ECG Interpretation, Arrhythmias, and Cardiac Pathologies

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Practice vocabulary flashcards covering ECG rate calculations, arrhythmias, conduction blocks, ischemic heart disease, myocardial infarction localization, and electrolyte disturbances.

Last updated 1:33 PM on 10/14/26
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31 Terms

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heart rate - R-R interval

a measurement used in electrocardiography (ECG) to determine the time interval between successive R-wave peaks represents the duration of one cardiac cycle

  • heart rate (HR)=60R-R interval\textcolor{green}{\text{heart rate (HR)}}\textcolor{green}{\bm{=\frac{60}{\text{R-R interval}}}} in beats per minute (bpm)

    • R–R interval = small boxes × 0.04 seconds OR large boxes × 0.20 seconds


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heart rate (HR)=60R-R interval\text{heart rate (HR)}\bm{=\frac{60}{\text{R-R interval}}}

heart rate - R-R interval

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heart rate - rule of 300

a rapid method for estimating heart rate in regular rhythms by dividing 300 by the number of large boxes between consecutive QRS complexes

  • (300Number of large boxes\frac{300}{\text{Number of large boxes}})


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rule of 300

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heart rate - small box method

a calculation to assess heart rate on an ECG: 1500Number of small boxes\frac{1500}{\text{Number of small boxes}}, derived from the paper speed calculation (25 mm/s×60 s/min=150025\,\text{mm/s} \times 60\,\text{s/min} = 1500)

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heart rate - 10 second rule

a heart rate calculation method ideal for irregular rhythms where one counts the total number of beats present on a 10-second ECG strip and multiplies that count by 6 to calculate beats per 60 seconds

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Normal Sinus Rhythm

a regular cardiac rhythm originating from the SA node

  • physiological heart rate ranging between 60 - 100 bpm


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sinus tachycardia

a regular cardiac rhythm characterized by a heart rate greater than 100 bpm

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sinus bradycardia

a regular cardiac rhythm characterized by a heart rate less than 60 bpm

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<p>Regular Cardiac Rhythm</p>

Regular Cardiac Rhythm

A rhythm where the SA node generates impulses and conducts them through the AV node at uniform intervals, manifested on an ECG by a constant R-R interval from cycle to cycle.

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Heart Block

A type of arrhythmia encompassing diseases characterized by irregularity in the initiation and conduction of cardiac electrical impulses, resulting in an irregular cardiac rhythm.

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<p>First Degree Heart Block</p>

First Degree Heart Block

An atrioventricular conduction disorder defined by slowed conduction through the AV node, resulting in a prolonged PR interval greater than 0.22 s0.22\,\text{s} (>0.2 s> 0.2\,\text{s}), where a P wave still precedes every QRS complex.

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Mobitz I (Wenckebach Phenomenon)

A subtype of second degree heart block marked by progressive lengthening of the PR interval until an impulse fails to transmit, resulting in a dropped QRS complex.

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Mobitz II

A subtype of second degree heart block where some impulses fail to conduct through the AV node without any preceding progressive lengthening of the PR interval.

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<p>Third Degree (Complete) Heart Block</p>

Third Degree (Complete) Heart Block

A complete failure of conduction where no impulses transmit from the atria to the ventricles; the atria and ventricles beat independently (with the intrinsic rate of the Purkinje fibers/ventricles being slower), showing no correlation between P waves and QRS complexes and a higher P wave frequency than QRS frequency.

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<p>Atrial Fibrillation (A-fib)</p>

Atrial Fibrillation (A-fib)

The most common chronic arrhythmia caused by multiple atrial ectopic foci; defined by an irregularly irregular rhythm, absent P waves with a wavy baseline, irregular R-R intervals, an atrial rate of 350–500 bpm350\text{--}500\,\text{bpm}, variable ventricular rate, and zero atrial contribution to ventricular filling.

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Atrial Flutter

An atrial arrhythmia wherein the atria beat rapidly but regularly, displaying a characteristic sawtooth wave appearance on the ECG.

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<p>Cardioversion Principle</p>

Cardioversion Principle

The delivery of an electrical current designed to depolarize a critical mass of myocardial cells simultaneously so that upon repolarization, the SA node can recapture its role as the pacemaker of the heart.

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<p>Ventricular Fibrillation</p>

Ventricular Fibrillation

A fatal, life-threatening arrhythmia caused by multiple ectopic ventricular foci; the ventricles fail to pump and quiver like a bag of worms, producing an erratic, chaotic ECG tracing devoid of distinct QRS complexes.

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<p>Angina Pectoris</p>

Angina Pectoris

Episodes of temporary chest pain, pressure, or discomfort caused by reduced coronary blood supply and myocardial ischemia, typically provoked by greater than 75%75\% narrowing of the coronary arteries.

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<p>ST Depression</p>

ST Depression

An ECG manifestation of transient myocardial ischemia seen during episodes of angina pectoris, which typically reverts to baseline within 5 minutes5\,\text{minutes} following nitroglycerin administration.

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Myocardial Infarction (MI)

Irreversible damage and death of myocardial tissue caused by prolonged hypoxia and complete (100%100\%) occlusion of a coronary artery by atherosclerosis, thrombus, or embolus.

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<p>Left Anterior Descending Artery (LAD) Occlusion</p>

Left Anterior Descending Artery (LAD) Occlusion

The site of occlusion responsible for 40–50%40\text{--}50\% of myocardial infarctions, causing damage to the anterior and apical regions of the left ventricle and the anterior two-thirds (23\frac{2}{3}) of the interventricular septum (IVS).

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Right Coronary Artery (RCA) Occlusion

The site of occlusion responsible for 30–40%30\text{--}40\% of myocardial infarctions, causing necrosis to the posterior wall of the left ventricle and the posterior one-third (13\frac{1}{3}) of the interventricular septum (IVS).

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Left Circumflex Artery (LCX) Occlusion

The site of occlusion responsible for 15–20%15\text{--}20\% of myocardial infarctions, affecting the lateral wall of the left ventricle.

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<p>Evolutionary ECG Changes in MI</p>

Evolutionary ECG Changes in MI

A sequential progression of ECG waveforms following myocardial infarction: acute marked ST elevation, development of pathologic Q waves with T wave inversion within hours, and the lifelong persistence of pathologic Q waves indicating an old infarct.

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<p>J Point</p>

J Point

The distinct ECG point marking the transition and junction from the end of the QRS complex to the beginning of the ST segment.

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<p>ECG Tombstoning</p>

ECG Tombstoning

An acute STEMI pattern characterized by prominent J point elevation combined with severe ST segment elevation, forming a dome-shaped waveform resembling a cemetery tombstone.

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<p>Cardiac Markers (AMI)</p>

Cardiac Markers (AMI)

Circulating biomarkers measured following acute myocardial infarction: Myoglobin (early rapid elevation peaking under 1 day1\,\text{day}), CK-MB (peaks at approximately 1 day1\,\text{day} and clears in 2–3 days2\text{--}3\,\text{days}), and cardiac Troponin I (cTnI, rises to the highest multiples of normal and remains elevated past 7–10 days7\text{--}10\,\text{days}).

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<p>Hyperkalemia</p>

Hyperkalemia

An electrolyte disorder defined by a serum potassium concentration exceeding 5 mEq/L5\,\text{mEq/L}; it causes defective repolarization and altered excitability, manifests with tall peaked T waves, prolonged PR interval, and widened QRS complexes, and can arrest the heart in diastole.

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<p>Tall Peaked T Wave</p>

Tall Peaked T Wave

The classic initial ECG sign of hyperkalemia resulting from defective myocardial repolarization.