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obsessions
intrusive thoughts, images, urges that one tries to resist or eliminate
-individual tries to suppress and ignore the thoughts
-out of the blue with certain themes
-random
compulsions
mental acts or behaviors a person feels driven to carry out, often in response to an obsession
individual uses this to soothe the intrusive thoughts
-repetitive behaviors
-washing hands, checking if you locked door certain times, blinking amount, praying, counting in your head, thinking certain thoughts in your head
-rules that are felt and must be applied
-in aims to prevent distress and decrease anxiety, gives relief but they may not want to, it could be distressful
- not connected in a realistic way
examples of obsessions and compulsive behaviors
E.g. obsessive thought that family will die, and she has to blink a certain amount of times to make her feel better about the thoughts
E.g. someone has a rule that both sides of her body had to touch something in the same way or time, germinal-wash hands multiple times until skin is raw and cracked
E.g. perfectionism or symmetry, obsessed over other people being harmed
typical obsessions
-need things to be in order
-fear of losing control
-possible need - hoarding
-violent themes: acting on impulse and hurting someone
-responsibility obsession
-religious or moral obsession
-very excessive concern of right or wrong / morality
etiological mechanisms that contribute to and explain OCD
Thought suppression and the rebound effect
Born with a predisposition to be very emotionally reactive
Emotional trigger / event
Try to suppress emotional response
But the more you suppress, the more aware you become
orbiotofrontal cortex
Overactive in signaling threat, salience, and persistent worry ("what's important").
Anterior cingulate cortex
deals with conscious internal emotional control “something is wrong”
rebound effect
is the loop and controls our, more attempts to suppress makes you more anxious
-a phenomenon where suppressed thoughts, emotions, or behaviors return with greater frequency or intensity once you stop trying to suppress them.
exposure and response prevention (ERP)
is the most successful for OCD, helps manage severe anxiety disorders and obsessive-compulsive disorder (OCD) by breaking the cycle of fear, intrusive thoughts, and compulsive avoidance
why is it effective for treating OCD
There’s target as EXPOSURE and RESPONSE, to expose people to their anxious cues and try to cause them to avoid doing their compulsions
-expose to obsessional cue: dirty things or obsessed about being able to sleep the exposure may be articles or videos of people with insomnia, and prevent them in erp from engaging in compulsions and just let anxiety be there
-patient given homework to provoke themselves to anxiety stimulus and see how long they can
-build up tolerance
-response prevention: push them farther than they would go, put stove on and walk out of house
-a problem is people drop out of this treatment at high rates because they are in trouble
what is trauma (as defined by the DSM-5)?
Trauma: an event that involves actual or threatened death, serious injury or sexual violence in one of the following ways:
directly experiencing the event
Witnessing the event (eye witness, not usually through representation or reproduction of the event)
-Vicarious trauma
Learning that an event occurred to a close friend or family member
Repeated exposure to details of a traumatic event (first responders and getting info and peoples deaths)
-doesn’t include television or media only work
criterion A and changes in this criterion
DSM- III-R (1987): An event “outside the range of usual human experience that would cause “significant symptoms of distress in almost anyone”
DSM-IV (1994):
Person experienced, witnessed, confronted with event that involved actual or threatened death / serious injury or a threat to the physical integrity of others
-required a specific response
-experience in PTSD
Person’s specific response involved intense fear, helplessness, or horror
DSM - V (2013): an event that involves actual or threatened death, serious injury, or sexual violence – focus on event itself
-don’t have person response to it like in DSM 4
*DSM 3 is nature and trauma and dsm 4 added the specific response to the traumatic event, added that trauma could be learning about the death of close friend or relative
What experiences are included vs. excluded based on this definition? Should the definition change to be more inclusive? Why or why not?
Right now it’s directly experiencing, witnessing event,, learning it happened to family member, and detailed know exposure to family or friend
*situations not included: repeated exposure to details of traumatic events, doesn’t apply to exposure unless it’s work related, If grow up in environment where you hear reoccuring stories about mass stories
*people who are a part of your demographic group that have crimes or discrimination – doesn’t have to be close
* emotional abuse and neglect: chronic crtiscism, rejection and humiliation with trauma event
*bullying, financial hardship, discrimination and hardship – bullying with physical violence or threat of death could account for being traumatic event
The definition should be expanded because there are traumatic life events that don’t necesarily slways fit the criterion but have long lasting psychological effects
-people who go through these other traumatic events may feel that their suffering is being minimized or may have difficulty receiving an appropriate assessment. For this reason, I would favor a more inclusive approach that recognizes a wider range of potentially traumatic experiences while maintaining specific requirements for diagnosing PTSD. Clinicians should be able to acknowledge and treat the effects of emotional abuse, discrimination, and other chronic stressors even when those experiences do not meet Criterion A.
