1/66
Looks like no tags are added yet.
define osmolality
measurement of solute concentration in fluid
normal urine specific gravity
1.010 - 1.025
Plasma / Serum Osmolality (normal range)
275-295 mOsm/kg
sodium normal levels
135-145 mEq/L
lab values in fluid volume excess
decrease - because the fluid is diluting the labs
-↓ Na, serum/plasma osmolality, Urine spec. gravity, Hct, BUN
manifestations of FVE
• Headache, confusion, lethargy, seizures, coma (Neurological)
• Peripheral edema: fluid leaking out (hydrostatic pressure)
• Jugular venous distention (JVD)
• S3 heart sound
- HTN
• Bounding pulse, ↑ BP, ↑ CVP
• Polyuria (with normal renal function)
• Dyspnea, crackles, pulmonary edema (Primary Concern HYPOXIA)
• Muscle spasms
• Weight gain
• Tachycardia because it is overworking
nursing management of FVE
Fluid Restrictions
Daily Weight: best indicator, important to be accurate
I & O
Lab Findings: What should you
Cardiovascular Care, Vital Signs, JVD, Edema
Respiratory Care; Crackles, congestion, hypoxia
Skin Care: Risk for breakdown
Patient Safety
R.E.S.T.R.I.C.T
management of FVE
for FVE
Reduce/stop IV
Evaluate breath sounds & ABGs
Semifowlers
Treat w/ Oxygen & diruetics
Reduce fluid
I & Os
Circulation, color, presence of edema
Turn - reposition q2
What is a cornerstone treatment for FVE?
Diuretics
-gets fluid out of the body
expected outcomes of nursing interventions w/ FVE
-Pulse Ox: ↑
-Resp rate: ↓
-Lung sounds: No crackles
-Skin: no edema, pink, warm, dry
-Weight: ↓
-BP: ↓
-HR: ↓
-LOC: ↑
Osmosis / Osmotic Pressure
the ability of the solute concentration to pull water
- the concentration (osmolality) of the solution determines the strength of the osmotic pull
- The higher the concentration, the greater the solution's PULL, or osmotic pressure
Hydrostatic pressure
the force exerted by a fluid against a wall
- causes movement of fluid between compartments
- The hydrostatic pressure of blood is the pressure exerted by blood against the walls of the blood vessels by the pumping action of the heart
3 functions of the Renin-Angiotensin-Aldosterone pathway
1) maintain a proper BP / Blood flow
2) maintain the right concentration of Na in the blood
3) maintain the right amount of water in the blood
aldosterone
"salt-retaining hormone" which promotes the retention of Na+ by the kidneys.
- Na+ retention promotes water retention, which promotes a higher blood volume and pressure
-secreted by adrenal cortex
antidiuretic hormone (ADH)
Hormone produced by the neurosecretory cells in the hypothalamus that stimulates water reabsorption from kidney tubule cells into the blood and vasoconstriction of arterioles.
- retain / hold onto water
A 78 year-old client is admitted with dehydration and urinary tract infection. After IV infusion of 750 mL normal saline, the client begins to cough and asks for the head of the bed to be raised to ease breathing. The nurse's assessment notes jugular vein distention (JVD) and increased respiratory rate. How should the nurse interpret this data?
a.The fluid volume deficit is worsening.
b.The patient is developing hypervolemia.
c.Hypotonic water intoxication is beginning.
d.The infection is causing respiratory compromise.
B
- we have over-corrected
- Hypervolemia = extra volume
- Normal saline is isotonic
Which client to be at highest risk for developing fluid volume deficit?
a.A 52 year-old female receiving corticosteroid therapy for bronchitis.
b.A 60 year-old male S/P left inguinal herniorrhaphy 12 hours ago.
c.A 76 year-old male who has a nasogastric (NG) tube to intermittent suction following colon resection.
d.A 68 year-old female who is NPO and receiving bowel prep for a flexible sigmoidoscopy procedure.
c
3 reasons
-75 + y/o = elderly
-Has NG tube
-and lost part of colon (resorbs water)
A nurse is caring for an elderly adult who is weak and confused; temperature is 38.3C (100.9F), pulse, 92, respirations 20/min, and blood pressure 108/60. He has lost ¾ of a pound and reports dizziness when assisted to the bathroom. Which action should the nurse take?
