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what is a head injury
Any trauma to the
Skull
Scalp
Brain
Head trauma includes an alteration in consciousness, no matter how brief.
A serious form of head injury is traumatic brain injury (TBI)
what factors can increase you rick for a poor outcome
Intracranial hematoma
Older age
Abnormal motor responses
Impaired or absent eye movements/pupillary light reflexes.
Early sustained hypotension
Hypoxemia or hypercapnia
ICP levels >20 mm Hg
GCS on arrival <8
types of head injuries
Scalp lacerations
Scalp is highly vascular → Profuse bleeding
Major complications: Infection & Blood loss
Skull Fractures
Closed (only skull, skin intact)
Open (skull and skin broken)
skull fractures and hemorrhage clinical manifestations
Facial paralysis
Battle’s sign (postauricular ecchymosis)
Periorbital ecchymosis (racoon eyes)
Cranial nerve deficits eyes and face
Conjugate deviation of gaze (eyes gazing same direction)
Change in LOC—report immediately
Rhinorrhea or otorrhea
Leaking fluid should be tested to determine if fluid is CSF.
CSF fluid gives a positive reading for glucose (use Halo sign or Dextrostix to test for glucose)
Place sterile gauze under nose to catch drainage
types of head trauma
concussion
lacerations
contusion
concussion
A sudden transient mechanical head injury with disruption of neural activity and a change in LOC
May or may not lose consciousness.
Brief disruption in LOC (<5 min)
Amnesia (possibly retrograde amnesia)
Headache
TX: Watch for further developments.
If grows into “stroke” symptoms, report immediately. Monitor LOC and neuro.
Post concussion syndrome: 2 weeks to 2 months
Headache, lethargy, behavior change, shortened attention span, decreased short-term memory, changes in intellectual ability
laceration
Tearing of the brain tissue
Often occur in association with depressed and open fractures and penetrating injuries
Intracerebral hemorrhage is generally associated with cerebral laceration.
Surgical repair of laceration is impossible.
Prognosis is poor with large intracerebral lacerations.
Clinical manifestations: Bleeding, Seizures, Cerebral Edema
contusion
Bruising of brain tissue within a focal area
Associated with closed head injury
Coup-contrecoup injury
Can cause hemorrhage, infarction, necrosis, edema
Seizures common complication
Anticoagulants: Potential for ↑ hemorrhage
Tx: Surgery
dxc studies for a head injury
CT scan: Best diagnostic test to determine craniocerebral trauma
MRI
PET
Glasgow Coma Scale (GCS)
intracerebral hemorrhage clinical manifestations
Neurologic: Altered level of consciousness, seizure activity, pupil dysfunction, cranial nerve deficit(s), ¯ GCS, headache
Skin: Lacerations, contusions, abrasions, hematoma, Battle’s sign, periorbital edema and ecchymosis, otorrhea (check for glucose), exposed brain matter
Respiratory: Rhinorrhea, impaired gag reflex, inability to maintain airway, altered/irregular respiratory rate and pattern
Cardiovascular: Cushing’s triad (systolic hypertension with widening pulse pressure, bradycardia, irregular resps)
Musculoskeletal: Motor deficit/impairment, weakness, palmar drift, paralysis, spasticity, decorticate or decerebrate posturing, muscular rigidity/increased tone, flaccidity, ataxia
intracerebral hemorrhage collaborative care
Treatment of life-threatening conditions will take priority
Major focus of nursing care relates to increased ICP
Maintain cerebral perfusion and oxygenation
Frequent monitoring for changes in neurologic status
Frequent GCS and neuro checks
Correct any electrolyte imbalances
Eye problems
Lubricating eye drops
Cold, later warm compresses
Eye patch
Treat Hyperthermia
Goal: 36ºC (96.8 F) to 37º C (98.6F) to Prevent shivering
Leaking CSF? Check for glucose.
Call provider immed, raise the head of bed, loose collection pad under nose, NO sneezing or blowing nose, NO NGT and NO naso-tracheal suctioning
Seizure precautions
Care of immobilized patient
Bladder and bowel function, skin care, infection
Pain (headache)
Acetaminophen (not NSAIDs) or small doses of codeine
If opioid used, pupils would constrict and alter neuro check
Patient/family education and support
complications of intracerebral hemorrhage
Epidural Hematoma
Bleeding between the dura and the inner surface of the skull
Neurologic emergency
Subdural Hematoma
Chronic SH in elderly are misinterpreted/misdiagnosed as vascular disease (stroke, TIA) and dementia.