the differences and similarities between trauma and adversity
-Adversity can help you grow where trauma can't necessarily be turned into a positive experience. Trauma psychologically affects person where adversity is more broad
Trauma is 1 event, outcome from adversity, adversity is from environmental context and more general, chronic
differences and similarities between adversity and stressful life events
Both are environmental toxins. both are negative life events. But adversity is unexpected where life stressors can be expected to go throug it in development (e.g. college, breakuo, hard exam)
PTSD in Dsm has 4 broad symptom category criteria
Intrusion (1+) - Criterion B:
Avoidance (1+) - criterion c
Cognition / Mood (2+) - criterion D
Arousal (2+) - criterion E
PTSD summary
Core symptom: experience of trauma - criterion A
At least the following symptoms:
-intrusion (1+)
Avoidance (1+)
-negative alterations in cognition mood (2+)
Alterations in arousal & reactivity (2+)
Significant distress and impairment: can’t do whats needed in daily life
Duration: more than 1 month
Intrusion symptoms
Intrusion (1+) - Criterion B: distressing memories of insistence, someone exposed to interpersonal violence and may have flashbacks. Terrifying dreams or nightmares, dissociative reactions in which the person feels and acts that it’s recurring. Psychological and physiological stress or cues from environment; triggers: someone in a house fire can smell smoke at barbeque and it can trigger the distress.
-need at least 1 one symptom of intrusion
avoidance symptoms
Avoidance (1+) - criterion c: persistent avoidance of stimuli from the trauma
ways to avoid distressing memories that are closely related to the event. Might distract or repress
avoiding physical things / external reminders as people or places or activities that remind a person of trauma and memories of the traumatic events pop up.
- one of these
cognition & mood symptoms
Cognition / Mood (2+) - criterion D
Negative changes in cognitions or mood in 2 or more of following
-inability to remember aspectects of an event, remember the child abuse they went through but forgot certain things
-inability to remember events
-persistent and negative beliefs of ones self: I’m bad, I’m worthless
-negative belief about others - world dangerous
- Persistent distorted cognitions of cause or consequences of event that lead individual blame himself or others, false memories in a way
-persistent negative emotional state: fear, worry, anger, shame,
- diminished interest in activities you used to enjoy like exercise or being w friends, after traumatic event might withdrawal
-feelings of detachment from others
-persistent inability to experience positive attitude feel joy or happiness
arousal & reactivity symptoms
Arousal (2+) - criterion E
Changes in arousal and reactivity in 2 or more of following:
-irritability
-reckless, self destructive behavior
-hypervigalence
-exaggerated startled response: startled easily
-problems with concentration: brain fog
-sleep disturbances: sleeping too little or too much
People who experience trauma have higher anxiety and easily more angry or irritable, excessive fear
Soldier might be sensitive to traffic or fireworks
acute disorder symptom clusters
Core symptom: experience of trauma Symptoms: some of the four categories: -intrusion -avoidance -cognition & mood symptoms -arousal & reactivity symptoms -dissociative symptoms (9 or more from any of the 5 categories) Significant distress and impairment
acute stress disorder duration
3 days - 1 month after trauma, after they experience a traumatic event people may have responses to symptoms. Immediate traumatic response PTSD is a month or longer, acute stress disorder is a risk factor
What are the main responses to potentially traumatic events (PTE)? And what are factors that affect those responses to PTEs?
