pH, normal PaCO2, increased HCO3-

Metabolic alkalosis

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Treatment for respiratory acidosis

Ventilation

Correct electrolyte imbalance

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Treatment for respiratory alkalosis

Increase CO2 retention via rebreathing, sedation

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Treatment for metabolic acidosis

Correct electrolyte imbalance; use NaCO3 for severe acidosis — pH < 7.2

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Treatment for metabolic alkalosis

Administer potassium chloride

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COPD, respiratory failure, GBS, MG, respiratory center depression, CNS lesions, hypermetabolism, sepsis, burns are causes for …

Respiratory acidosis (causes)

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Hyperventilation, increased excretion of CO2 by the lungs, pregnancy, high altitude, PF, ascites, scoliosis, CHF, anxiety and severe pain are causes for …

Respiratory alkalosis (causes)

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Acid retention, deficit of base in the blood stream are causes for …

Metabolic acidosis (causes)

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Abnormal loss of acid or excess accumulation of bicarbonate are causes for …

Metabolic alkalosis (causes)

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Ideal ventilation (V) and perfusion (Q) are seen as what V and Q ratio?

1:1

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Dead space

Well ventilated but no gas exchange (respiration) occurs

  • anatomical: blood and O2 present, vessels wall too thick; no exchange ie: nose, trachea and bronchi


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Physiological dead space

Not enough blood flow (perfusion)

  • PEs

  • Air that doesn’t take part in gas exchange


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Shunt

Complete atelectasis of respiratory unit, allows blood to travel through pulmonary capillaries without gas diffusion

  • pneumonia, PE, alveolar collapse


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Class 1 lymphedema garment compression …mmHg

20—30mmHg

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Class 2 lymphedema garment compression …mmHg

30—40mmHg

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Class 3 lymphedema garment compression …mmHg

40—50mmHg

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Class 4 lymphedema garment compression …mmHg

50—60mmHg

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Class 1 lymphedema garment compression indications

  • Mild Lymphedema

  • Typically UE, not LE

  • Fragile skin or elderly


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Class 2 lymphedema garment compression indications

  • Stage II UE lymphedema

  • Minimum compression of LE lymphedema


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Class 3 lymphedema garment compression indications

  • Rarely used for UEs

  • Typically for stage II LE lymphedema

    • high intensity, repetitive activities


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Class 4 lymphedema garment compression indications

  • Rare

  • Only for LE lymphedema

  • Custom made


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Left: chap8 + pg 204—208