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normal periodontium
group of tissues that surround, support, and attach the tooth to the jaws
consists of gingiva, periodontal ligament, cementum, alveolar bone
gingiva
portion of oral mucosa that covers the alveolar processes of the jaws and surrounds the necks of the teeth
usually pinker, more keratinized, and firmer
gingival anatomy (coronal to apical) —
free/marginal gingiva → unattached gingiva surrounding tooth
approximately 1mm wide
free gingival groove separates it from attached gingiva
attached gingiva → continuous with marginal gingiva
firm, resilient, tightly bound to underlying periosteum / alveolar bone
apical border is mucogingival junction
mucogingival junction → separates attached gingiva from alveolar mucosa
alveolar mucosa → redder, less keratinized, more movable
gingival sulcus
small space between tooth and free gingiva, reaching to junctional epithelium
healthy sulcus = 1-3mm; mean of 1.8mm
facial = 2mm, lingual = 2mm, interproximal = 3mm
interproximal probing is deeper because gingival architecture between teeth rises coronally and includes the COL area
probing affected by many factors → inflammation, morphology, angulation, interproximal anatomy, tissue resistance
attached gingiva width
amount of attached gingiva varies depending on location and between individuals
least attached gingiva occurs around first premolars
maxillary anterior → 3.5-4.5 mm
mandibular anterior → 3.3-3.9 mm
maxillary first premolar → 1.9 mm
mandibular first premolar → 1.8 mm
keratinized gingiva
keratinized gingiva = free gingiva + attached gingiva
hard palate consists of firmly attached keratinized tissue
interdental gingiva
pyramidal or col-shaped gingiva that occupies space between adjacent teeth
col conforms to shape of interproximal contact
col → depression or valley between facial and lingual portions of interdental papilla
shape of interdental gingiva is a function of the contact point
embrasures
space created around contact area between adjacent teeth
buccal, lingual, incisal, gingival
in healthy gingiva → gingival embrasure normally filled by soft tissue
important in restorative and prosthetic treatment
if contact-to-bone distance is ≤ 5mm, the papilla is more likely to fill the embrasure
as distance becomes > 5mm, probability of papilla to fill embrasure decreases and leads to an open gingival embrasure or “black triangle”
diastema
space between adjacent teeth
no normal interdental papilla within open contact area
gingiva becomes firmly bound over underlying interdental bone
clinical features of healthy gingiva
color → coral pink
depends on blood supply, thickness of tissue, degree of keratinization, pigmented cells
alveolar mucosa = red, smooth shiny
attached gingiva = pink, stippled
color alone does not diagnose periodontal disease, but is useful for monitoring gingivitis and response to therapy
pigmentation → melanin is a non-hemoglobin-derived brown pigment responsible for pigmentation of skin, gingiva, oral mucosa
size → depends on cellular components, intercellular components, blood supply, genetic tissue thickness
enlargement due to inflammation and edema or fibrosis
gingival overgrowth produced by some medications
can be indication of health or disease following therapy
contour → depends on shape of teeth, alignment, contact, embrasures
healthy gingiva described as scalloped with knife-edge margin
parabolic when interproximal more coronal than direct facial or lingual
shape → shape of interdental gingiva depends on proximal tooth contour, embrasures, contact relationship
broad vs narrow papilla
bone loss or disease if teeth are in contact but papilla is blunted or craters
consistency → firm, resilient in health
gingival fibers contribute to firmness; diseased if spongy
free gingiva not bound to underlying bone
texture → related to keratinization that protects against friction, microbial invasion, desiccation
stippling is small dimpled surface texture on attached gingiva, produced by protuberances and depressions in gingiva
in health, free gingiva is smooth or stippled
edema caused by inflammation forms a glossy surface and loss of stippling
position → relationship between gingival margin, sulcus, junctional epithelium, tooth changes as teeth erupt
margin normally at CEJ, but moves coronal to form pseudopocket in hyperplasia or edema
position of mucogingival junction influences amount of attached/keratinized gingiva
continuous tooth eruption
when teeth reaches their functional antagonists, gingival sulcus and gingival epithelium are still positioned on enamel and clinical crown is about 2/3 of anatomic crown
active eruption → tooth moves towards the occlusal plane
compensates for attrition to preserve vertical dimension of occlusion
cementum deposited around root apices and furcation areas, with bone forming at crest
as crown structure wears down, eruption/tooth-related changes maintain tooth’s functional relationship
passive eruption → gingiva moves apically, exposing more tooth structure
normal apical migration doesn’t occur appropriately, resulting in gingival attachment on enamel and clinical crown appearing shorter
anatomic crown is covered by enamel; clinical crown is clinically exposed or visible
anatomic root is covered by cementum; clinical root is covered by PDL
bleeding on probing
healthy sulcus will not bleed upon probing
with inflammation:
sulcar epithelium becomes ulcerated
underlying connective tissue undergoes vasodilation
bleeding is an indicator of gingival inflammation
purulent exudate
no purulent exudate in health
purulent → containing or accompanied by pus
exudate → material that escapes blood vessels, usually as part of inflammation