-4 prototypical response patterns
-pre, peri, and post-trauma factors affecting PTE responses
-latent vulnerability approach and 3 threat biases
4 prototypical response patterns
Resilience
Recovery
Delayed:
chronic
resilience
Low symptoms of distress after exposure to PTE
recovery
elevated distress after pte that decreases over time especially after therapy
delayed
low distress after PTE and becomes elevated after some time
chronic
t’s stable from when it happened to years later, elevated distress
pre factors affecting PTE response
demographic: age, gender, previous trauma, personality traits, SES, copying styles, access to resources family history, prior mental health: depressive or aggressive behavior, family history of mental illness, intergenerational trauma
-families who have been through korean war may have lower function, lower emotional expression and it affects their children and grandchildren in how they share emotions
peri-trauma factors affecting PTE responses
(those during or immediately after PTEs): nature or subtype of trauma, interpersonal, abuse, domestic violence, neglect, associated with ptsd symptoms than with non-interpersonal traumas (natural disasters, accidents), interpersonal events can affect, early onset, childhood trauma is later risk factor to PTEs and brain development, chronicity and frequency: trauma and xposure to multiple,examined and duration of exposure over periods of time: early childhood to 18 yrs old. Frequency: experience to chronic or multiple traumas
post-trauma factors affecting PTE responses
level of social support, do they have support system, do they have good resources like counseling, ongoing stressors: is the same or similar thing still going on
-social support
-access to resources
-mental health care
-hospitals
Latent vulnerability approach
approach: PTEs exert influence by creating lasting change in specific vulnerabilities across multiple units of analysis (e.g. neural, cognitive behaviroal)
-measurable challenges that follow potential traumatic event disposure that are latent
-latent: doesn’t have immediate clinical manifestation, or impact on person that you can see
-increase risk or vulnerability to psychiatric disorders
-affect persons well being over time and later on
latent vulnerability 3 biases
threat biases
reward biases
social-emotional defecits
threat biases
dysregulated stress response system, post traumatic cognition, avoidant behaviors, hypersensitivity, or blunted response to threats in general,
-the interventions are targeted through exposure based trauma focused interventions, related trauma exposed interventions, can alleviate symptoms of PTSD by reducing the biases
reward biases
deficits in reward learning, experiencing or dissipating awards Salient within context of emotionally numb presentations of PTSD
-poverty, neglect - physical resource, can be seen in emotionally numb presentations of PTSD, interventions with healthy reward process, empower people to do behavior
-promote healthy reward processing, behavioral activation
social-emotional deficits
difficulties identifying and demonstrating appropriate responses to own and other emotions around you, social cognitive processes like trust after traumatic experience and find it difficult to trust others or themselves emotion dysregulation, have problems with appropriate emotions and those around them
-diff cognitive processes like trust
PTE and PTSD epidemiology. How common are PTEs?? PTSD?
Epidemiology of potentially traumatic events (PTEs)
Prevalence of PTEs and PTSD
Experience of PTEs is quite common
Lifetime prevalence: 40-90%
Higher prevalence for female - identifying, non-binary individuals, and trans individuals
Still PTSD is rare:
People who have PTEs may not get diagnosed with ptsd
Research suggests that black and hispanic have higher exposure but white have higher. SES, poverty factors
Lifetime prevalence 6.8%
12 month prevalence: 3.5%
main etiological risks Trauma-related disorders.
Etiology **(ON THE EXAM AND ESSAY): unlike many disorders, PTSD / acute stress disorder have a clear etiology
-people who are more like to have PTSD
Vulnerable communities:
Children
Veterans
Those who live in low SES communities: more likely to include minority populations, high exposure can experience discrimination, community violence and is chronic can have effects on well-being when growing up
Resilience: age, identities, stressors
main etiological models for trauma related disorders
intersectionality
racial trauma
intersectionality
Intersectionality: can play a big role in determining risk
Response to trauma where multiple stressors in response to
-natural resilience
-groups can be more at risk
-a group you identify as can have initial vulnerabilities,
-influence likelihood of experiencing PTSD and acute stress disorder
Conceptualized by black feminist that was responding to racism in feminist movement
The term was officially by Crenshaw, black women said they were facing discrimination and court dismissed this and said there was no discrimination and wouldn’t consider
Explains how multiple identities leads to unique experiences of depression and privilege, by examining single identities it shows impact of one’s collective identity of how they navigate the world and result of system
racial trauma
A real or perceived racial discrimination
-threats of harm or injury
-humiliating or shame events
-witness harm or racism to others
-experiences of micro aggressions
know the main, evidence-based treatments for Trauma related disorder
medication, prolonged exposure, cognitive processing, therapy, EMDR
medication
can be used to provide relief from intense PTSD symptoms
-anti-depressants: ssris to alleviate depression, intrusive thoughts, flashbacks, avoidance-behaviors, intrusion behaviors
-antipsychotic medications
psychotherapeutic treatments
To help with PTSD and acute stress disorder
Exposure therapy: like with OCD, brings something up that person doesn’t want to experience and same thing with PTSD
-try to relive and experience it and not block it out
Prolonged Exposure, Cognitive Processing Therapy, EMDR.
