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rapport
the development of a trusting, therapeutic relationship through warmth, removing biases, and understanding; decreases anxiety and increases recovery
empathy
the ability to understand the patient's perspective and share their feelings
empathetic linkages
feelings, usually anxiety, that the nurse can directly share with the patient and must be aware of
introduction phase of the therapeutic relationship
when the nurse explains their role and task, must put initial bias aside and keep it patient-centered
orientation phase of the therapeutic relationship
the nurse and patient get to know each other, establish trust, set boundaries, get to know problems, and discover themes
first meeting of the orientation phase
nurse and patient set boundaries, outline responsibilities, discuss rules of the unit, address anxieties, and observe problems
confidentiality of treatment in the orientation phase
families may be better historians than patients, but nurses must get permission first
testing the relationship in the orientation phase
patients will test the relationship to gauge if they should trust you: forgetting scheduled session, being late, getting frustrated, making small issues big ones
working phase of the therapeutic relationship
exploring the problem and identifying possible strategies; the patient becomes more trusting and the nurse educates and encourages the patient; phase where transference and countertransference can arise
resolution phase of the therapeutic relationship
problems are resolved and the relationship comes to a close; doesn't mean the problems are gone; ideally the patient takes responsibility, but termination can be stressful
nontherapeutic relationship
nurse and patient both are trying to find a solution but keep failing, thus they get frustrated with each other and can lead to mutural withdrawal
deteriorating relationship
starts with withholding where the patient doesn't feel seen, moves to avoiding and ignoring where the patient avoids the nurse but thinks the nurse is avoiding them and then the nurse starts to ignore, moves to the patient struggling to make sense of the situation which can lead to blaming, taking personally, and internalizing
strategies to support a therapeutic relationship
motivational interviewing, collaboration, client-centered counseling, client change talk, negotiating change plans, empowering goals and decisions
empathy vs sympathy
empathy is feeling with someone and recognizing and not judging their emotion, while sympathy is feeling for someone and feeling bad for them
emotional vs cognitive empathy
emotional is where you absorb emotions as if they were your own and can contribute to compassion fatigue, while cognitive is based on reasoning and keeps you grounded because it is empathy for the purpose of understanding how to help
WBC lab results
decreased from medications like phenothiazine, clozapine, and carbamazepine; increased from lithium and NMS
platelets lab results
decreased from medications such as phenothiazine, clozapine, and carbamazepine
vitamin B12 lab results
deficiency can result in psychosis, paranoia, fatigue, agitation, personality changes, dementia, and delirium
folate lab results
deficiency can be caused by alcohol, phenytoin, oral contraceptives, and estrogens
CK lab results
elevated from muscle injury, drug use, IM injections, NMS, manic episodes, and restraints
sodium lab results
deficiency caused by Addison's, SIADH, polydipsia, and carbamazepine and can increase lithium toxicity
potassium lab results
can lead to weakness, fatigue, and EKG changes
BUN/creatinine lab results
elevated from dehydration or lithium and amantadine toxicity, can cause AMS, lethargy, and delirium
concepts of psychiatric nursing process
assessment, diagnosis, planning, implementation, evaluation
what factors are included in a recovery-oriented assessment?
patient interview, trauma-informed assessment, physical assessment, psychological assessment, self-concept, stress/coping assessment, risk assessment, suicide/homicide assessment, social/cultural/spiritual assessment
what are recovery-oriented nursing interventions?
self-care, activity/exercise, sleep strategies, nutrition, relaxation, hydration, thermoregulation, medications, CBT, conflict resolution, cultural brokering, bibliotherapy/social media, reminiscence, psychoeducation, health teaching, milieu therapy, privilege systems
describe the physical response to stress
increased cortisol, epinephrine, and adrenaline results in hyperglycemia, tachycardia, HTN, and increased blood supply
role of cortisol in stress
fight or flight triggers increased release of stress, the body attempts to adapt, stress continues, leads to burnout, weak immunity, and illness
priorities related to stress
depend on the person but usually entail reestablishing basic functional patterns, addressing psychological issues, and developing coping skills
interventions related to stress
reinstating daily routine, adequate diet education, regular exercise routine, relaxation techniques, problem-solving strategies, reflection of past coping skills, family unit functioning
medications used to treat stress
benzodiazepines, SSRIs, SNRIs, beta-blockers (physical s/s), sedative-hypnotics (stress-related insomnia), buspirone
what is included in the DSM-V criteria for depression?
