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How to treat open chest wound/open pneumothorax
treat immediately
convert wound to a closed injury
place on high flow O2 via NRB/BVM
open doesn’t usually progress to tension
Asphyxia
induced by traumatic injury that forcefully compresses the thoracic cavity
does not allow for space for the thorax to expand
Simple pneumothorax
frequent DX in blunt chest trauma pts
accumulation of air/gas in pleural cavity
air enters through a hole in the chest wall or lung
causes lung collapse on affected side
in hospital treatment — chest tube
Open pneumothorax
occurs when a chest wall defect allows air into thoracic space
results from penetrating and blunt chest trauma
negative pressure draws air into pleural space
as size increases, lung loses ability to expand
if hole is larger than glottis opening, air is more likely to enter chest wall
creates a sucking chest wound
open pneumothorax physical assessment shows
chest wall defect
impaled object
sucking chest wound
bubbling wound
subcutaneous emphysema
Hemothorax
occurs when potential space between the parietal and visceral pleural is violated
commonly caused by lung parenchuma tearing
collection of blood compresses and displaces lung
hemopneumothorax
blood and air in the pleural space
massive hemothorax
accumulation of more than 1500ml of blood in pleural space
Tension pneumothorax
life threatening condition from air accumulation within pleural space
as air accumulates, pressure builds against surrounding tissue
compresses the lung, diminishing ability to oxygenate blood and eliminate CO2
Signs of tension pneumothorax
absence of breath sounds on affected side
unequal chest rise
pulsus paradoxus
tachycardia
ventricular fibrillation
JVD
narrow pulse pressure
tracheal deviation
hypotension in a late finding
Commotio cordis
cardiac arrest caused by a direct blow to the thorax during the repolarization period
result of chest wall impact directly over the heart
sports where contact with high speed objects to occurs
Mediastinum
important anatomy of the thorax that is vital to life
heart
lungs
bronchi
great vessels
Heart in mediastinum
pericardium — membranous lining; surrounds heart and space containing it
pericardial fluid — functions as lubricant; permits heart to move easily against lungs during contractions
epicardium — covers heart’s outer surface
endocardium — inner most layer of the heart lining the chambers/valves of the heart
Great vessels in mediastinum
large arteries and veins that enter and leave heart
aorta
superior and inferior vena cava
pulmonary veins and arteries
during injury to these vessels pt’s will suffer a quick death
Rib fractures
most common thoracic injury
pain contributes to
inadequate ventilation
self-splinting
atelectasis
pneumonia from inadequate respiration
rib fractures treatment
treat the pain
breathing exercises to ensure inflation of lungs
rib plating — sx procedure to realign and attach broke fragments of ribs
which ribs are the most commonly fractured ribs?
4-9
Compensated shock
agitation, anxiety, restlessness
sense of impending doom
weak, rapid (thready) pulse
clammy (cool, moist) skin
pallor with cyanotic lips
shortness of breath
nausea, vomiting
delayed capillary refill in infants and children
thirst
normal BP
decompensated shock
altered mental status (verbal to unresponsive)
hypotension
labored or irregular breathing
thready or absent peripheral pulses
ashen, mottled, or cyanotic skin
dilated pupils
diminished urine output (oliguria)
impending cardiac arrest
Treating hemorrhagic shock
XABCD
stop the bleeding
replace the blood: IV fluid— use cautiously
oxygen
warm the pt
rapid transport to appropriate trauma center
prevent further injury
tourniquets
If commercial torniquet not available
apply a triangular bandage and a stick or rod— bp cuff can be used as well
junction torniquet
used when the hemorrhage is inguinal or axillary
belt system holds the device in place
pump inflates a compression device to put pressure on the deep vessels
Class 1 hemorrhage
Mentation: slightly anxious
Ventilatory rate: 14-20 breaths/min
Pulse: < 100 beats/min
BP: normal systolic/diastolic
Pulse pressure: normal
Skin: warm, dry
Urine output: >20 ml/hr
class 4 hemorrhage
Mentation: difficult to arouse
Ventilatory rate: >35 breaths/min
Pulse: >140 beats/min
BP: decreased
Pulse pressure: decreased
skin : cool, diaphoretic, pale
