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precordium
area of chest overlying heart/ great vessels
base of heart
broader area of heart’s outline located at 3rd R/L ISC
apex of heart
tip of heart pointing down toward 5th L ICS
pericardium
tough, fibrous, double walled sac that surrounds/protects heart
myocardium
muscular wall of heart
endocardium
thin layer if endothelial tissue that lines inner surface of heart chambers/ valves
heart structure
4 chambers seperated by valves
atrium
thin walled reservoir for holding blood
ventricle
thick-walled, muscular pumping chamber
valves
prevents backflow of blood; unidirectional; open/close passively in response to pressure gradients
atrioventricular valves (AV valves)
open during diastole/ allows for ventricles to fill
tricuspid valve
R AV valve
mitral valve
L AV valve
semilunar valves (SL valves)
open during systole/ blood ejected from heart
pulmonic valve
R SL valve
aortic valve
L SL valve
L sided HF
pulmonary congestion
R sided HF
JVD/ distended abdomen
unoxygenated blood to RA
SVC/ IVC
RA to RV
tricuspid valve
RV to pulmonary artery
pulmonic valve
lungs
blood oxygenated
pulmonary vein to
LA
LA to LV
mitral valve
LV to aorta
aortic valve
aorta to
body
preload
volume/ amount of stretch of heart muscle before contraction
greater the strecth
stronger the contraction
afterload
resistance/ force heart has to pump against to eject blood
arteries
thick, elastic, high pressured; carry blood away from heart; oxygenated blood; deep in body
veins
thin, less elastic, valves; carry blood to heart; deoxygenated blood; more superficial
causes vasconstriction
cold temp, nicotine
causes vasodilation
warm temp, alcohol, fever
conduction pathway
SA node —> AV node —> Bundle of His —> L/R bundle branches —> ventricles
cardiac cycle
systole/ diastole
systole
ventricular contraction
diastole
relaxation/ ventricular refilling
cardiac output (CO)
volume of blood ejected by heart each minute; L
normal adult CO
4-6 L
CO =
SV x HR
SV
preload, afterload, contractility
aortic valve
2nd ICS R sternal border
pulmonic valve
2nd ICS L sternal border
Erb’s point
3rd ICS L sternal border
tricuspid valve
4th ICS L sternal border
mitral valve
5th ICS midclavicular line
mitral valve (AKA)
point of maximal impulse (PMI)
inspection of heart
inspect valves/ PMI
normal findings of inspection of heart
no visible pulsations except for PMI
palpation of heart
assess for pulsations, thrills, heaves
thrills
purr of cat
heaves
forceful thrusting of ventricle during systole
normal findings for palpation of heart
no pulsations (except at PMI). thrills, heaves
position for auscultation of heart
supine, upright/ leaning forward, L lateral
S1 heart sound
“lub”; AV valves close (start of systole)/ coincides w/ pulse
where S1 is loudest at
mitral + tricuspid areas/ apex
S2 heart sound
“dub”; when SL valves close (start of diastole)
where is S2 loudest at
aortic + pulmonic areas/ base of heart
murmurs
gentle, blowing/ swooshing sound that can be heard on chest wall; caused by turbulent flow
how to document murmurs
radiation, timing, pitch, pattern
innocent murmur
always systolic/ not associated w/ heart abnormalities
functional murmur
high out-put states/ increased blood flow in heart
pathological murmur
structural abnormalities
Grade I (murmur)
barely audible
Grade II (murmur)
clearly audible but faint
Grade III (murmur)
moderately loud/ easy to hear
Grade IV (murmur)
loud w/ thrill
Grade V (murmur)
very loud w/ thrill
Grade VI (murmur)
loudest w/ thrill
S3 heart sound
Ken-tuc-ky; place bell over mitral landmark, L lateral position/ exhaling
S3 normal in
children/ young adults
S3 early sign of
fluid volume overload; HF
S4 heart sound
Ten-nes-see; place bell over mitral/tricuspid; L lateral position exhaling
S4 heart sound indication
cardiac decompensation
pericardial friction rub
apex/ L lower sternal border; systolic/diastolic; radiating from anywhere; scratchy/ like sandpaper; high pitched; sitting up/ breath held
modifiable risk factors for CVD
HTN, hyperlipidemia, tobacco use, physical inactivity, diet, T2DM, stress, sedentary lifestyle, abdominal obesity
non-modfiable risk factors for CVD
age, gender, race, family history
DASH diet
< 2,400 mg sodium; < 30% total fat intake; 55-60% carbohydrates
exercise/ cardiovascular health
consult provider before starting program; avoid strenuous activity; stop if there is chest pain, dizziness, faintness
inspection of CV system
observe facial skin color; examine eyes/ periorbital area; inspect lips, buccal mucosa, gums, tongue for color changes; pursed lips/ nasal flaring; use of accessory muscles; JVD
inspection (general appearance)
apple vs. pear; color of skin, nails, gums, conjunctiva; clubbing/ spooning of nails; temperature of skin
Levine’s sign
cleanched fist over chest, sign of MI
apple shape
central obesity; linked to T2DM/ CVD
pear shape
stores fat in hips/ thighs; carries lower CVD risk
+1 (edema)
2 mm; ASAP
+2 (edema)
4 mm; 10-15 sec
+3 (edema)
6 mm; > 1 min
+4 (edema)
8mm; > 2 min
palpation of carotid artery
palpate one at a time
auscultation of carotid artery
use bell to listen for a bruit
jugular vein exam
supine at 30-45 degrees; note distention (R sided HF)
developmental considerations for infants/ children
apical pulse 4th ICS (up to 4 years); HR slows as child gets older; physiologic S3 is commmon
hemodynamic changes w/ aging
gradual increase in systolic BP; decrease in diastolic BP after 40 years; thickening of L ventricular wall; arteriosclerosis
vascular system
transport fluid, deliver oxygen/ nutrients, eliminate waste
arterial circulation
system of thick muscular walls for high speed/ pressure; away from heart
venous circulation
system of thinner walls, low pressure; to heart; valves
subjective data for vascular system
leg pain/ cramps, skin changes/ swelling in arms/legs, lymph node enlargement, medications, smoking history
objective data for vascular system
inspection/palpation, symmetry, check capillary refill
inspection/ palpation of upper extremities
skin (color, temp, texture, turgor), nails (color, clubbing), pulses, capillary refill
modified Allen test
checks ulnar patency