a. Initiate fluid restrictions and monitor urine output.
b. Observe for signs of peripheral edema.
c. Encourage the client to ambulate to promote oxygenation.
d. Monitor for orthostatic hypotension
d
A nurse is admitting a client experiencing dyspnea, weakness, weight gain of 2 lb. and 1+ bilateral edema of the LE. The client's temp. is 37.2C (99.0F), HR 96, BP 152/96, RR 26, O2 Sat. 94% on O2 at 3L N/C. Which manifestations of FVE should the nurse expect. (Select all that apply)
a. Serum osmolality of 301 mOsm/L
b. Auscultation of crackles
c. Urine specific gravity of 1.003
d. Distended neck veins
e. Cool clammy skin
f. Hematocrit 55%
g. Thready pulse
b, c, d
- in fluid excess lab values go down
a. Serum should be below 275 in FVE
b. True
c. True (normal UGS 1.010-1.025)
d. True
e. cool, clammy caused by vasoconstriction d/t FVdeficit
f. Hct should be low (below 35ish)
g. Pulse would be bounding in FVE
What do we need sodium for?
- Regulate body fluids
- Regulate BP
- Absorption of nutrients
- Nerve and muscle functions
Which interventions does the nurse complete when caring for an elderly client admitted with a sodium level of 152 mEq/L? (Select all that apply)
a.Provide extra blankets for warmth
b.Observe client for nausea and malaise
c.Observe and prepare for possible seizures
d.Restrict additional fluids to 1000 mL per day
e.Monitor serum osmolality
f.Daily weights
b, c, e, f
In caring for a client who is experiencing hyponatremia, which factors probably contributed to this electrolyte imbalance. (Select all that apply)
a.Diuretic therapy
b.Fever
c.Fluid retention
d.Excessive intravenous infusion of hypertonic fluid
e.Heart Failure
f.Nasogastric tube connected to low-intermittent suction.
a, c, e, f
- hypertonic fluid would pull more sodium out of cells increasing Na more
The nurse is caring for a client with heart failure who has received large doses of diuretic therapy. Assessment reveals flat neck veins, generalized muscle weakness, and fatigue. The nurse suspects hyponatremia, what other clinical manifestations would you anticipate?
a. Hyperactive bowel sounds
b. Increased thirst
c. Decreased urine output
d. Increased urine specific gravity
a
-think FVE
-increased urine output and decreased urine specific gravity
The nurse anticipates the client with which condition would be most at risk to develop hyperkalemia? (Select all that apply)
a.Chronic renal failure
b.Newly diagnosed cirrhosis
c.A client involved in a trauma in which a crush injury was sustained
d.A client with a severe MRSA infection of the skin
e.Partial bowel obstruction requiring nasogastric suctioning
f.Diarrhea for the last 4 days
a, c, d
d- bacterial cells die and release potassium when antibiotics are given
Which of the following statements by the client indicates a need for further instruction regarding treatment for hypokalemia?
a."I will eat more bananas and cantaloupes for breakfast."
b."I will eat more bran flakes to increase my potassium level."
c."I will take my potassium in the morning after breakfast so it does not upset my stomach."
d."I will tell my primary care provider if I start having muscle cramps or weakness."
b
A client with hypocalcemia has been started on intravenous (IV) corticosteroids. Which findings indicate to the nurse a further decrease in calcium level in the client?
(Select all that apply)
a.Absence of Trousseau sign
b.Tetany
c.Muscle weakness
d.ECG Changes
e.Abdominal distension and hypoactive bowel sounds
B, D
a. Trousseau sign should be there
b. true
c. muscles are excited not weak
d. true
e. hypOactive bowel sounds = hypERcalcemia
-HypOcalcemia = excitability
The nurse determines that a client with a serum calcium level of 12 mg/dL understands client teaching when the client makes which statement?
a."If my stomach becomes upset, I can just take more Tums."
b."I will need to take my phosphorus supplements once a day."
c."I will need to be on strict bed rest to help with this problem."
d."I will need to drink many more fluids than I have been, even up to two to three liters each day."
d
- to get rid of the excess Calcium and to prevent kidney stone formation
potassium normal levels
3.5-5
hematocrit normal levels
M: 39- 50%
W: 35- 47%
Normal BUN
6-20
Normal Calcium levels
8.6-10.2 mg/dL
lab values in fluid excess
decrease
- all go down cuz they're diluted
lab values in fluid deficit
increase
- all go up cuz they're more concentrated
Causes of FVD
· Fluid intake
· Polypharmacy
· Burns
· Diarrhea
· Trauma, large wounds
· Bleeding
· Surgery
· Chemotherapy
· NG tubes
Diuretics
what drives the plan of care in FVD?
