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Systems of Voice Production
Articulatory system, Laryngeal system, Respiratory system
Articulatory system
resonance
Laryngeal system
vocal fold vibration
Respiratory system
outgoing air stream; relatively constant Psg
Laryngeal system location
suspended (below) from the hyoid bone; sitting on top of the trachea
Laryngeal system primary/biological function
controls airflow into and out of lungs; prevents food and liquid from entering airway
Laryngeal system secondary function
voice production
Voice tells us about…
Laryngeal pathology: functional, organic, neurogenic
Whole person: emotions
Functional
caused by something we do to our own voice
Organic
a structural change in the vocal folds that we did not cause
Neurogenic
a neurological issue that causes a voice disorder
Larynx is built of…
bone, cartilages, and muscles
Laryngeal bone
Hyoid bone: horseshoe shaped, not attached to any other bone or cartilage, supportive structure for tongue root, inferior attachment for tongue muscles, superior attachment for extrinsic laryngeal muscles
Laryngeal individual cartilages
Cricoid: most inferior laryngeal cartilage, full ring which is wide posteriorly and narrow anteriorly
Thyroid: largest laryngeal cartilage, shield-like shape, closed anteriorly and open posteriorly
Epiglottis: attached to thyroid and arytenoids, prevents food/liquid from entering larynx
Laryngeal paired cartilages
Arytenoids: positioned posteriorly on top of the cricoid, 3 processes: muscular, vocal, apex
Corniculates: on top of arytenoids
Cuneiforms: within aryepiglottic folds, provide structural support
Arytenoids determine…
adduction or abduction
Ventricular folds (false vocal folds)
Cover the true vocal folds
On the outer edges; we see the white tissue of the true vocal folds
Glottis
horizontal space between the vocal folds
Intrinsic Laryngeal Muscles
Adductors
Abductor
Tensors
Intrinsic Laryngeal Muscles ADDUCTORS
Lateral cricoarytenoids
Interarytenoids (transverse and obliques)
Intrinsic Laryngeal Muscle ABDUCTOR
Posterior cricoarytenoid (rotates away)
Intrinsic Laryngeal Muscles TENSORS:
Cricothyroid (pars recta and pars oblique) (rotate downward)
Thyroarytenoids (inside vocal folds)
Every muscle has…
…an origin (start) and an insertion (end)
When a muscle contracts/shortens…
… the insertion moves towards the origin
Extrinsic Laryngeal Muscles
Elevators
Depressors
Extrinsic Laryngeal Muscles ELEVATORS
Digastric
Geniohyoid
Mylohyoid
Stylohyoid
Genioglossus
Hyoglossus
Extrinsic Laryngeal Muscles DEPRESSORS
Sternohyoid
Omohyoid
Sternothyroid
Other extrinsic laryngeal muscles
Thyrohyoid/Hyothyroid: elevator and depressor; when it contracts it simply reduces space between the two structures
Cricopharyngeus: not an elevator or a depressor; source of vibration in esophageal speech production
Digastric
Extrinsic elevator
Anterior: inner front surface of mandible to hyoid
Posterior: Hyoid to mastoid
Geniohyoid
Extrinsic elevator
Mandible to hyoid
Mylohyoid
Extrinsic elevator
Floor of mouth
Stylohyoid
Extrinsic elevator
Styloid process of the skull to hyoid
Genioglossus
Extrinsic elevator
Mandible to tongue
Hyoglossus
Extrinsic elevator
Hyoid to tongue
Sternohyoid
Extrinsic depressor
Sternum to hyoid
Omohyoid
Extrinsic depressor
Shoulder blade to hyoid
Sternothyroid
Extrinsic depressor
Sternum to thyroid
What part of the vocal folds does vibration
Anterior two thirds are muscular; vibrates
Posterior third is cartilaginous; does not vibrate
The muscle or cartilage is what is under the actual vocal fold tissue
Vocal fold anatomy
Cover: epithelium (encases the entire vocal fold), superficial lamina propria, Reinke’s space is between these two layers
Transition: intermediate lamina propria, deep lamina propria
Body: thyroarytenoid muscle
How the vocal fold anatomy is categorized
Outer layer is most moveable, inner layer is least moveable
Grouped based on how much they move during vibration
Vocal fold physiology
Two types:
Abduction and adduction
Vibration
Paramedian
Anywhere between adduction and abduction
Myo
Muscle contraction (adductors and abductors)
Elastic
Vocal fold elasticity
Aerodynamic
Air pressure and airflow
Myoelastic-aerodynamic theory of phonation
Adduction: adduction muscles contract, vibration begins
Vibration: Psg increases and blows vocal folds apart, air flow is released into the articulatory system, vocal fold elasticity and Bernoulli effect return vocal folds to midline
Abduction: abduction muscles contract, vibration ends
Vertical phase difference
How the vocal folds vibrate
Vocal folds open from bottom to top and close from bottom to top
Bernoulli effect
Fast moving air passing through the vocal folds creates a drop in pressure that pulls tissue inward
Vagus nerve (X)
Wanderer: distribution through neck, throax, abdomen
Many branches: e.g., pharyngeal nerve, superior laryngeal nerve, recurrent nerve
Innervation
Motor command is brought to a muscle
Recurrent nerve
Goes past the larynx, into thorax, then loops back up to larynx
Afferent
Sensory
Efferent
Motor
Pharyngeal nerve
Afferent component: tongue base, upper pharynx