Urine output: <5 ml/hr
Force of trauma and energy that related to traumatic injury mechanisms
Someone falls on an air mattress from 10 ft, wont sustain a lot of injury, air would absorb energy// as opposed to concrete surface, our body cannot handle the energy transition
Energy dissipation
process by which KE is transformed into mechanical energy
factors affecting energy dissipation in a crash
vehicles angle of impact
differences in sizes of the two vehicles
restraint status and protective gear of occupants
protective devices can manipulate the way energy is dissipated
rapid deceleration dissipates tremendous force
energy
different forms of energy produce different kinds of trauma
mechanical
chemical
thermal
electrical
barometric
Preferred fluid resuscitation for pt in hemorrhagic shock
blood
How does trauma cause anaerobic metabolism
an injury that affects any of the components that aerobic respirations depends on will affect energy production
Anaerobic metabolism
metabolic process that functions in the absence of oxygen
instead uses stored glucose in the form of glycogen for energy production
capable of sustaining energy requirements only for a short time
produces only small amounts of energy
19 fold decrease in ATP
increased lactic acid as a by-product
best treatment for anaerobic metabolism
ensure patent airway
stop hemorrhage
replace blood
rapid transport
1st sign of anaerobic metabolism
tachypnea
aerobic metabolism
most efficient method of energy production
uses oxygen and glucose to produce energy via chemical reactions
produces large amounts of energy (ATP)
waste products
carbon dioxide
water
aerobic metabolism is dependent on
adequate and continuous supply of oxygen
patent airway
functioning lungs (pulmonary system)
functional heart
pump blood to the cells
intact vascular system
adequate supply of RBCs
carry and transport oxygen
removes waste
Neurogenic shock
associated with spinal cord injury
interruption of the sympathetic nervous system resulting in vasodilation
patient has normal blood volume but vascular container has enlarged, thus decreasing blood pressure
Level I trauma center
highest and most comprehensive, 24/7 care (specialized surgery, research, and rehab); must have robust research,education and surgical residency programs
Level II trauma center
similar to Level I but is not required to have the same research or teaching obligations
Level III trauma center
focuses on initial stabilization and resuscitation of severely injured patients before transferring them to higher-level centers
Level IV trauma center
provides advanced trauma life support, stabilized patients in rural or remote areas, and arranges for transfer to higher-level facilities
Scenarios talking about pt in traumatic injury, and which one will have the most serious injury based on the energy transfer
a fast traveling car that crashes into a wall will experience more traumatic injuries than a truck traveling at a slower speed that crashes into a wall
energy/speed matters more than mass in a crash
Golden hour
prehospital care provider responsibilities
spend as little time on scene as possible
expedite field care and transport patient to definitive care
platinum 10
EMS guidelines recommend that trauma center be less than 10 minutes
Seatbelts
stop the motion of an occupant traveling at the same speed as the vehicle
limit contact with the interior of the vehicle
prevent ejection
associated injuries include cervical fractures and neck sprains
seatbelts absorb energy — especially important in the back of a car
airbags
reduced deaths in direct frontal MVC by 30%
secondary injuries
direct contact
chemical
frontal
Front end of vehicle distorts
Passengers decelerate at the same rate as vehicle
Abrupt deceleration injuries are produced by a sudden stop of a body’s forward motion
frontal collision common injuries
head
torn aorta
tearing or shearing injuries to internal organs
crush and compression injuries
down and under pathway
one of the trajectory in a frontal collision
all the force is being put on the dashboard and lower extremity
up and over pathway
one of the trajectory in a frontal collision
leads to head injuries
rear collision
have the most survivors
whiplash injury is common
energy is imparted to the front vehicle
What happens to a car/ how to be able to stop a car
