MEDSURG WEEK 5

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Last updated 12:12 AM on 10/3/26
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149 Terms

1
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Which findings mean the patient may be failing?
Altered LOC + exhaustion + ↑ oxygen need + hypotension + silent/absent breath sounds.
2
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Why is mental status a priority respiratory assessment?
Restlessness/confusion = early hypoxemia; drowsiness = CO₂ retention or respiratory fatigue.
3
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Why can a lower respiratory rate be dangerous?
RR dropping + drowsiness/weak effort = exhaustion
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What should the nurse ask before choosing an answer?
What will kill this patient first? What can I reverse now?
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What should happen after every intervention?
Reassess breathing effort + SpO₂ + mental status + BP.
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Oxygen-Delivery Devices
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When is a nasal cannula used?
Stable patient with mild hypoxemia; provides low-flow oxygen.
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When is a Venturi mask used?

When a precise FiO₂ is needed, especially for COPD.


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When is a nonrebreather mask used?
Severe hypoxemia or emergency while preparing further support.
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What must the nurse check with a nonrebreather?
Reservoir bag stays partially inflated + mask seals well + oxygen flow is adequate.
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What should the nurse do if the nonrebreather bag collapses?
Increase oxygen flow + check tubing/mask.
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What does high-flow nasal oxygen provide?
Higher flow + humidification + more reliable oxygen delivery.
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Who can use CPAP/BiPAP?
Alert/cooperative patient who can protect the airway and clear secretions.
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When is CPAP/BiPAP unsafe?
Vomiting + excessive secretions + inability to protect airway + severe instability.
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When is intubation needed?
Refractory hypoxemia + worsening acidosis + exhaustion + inability to protect airway.
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Oxygen-device recall?
Cannula = mild; Venturi = precise; Nonrebreather = severe.
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Postoperative Atelectasis
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What is atelectasis?
Collapsed alveoli → ↓ lung expansion → ↓ oxygenation.
19
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Why does surgery cause atelectasis?
Pain + sedation + immobility → shallow breathing/weak cough → alveoli collapse.
20
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What findings suggest postoperative atelectasis?
↓ SpO₂ + tachypnea + diminished bases/fine crackles + shallow breathing.
21
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A postoperative patient refuses to cough because of pain. What is the priority?
Give prescribed analgesic → splint incision → deep breathe/cough.
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What prevents atelectasis?
Incentive spirometry + early ambulation + turning + deep breathing/coughing.
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Why is pain control important?
Less pain → deeper breaths + stronger cough → better lung expansion.
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How does incentive spirometry help?

Slow, deep inhalation reopens collapsed alveoli.


25
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What complication can atelectasis cause?
Retained secretions → pneumonia or respiratory failure.
26
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How does the nurse know interventions worked?
↑ SpO₂ + improved basilar sounds + easier breathing.
27
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Postoperative complication chain?
Surgery → pain/shallow breaths → atelectasis → retained secretions → pneumonia.
28
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Atelectasis prevention mnemonic?

“DICE” = Deep breathe/cough, Incentive spirometer, Change position, Early ambulation.

29
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Pneumonia and Hospital Prevention
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What happens in pneumonia?
Infection fills alveoli with fluid/inflammatory material → impaired oxygen exchange.
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What is hospital-acquired pneumonia (HAP)?
Pneumonia beginning ≥48 hours after admission.
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How may pneumonia appear in an older adult?

New confusion + weakness + functional decline, sometimes without high fever.


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What should the nurse assess in pneumonia?
RR/effort + SpO₂/oxygen need + mental status + BP + breath sounds + sputum.
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What are priority pneumonia interventions?
Oxygen + prescribed antibiotics + mobility + hydration + airway clearance.
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When should cultures be collected?
Before antibiotics when ordered—but do not delay emergency treatment.
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Which pneumonia patient needs immediate escalation?
Patient with ↑ RR + falling SpO₂ despite oxygen + confusion + hypotension.
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Why is a rising oxygen need dangerous?
It shows worsening gas exchange despite treatment.
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What are serious pneumonia complications?
Sepsis + acute respiratory failure + pleural infection + ARDS.
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How should a hospital reduce pneumonia rates?
Early mobility + oral care + aspiration precautions + coughing/deep breathing—not blanket antibiotics.
41
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Why does immobility increase pneumonia risk?
↓ lung expansion + retained secretions → bacterial growth.
42
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Pneumothorax
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What is a pneumothorax?
Air enters pleural space → lung cannot fully expand.
44
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What findings suggest pneumothorax?
Sudden unilateral chest pain + dyspnea + decreased/absent breath sounds on one side.
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What causes tension pneumothorax?
Trapped pleural air increases pressure → lung collapse + ↓ venous return → obstructive shock.
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What findings suggest tension pneumothorax?
Severe distress + unilateral absent sounds + tachycardia + hypotension ± JVD.
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What separates simple from tension pneumothorax?
Tension pneumothorax causes shock/hypotension.
48
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Is tracheal deviation an early sign?
No—it is a late sign; do not wait for it.
49
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A patient develops absent right breath sounds and BP 76/44 after central-line placement. What is the priority?
Suspect tension pneumothorax → oxygen + emergency response + prepare decompression/chest tube.
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Should an unstable patient wait for imaging?
No—tension pneumothorax is treated from the clinical pattern.
51
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Collapse comparison?
Atelectasis = gradual/postoperative; pneumothorax = sudden/unilateral.
52
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Chest-Tube Safety
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Where should the chest drainage system stay?
Upright + below chest level.
54
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What does new continuous bubbling in the water-seal chamber mean?
Air leak.
55
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What is the first action for continuous bubbling?
Assess the patient → then trace tubing and connections.
56
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What does tidaling mean?
Pressure changes occur with breathing; this may be expected.
57
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Why can sudden loss of tidaling be dangerous?