Efferent component: innervates almost all velopharyngeal muscles (causes velum to elevate)
Pharyngeal nerve damage symptoms
Hyper nasality
Nasal regurgitation
Swallowing difficulty (when afferent component is damaged)
Superior Laryngeal Nerve
Afferent component: internal branch, upper (supraglottal) larynx
Efferent component: external branch, cricothyroid muscle (main muscle of V.F. tension)
Superior laryngeal nerve damage symptoms
Loss of sensation in upper larynx
Monopitch (cannot lengthen or shorten vocal folds)
Recurrent nerve
Efferent component: all intrinsic muscles except cricothyroid
Recurrent nerve damage symptoms
Unilateral damage: breathiness
Bilateral damage: aspiration, laryngeal stridor (phonation on inhalation), limited pitch range
Infant larynx
High in neck; cricoid at C1-3 level
Cartilages more elastic
Shorter VF (3mm)
Higher F0 (500 Hz for cry at birth)
Adult larynx
Lower in neck; cricoid at C4-6 level
Cartilages more osseous
Longer VF (15-20 F, 20-25 M)
Lower F0 (200Hz F, 100Hz M)
Objective measures
Frequency (Hz)
Jitter (%)
Intensity (dB)
Shimmer (dB)
Subjective descriptors
Pitch
Loudness
Quality (breathiness, hoarsness, harshness, tremor, etc)
Pitch
Perceptual (subjective) correlate of frequency
F0 (fundamental frequency)= frequency of V.F. vibration
Thinner and tense vocal folds…
… higher pitch
Thicker and relaxed vocal folds…
… lower pitch
Voice registers
Modal register (chest register): comfortably produced frequencies
Pulse register (glottal/voice fry): below comfortable register
Loft register (falsetto): increased voice pitch above comfortable registers
Frequency Perturbation
Jitter:
objective: measured by % difference (1% is normal range)
Amount of variation in the duration of a glottal cycle
Not healthy if used at all times
Loudness
Perceptual (subjective) correlate of intensity
Higher Psg…
… increased loudness
Lower Psg…
… decreased loudness
Intensity perturbation
Shimmer:
Cycle to cycle variation of amplitude/voice intensity
Expressed in dB
Quality
Distinctive characteristics of voice, exclusive of pitch and loudness
Effects of resonance on voice quality
Hyponasality (too much nasality)
Hypernasality (cannot fully elevate velum)
Effects of vocal folds on voice quality
breathiness
hoarseness
harshness
strain-strangle
tremor
Voice disorder
a condition in which voice functioning is unacceptable to the user in social, professional, or other contexts, and for which the SLP or other practitioner generally finds some corroborative evidence
Incidence
Within a certain time frame
Prevalence
In this specific moment in time
Incidence of voice disorders
Children: 6-9%
Adults: 3-9%
Elderly: 47%
Professional voice users: 30%
Pitch disorder
monopitch
reduced pitch range
inappropriate pitch
pitch breaks
Loudness disorder
monoloudness (cannot increase loudness)
reduced loudness range (can increase volume, but not as much as usual)
Perceptual signs of voice problems
Pitch
Loudness
Quality
Causes of voice disorders
3 types of voice disorders:
functional: how we use our voice
organic: pathological problem
neurogenic: neurological
Cannot treat a voice disorder without knowing…
… the cause
determining the cause is essential for treatment
two people can have the exact same symptoms with two completely different causes (voice overuse vs. laryngeal cancer)
Functional voice disorder types
Muscle tension dysphonia (MTD)
Psychogenic voice disorders (PVD)
Phonotrauma
Expressive talking/shouting/cheering. prolonged loudness, speaking in noisy environment, inappropriate pitch level, excessive coughing/throat clearing
Functional voice disorder series of events
Phonotrauma→ Structural changes→ Voice disorder→ Social, emotional, occupational problems
Muscle tension dysphonia example
Vocal nodules
Vocal polyps
Contact ulcers
Psychogenic voice disorder examples
Puberphonia
Functional aphonia
Vocal nodules
most common benign growths in children and adults
caused by continuous phonotrauma
typically bilateral, at anterior middle 3rd
initially soft, later hard
glottal hourglass figure during phonation
Vocal nodule symptoms
Breathy and hoarse voice
Sensation of something on vocal folds; need to clear throat
Could lead to lowering of pitch or reduced pitch range
Vocal polyps
masses on vocal folds
single traumatic vocal event→ hemorrhage→ polyp
typically unilateral, at anterior-middle third junction
softer than nodules; may be filled with fluid
2 types of vocal polyps
Sessile
Pedunculated
Sessile
Broad based
Pedunculated
Narrow based
Vocal polyp symptoms
Breathy and hoarse voice; possible diplophonia (2 pitches at once)
Could affect pitch
Severe cases: barely any pitch, monoloudness, aphonia
Contact ulcers/granuloma
ulcerations along posterior third of glottis
chronic inflammatory disease of the larynx
Three most common causes of contact ulcers/granuloma
Slamming together of arytenoids (functional disorder)
Reflux (organic disorder)
Intubation (organic disorder)
Contact ulcers/granuloma symptoms
Hoarse voice
Vocal fatigue
Pain in laryngeal area
Pitch breaks
Trouble manipulating loudness
Build up from ulcers
Granulomas