must include at least one of either depressed mood OR loss of interest or pleasure
additionally, must include 4/7: appetite or weight changes, sleep disturbances, psychomotor changes, fatigue, worthlessness or guilt, concentration problems, suicidal thoughts
neurobiological theory of depression
depression is linked to abnormalities in serotonin, norepinephrine, and dopamine
self-care strategies for depression
stress management, journaling, positive self-talk, goal-setting, contacting friends/family, participating in support groups, prayer, meditation, volunteering, problem-solving skills
family teaching on depression
triggers of depression and steps to take, possibility of depression-related symptoms, stress-management techniques, coping skills, options and support services
patient teaching on depression
illness isn't their fault, full range of tx options, incorporating wellness strategies, pharmacologic agents, risk factors and signs of recurrence, adherence, recovery strategies, nutrition and sleep measures, self-care, goal setting, problem solving, social interaction skills, follow-up, support services
medications used to treat depression
SSRIs, SNRIs, MAOIs, TCAs, NDRIs
nursing priorities for depression
safety from self harm, nutrition, sleep, activity, self-care, social functioning, hopefulness
risk factors for depression
prior episode, family history, lack of social support, lack of coping skills, presence of life stressors, current substance use/abuse, medical and/or mental illness comorbidities
addiction
chronic, relapsing condition which involves compulsive drug seeking and continuation despite the harmful consequences and long-lasting brain changes
abuse
using a substance for intoxication
detoxification
the process of safely withdrawing from a substance under medical supervision
recovery
overall goal, partnership with patient, family, and provider
withdrawal
physical and psychological symptoms that can occur when a substance is no longer used
relapse
using a substance again after time of sobriety, considered part of the disorder
tolerance
needing to ingest an increasing amount of drug to feel intended effects; levels are still the same in the body which can damage organs
DSM-V for SUD
impaired control, social problems, risky use, physical dependence
CAGE questionaire
self-report on alcohol use
C - have u tried to CUT down on your drinking
A- have you ever felt ANNOYED by criticism about drinking
G- have you felt GUILT about your drinking
E- have you ever had an EYE opener
CIWA withdrawal assessment of alcohol
assesses for N/V, tremors, paroxysmal sweats, anxiety, panic, agitation, tactile disturbances, auditory/visual disturbances, headache, and decreased LOC
effect of alcohol
short-term: enhances GABA to relax inhibitions and heighten emotions
withdrawal: enhances glutamate and causes anxiety, tremors, hallucinations, increased bp/HR
what do you give for alcohol withdrawal?
chlordiazepoxide if high CIWA score (done every few hours), lorazepam for older people or those with liver conditions; may also need to give antidepressants, anticonvulsants, and antipsychotics
complications from long-term alcohol use
thiamine/folic acid/magnesium deficiency, Wernicke encephalopathy, Korsakoff Amnestic Syndrome, esophagitis, pancreatitis, cirrhosis, stroke, cardiomyopathy, dysrhythmias
what do you give for alcohol recovery?
naltrexone or disulfiram
cocaine effect on NTs
increases dopamine (euphoria), increases norepinephrine (tachycardia, HTN, dilated pupils, increased temp), and increases serotonin (sleep disturbances and anorexia)
amphetamine effect on NTs
increases dopamine, norepinephrine, and serotonin
methamphetamine effect on NTs
releases excess dopamine
club drugs (MDMA, GHB, ketamine) effect on NTs
increases dopamine, norepinephrine, and serotonin
nicotine effect on NTs
increases release of norepinephrine and acetylcholine
why are opioids so addictive?
they bind to opioid receptors all over the body to block pain and cause a euphoric high; they increase dopamine so the body overcompensates by stopping dopamine production, thus people seek out drugs to go back to a normal dopamine state
what is given for opioid detoxification?
methadone, buprenorphine, or naltrexone
FRAMES model (motivational interviewing technique)
Feedback, Responsibility, Advice, Menu of options, Empathy, Self-efficacy
diathesis-stress model
explains addiction as a result of the interaction between a person's underlying genetic/biologic vulnerability (diathesis) and environmental triggers (stress)
akathisia
inability to sit still, restless, rocking in chair, pacing, marching in place, repetitive motions; can be misdiagnosed as psychiatric symptoms; treatment is to change the antipsychotic
tardive dyskinesia
repetitive movements of mouth, face, and tongue like lip smacking, lip puckering, and chewing; irreversible so the best treatment is to monitor
neuroleptic malignant syndrome
high fever, diaphoresis, unstable BP, muscular rigidity; must monitor temp and vitals
pharmacodynamis
medications act at receptors, ion channels, enzymes, and carrier proteins
why do drugs work?