breaks: once something is in motion, it cannot be stopped unless there is an equal force that stops momentum – breaks have calipers and pads, when we hit break pedal that squeezes break pads against the rotating portion of the wheel, there is a heat transfer into the breaks (that is where the energy goes) → as we squeeze harder, the more compressed those breaks go onto the caliper of the wheel, that’s what stops the car
Heat is transferred onto the breaks and that’s how the energy is created into thermal energy – he wants us to understand how a vehicle stops
Pregnancy and trauma
changes in anatomy effect injuries of both mom and the fetus
at 38 weeks uterus and fetus are most susceptible to injury
placenta and uterus highly vascular
hemorrhage can be hard to determine
increased falls
increased violence
injury during pregnancy
trauma #1 killer of pregnant females
penetrating abdominal trauma, 36% overall maternal mortality
gunshot wounds: fetal mortality rates btw 40-70%
blunt trauma: auto collisions leading cause of maternal and fetal mortality
% of blood loss before s+s in pregnant pts
pregnant pts may lose 30-35% of their blood volume before signs and symptoms become apparent
if this happens in third trimester, it may induce premature labor
Mechanism of injury
understanding the effects of forces + energy transfer will help assess MOI
kinetics of trauma refers to the study of forces involved in MOI
Primary brain injury
occurs at the time of injury
brain bleeds, contusions, and damage to nerve and brain vessels
neural tissue does not regenerate well, rarely can repair, function of damaged structure of brain is usually permanently lost upon injury
Secondary brain injury
additional injury that occurs as a result of the progression of an untreated primary injury
the secondary injury is uninjured at the time of the injury
can happen from hours to weeks after primary injury
rapid treatment at skilled trauma facilities gives the best outcomes and prevents worsening secondary injury
coup
injury at site of impact
countrecoup
injury on opposite side of impact
epidural hematoma
bleeding btw dura mater and skull
involved arteries
middle meningeal artery most common
rapid bleeding* and reduction of oxygen to tissues
patients will have a lucid interval
acute: sudden onset
time sensitive emergency
rarer in older adults, usually originating from arterial damage
subdural hematoma
bleeding within meninges
beneath dura mater
above arachnoid
slow bleeding
superior sagittal sinus
signs progress over several days
slow deterioration of mentation
far more common in older adults due to brain atrophy
X
immediate control life threatening bleeding first (TQ’s, pressure bandages, hemostatic gauze)
Airway
is it open? will it stay open? suction? airway adjunct needed
crying or talking indicates airway adequacy
move from simple to complex
position
obstruction
Breathing
exposed chest, listen to breath sounds, palpate chest, seal and stabilize wounds to chest, deliver oxygen if needed
consider minute volume — the total volume of air inhaled or exhaled from the lungs in one minute
absent = apnea
difficulty = dyspnea
very fast = more than 30 respirations is bad and will lead to acidosis
Circulation
pulse (15×4 or 30×2), skin color, temperature, and condition
normal is 60-10 bpm
causes of pallor
excessive blood loss
anaphylaxis
hypoglycemia
anxiety
Disability
perform a neurologic evaluation
AVPU scale + pupils
quick assessment for neurologic deficits
GCS
assess for any gross neurologic deficits
have the pt move all extremities
assess for motor strength + weakness
assess grip strength
assess for loss ofsensation
Decision
transport decision
identify priority patients — do only what is necessary at the scene, and handle everything else enroute
most severe pneumothorax
tension pneumothorax
Retroperitoneum
area of the abdominal cavity that is located behind the peritoneum and includes
kidneys
ureters
inferior vena cava
abdominal aorta
pancreas
duodenum
colon
rectum
visceral + parietal peritoneum
peritoneal cavity
Peritoneum
membrane that lines the abdominal cavity and covers the abdominal organs
the true abdominal cavity
contains liver, spleen, gallbladder, stomach, part of the large intestines, majority of the small intestines, and the female reproductive organs
Cerebral perfusion pressure
the amount of pressure that is needed to push blood through the cerebral circulation (makes sure that there is enough pressure allowing blood flow through the whole brain)
