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What are common risk factors of electrolyte imbalance?
Elderly
Diuretic therapy
Kidney disease
Dehydration
GI fluid losses
What are common manifestations of electrolyte imbalances?
Decreased respiratory muscle function (hypokalemia)
Changes in cardiac function
Changes in neurological function
Changes in GI motility
Decreased skeletal muscle function
What are common interventions for electrolyte imbalances?
Address cause of imbalance
Monitor level of consciousness
Daily weight
skin turgor
Intake and output
Monitor cardiac status (BP, P, heart sounds, ECG if appropriate)
Fall risk safety
Monitor intestinal status (abdominal assessment, N/V/D)
Monitor labratory values
Administer appropriate medication to correct imbalance
Patient education
How do you treat hyponatremia?
Increase sodium intake (slowly)
Restrict free water (for dilutional hyponatremia)
Severe cases: cautiously administer 3% saline
If caused by FVE: admin diuretics that promote water excretion while maintaing sodium
If caused by excess diuretics promoting sodium loss: decrease dose of diuretic
SEIZURE precautions!
How do you treat hyperkalemia?
Provide IV fluids if imbalance is caused by fluid loss
Limit sodium intake and/or increase fluid intake (depends on cause)
Teach to read food lables and use salt substitutes
What are examples of high sodium foods?
Table salt
Bouillon cubes/soups
Soy sauce
Cheese
Cottage cheese
Carrots
Dried fruits
Canned foods
Snack food/pretzels
Pickled foods
Spinach
Lunch meats
Bacon, Salami
Dried cereals
Graham crackers
Popcorn
Potato chips
Ketchup
Salad dressing
How do you treat hypokalemia?
Ask about medications (diuretics, digoxin)
Cardiac monitor!
Mild: potassium oral supplements (give w/food)
Severe: IV replacement, max 20meq/hr, tissue irritant (monitor respiratory status)
How do you treat hyperkalemia?
Avoid salt substitutes, which are high in K+
Eat low potassium foods
Use K+ wasting diuretics
Give IV insulin to bring K+ into cell (in addition to IV D5 to normalize BGL)
Cardiac monitor!!
What are high potassium foods?
APricots
Avocados
Bananas
Melons
Kiwi
Dates/figs
Oranges
Beans
Potatoes
Squah
Tomatoes
Sweet potato
Beets
Yogurt
Carrot juice
Milk
Cantaloupe
Orange juice
Spinach
How do you treat hypocalcemia?
Drug therapy: calcium replacement IV
Oral replacement: may interfere with absorption of other medications if given at the same time
Nutrition: high calcium diet
Injury prevention measures if chronic hypocalcemia
Cardiac monitor!
Neuromuscular changes: numbness/tingling and tetany
How do you treat hypercalcemia?
Stop meds that contain or increase calcium
Encourage fluids for renal excretion of calcium
Diuretics such as furosemide excrete calcium
Cardiac monitor!
Neurmuscular changes: severe weakness and decreased deep tendon reflex
What are foods high in calcium?
Cheese
Yogurt
Milk
Sardines
Dark leafy greens
Fortified orange juice
Soybeans
Tofu
Salmon
Enriched breads and grains
What are causes of fluid volume deficit (FVD)/DEHYDRATION?
Hemorrhage
Vomiting
Diarrhea
Fluid leakage: fistulas
Ileostomy
Burns
Severe wounds
NPO
Diuretic therapy
GI suction
Diabetes insipidus
Difficult swallowing
Fever
What are causes of Fluid Volume Excess (FVE)/FLUID OVERLOAD?
IV fluids: too much or too fast
Blood transfusion
Failure to excrete fluids (kidney failure)
Heart failure
Obstructed lymph system
Long-term corticosteroid therapy
Syndrome of inappropriate antidiuretic hormone (SIADH)- too much of not diuresing
For Fluid Volume Deficit, what would you assess/see with the different systems?
