aerobic exercise

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1
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What are the main cardiorespiratory benefits of aerobic training?

Higher VO₂max; lower submaximal HR, BP, ventilation and myocardial oxygen demand.

2
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What central and peripheral changes improve VO₂max?

Central: higher maximal cardiac output. Peripheral: improved capillarisation and oxygen extraction.

3
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How does aerobic training change skeletal-muscle capillary density?

It increases capillary density and improves oxygen delivery.

4
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How does aerobic training affect resting blood pressure?

It generally reduces resting systolic and diastolic blood pressure.

5
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How does aerobic training affect HDL and triglycerides?

It often raises HDL cholesterol and lowers triglycerides.

6
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How does regular aerobic activity affect adiposity?

It can reduce total body fat and visceral (intra-abdominal) fat.

7
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How does aerobic exercise change glucose regulation?

Improves insulin sensitivity and glucose tolerance; may reduce insulin requirements.

8
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What six variables describe acute and chronic aerobic exercise responses?

Heart rate, stroke volume, cardiac output, blood pressure, blood flow and ventilation/VO₂.

9
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Why does heart rate rise just before and at exercise onset?

Parasympathetic withdrawal and increased sympathetic stimulation.

10
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What is the approximate HR increase per MET during incremental exercise?

About 10 beats/min per MET (rough guide; varies across people).

11
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How are HR and VO₂ related in moderate incremental exercise?

They rise approximately linearly together over much of the submaximal range.

12
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What chiefly determines age-predicted maximal heart rate?

Age; formulas are rough estimates with substantial individual variation.

13
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How does resting HR change after aerobic training?

It generally decreases.

14
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Why does resting HR fall with endurance training?

Increased vagal tone, reduced sympathetic drive and higher resting stroke volume.

15
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What intrinsic pacemaker change can contribute to athlete bradycardia?

A lower intrinsic sinoatrial-node firing rate.

16
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How does HR at a fixed submaximal workload change after training?

It decreases, often more noticeably at higher submaximal workloads.

17
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How does HRmax change with endurance training?

Usually little change; it may decrease slightly.

18
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What does a slower-than-expected HR recovery suggest?

Potential autonomic/cardiovascular concern; interpret using test protocol and clinical context.

19
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What benchmark is often used for first-minute HR recovery?

A drop of about 12 bpm or more, depending on the recovery protocol; not a universal cutoff.

20
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What cardiac structural adaptations occur with endurance training?

Physiological LV enlargement/hypertrophy, possible RV dilation and atrial enlargement.

21
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What functional cardiac adaptations support endurance performance?

Better diastolic filling, stronger contraction and higher maximal stroke volume.

22
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Define stroke volume (SV).

Volume of blood ejected by one ventricle per heartbeat.

23
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Name three principal determinants of stroke volume.

Preload (filling), myocardial contractility and afterload.

24
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What is preload?

Ventricular wall stretch near end-diastole, linked to venous return and end-diastolic volume.

25
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What is afterload?

Pressure/resistance the ventricle must overcome to eject blood.

26
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State the Frank–Starling mechanism.

Within physiological limits, greater end-diastolic filling/stretch produces stronger contraction.

27
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Why does venous return help increase stroke volume during exercise?

Greater ventricular filling increases preload and contraction through Frank–Starling.

28
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When does stroke volume increase most during incremental exercise?

During the transition from rest to moderate workloads; later response varies.

29
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How does endurance training affect stroke volume at rest and exercise?

Generally increases SV at rest, submaximal exercise and maximal exercise.

30
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How do untrained people compensate for lower SV during exercise?

They depend relatively more on increased HR to raise cardiac output.

31
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What is the formula for cardiac output?

Q = HR × SV.

32
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Calculate Q for HR 150 bpm and SV 100 mL/beat.

15,000 mL/min = 15 L/min.

33
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What happens to cardiac output during incremental aerobic exercise?

It rises with intensity as heart rate and stroke volume increase.

34
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How does Q respond during short, constant-load moderate exercise?

It usually approaches steady state within a few minutes.

35
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What does cardiovascular steady state mean?

Oxygen transport approximately matches a stable exercise metabolic demand.

36
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Why is maximal cardiac output higher after endurance training?