CPP
MAP (mean arterial pressure) - ICP
As ICP increases
CPP decreases (ICP is the main factor that changes CPP)
Normal intracranial pressure in adults
< 15 mmhg
MCI Start triage
Prioritizing sick and injured
Green – minor
Yellow - delayed
Red - immediate
Black - deceased
30-2-can do
30 respirations
2 is for capillary refill and pulse
EMS scene size up
starts at dispatch
look, listen, smells
rapid 5 second “sick or not sick” assessment
LOC
color of skin
work of breathing
safety (info that should be communicated with dispatch)
traffic — highway design/vehicle positioning
crowds — mitigating them
hazards — minimize them
weather — protect yourself
light — flashlight
call for additional resources
First impact MOI
vehicle strikes body with its bumpers
second impact MOI
adult is thrown on hood/grille of vehicle
third impact MOI
body strikes the ground or some other object
Index of suspicion
medical term for the level of awareness or concern a clinical has for potentially serious, hidden, or underlying injuries or illnesses in a patient, based on initial findings
high index of suspicion
the provider considers a serious diagnosis likely and initiates further investigation, even if symptoms seem minor or vague
a paramedic has a high index of suspicion for internal bleeding in a patient who was in a high impact car crash, even without obvious external injuries
Eupnea
normal breathing rate and pattern
Tachypnea
increased respiratory rate
fever, anxiety, exercise shock
Bradypnea
decreased respiratory rate
sleep, drugs, metabolic disorder, head injury, stroke
Apnea
absence of breathing
deceased patient, head injury, stroke
Hyperpnea
normal rate, but deep respirations
emotional stress, diabetic ketoacidosis
Cheyne-stokes
gradual increases an decreases in respirations with periods of apnea
increasing intracranial pressure, brain stem injury
Biot’s
rapid, deep respirations (gasps) with short pauses between sets
spinal meningitis, many CNS causes, head injury
Kussmaul’s
tachypnea and hyperpnea
renal failure, metabolic acidosis, diabetic ketoacidosis
Apneustic
prolonged inspiratory phase with shortened expiratory phase
lesion in brain stem
GCS
Neurological tool used to objectively assess and monitor the level of consciousness in patients with acute brain injury, trauma, or non-traumatic coma
Lowest total GCS value is 3 (completely unresponsive patient)
Maximum GCS value is 15 (conscious, but not necessarily fully oriented patient)
Report each best response for GCS
Evaluates eye response, verbal response and motor response
flail chest
may result from blunt force mechanisms
two or more adjacent ribs fractures in two or more places
location and size affect degree that chest wall and air movement are impaired (flat sternum = most extreme)
underlying pressure causes paradoxical movement of segment and rest of chest wall
may not be initially apparent
palpate rib cage fractures and crepitus
pneumothorax or hemothorax may occur if bone fragments are driven into the body
pain may prevent adequate tidal volume
flail chest management
positive pressure ventilation (PPV)
positive end-expiratory pressure
flail chest assessment and management
palpation may reveal
crepitus
tenderness
dissection of air into tissue
ausculation
decreased or absent breath sounds
poses a threat to pt’s ability to breathe
intubation and PPV are indicated
goal is an SpO2 of at least 95% with supplemental oxygen and positive pressure ventilations
Upper airway
from mouth to nose to larynx
includes nasal cavity, oral cavity, pharynx
larynx joins upper and lower airways
pharynx - nasopharynx, oropharynx, hypopharynx
lower airway
everything below larynx
parenchyma
two pulmonary lobules; anatomic division of lungs
further divided into lobes
alveoli
respiratory bronchioles divide into alveolar ducts
alveolar ducts terminate into alveoli sacs
individual alveoli are sites of primary gas exchange
surfactant layer to prevent atelectasis
Bronchi
larger, cartilage-supported airways branching directly from the trachea
gives rise to bronchiole structures supported by smooth muscle
bronchioles
smaller, cartilage-free branches arising from the bronchi that lead to the alveoli
after about 22 division, give rise to respiratory bronchi that have limited capacity for gas exchange
Trachea bifurcates at…
carina into two mainstem bronchi
Right mainstem bronchi is often the site of…
aspirated foreign bodies