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1st stage pain
type: visceral
why it hurts: cervical changes, uterine distention/ischemia
what it feels like: diffuse lower abdominal cramps; may refer to back, hips, thighs
2nd stage pain
type: somatic
why it hurts: perineal/pelvic-floor stretching
what it feels like: sharp, burning, localized
3rd stage pain
type: -
why it hurts: uterine contractions during placental delivery
what it feels like: similar to first stage pain
morphine, fentanyl (opioid agonists)
assess RR
sedation
BP
FHR before/after
placental transfer → neonatal resp depression risk
butorphoanol/stadol; nalbuphine/nubain (mixed agonist-antagonists)
may precipitate withdrawal with dependence
opioid antagonist (naloxone)
reverses opioid related resp dep
most important immediately after edpidural/
maternal bp
intermittent auscultation
periodic FHR checks; doppler is most common device listed
external ultrasound transducer
FHR; place over fetal back
external TOCO
contraction timing; cannot accurately measure intensity
internal scalp electrode
more direct FHR; invasive, requires ruptured membranes
IUpressure catheter
contraction pressure and resting tone; enables montevideo units
baseline FHR - absent/minimal
(0BPM) and (<5BPM)
abnormal or indeterminate
fetal hypoxemia or metabolic acidemia
baseline FHR - moderate
(6-25BPM)
normal
baseline FHR - marked
(>25BPM)
unclear significance
fetal anemia, sepsis, etc.
sinusoidal
a true sinusoidal FHR pattern is category III, not normal variability
IV opioids can cause a transient pseudo-sinusoidal appearance
IV analgesia
systemic and sedates baby
epidural is high risk for :
hypotension
dec placental perfusion
ineffective breathing pattern
nursing actions before epidural placement
VS and fetal monitoring prior to administration
assess fluid balance
give IVF bolus 15-30 min before to dec hypotension
nursing actions after epidural placement
assess VS Q5min for 15 min
woman may/may not feel contractions, needs to be instructed when to push
nitrous oxide
quick half life
pt controlled: only the client can hold the mask. start when a contraction begins
fetal oxygen supply can dec due to
reduction of BF through maternal vessels as a result of HTN and hypotension
reduction of oxygen content in maternal blood as result of hemorrhage or severe anemia
alterations in fetal circ w compression of umbilical cord
reduction in BF to intervillous space in placenta
uterine tachysystole
more than 5 contractions in 10 mins, averaged over a 30 min window
variable deceleration
cord compression
early deceleration
head compression
acceleration
okay!
late deceleration
placental insufficiency
early decels
FHR goes down at the same time as a contraction and comes up/recovers after the contraction is over
NOT related to fetal oxygenation
causes:
uterine contractions/compression of fetal head
nursing care: check patients dilation/effacement
late decels
FHR begins after the contraction starts, lowest point is AFTER the peak of the contraction, does not return to baseline until after contraction ends
*related to fetal hypoxemia
causes:
reduced oxygen delivery to fetus during contraction
nursing care:
stop pit
side lying
administer 10 O2 by nonrebreather
nursing management of abnormal patterns
assist the client to the side-lying position
inc maternal BV by inc the rate of the primary IV infusion
administer O2 10L/min x 15-30min
if there are any meds running to induce labor, turn off
notify physician
fetal tachycardia is most common during which of the following situations/conditions?
maternal fever