Orthostatic Hypotension
What drives the plan of care in FVE?
Weight gain
manifestations of FVD
· Orthostatic Hypotenion - drive for POC
· Cool extremities - blood shift to organs
· Mental status changes
· Decreased skin turgor
· Dry mucous membranes
· Low-grade fever
· Tachycardia
· Weak pulses
· Increased capillary refill (>3 sec)
· Increased Thirst
· Confusion, dizziness
· Weight loss
- Dark, amber urine
Lab values associated w/ FVD
Concentrated - Increased levels
Sodium = >145
Serum Osmolality = >295
Urine Specific Gravity = >1.025
Hct = ↑ (M>50%; F>47%) [look for number in the 50s]
Hgb = ↑
BUN = >20
F. L. U. I. D. S.
management of FVD
For FVD
Fluid ↑ orally & IV
LOC
Urine - want it > 30 mL/hr
IV fluids & I&Os
Document & monitor VS
Shock - hypovolemic shock
S. A. L. T.
manifestations of hypernatremia/FVD
skin flushed
agitation
low grade fever
thrist
When we see sodium we should think?
Neuro
- neuro changes
Causes of Hypernatremia
Think Fluid volume Deficit
· NPO
· Excess Sodium intake
· Inadequate water intake
· Excess Water loss (↑ sodium concentration)
· Diseases (Diabetes insipidus, primary hypoeraldosteronism, Cushing's, uncontrolled diabetes)
· Infection, fever
· Coma
- Kidney failure
Manifestations of hypernatremia
· Restlessness, Agitation, Lethargy, Coma
· Seizure
· Weakness, muscle cramps
· Intense thirst, dry swollen tongue, sticky mucous membranes
· Postural hypotension, weight loss, tachycardia
· Confusion
· Irritable
· Nausea & vomiting
· Tremors
Hyperreflexia
Nursing interventions for hypernatremia
· Freq neuro assessments
· Document seizures, if happen
· Daily weights, I&Os
· Replace fluids slowly so you don't cause increased shift (avoid shock)
· Restrict salt if it was caused d/t increased salt intake
When we see hyponatremia we think
Fluid volume Excess
causes of hyponatremia
· Excess sodium loss
· GI loss (diarrhea, vomiting, fistulas, NG suctioning)
· Renal loss (diuretics, adrenal insufficiency, Na+ wasting renal disease)
· Skin loss (burns, wound drainage)
· Inadequate sodium intake (fasting diets)
· Excess water gain (↓ sodium dilution) (Excess hypotonic fluids, primary polydipsia)
· Diseases (SIADH, heart failure, primary hypOaldosteronism, cirrhosis)
manifestations of hyponatremia
think Neuro
· Neuro (irritability, confusion)
· Muscle twitching, tremors,
· Coma
· Delirium
· Possible seizures
· Lethargy
· Headache
· Apprehension
· Dry mucous membranes
· Postural hypotenion
· Tachycardia, thread pulse
nursing interventions for hyponatremia
· Neuro assessments
· Document seizures
· Daily weights
· I&Os
· Check for bounding pulses or bulging neck veins
· Check for BP and Resp changes
· Changes in sensorium and signs of cerebral edema
Check for pitting edema
when we see potassium we think the
heart
Causes of hyperkalemia: MACHINE
M: meds (ACE inhibitors, NSAIDs)
A: acidosis (in acidosis, there is cell death → ↑K+)
C: cellular destruction (burns, injury)
H: hypoaldosteronism, hemolysis
I: intake excessive
N: nephrons (renal) failure
E: excretion impaired
manifestations of hyperkalemia
· Muscle twitching (stomach, heart) → cramps (abdominal cramps, diarrhea) → Paresthesia
· Irritability, fatigue, & anxiety
· Decreased reflexes
· ↓BP
· EKG changes - peaked T waves
· Dysrhythmias - irregular rhythm
· Abdominal cramping
· Diarrhea - stomach trying to get rid of excess K+
Confusion, tetany
nursing interventions for hyperkalemia: KIND
· Kayexalate* (binds to excess K+ and is secreted via diarrhea)
· Insulin - given to push K+ back into cells
· Na & Bicarb (Pt at risk for acidosis)
· Dialysis is sometimes needed
- Avoid foods high in K+ (banana, avocado, dried fruit, cantaloupe, broccoli)
- Telemonitor
Which hormones are associated with calcium?