If the patient worsens, the tube may be obstructed.


58
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Should a chest tube be routinely clamped or stripped?
No—this can trap air or create harmful pressure.
59
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Chest-tube mnemonic?
“BKN” = Below chest
60
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COPD Pathophysiology and Findings
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What is COPD?
Persistent airflow obstruction → difficult exhalation + air trapping.
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What happens in chronic bronchitis?
Airway inflammation + excess mucus → narrowed airways + productive cough.
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What happens in emphysema?
Alveolar destruction + loss of elastic recoil → air trapping + ↓ gas-exchange area.
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Why is expiration prolonged in COPD?
Small airways narrow/collapse during exhalation → air cannot fully escape.
65
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COPD pathophysiology chain?
Narrowed airways/lost recoil → difficult exhalation → air trapping → hyperinflation → impaired gas exchange.
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What are expected baseline COPD findings?
Exertional dyspnea + chronic cough ± sputum + prolonged expiration + ↓ activity tolerance.
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What findings suggest COPD exacerbation?
↑ dyspnea + ↑ sputum amount/purulence + new oxygen need + fever/confusion + ↓ activity.
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What commonly worsens COPD?
Infection + smoke/irritants + missed medications + poor inhaler technique.
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What complications can mimic a COPD exacerbation?
Pneumonia + pneumothorax + pulmonary embolism + heart failure + dysrhythmia.
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What confirms persistent COPD obstruction?
Post-bronchodilator FEV₁/FVC <0.70.
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COPD Priorities and Treatment
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A COPD patient has RR 28
SpO₂ 86%
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What are immediate COPD-exacerbation interventions?
Sit upright + controlled oxygen + short-acting bronchodilator + prescribed steroid ± antibiotic.
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What is the foundation of COPD medication treatment?
Bronchodilators.
75
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When are antibiotics used during COPD exacerbation?
When bacterial infection is suspected
76
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How does pursed-lip breathing help?
Keeps small airways open longer → releases trapped air.
77
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How is pursed-lip breathing performed?
Inhale through nose → exhale slowly through pursed lips longer than inhalation.
78
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Should oxygen be withheld from a severely hypoxemic COPD patient?
No—treat hypoxemia while monitoring ventilation.
79
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What SpO₂ range is often ordered for patients at risk for hypercapnic failure?
88%–92%
80
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A COPD patient reaches the SpO₂ target but becomes drowsy. What is the concern?
CO₂ retention and worsening ventilation.
81
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What should the nurse assess when a COPD patient becomes drowsy?
Mental status + work of breathing + ABG/PaCO₂.
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When may BiPAP be used in COPD?
Acute hypercapnic respiratory failure when the patient can protect the airway.
83
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When may COPD require intubation?
Exhaustion + worsening acidosis + refractory hypoxemia + inability to protect airway.
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COPD Prevention and Teaching
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What slows COPD progression the most in a patient who smokes?
Smoking cessation.
86
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What nonmedication treatments improve COPD?
Vaccines + pulmonary rehabilitation + activity + nutrition + energy conservation.
87
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What should be checked before deciding COPD medications are ineffective?
Adherence + correct inhaler technique.
88
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What should COPD discharge teaching include?
Rescue vs maintenance medications + inhaler technique + oxygen safety + early exacerbation signs.
89
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Which changes should a COPD patient report early?
↑ dyspnea + sputum amount/color change + fever + reduced activity tolerance.
90
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Asthma Pathophysiology and Control
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What happens during asthma?
Bronchospasm + airway swelling + mucus → narrowed airways.
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What commonly triggers asthma?
Infection + allergens + smoke + exercise + cold air + some medications.
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What shows controlled asthma?
Few daytime symptoms + no night waking + normal activity + little reliever use.
94
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What shows uncontrolled asthma?
Frequent symptoms + night waking + activity limits + frequent reliever use.
95
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What does frequent rescue-inhaler use mean?
Poor control + increased risk for a severe attack.
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Why are inhaled corticosteroids used?
↓ airway inflammation → ↓ future attacks.
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Can LABA be used alone for asthma?
No—LABA must be combined with an inhaled corticosteroid.
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Should controller medication stop when symptoms improve?
No—continue it to prevent inflammation and attacks.
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Acute Asthma Attack
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What findings show a severe asthma attack?
Cannot speak full sentences + accessory muscles + low SpO₂/peak flow + poor air movement.