trauma narrative
Approach that helps process clients instead of avoid, use a narrative for gradual exposure therapy since you’re talking about your trauma in small doses in safe controlled environment
-sense of self
Able to think and talk about the trauma, particularly the worst moments
-helping people approach and talk about their feelings, thoughts, memories
-gradual exposure so people feel comfortable to say it’s a part of my history to say i experienced this and it’s a part of my history
Identify unhelpful or inaccurate cognitions (e.g., “it was my fault”) and altered views of self, others, or the world
-cognitive distortions about themselves or the world
-identify when the person is trying to blame themselves
-combat that by giving them
Identify more helpful and accurate ways of thinking about traumatic events, self, others, the world, the future
prolonged exposure
Exposure based program for clients to process trauma events and reduce trauma induced psychological disturbances
Talk about traumatic event in first person as if it were happening in the moment
use psychoeducation, imaginable exposure, in vivo (live)) exposure
psychoeducation
teaching people about mental health, reactions to trauma, helps normalize and validate experiences, help post trauma difficulties
imaginable exposure
repeated recounting of traumatic memory: emotional reliving, talk about it with therapist, write about it, talk and record yourself and then hearing it play back
In (vivo) live exposure
-gradually approaching reminders of event, situations or objects
-despite being safe you're avoidant and fearful
-approach trauma of objects and situations
-if got attacked at restaurant you might try to approach other restaurants that bring those traumas and reminders - might avoid restaurant
-talk about it in the first person
cogntive processing therapy
The focus is on cognitive restructuring:
Self blaming thoughts
Meaning of the event
Implications of traumatic events, trauma on their lives
Beliefs about safety trust, power, control, self-esteem, intimacy
eye movement desensitization and reprocessing (EMDR)
*some clinicians thinks this works and others don’t, some people are suspicious
*form of exposure with strong cognitive component and rapid eye movements
Exposure: have the patient imagine scene for the trauma or sequence of what happened
Strong cognitive component: patient or client imagines trauma
Accompanied by rapid eye movements: have patient track my finger or moving object
-use finger or pencil with fuzzy
-think of traumatic event and eyes follow the stimulus, the sequence repeated until distress decreases
-patient is focusing on negative and can make a positive thought
-eye movement is irrelevant but it is exposure and cognitive restructuring, which is why it might work
Anxiety disorders
fear, anxiety, GAD, Specific Phobia, SAD
fear
a stress response from immediate danger
anxiety
stress response just from your thoughts e.g. “What if the big bad fish comes out today?”
fear duration
Fear is present focused: immediate threats or stimuli
-e.g. Spiders come out of nowhere and freak out
anxiety duration
Anxiety is Future focused: future threat, don’t know what the future will look like, distant or less concrete stimuli or worries
-something that may or may not happen
fear immediacy
Fear is Brief: short term acute response specific stressor, builds intensity in the moment and panic
anxiety immediacy
Anxiety is Sustained: concerned longer than a moment with future-oriented stimuli or worries
fear specificity
Fear is a Specific Threat: “This Viper!”
anxiety specificity
Anxiety is a Diffuse Threat: “something bad happening” feel the anxiety everywhere and that something bad is happening
fear adaptivity
Fear is generally adaptive: a spike in ability to cope.
-flight or fear response
anxiety adaptivity
Anxiety is only adaptive in moderation: wears down your system over time
-worrying about a trip signals you to prepare but in anxiety you overestimate the risk and fixate on the out comes
-worry about an exam is okay because it motivates you, but at a certain point its not helpful, anxiety is helpful in moderation
anxiety epidemiology
Prevalence
-anxiety is most common: trauma related, ocd,
Lifetime Prevalence: Up to 33.7% of the general population will experience an anxiety disorder during their lifetime. [1]
Annual Prevalence: Past-year rates range from roughly 8.4% to 24.9% globally, with US adult past-year prevalence around 19.1%. [1, 2]
Sex and Gender: Women are affected approximately twice as often as men (roughly 1.6 to 2 times higher rates). [1, 2]
Age of Onset: Anxiety disorders frequently begin early in life, with a median age of onset around 11 years old. Specific phobias often start even earlier (average age 7), while social anxiety typically emerges around age 13. [1, 2]
anxiety comorbidity
-anxiety is comorbid with other disorders or anxiety disorders itself e.g. GAD, Social Anxiety, Separation Anxiety, Phobia
-high comorbidity between anxiety and substance abuse, mood disorders: depression, bipolar disorders
-some researches think GAD is preexisting for other anxiety disorders
what do anxiety and depression have in common
