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Dysphagia
Difficulty swallowing, often secondary
Unaddressed dysphagia may cause
airway obstruction, dehydrating, malnutrition, aspiration pneumonia
conditions associated with dysphagia
CVA, MS, PD, ALS, CP, tumors of head/neck/esophagus
4 stages of swallowing
oral preparatory phase
Oral phase
Pharyngeal stage
Esophageal stage
Oral preparatory phase
food is recieved
Oral phase
Tongue propels food back into the pharynx (1 second)
Pharyngeal phase
Food is carried through pharynx with laryngeal closure (epiglottis), also 1 second
Esophageal stage
esophageal peristalsis carries the bolus through esophagus into the stomach (5-15 seconds)
What needs to halt during swallowing
respiration
Penetration
entry of material into laryngeal vestibule (above vocal cords)
Aspiration
Entry of material into the airway below the level of vocal cords
Silent aspiration
no cough with aspiration
Signs of aspiration
fever, SOB, elevated WBC
Bedside swallow examination
SLP will bring foods of different consistencies and evaluate what foods are safest to eat
Instrumental examination
done when there is evidence of aspiration, modified barium swallow evaluation, fluoroscopic view of the throat while someone is swallowing
Fiberoptic endoscopic exam
tube with camera down throat to evaluate the structure of the pharynx/larynx
NPO
nothing by mouth
If long term problem with swallowing, will get nutrition by
gastrostomy tube: direct to stomach
Jejunostomy tube: direct to intestines
Short term swallowing problem will be treated me
NG tube
Treatment of dysphagia
diet modifications
Postural changes
Strengthening/ROM
EMG/biofeedback/NMES
Function of pelvic floor muscles
close urinary and anal sphincters
Closing of vaginal opening
Support abdominal viscera
Sexual function
Incontinence is more common in
women
These normally prevent fecal incontinence
internal and external anal sphincters and puborectalis muscle
Defacation is initiated by
Rectal filling, PNS response, voluntary relaxation of puborectalis muscle and external sphincter, assisted by valsalva maneuver
What happens to PFM with aging
weakness, loss of elasticity
Most common cause of bowel incontinence
Fecal impaction, associated with constipation and diarrhea
Leakage can be associated with
sensation or muscle tone losses, cognitive decline
Chronic fecal impaction
caused by inadequate fluid intake, chronic laxative abuse, poor bowel habits. These things lead to liquid stool leaking from higher in the colon, draining at the anus
Dx of incontinence
patient history
Physical exam
Neuro exam
Mobility/hygiene
Mental function
Tax of incontinence
medications
NM reeducation
Strengthening PFM
Loperamide
treats diarrhea
Normally, urine remains in the bladder for as long as the pressure is
lower than urethral resistance, then the detrusor contracts forcing urine into the urethra
Urge incontinence
person feels the need to urinate, but is unable to get to the toilet before urination occurs. Most commonly triggered by an external stimulus, caused by detrusor instability
Stress incontinence
Cough/strain/sneeze causes urination, most common in middle aged women
Overflow incontinence
bladder is overly distended, causing bladder pressure to exceed urethral pressure, causing small and continuous urine loss
Functional incontinence
difficult mobility/cognitive function causing incontinence
DIAPPERS
Causes of incontinence
delirium
Infection
Atrophic urethritis/vaginitis
Pharmaceuticals
Psychological disorders
Endocrine disorders
Restriction mobility
Stool impaction
Pharmaceuticals causing incontinence
diuretics
Anticholinergic agents
Sedatives
LT alcohol abuse
Iatrogenesis
Injury that is a result of medical treatment that was done improperly
Iatrogenesis risk is increased
By 2x over age of 65
Cascade Iatrogenesis
development of multiple complications initiated by a seemingly innocuous initial event
This alters distribution
decreased total body water and lean body mass
This alters metabolism
decreased liver mass and blood flow
This alters elimination
Decreased renal plasma flow and GFR
Other causes of ADRs
T2DM, CRF, dementia
Antipsychotics can cause
Parkinsonism, tardive dyskinesia (involuntary movement of tongue and lips)
Testosterone replacement can cause
prostate cancer
May increase falls