Weight: loss
Cardiovascular: HR- tachycardia, pulses- weak, BP- hypotension (orthostatic, pressure can’t compensate to get fluid up to brain, dizziness/light headed), Veins: neck and hand flat
Respiratory: RR- increased (need for oxygen)
Skin: turgor- poor with tenting, mucous membranes (dry, cracks)
Neurological: confusion
Organs: Kidney- reduces blood flow to the kidneys, forcing them to conserve water, concentrate urine, and risk a sudden drop in function, Urine- specific gravity >1.030, dark, order, output below 500ml/day
For Fluid Volume Excess, what would you assess/see with the different systems?
Weight: gain
Cardiovascular: HR- tachycardia, Pulse- bounding, BP- elevated, Pulse pressure- decreased, Central venous pressure- elevated, Veins- distended, engorged
Respiratory: RR- increased, shallow, SOB, crackles
Skin: pitting edema, pale and cool to touch
Neurological: ALOC, HA, visual changes, muscle weakness
Organs: GI- increased motility, enlarged liver, Urine- specific gravity depends, output also depends
With FVD what do their lab values look like?
Hgb: Elevated (hemoconcentration)
Hct: Elevated
Serum osmolality: Elevated
Glucose: Elevated
Protein: Elevated
BUN: Elevated
Electrolyes: Elevated
With FVE what do their lab values look like?
Hgb: Decreased (hemodilution)
Hct: Decreased
Serum osmolality: Decreased
Glucose: Decreased
Protein: Decreased
BUN: Decreased
Electrolyes: Decreased
With FVD what is the goal, treatment, and priority?
Goal: prevent further fluid loss, return fluid volume to normal, and prevent complications from FVD
Fluid replacement: begin with oral fluids (offere every 2hrs), IV fluids (assess cardiac, pulmonary or kidney problems), Do not withhold fluid for incontinence
Medications: antiemetic, antidiarrheal, antipyretics, antibiotics
Monitor: VS, I&O, daily weight
Priority: patient safety (Fall risk, Risk for adverse effects from parental therapy)
With FVE what is the goal, critical rescue, and priority?
Goal: restore normal fluid balance, prevent complications of FVE, and prevent future fluid overload
Cirtical rescue: pulmonary edema can occur very quickly and lead to death
At risk: elderly or adults with cardiac, kidney, pulmonary or liver problems
Priority: patienty safety
Monitor VS and lungs sounds, accurate I&O/daily weights, at risk for developing AE from treatment of FVE (might do it too well)
With FVE what is the treatment?
Drug therapy: loop diuretics- furosemide (lasix)
Increases urine output, monitor potassium and sodium levels, monitor ECG changes
Nutrition therapy: restirct water and sodium (2-4 g/day)
Weight: call HCP if gain more than 3 lbs/week or 2 lbs in 24 hrs
Assess respiratory and CV status often: administer oxygen as prescribed
Assess for skin breakdown
FVD evaluation
No longer experiencing FVD → BP increased, urine output increases, electrolyte balance, skin turgor normal, moist mucous membranes
AE from treatment → FVE (crackles in lungs, pitting edema, VS changes)
FVE evaluation
No longer experience FVE → lung sounds normal, decreased edema, decreased SOB, VS improvement, decreased weight
AE from treatment → FVD
Types of IV Fluids
Hypotonic
Tonicity: <270 mOsm/L
Moves water into cells
.45% NS
Isotonic:
Tonicity: 270-300 mOsm/L
Water neither moves in/out of cells
.9% NS, LR, D5W in bag (hypo in body)
Hypertonic:
Tonicity: >300 mOsm/L
Moves water out of cells
D5LR, D%NS, D5 ½ NS in bag (hypo in body), TPN
What are the different vascular access devices- peripheral IVs?
Short peripheral catheters: usually inserted into superficial veins of forearm, intended for short term use (<6days)
Midline catheter: usually inserted into median antecubital vein, catheter tip located in upper arm, intended for vascular access (>6 days)
Cannot be used for high-osmolarity infusates or vesicants!!
What is the difference from Peripheral IV therapy with Central Vascular Access Devices?
