DPT 652 Midterm Study Guide: Balance, Gait, Orthotics, CVA, SCI, Cerebellar Ataxia, and MS

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Vocabulary practice flashcards covering postural balance strategies, sensory organization, gait deviations, lower-limb orthotics, stroke syndromes and recovery, spinal cord injury levels and syndromes, cerebellar ataxia, and multiple sclerosis clinical characteristics.

Last updated 10:37 PM on 10/8/26
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45 Terms

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Ankle Strategy

A postural motor strategy used for small, slow perturbations on a firm surface; moves the body as a single unit around the ankles with muscle activation firing distal to proximal.

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Hip Strategy

A postural motor strategy used for larger or faster perturbations, compliant surfaces, or when the base of support (BOS) is smaller than the feet; produces rapid movement at the hips with antiphase ankle movements and serves as the primary strategy for recovering stability in the mediolateral direction.

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Stepping Strategy

A postural motor strategy used when ankle and hip strategies are insufficient, involving taking a step to bring the base of support (BOS) back underneath the center of mass (COM).

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Feedforward Mechanism (Anticipatory)

Postural adjustments occurring before movement or disturbance to prepare and stabilize the body (e.g., activating trunk muscles before raising an arm).

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Feedback Mechanism (Reactive)

Postural responses occurring after an external disturbance to correct an error and regain stability (e.g., taking a step after being pushed).

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CTSIB Condition 2

Clinical Test of Sensory Integration and Balance condition consisting of standing on a firm surface with eyes closed; tests somatosensory/proprioceptive reliance as vision is eliminated.

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CTSIB Condition 5

Clinical Test of Sensory Integration and Balance condition consisting of standing on a foam surface with eyes closed; isolates and tests the vestibular system because somatosensory information is inaccurate and vision is unavailable.

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Foot Slap

A gait deviation at initial contact/loading response caused by dorsiflexor weakness or an impaired eccentric control of the plantarflexors.

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Knee Buckling (Loading Response)

Excessive knee flexion during loading response resulting from quadriceps weakness or poor eccentric quadriceps control.

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Anterior Trunk Lean (Loading Response)

A gait deviation during loading response used as a compensation for quadriceps weakness.

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Posterior Trunk Lean (Loading Response)

A gait deviation during loading response resulting from hip extensor or gluteal weakness.

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Early Heel Rise

A gait deviation during stance phase caused by limited dorsiflexion ROM, heel pain, gastrocnemius/soleus spasticity, or plantarflexor tightness/contracture.

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Knee Thrust into Extension

Knee hyperextension during stance caused by quadriceps weakness, glute/hip weakness, spasticity, or plantarflexor contracture.

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Trendelenburg Gait

Contralateral pelvic drop during single-limb stance caused by hip abductor weakness, specifically weakness of the gluteus medius.

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Hip Hiking (Swing Phase)

Elevation of the pelvis during swing to compensate for decreased foot clearance, often caused by inadequate knee flexion or ankle dorsiflexion.

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Posterior Leaf Spring AFO

An ankle-foot orthosis providing dorsiflexion assistance during the swing phase of gait while permitting normal plantarflexion during loading response.

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Carbon Posterior Leaf AFO

An orthosis designed to provide energy storage and return, assisting with dynamic swing-phase clearance and smoother transitions.

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Custom Solid AFO (Tuned)

An orthosis providing maximum ankle immobilization to control severe spasticity or instability.

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Anterior Shell AFO

An orthosis designed to limit dorsiflexion in stance, preventing the knee from buckling or collapsing forward during the stance phase of walking.

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Swedish Knee Cage

An orthotic device used to prevent knee hyperextension.

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Anterior Trimlines

Brace trimlines where material extends more anteriorly, providing increased control and allowing less motion.

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Posterior Trimlines

Brace trimlines where material provides less contact, providing decreased control and allowing more motion.

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Brunnstrom Stage 1

The post-CVA motor recovery stage characterized by flaccidity and no active limb movement.

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Brunnstrom Stage 3

The post-CVA motor recovery stage characterized by voluntary control of movement strictly within synergy patterns and maximized spasticity (peak tone).

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Brunnstrom Stage 4

The post-CVA motor recovery stage characterized by movement beginning outside of synergy patterns and a decrease in spastic tone.

