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umbilical cord prolapse classification
emergency, immediate intervention
umbilical cord prolapse is when
the umbilical precedes or lies alongside presenting part
risk factors for umbilical cord prolapse
malpresentation
polyhydramnios
multiple gestation
preterm
overt prolapse
palpable/visible
occult prolapse
alongside, not past
what to do with cord prolapse
elevate present part with gloved hand
maintain pressure
nuchal cord is
cord looped around fetal neck
loose nuchal cord intervention
slip over head before birth or shoulders
tight cord intervention
clamp and cut before the birth
nuchal cord classified as
usually not harmful, FHR monitored closely
placental abruption is
premature separation of placenta from uterine wall
placental abruption cuases
htn
preeclampsia
trauma
cocaine/tobacco use
partial placental abruption shows as
late decelerations
changes in variability
some placenta still hanging on
complete placental abruption is
emergency need
category 3
bradycardia
general anesthesia needed
placental abruption presents as
painful
dark red bleeding
board like abdomen
how can placental abruption be delivered
vaginally if hemodynamically stable
placenta previa presents as
painless bright red bleeding
placenta previa is
placenta covers os
c- section required
avoid digital exam
placenta accreta spectrum is
abnormal invasion of myometrium
accreta is
superficial invasion
increta is
into the muscle, might need hysterectomy
percreta is
invading organs
requires multiple surgeons and hysterectomy
uterine rupture indication
pop or sudden relief of pain during labor
uterine rupture risk factors
prior uterine surgeries
5+ deliveries
oxytocin hyperstimulation
uterine rupture symptoms
sudden abdominal pain
loss of FHR
maternal shock
uterine rupture treatment
stat section, possible hysterectomy
uterine tachysystole is
>5 contractions in 10 mins
uterin hyperstimulation is
hypersystole with adverse FHR changes
uterine tachysystole causes
oxytocin
misoprostol
cervical ripening agents
uterine tachysystole interventions
stop or reduce oxytocin
left lateral position
O2
IV fluid
terbutaline 0.25 mg SQ if ordered
uterine inversion is
uterine fundus collapses into uterine cavity thru cervix, rare but life threatening
uterine inversion risk factors
excessive cord traction
fundal pressure of uncontrolled uterus
uterine inversion symptoms
postpartum hemorrhage
severe pelvic pain
non palpable fundus
profound shock
amniotic fluid embolism is
catastrophic entry of amniotic fluid/fetal cells into maternal circulation
amniotic fluid embolism symtpoms
sudden hypoxia
hypotension
altered mentation/coagulopathy
amniotic fluid embolism occurs
during labor
delivery
immediately post partum
amniotic fluid embolism can lead to
respiratory arrest
cardiac arrest
DIC
amniotic fluid embolism is usually due to
induction of labor
A in amniotic fluid embolism treatment
atropine
increase contractility of heart
O in amniotic fluid embolism treatment
ondansetron
blocks more uptake of amniotic fluid
K in amniotic fluid embolism treatment
ketorolac
busts the clots
oligohydramnios can be caused by
post dates
IGUR
ROM
renal problems
oligohydramnios can cause
cord compression, variable decels
oligohydramnios treated with
amnioinfusion
polyhydramnios can be caused by
maternal diabetes
fetal GI obstruction
twin to twin
polyhydramnios is a what risk
cord prolapse
malpresentation
polyhydramnios can cause
preterm labor
placental abruption
shoulder dystocia is classified as
fetal hypoxia risk, urgent
shoulder dystocia is
anterior fetal shoulder behind maternal pubis
shoulder dystocia warning signs
turtle head
macrosomia, diabetes, prior shoulder, prolonged labor
HELPERR in shoulder dystocia
H - call for help
E - evaluate for episiotomy (E)
L - legs (mcRoberts)
supine, legs far back, heels tilting
P - suprapubic pressure
rock on top of shoulder, pubic bone pressure
E - enter
R - remove posterior arm
R - roll patient
intrauterine fetal demise is
fetal death >20 weeks
intrauterine fetal demise causes
placental abruption
infection
hypertension
intrauterine fetal demise diagnosed by
ultrasound, always need confirmation
intrauterine fetal demise delivery
vaginally if stable
intrauterine fetal demise risks
DIC over time after demise
intrauterine fetal demise bereavement care
sit w/ them. verify feelings, offer to call
DIC is
simultaneous clotting and bleeding
DIC causes
placental abruption
fetal demise
sepsis
AFE
severe preeclampsia
DIC labs
decreased fibrinogen
increase PT/PTT
increase d-dimer
decrease PLT
decrease H/H
tranexamic acid should be given
within 3 hours of delivery, for PPH >500 mL
tranexamic acid is a
antifibrinolytic agent
tranexamic prevents
fibrin degeneration
tranexamic window peak
6-10 hours post delivery
tranexamic dose
1 g IV over 10 mins
tranexamic reduces
blood transfusion need