selectivity - ability of a drug to be specific to a receptor
affinity - degree of strength of the bond
intrinsic activity - ability to produce a response
first-pass effect
metabolized in GI tract or liver before systemic absorption
bioavailability
amount of drug reaching systemic circulation
St. John's Wort
used for anxiety and depression, can contribute to serotonin syndrome
AE: GI upset, dizzy, confusion, sedation
Kava
used for anxiety
AE: liver injury, agranulocytosis, GI upset, drowsy, dizzy
Valerian
used for insomnia and nervousness
AE: headache, GI upset, dizzy, vivid dreams
electroconvulsive therapy (ECT)
used for severe depression, catatonia, and treatment-resistant disorders
a brief electrical current is passed through the brain imitating a seizure
AE: hyper/hypotension, brady/tachycardia, h/a, arrhythmia, nausea, amnesia
light therapy
used for SAD and depression
AE: eye strain, h/a, insomnia
transcranial magnetic stimulation (TMS)
used for depression
magnetic field thought to cause brain activation and brain inhibition in areas
AE: h/a, scalp discomfort
vagus nerve stimulation (VNS)
used for treatment-resistant depression and anxiety
sends electrical impulses to vagus nerve and linked to help changing NT levels
AE: voice changes, throat discomfort
SSRIs
includes Citalopram, Escitalopram, Fluoxetine, and Sertraline
MOA: inhibits reuptake of serotonin - improves mood, emotion, energy
Use: depression, anxiety, eating disorders
AE: sexual dysfunction, diastolic HTN, sedation, serotonin syndrome
serotonin syndrome
Potentially life-threatening condition from excess serotonin; mental status changes, autonomic instability, neuromuscular problems, GI issues
SNRIs
includes duloxetine and venlafaxine
MOA: inhibits reuptake of serotonin and norepinephrine
Use: depression (especially those who sleep excessively), anxiety, insomnia, pain
AE: same as SSRIs with high BP
NDRIs
includes Bupropion
MOA: inhibits reuptake of norepinephrine, serotonin, and dopamine
Use: depression, SAD (higher dose), smoking cessation (lower dose)
AE: seizures, CNS stimulant, dry mouth, h/a, N/V, constipation, low risk for sexual dysfunction
NMDA antagonist
includes esketamine
MOA: psychodelic drug that can cause euphoric effects
Use: nasal spray for resistant depression and Alzheimer's
AE: dizzy, sedation, N/V, headache, nasal discomfort, throat irritation
TCAs
includes Trazodone
MOA: acts on several NTs
Use: depression
AE: sedation, OH, anticholinergic effects, tremors, restlessness, dysrhythmias, weight gain
MAOIs
includes phenelzine
MOA: inhibits breakdown of serotonin, norepinephrine, etc
Use: depression
AE: dysrhythmias, drowsy, dizzy, sexual dysfunction, OH, HTN crisis
atypical antipsychotics
includes aripiprazole, clozapine, olanzapine, quetiapine, risperidone, and ziprasidone
MOA: a bunch of receptor blockers
Use: schizophrenia, mania, autism, Tourette's
AE: EPS, NMS, hypo/hyperglycemia, hyponatremia, bronchospasm, agranulocytosis, anticholinergic effects
- clozapine causes constipation
- olanzapine causes increased prolactin/LFTs and decreased bilirubin
- risperidone causes weight gain in youth
- ziprasidone can cause QT prolongation
- aripiprazole can cause compulsive urges
typical antipsychotics
includes chlorpromazine and haloperidol
MOA: a bunch of receptor blockers, mainly dopamine
Use: schizophrenia, mania
AE: EPS, NMS, hypo/hyperglycemia, hyponatremia, bronchospasm, agranulocytosis, anticholinergic effects, QT prolongation, depressed cough reflex
mood stabilizer
includes lithium
MOA: increases dopamine, norepinephrine, and epinephrine, increases GABA, increases serotonin sensitivity
Use: acute mania in bipolar disorder (depression side too)
AE: severe thirst, metallic taste, polyuria, mild diarrhea, fine hand tremor, drowsy, memory problems, weight gain, hypothyroidism
anticonvulsants
includes valproic acid, carbamazepine, and lamotrigine
MOA: reduces repetitive firing of action potentials
Use: bipolar disorder, mania
AE:
- valproic acid causes GI upset, tremors, lethargy, weight gain, and alopecia
- carbamazepine causes dizzy, drowsy, tremor, N/V, and has several drug interactions with erythromycin, verapamil, and cimetidine
- lamotrigine causes a life threatening rash in kids
benzodiazepines
includes diazepam, lorazepam, and alprazolam
MOA: enhances GABA
Use: anxiety, lorazepam for acute alcohol withdrawal, insomnia
AE: sedation, memory impairment, ataxia, dependence, respiratory depression, confusion, low attention
non-benzodiazepine
includes buspirone
MOA: enhances GABA
Use: anxiety, insomnia, GAD
AE: dizzy, drowsy, nausea
antihistamine
includes hydroxyzine
MOA: crosses BBB to induce sedation and anticholinergic action
Use: anxiety
AE: deep sleep, incoordination, sedation, calmness, dizzy, anticholinergic effects
amphetamines
includes methylphenidate, amphetamine/dextroamphetamine, and dexmethylphenidate
MOA: produce alertness, wakefulness, vasoconstriction, and suppressed appetitie
Use: narcolepsy, ADHD in kids, obesity, adjunct to depression fatigue
AE: appetite suppression, insomnia, irritability, weight loss, nausea, headache, palpitations, blurry vision, constipation, dry mouth, dizzy
iron deficiency
fatigue, low mood, anxiety, irritability, anhedonia, sleep disturbances
folic acid deficiency
depression, irritability, confusion, memory issues, insomnia, psychosis
magnesium deficiency
anxiety, depression, mood instability, irritability, personality changes, mental fatigue
vitamin C deficiency
depression, irritability, fatigue, apathy, cognitive impairment
biotin deficiency
depression, fatigue, irritability, ataxia, hallucinations, cognitive impairment