Both defined by negative emotional experience: don’t feel could
Can be triggered by stressful experiences - could be anything: death of loved one, negative experiences, stressful experience
Comorbidity: Clark & Watson’s tripartite model
General distress is common in depression and anxiety
-irritability, upset, feeling unpleasant in general
Depression is characterized of low levels of positive affect: lack of energy, inability to experience pleasure, lack of drive, not interested in thinks
“Pure depression”
Anxiety: increased levels of somatic arousal: increased heartbeat, sweating, chills, stomach discomfort, aches
-”pure anxiety”
Both triggered by stress full experience shared and distinct etiological factors
affect
any experience of feeling or emotion
-positive: positive emotions
-negative: negative emotions, high negative affect is distress
Generalized Anxiety Disorder symptoms
Core symptom: excessive worry
-applies to two or more areas: family and friends at school home or relationships - find it difficult to control their worry in everyday activity or events, not unusual events
Other Symptoms - need 3 or more of the following
Restlessness or on edge
Tiredness and fatigue
Difficulty concentrating or mind going blank - can’t listen in class during notes
Irritability
Muscle aches, tension, soreness
-can’t pin where the ache in your body is coming from
Sleep problems related to insomnia
*for someone to be diagnosed with GAD must cause significant distress or impairment in their lives
GAD duration
Duration: 6 months, more days than not
Worry has to apply to two or more areas of life: school, family or friends, work
The person must find it difficult to control the worry
-may want to decrease anxiety but can’t
etiology of GAD
traigt anxiety runs in families, neurotransmitters, overactive brain circuits, chronic stress, stressful life events
intolerance of uncertainty
belief that worry is adaptive
avoidance of threatening information
cognitive appraisals associated with GAD: Intolerance of uncertainty
characteristic that is result of negativity beliefs of uncertainty , tendency to react negatively on emotional, cognitive, behavioral level to uncertain situations or events. Find uncertain situations stressful and upsetting e.g. always worried about the future and concerned because they can’t predict future
cognitive appraisals associated with GAD: belief that worry is adaptive
positive beliefs that worrying serves them, that it helps them cope with events or prevents events to not panic e.g. think that worrying about exam helped me do good on the exam
cognitive appraisals associated with GAD: avoidance of threatening information
may focus on threat, then avoid mild threat cues to regulate anxiety, however feelings of worry are likely to continue, which is not adaptive bc chronic worry has negative impacts on the body and affect parts of life
epidemiology of GAD
Prevalence: 2x likely for women to be diagnose
Gender differences: 2-1 female to male, 5% of population meet criteria for GAD,
*Onset: develops in adolescence, 10-19 years old or can be in response to a life stressor in women over 40
-high overlap of comorbidity
specific phobia symptoms
Excessive or unreasonable fear to specific object / situation Immediate fear / anxiety Avoidance or distress: avoids or endures situation with intense fear or anxieety Proportionality: fear of anxiety is out of proportion to actual threat and social cultural context -might start crying or screaming Somatic symptoms: see spider, might have heart palpitations, dizziness, nausea, sweating, chills, shaking, trembling, upset stomach, out of breath
specific phobia duration
must occur for 6 months or more, persistent level of fear / anxiety or avoidance
subtypes
animal type, natural environment type, blood / injection / injury type, situational type
animal type
fear or avoidance of animals, arises in childhood
natural environment type
fear of heights or water. Fear of storms, can arise in childhood
blood / injection / injury type
seeing blood, injections, watching surgery, bodily infections. Faints when get blood drawn. Runs in families or in early childhood
other type
doesn’t fall into the 4 categories, fear of falling down, fear of choking, vomiting or illness, fear of costume characters like clowns, random objects Snacks and heights are most common phobia and have associated panic attacks when seeing the object or situation
specific phobia etiology
preparedness modal, social learning, classical conditioning, operant conditioning
preparedness model
indicates there’s something we are naturally afraid of because it leads to evolutionary disadvantage. Less experience to gain response like fear of fark. Leads to fear and can become phobias, evolutionary advantage: afraid of insects or certain animals if our ancestors were killed by those and that’s why we’re scared -negative information from others like parents or TV, e.g. hear that walking alone at night can be dangerous and that’s why person has fear
social learning
you can learn fear by watching others behaviors and interactions to a stimulus, their response. You see it and then copy, direct observation -e.g. Baby monkeys learn from adult monkeys to be afraid of snacks - classical conditioning? -see parents scared to get blood drawn you might develop that fear
