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anaesthesia
absence or abolition of sensation
types of anaesthesia
general, spinal, epidural, topical
general anesthesia
Required for majority of surgical procedures, including abdominal, cardiothoracic, head and neck and vascular surgery
stages of general anesthesia
induction, maintenance, emergence
epidural
injected anesthesia into subarachnoid space in spinal cord, often in lower back
common use of topical anesthesia
kids before IVs
analgesia
the absence or abolition of pain
routes of analgesia
epidural, oral, intramuscular, peripheral block, IV
common use of epidural analgesia
lower abdominal surgery
common uses of peripheral block analgesia
thoracic, upper abdominal surgery
types of IV analgesia
patient controlled or continuous (nurse controlled)
effects of anaesthesia and surgery
pulmonary dysfunction, cardiovascular instability, deep vein thrombosis,
pulmonary dysfunction from anesthetic
pain and abdominal muscle splinting, respiratory depression, impaired cough, sleep, incision, diaphragmatic dysfunction
pain and abdominal muscle splinting
hypoventilation and immobility
respiratory depression
Lingering effects of anaesthesia or narcotic analgesics
Strong effect on the brainstem = decrease respiratory centre drive to breathe
impaired cough
Incision pain - can't cough at all
Decreased lung volume and expiratory flow rates - inefficient cough
Analgesics depress cough reflex - opioids, morphine
sleep
may cause abnormal chest wall function
diaphragmatic dysfunction
Incision of diaphragm
Deliberate of accidental
Distension
Distended abdomen = pushes diaphragm superiorly
Compresses the bottom of the lungs
Position
Flat on their back = diaphragm moves up
cardiovascular instability after anesthesia
decreased cardiac output and blood pressure
causes of decreased cardiac output and blood pressure
blood volume deficit, myocardial depression, abnormal rates and rhythms, effects of epidural anesthesia
blood volume deficits
Inadequate management of blood volume intra-op
Rapid bleeding
Need to determine if this is a big enough effect to not mobilise them
myocardial depression
drugs = decrease contractility and cardiac output
abnormal rates and rhythms
Worse if myocardium has been cut into - valve repair or replacement
Changes in electrolyte balance = changes in action potential = abnormal contraction
Hypo- or hyper-volumaemia
risk factors of deep vein thrombosis
Age, obesity, immobility >4 days, varicose veins, pregnancy, post-partum, high dose oestrogen therapy, smoking history, malignancy, previous DVT/PE
Fracture/major ortho surgery of pelvis, hip or LL
Pelvic or abdominal surgery for cancer
identification of deep vein thrombosis
identify hgih risk group, eary ambulation and avoid leg crossing, cinical diagnosis, official diagnosis
clinical diagnosis of DVT
fever, temp of limb, pain, swelling
diagnosis of DVT
venogram, doppler ultrasound
other effects of analgesia
fluid imbalance, wound problems, hypothermia, neurological changes, haemorrhage, nausea and vomiting, shivering, amnesia, fatigue, hiccups
post operative pulmonary complications
atelectasis, hypoxaemia, chest infections, pulmonary embolism
clinically significant PPCs
alter the patient’s clinical course following surgery
length of stay
need extra medication
requires more treatment
signs and symptoms of PPCs
arterial oxygen desaturation, radiological evidence of atelectasis or pneumonia, raised oral temp, yellow/green sputum, bacterial growth on sputum microbiology, raised WCC, prescription of antibiotics, abnormal lung uscultation
risk factors of developing a PPC
anaesthesia >180 minutes, incision site above umbilicus, pre-existing respiratory and cardiac disease, smoking, reduced level of preoperative activity, severe malnutrition, nasogastric tube for >5 days, age >60
additional risk factors of PPCs for thoracic surgery
anemia, obesity, excessive lcohol intake
non-risk factors of PPCs
obesity, mild/moderate asthma, lower abdominal surgery
average length of atelectasis after surgery
2 days
causes of post-surgery atelectasis
induction of anesthesia, pain, prolonged recumbency, drowsiness and immobility, diaphragm dysfunction, absorption atelectasis, respiratory depression
relationship between functional residual capacity and closing capacity in atelectasis
CC exceeds FRC
closing capacity
the volume in the lungs at which point the smallest airways begin to collapse
pathology of post-surgery hypoxaemia
localised atelectatic alveoli causes decreased ventilation which decreases v/q mismatch
causes of post-surgery hypoxaemia
depression of respiratory centre, inhalation of dry gases, atropine, irritation by the endotracheal tube
atropine
commonly used medication in cardiac surgery to maintain heartbeat, causes dry secretions
causes of post-surgical chest infection
Intubation overrides defence mechanisms
Anaesthetic agents impair ciliary action
Cough reflex diminished
Hospitalisation
Persistent atelectasis may be associated
physiotherapy problems in patients following surgery
reduced lung volume, impaired gas exchange, impaired airway clearance
pathophysiology of reduced lung volume after surgery
Atelectasis
Compression/contraction of lung tissue
Decreased compliance
Fibrosis
Abnormal posture
Pain
evidence of reduced lung volume after surgery
Reduced chest expansion
Abnormal
Auscultation
Diminished breath sounds, usually in the bases
Bronchial breathing
CXR
SpO2
ABGs
Weak cough
prevention and treatment of post-operative atelectasis
ensure adequate pain relief, increase FRC and VT
management of impaired gas exchange in patient following surgery
treat atelectasis with positioning, ambulation, SMIs, CPAP
monitoring during intervention
subjective report of symptoms, observation, SpO2, manual pulse palpation
short term outcome measures
auscultation, chest expansion, oxygenation (SpO2), cough, mobility
medium term outcome measures
CXR, LOS, prevent clinically significant PPCs