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Middle Cerebral Artery (MCA) Syndrome

A vascular stroke presentation with contralateral face and upper extremity weakness greater than lower extremity weakness (face+arm>leg\text{face} + \text{arm} > \text{leg}), contralateral sensory loss, aphasia (if dominant hemisphere), or neglect (if non-dominant hemisphere).

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Anterior Cerebral Artery (ACA) Syndrome

A vascular stroke presentation with contralateral lower extremity weakness greater than upper extremity weakness (leg>arm\text{leg} > \text{arm}), lower extremity sensory loss, possible behavioral/personality changes, and urinary incontinence.

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Posterior Cerebral Artery (PCA) Syndrome

A vascular stroke presentation primarily characterized by contralateral visual field deficits, possible sensory deficits, and memory/language impairments if the dominant hemisphere is involved.

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Head/Hip Relationship

A functional transfer and mobility principle in spinal cord injury rehabilitation stating that the head must move in the opposite direction of the intended movement of the hips.

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Tenodesis Grasp

A functional passive grip observed at the C6 SCI level where active wrist extension naturally produces passive finger flexion.

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C7 Spinal Cord Injury Level

A major functional transition level in SCI marked by triceps innervation, allowing independent transfers without a sliding board, independent wheelchair push-ups for pressure relief, and manual wheelchair mobility.

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Anterior Cord Syndrome

An incomplete cord injury syndrome typically caused by flexion trauma damaging the anterior spinal artery, corticospinal tract, and spinothalamic tract; results in loss of motor, pain, and temperature function below the lesion with preserved dorsal column function (vibration, conscious proprioception, and discriminative touch).

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Central Cord Syndrome

An incomplete cord injury typically caused by hyperextension trauma; characterized by motor and sensory loss affecting the upper extremities greater than lower extremities (UE>LE\text{UE} > \text{LE}) with sacral sparing.

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Brown-Séquard Syndrome

An incomplete spinal cord hemisection injury caused by penetrating trauma; produces ipsilateral loss of motor function, light touch, vibration, and proprioception below the lesion, along with contralateral loss of pain and temperature starting 1–21\text{--}2 segments below the lesion.

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Autonomic Dysreflexia

A life-threatening medical emergency occurring in spinal cord injuries at or above T6T6 triggered by noxious stimuli (e.g., full bladder, blocked catheter, UTI, constipation, pressure sore) causing sudden hypertension; managed by immediate removal of the noxious trigger.

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Heterotopic Ossification (SCI)

Abnormal bone formation in soft tissue typically arising within approximately 4 months4\text{ months} post-injury around the hips and knees; manifests with redness, heat, decreased ROM, and a bony end-feel.

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Dysmetria

An impaired ability to accurately judge distance or force during movement, commonly observed with cerebellar pathology.

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Dysdiadochokinesia

Difficulty in performing rapid, alternating movements, indicative of cerebellar dysfunction.

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Intention Tremor

A tremor that increases in amplitude and worsens as the limb approaches a targeted destination, characteristic of cerebellar ataxia.

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Sensory Ataxia vs. Cerebellar Ataxia

Sensory ataxia stems from lost proprioceptive/sensory input, displays a positive Romberg test, and improves significantly with visual guidance; cerebellar ataxia is an intrinsic motor coordination deficit that does not resolve with visual input.

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Relapsing-Remitting MS (RRMS)

The most common form of multiple sclerosis (70%70\% of cases), characterized by temporary flare-ups (relapses) followed by periods of recovery without disease progression (remissions).

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Secondary-Progressive MS (SPMS)

A clinical course of multiple sclerosis that begins as relapsing-remitting MS and gradually shifts into continuous, worsening disease progression (developing in approximately 80%80\% of RRMS patients).

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Primary-Progressive MS (PPMS)

A form of multiple sclerosis accounting for 10–15%10\text{--}15\% of cases, characterized by continuous symptom worsening from disease onset without distinct relapses or remissions.

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Lhermitte's Sign

A neurological symptom seen in multiple sclerosis characterized by an electric-shock sensation radiating down the spine and into the limbs upon neck flexion.

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4 P's of Energy Conservation

Pacing, planning, prioritizing, and positioning; principles utilized in multiple sclerosis rehabilitation to manage fatigue.