classical conditioning
learned from repeated pairing of specific stimulus, little albert, racism can be classically conditioned in children if fear of certain racial group
operant conditioning
behavior learned by the reduction of anxiety, learned through negative reinforcement and that a fear of a stimulus is continually reinforced by a stimulus because it avoids anxiety .e.g fear of dogs and avoid them when crossing the street, avoid at all costs
epidemiology for specific phobia
Prevalence: 12 month prevalence is 9%, lifetime prevalence is 12%
Gender differences: women are 2-3x more likely to have than man, but different for each phobia -animal, and specific situation is common in women but blood is common in men and women -animal most common
Age of onset: can develop throughout lifetime. Can begin in childhood, see how people around you act. Mostly arrive unexpectedly in early adolescence or adulthood
situational type
fear of particular situation e.g. airplane, elevator, enclosed space. Arises in childhood but can have onset in mid 20s
social anxiety disorder
Fear / anxiety of social or performance situations; exposed to possible scrutiny by others -people being socially anxious can be debilitating -persistant fear of anxiety of 1 or more -e.g having a conversation, meeting unfamiliar people, talking to people you know well, being observed -e.g. Anxiety in restaurants and cafeteria, performing infront of others like a speech
-fear of negative evaluation
-avoidance or distress
SAD epidemiology
Prevalence: lifetime is 12%, more common in women and stronger in childhood / adolescence
Gender differences: women's sense of self is more dependent on others compared to men, care about interpersonal connections, women have more anxiety and got to health provider, women experience more than men
Onset: childhood or adolescence - 15 years old
SAD etiology
classical conditioning applied here: social negative experience as teasing or bullying, a bad response to social situation,
-classical conditioning from early on preparedness: there’s somethings were naturally afraid of and this can be a cause, we need others to survive,
other researches say this doesn’t make sense because we need social bonds to survive and shouldn’t be avoiding it
panic attacks & panic disorder
just because you have panic attacks doesn’t mean you have panic disorder
panic attacks
By an abrupt intense fear that appears, symptoms are Sudden and quickly peak symptoms, Reaches peak within 10 minutes
4 or more of following symptoms
Heart palpitations
Sweating
Trembling or shaking
Sensations of shortness of breath Can’t breathe
Choking
Chest pain
Nausea / abdominal distress
Dizzy / faint
Chills / hot flushes
Numbness or tingling
Derealization / depersonalization - outside of your body
Fear of losing control / going crazy
Fear of dying
panic disorder
panic attacks are necessary but insufficient for panic disorder recurrent unexpected panic attacks, history of 1 panic attack, more than one month of fear of panic attack / behavioral changes
Recurrent, unexpected panic attacks
Fear of having panic attacks (or its consequences) And /OR behavioral changes to avoid panic attacks
IMPORTANT:
Panic disorder is reoccurent, they show up over and over again in situations you wouldn’t expect them too, in one or more panic attacks experience
Persistent concern or worry of attack happening again/ consequences of the attack
-worry certain feelings will bring attack
2. Behavioral changes to avoid panic attacks
-avoid elevators or enclosed spaces, take stairs, stop exercising
* panic attacks are necessary but insufficient for panic disorder
panic attack vs. panic disorder
Panic Attacks VS. Panic Disorder
Panic Attack:
physical symptoms
often isolated: NOT TIED TO EMOTIONAL AND BEHAVIORAL SYMPTOMS
Panic disorder: emotional / behavioral symptoms:
Either fear of further panic attackers AND / OR changing behavior to avoid further panic attacks
-stop working out so heart rate doesn’t increase
Involves a (potential) pattern!!
agoraphobia
Can be related to panic disorder, used to be related
Anxious apprehension about being in places or situations from which:
escape might be difficult or embarrassing e.g. can’t get out of elevator and they’ll suffer inside
E.g. crowded streets, shops, enclosed spaces (theatre, church, concert, public transit), bridges, tunnels
help may not be available if one has a panic attack
agoraphobia situations
Need marked fear or anxiety of 2 or more of the situations
Public transportation
Open spaces
Enclosed spaces
Being in crowd / standing in line
Being outside of the home alone
Example: fear is out of proportion of what the situation is. Someone who’s in a grocery store may be scared they’re going to die there, fear of places of assembly, different situations of threat stimuli
Actual exposure to public spaces
People avoid these places
agoraphobia cognitive theories
Focus on how individual interprets bodily sensations
-internal stimuli provokes panic attacks
Anxiety sensitivity:
Interoceptive awareness
panic disorder etiology
-stressful life events
-biological