No limitations on pH or osmolarity of infusate
Ex’s: Non tunneled catheter, tunneled catheter, PICC line, implanted port
What is the difference between non-tunneled and tunneled?
Non-tunneled CVAD: inserted by provider, usually into subclavian vein in upper chest or jugular vein in neck, usually inserted in emergent situations only
Tunneled CVAD: surgical insertion by provider; portion of catheter lies in sub-q tunnel to prevent infection, intended for frequent/long-term infusion therapy (months to years)
What are Peripherally Inserted Central Catheters (PICC)?
Indications: lengthy courses of antibiotics, chemotherapy agents, parenteral nutrition, vasopressors
Intended for months of vascular access or when peripheral site is not possible
What are implanted port-a-cath (port)?
Inserted into subclavian or jugular vein with access port in sub-q pocket in chest/upper extremity
Intended for vascualr access >1 year
What is a hemodialysis catheter?
Used for dialysis only
Very large lumens accommodate hemodialysis
DO NOT use for adminstering other fluids/medications (except in emergency)
What are complications of peripheral line IV therapy?
Local Complications: infiltration, extravasation, phlebitis, thrombosis and thrombophlebitis, ecchymosis and hematoma, site infection, venous spasm, nerve damage
What are complications of Central Line IV therapy?
Central Line Associated Blood Stream Infection (CLABSI) → biggest concern
Occlusion
Catheter dislodgment
What are complications of both peripheral lines and central lines?
Fluid volume overload
Catheter embolism
How can you prevent complications of peripheral VADs?
Select appropriate site and catheter size
Monitor site frequently
Educate pts about symptoms of complications
Administer meds into appropriate sites at correct rates
Adequately secure catheter
Use aseptic technique for insertion
Rotate sites per facility policy
What is treatment for complications of peripheral VADs?
Remove catheter for all complications
Redness or inflammation → cool compress
Swollen → elevate
Cool to touch → warm compress
Extravasation → administer antidote
Bleeding → apply pressure
Infection → clean site, send catheter for culture
How can you prevent complications of CVADs?
Hand hygiene before and after all contact with the CVAD
Change tubing at least every 96hrs
Skin asepsis: daily chlorhexidine (CHG) baths, sterile dressing, CHG-impregnated patch dressing, change dressing 24hrs after insertion, change dressing on a scheduled basis (depending on dressing)
Daily review of necessity with removal of unnecessary lines
Scrub the hub for a minumum of 15 sec before accessing
How can you prevent and treat complications of CVADs?
Catheter dislodgment:
Prevent: secure catheter properly, advise the pt to avoid physical exertion
Treatment: stop infusion, advise provder
Occulusion:
Prevent: flush before/between/after each med, use positive pressure flush (push, pause)
Treatment: may administer thrombotic enzymes
What is Total Parenteral Nutrition (TPN)?
Daily nutrients given in IV form if GI tract not functional
May include lipids (only thing that can be y-sided), electrolytes, proteins and dextrose
Must be given through CVAD
What are SE and AE of TPN?
Fluid overload- consistency and quantity
Electrolyte imbalance
Infection
Blood glucose imbalance
have deficit → hypertonic fluid w/ lots of sugar/electroyltes → causes BG to spike initially → often resolves themselves once body becomes regulated
What is BMP/BMET?
Why: checks electrolytes, glucose, kidney function, acid-base status
Assess if: weakness, confusion, dehydration, vomiting/diarrhea, kidney disease, dysrthmias
Measures: sodium, potassium, chloride, carbon dioxide/bicarbonate, glucose, BUN, creatinie, calcium
Considerations: blood draw- fasting may be required
Normal: within ranges, abnormal: high/low values
What is CMP/CMET?
Why: same as BMP + liver/protein assessment
Asess if: suspected liver/kidney disease, jaundice, fatigue, abdominal symptoms
Measures: BMP, albumin, total protein, AST, ALT, ALP, bilirubin
Considerations: blood draw- fasting may be required
Normal: within ranges or abnormal: depends on specific value/severity