analgesics and antidepressants
Used to reduce menopausal symptoms
limiting caffeine/alcohol, stress reduction, light colored therapy, SSRIs, estrogen replacement (vaginal ring)
Dizziness increases risk of
Falls
Most common cause of dizziness
medication changes
Nystagmus
Involuntary rhythmic oscillation of the eye
Vertigo
illusion of movement, rotary component (the room is spinning), caused by inner ear dysfunction or cerebellar stroke
Most common cause of dizziness
BPPV, when otoconia within otolith become dislodged and symptoms are caused with a certain change in head position
Treat BPPV
epley and seamount
Can be used to dx peripheral vestibular disorder
Hallpike did, head thrust test or head impulse test
DHI
dizziness handicap inventory, over 60 increases risk of falls
Most common medication used to treat dizziness
meclizine, best to reduce during PT
Do not respond well to conservative treatment
menieres disease, perilymphatic fistula
Three categories of unsteady patients
dizzy
Balance disorders
Both
Frequent falls mean
over 2 within the past 6 months
This indicates exercise was too difficult
dizziness lasts over 20 minutes after exercise
Self reported balance assessment
FES, ASBCS
Balance assessment
Berg, Functional reach test, SLS, tandem
SOT
expensive version of foam and dome
CTSIB
foam and dome
CSTIB and SOT are used to determine
if the problem is visual, somatosensory or vestibular
Bone healing phases
inflammatory phase (24-48 hrs)- bleeding
Reparative phase (4-6 weeks)- collar formation
Remodeling phase- bony trabeculae forms, osteoclasts remove necrotic bone
Can impair bone healing
osteoporosis, DM, decreased vitamin D and Ca, ORIF
2-4 weeks
callus forms
6 weeks
bony bridging
Stress fractures are typically
intramedusllary and non-displaced
Microfractures
often the patient is unaware, silent fracture
Pathological fracture
Occur from metastasis, commonly have night pain/pain with rest
Osteoporosis related fractures are typically caused by
increased osteoclast activity and decreased osteoblast activity, affecting the cancellous (trabecular) bone
Contractures and stiffness are caused by
fibrin buildup in extracellular space, which is then deposited in the tissues, impacting functional grasp and use of the hand. Could also be after a trauma/surgery
Myofibroblasts
damage to tissue causes cell multiplication and increased cellular secretions (hyperplasia) that creates excessive productive of actomyosin. This results in 30% strength loss
Changes to collagen with aging
collagen loses its elasticity and there is a loss of ground substance volume (extracellular matrix) causing difficulty of fiber movement over one another. Fibers are in closer contact with one another.
Newly developed contractures are
fibrous adhesions
Chronic contractures are
collagenous, requiring heat and progressive low load stretching
Hyaluronic acid
functions to lubricate the joint, this decreases with age and can contribute to stiffness
Cartilage needs — in order to move nutrients in and out of the chondrocytes
loading and unloading, this decreases with age
What changes occur to cartilage with aging
hyaline to fibrocartilage d/t decreased compressive forces
Hypokinesis
slowed movement placing joints in a lengthened/shortened state for prolonged periods causing collagenous adhesions
Elasticity
tissue will stretch and then recoil to original length
viscous component
permanent deformation to connective tissue
avoid compression to vertebral artery by
avoid hyperextension of cervical spine, esp with rotation
peripheral fatigue
reduction in maximum muscle force or motor output, d/t physical activity
central fatigue
general tiredness, weakness, languor or sleepiness
normal fatigue goes away with
rest
fatigue is caused by
chronic conditions
increased fatigue is correlated with
higher mortality
Strategies that may be used for fatigue
antidepressive tx
nutrition
exercise
limit functional decline
Changes to skin at the hand
loss of dermis/epidermis on dorsum of hand
collagen breakdown, making skin fragile
skin more vulnerable to injury, slowed healing
clubbing of nails
inadequate oxygen
spoon shaped nails
anemia
Colles fx
dorsal displacement
Smith fx
palmar displacement
commonly missed fx with FOOSH
scaphoid
Changes to cartilage with aging
loss of glycosaminoglycans, causing increased breakdown of cartilage