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Flaccid Dysarthria
Damage: lower motor neurons OR PNS
Flaccid: weak, limp
characteristics: weakness, hypotonia, atrophy, fasciculations, breathiness, hypernasality & imprecise consonants
Dysarthria
speech disorder caused by impaired control of the muslces used for speech
affects breathing, voice, articulation & prosody
slurred, weak, strained, slow, abnormal
Lower motor neuron
carries signals from the spinal cord or brainstem directly to muscles
activate muscle movement
damage: weak, floppy, atrophy, reduces tone, reflexes & causes muscle twitching
FLACCID DYSARTHRIA
Peripheral nervous system PNS
nerves outside the brain and spinal cord
connect the CNS to muscles, skin, and organs
damage: communication b/w brain and muslces disrupted, leading to weakness, sensory loss or impaired movement
FLACCID DYSARTHRIA (weak/breathy)
Hypotonia
reduced muscle tone
poor posture, weak facial expression, difficulty w/ speech & swallowing
FLACCID - reduced speech strength/control
Atrophy
wasting away of body tissues (muscle) b/c it is not being used normally OR lost its nerve supply
nerve damage, aging, or chronic disease
FLACCID DYSARTHRIA (LMN, PNS)
Fasciculations
small involuntary muscle twitches that often seen under the skin
damage to motor nerve supply LMN
FLACCID
Spastic Dysarthria
UMN on both sides of the body
spastic: stiff, tight, overly active, increased tone and resistance to movement
speech: strained, slow, effortful
BILATERAL umn damage
increased muscle tone, slow speech, strained-strangled voice, imprecise consonants & hypernasality
upper motor neuron
nerve cell that sends movement commands down to the LMN
UMN: spastic & unilateral UMN dysarthria
severe spastic pattern = bilateral
milder = unilateral
they plan & control voluntary movement
Cerebellar system
cerebellum & connections
regulate coordination, timing, balance & smooth movement
DOES NOT INITIATE MOVEMENT
fine-tunes it so its accurate/well-times
uncoordinated or poorly timed movements
ATAXIC dysarthria
characteristics: coordination, timing, artic, prosody
Ataxic dysarthria
damage to cerebellar system
ataxia = lack of coordination
irregular, uneven, poorly timed
coordination timing articulation prosody
basal ganglia
regulate movement
starting, stopping, smoothing, scaling movement
too slow & reduced or too excessive and involuntary
HYPO & HYPERkinetic dysarthria
HYPOkinetic dysarthria
basal ganglia dysfunction results in reduced movement
smaller/less forceful, or diminished
speech: quiet, monotone, rushed —> PD
reduced loudness, monopitch, reduced stress, imprecise consonants and short rushes of speech
HYPERkinetic dysarthria
basal ganglia dysfunction leads to excessive involuntary movement
excessive uncontrolled abnormal
HUNTINGTONS
Mixed dysarthria
two or more dysarthria types
ALS, MS
ALS
amyotrophic lateral sclerosis
progressive neurodegenerative disease that affects motor neurons
upper and lower
MIXED flaccid & spastic dysarthria
PD
neurological condition that affects movement, usually causing
tremor, slowness & rigidity (basal ganglia)
softer, flatter less expressive speech
HYPOKINETIC DYSARTHRIA