Parathyroid hormone and Calitonin
PTH
pulls Calcium from bones
Calcitonin
keeps Calcium inside bones
causes oh hypokalemia
· GI loss: diarrhea, vomiting, fistulas, NG suctioning, ileostomy drainage
· Diaphoresis
· Diuretics (loop, thiazide, antibiotics, insulin, over-use of laxatives)
· Dig toxicity - causes bradycardia, dysrhythmias
· Hyperventilation
· Severe vomiting & diarrhea
↓intake of K+ rich foods
manifestations of hypokalemia: SUCTION
Skeletal muscle weakness
U waves - Dysrhythmias
Constipation
Toxic effects of digoxin
Irregular, weak pulse
Orthostatic hypotension
Numbness; paresthesia
· Shallow respirations - weakness in diaphragm
· ↓ intestinal motility, ↓bowel sounds, possible ileus, nausea, vomiting
· Tachycardia, tachypnea, anorexiA
Nursing interventions for hypokalemia
· Oral or IV potassium chloride (KCl) supplements and increased intake of K
· Never given IV push
· Monitor urine output - to ensure kidney function
Pt on cardiac monitor
Calcium plays a key role in
- skeletal muscle contraction
- Cardiac muscle contraction
- Smooth muscle contraction
- Blood clotting
- Transmission of nerve impulses
causes of hypocalcemia
Increased Excitability
· HypOparathyroidsim
· Decreased intake
· Excess laxatives
· Crohn's disease
· Some meds (dilantin)
· Chronic renal disease/failure
· Loop diuretics
· Lack of Vit. D
· Lactose intolerance
· Tumor lysis syndrome
· Chronic alcohol use
Alkalosis
Manifestations of hypocalcemia
· Increased excitability
· Paresthesias fingers & toes
· Chvostek & Trousseau's sign
· Anxiety, muscle twitching
· Confusion, depression, irritability
· Dementia
· Increased risk for fracture if prolonged hypocalcemia
· At risk for laryngospasms and bronchospasms - can compromise the airway
Convulsions
Arrhythmias
Tetany
Spasm & Stridor
Nursing management of hypocalcemia
· Increase dietary Ca and Vit d (canned sardines, salmon, green leafy veggies, beans)
· Monitor for bleeding (gums, mucous membranes, not clotting appropriately)
· IV calcium Gluconate if symptomatic
· Calcium oral replacement (w/ milk if GI upset)
· Exercise if able
· Seizure precautions
· Oxygen, trach tray & suction, bag mask → for Laryngospasm & bronchospasm
· Cardiac monitor
· Respiratory status
Tetany
causes of hypercalcemia
· Primary hypERparathyroid hormone (too much pulling from bone into blood)
· Lack of weight bearing
· Immobility
· Meds (Ca-containing antacids)
· Cardiac arrest
· Excess Ca supplements
· Hematologic cancer
· Cancers w/ bone metastasis
Acidosis
manifestations of hypercalcemia
Decreased excitability
· HypOactivity
· HypOreflexia
· Fatigue
· Lethargy
· Confusion
· Depression
· Px changes
· Bone pain, fractures
· HTN
· Bradycardia
· Arrhythmias
· 1st complain of GI issues - ↑ thirst, nausea, vomiting, ↓bowel sounds
Polyuria, dehydration, kidney stones - kidneys working to get rid of Ca - can sometimes lead to renal failure
Nursing interventions for hypercalcemia
· Fluids 2 - 4 L, to promote excretion of Ca and ↓ chance of kidney stone formation
· Encourage weight bearing exercise
Fall safety
increased excitability is associated with
hypOcalcemia
decreased excitability is associated with
hypERcalcemia