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Cataracts
Lenticular opacification
Leading cause of preventable blindness
Cataracts risk factors
50-70s
UV light exposure
Steroid use (typically oral)
Smoking
When should you refer out for cataract Sx?
20/40 VAs
Pt daily life is affected
Can’t see retina
Typically refer out 70-80s
What does vision with cataracts look like?
Loss of details
Blurry vision
Glare/halos
Nuclear Sclerotic Cataract (NSC)
Yellowing of the lens from urochrome pigment deposition in the nucleus
NSC clinical findings
Myopic shift due to changes in refractive index of lens
Becomes more brown with age
Cortical cataracts
Opacities start in periphery and grow central
Pt experiences glare, decreased vision for dist (driving at night when pupil is dilated) and near (if in visual axis), and decreased contrast sensitivity
Cortical cataracts clinical findings
Clefts: Accumulation of fluid between fibers
Wedges/spokes: Clefts that collect cellular debris and become opaque
Vacuoles: Fluid filled bubbles within cortex
Posterior subcapsular cataract (PSC)
Associated with long term oral steroids and diabetes, appears central
Found in younger pts (50-60s)
Pts complain of glare, difficulty seeing in bright light, or reduces near VA
PSC clinical findings
Granular, vacuolated, or plaque like opacity that is on visual axis
Push through on retro because on posterior capsule
Hypermature cataract
Age related cataract that kept worsening because it was never removed
Pt will have severe reduction in VA (20/400 or hand motion)
Hypermature cataracts clinical findings
Wrinkling of ant capsule
Liquification of cortex
Shrinking of nucleus
Hypermature cataracts complications
Secondary glaucoma
Morgagnian cataract
Even more advanced hypermature cataract
Pt will have significant reduction in VA
Morgagnian clinical findings
Progressive liquification of the cortex → Milky white
Nucleus will become free floating and drop inferior
Morgagnian complications
Secondary glaucoma