manifestations of epidural hematoma
Initial period of unconsciousness
Brief lucid interval followed by decrease in LOC
Headache
Nausea, vomiting
Focal neurologic deficits (ie. visual field cuts, aphasia, weakness, numbness)
manifestations of subdural hematoma
Acute signs develop within 24 to 48 hours of the injury
Symptoms related to increased ICP
↓ LOC
Headache
Drowsy and confused
Ipsilateral pupil dilates and becomes fixed
txmnt for epidural hematoma
Tx: evacuate hematoma and decrease ICP
Emergency treatment
Maintain oxygenation
Assume neck injury
Administer fluids cautiously
Surgical intervention
Craniotomy, Craniectomy, Cranioplasty, Burr hole
Carefully monitor O2 after sx
complications of cranial surgery
Intracranial hypertension
Surgical hemorrhage
Fluid imbalance
CSF leak
Venous Thromboembolism
Pneumonia
caregiver instructions following a head injury
No alcohol
Have someone stay with you until cleared by MD
Avoid driving or using heavy machinery
Avoid contact sports
Return to hospital or call provider if:
Increased Drowsiness or confusion
Inability to be awaken (dec LOC)
Vomiting or nausea (increased ICP)
Seizures
Bleeding from the ear or nose
Motor problems or weakness
Blurred vision slurred speech
what are intracranial tumors
Tumors of the brain result from a metastasis elsewhere
Classified according to the tissue from which they arise
Gliomas: arise inside brain tissue: Astrocytoma, Glioblastoma, or Multiforme
Meningioma: arise outside (the layers surrounding) of brain tissue, meninges, which are the membranes that cover and protect the brain and spinal cord
intracranial tumor clinical manifestations
Depends on size & location of tumor
Increased ICP
Headache; dull, throbbing, worse at night
Seizures
N, V
Cognitive dysfunction, decreased LOC
Muscle weakness
Sensory losses, aphasia
Visual changes
dxc studies for intracranial tumors
CT
MRI
PET
intracranial tumors collaborative care
Surgical Intervention
Shunt for hydrocephalus
Radiation. If cerebral edema, use corticosteroids
Chemotherapy: Nitrosoureas class (ie. carmustine, lomustine) are effective to cross blood brain barrier.
Chemo drugs such as methotrexate, can be given intrathecally (in the subarachnoid space of the spinal cord).
Temozolomide is oral chemo
inflammatory conditions of the brain
Meningitis, Bacterial and Viral
Encephalitis
Brain abscesses
dxc studies for inflammation of the brain
Blood test and culture
CT scan
Diagnosis verified by lumbar puncture and analysis of CSF
Specimens of CSF, sputum, nasopharyngeal secretions to identify causative organism
Gram-stained smear to detect bacteria
what is bacterial meningitis
Medical emergency.
Untreated bacterial meningitis has a mortality rate near 100%
Usually follows viral respiratory disease-may enter brain through sinuses
bacterial meningitis clinical manifestations
Fever
Severe headache
Nausea, vomiting
Nuchal rigidity
Increased ICP: Photophobia, Decreased LOC
bacterial meningitis collaborative care
Respiratory isolation until cultures are negative, Bacterial Meningitis is highly contagious (resp prec)
Vitals, neuro, and LOC every hour
Antibiotics
Codeine for neck pain
Dark room and cool cloth for photophobia
Minimize environmental stimuli
Antiseizure medication (phenytoin, levetiracetam)
Manage increased ICP
Acetaminophen or ASA for fever, cooling blanket, or tepid sponge bath
Check I/O and encourage fluids
viral meningitis: what is it, s/s, dxc, tx
Most common causes are enterovirus, arbovirus, HIV, and HSV. Most often spread through direct contact with respiratory secretions
Clinical manifestations:
Headache, fever, photophobia, stiff neck
Diagnostics:
Lumbar puncture: cerebrospinal fluid (CSF) shows lymphocytosis
Blood test for viral specific DNA or RNA
Self-limiting virus
what is encephalitis & s/s
Acute inflammation of brain
Caused by a number of viruses. Ticks or mosquitoes can transmit epidemic encephalitis
Clinical Manifestations: Fever, headache, N, V. Later, if no tx, mental status changes.
encephalitis: dxcs, tx
Diagnostic: Blood test for West Nile Virus
Tx: Antiviral, antibacterial, antifungal, or anticonvulsant medications can help treat the underlying cause and manage symptoms. Corticosteroids can reduce brain swelling, and sedatives can help with irritability or restlessness
what is a brain abscess; s/s, dxcs, tx
Accumulation of pus within brain tissue from a local or systemic infection
Clinical manifestations: Headache, fever, N, V, signs of increased ICP, and symptoms of area abscess is located in.
Diagnostic: CT, MRI
Tx: Antimicrobial and symptom management