Mainly because maximal stroke volume increases.

37
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Compare maximal Q and Q at a fixed submaximal workload after training.

Maximal Q rises; Q at a given absolute submaximal workload often changes little.

38
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How can endurance-trained people achieve similar submaximal Q at lower HR?

Their higher stroke volume offsets their lower heart rate.

39
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What happens to SBP during dynamic aerobic exercise?

It normally increases with rising exercise intensity and cardiac output.

40
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What happens to DBP during dynamic aerobic exercise?

Usually stays similar or decreases slightly owing to lower peripheral resistance.

41
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How does SBP respond during a prolonged constant-load session?

It rises initially then usually plateaus; it may drift with conditions.

42
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How does heavy resistance exercise differ from aerobic exercise in BP response?

Can produce much larger transient SBP and DBP increases.

43
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Why should breath-holding be avoided during heavy exercise?

Valsalva-like straining can greatly increase intrathoracic pressure and BP swings.

44
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How may upper-limb resistance work affect BP versus leg work?

For comparable relative tasks, upper-limb exercise may provoke higher BP responses.

45
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How does sustained endurance training generally affect BP?

Can lower resting BP, particularly in people with elevated baseline BP.

46
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What chest symptom is abnormal during exercise?

Chest pain, pressure or tightness suggestive of angina; stop and assess.

47
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Why should one fixed exercise BP limit not be applied to everyone?

Thresholds depend on setting, protocol, baseline BP and medical guidance.

48
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What happens to arterioles in active skeletal muscle during exercise?

They dilate, increasing local blood delivery.

49
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What happens to blood flow to many nonessential vascular beds during exercise?

Sympathetic vasoconstriction redirects a greater share of flow to working muscles.

50
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How does the proportion of Q reaching skeletal muscle change with exercise intensity?

It markedly increases.

51
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What happens to absolute cerebral blood flow in typical dynamic exercise?

It is relatively well maintained, despite shifts in percentage of total Q.

52
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How does capillarisation change after endurance training?

The capillary-to-fibre ratio increases in trained muscles.

53
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How does endothelial vasodilator function adapt to aerobic training?

Improves, partly through nitric-oxide-mediated dilation.

54
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What long-term vascular structural change may occur with training?

Larger arterial diameter and improved vascular capacity.

55
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Why does ventilation increase during aerobic exercise?

Higher oxygen demand and CO₂ production require increased gas exchange.

56
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How does minute ventilation relate to tidal volume and breathing frequency?

Minute ventilation = tidal volume × respiratory rate.

57
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What happens to tidal volume early in incremental aerobic exercise?

It rises substantially; it later tends to plateau.

58
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How does respiratory rate behave near maximal exercise?

It continues rising after tidal volume starts to plateau.

59
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What is end-expiratory lung volume (EELV) at rest?

Approximately equal to functional residual capacity (FRC).

60
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How does EELV typically change from rest to moderate exercise?

It usually decreases as expiration encroaches on expiratory reserve volume.

61
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What lung-volume reserves accommodate deeper exercise breathing?

Inspiratory reserve volume (IRV) and expiratory reserve volume (ERV).

62
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What do VO₂ and VCO₂ mean?

VO₂ is oxygen uptake; VCO₂ is carbon dioxide output.

63
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How does ventilation at a fixed submaximal workload often change with training?

It may be lower and more efficient, with deeper/slower breathing.

64
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How can aerobic training reduce respiratory muscle work at a given workload?

Through a more economical breathing pattern and improved ventilatory efficiency.

65
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In pulmonary gas exchange, where does O₂ diffuse?

From alveoli into pulmonary capillary blood.

66
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In pulmonary gas exchange, where does CO₂ diffuse?

From pulmonary capillary blood into alveoli.

67
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What should be reviewed before prescribing aerobic exercise?

Goals, screening, medical history, limitations and physical activity history.

68
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What constraints shape an aerobic exercise plan?

Time, equipment, availability, setting and preferences.

69
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What major priorities may drive aerobic exercise programming?

Health, general fitness or sport/performance.

70
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What does training age mean?

The person’s prior experience and adaptation to structured training.

71
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Why should aerobic modality resemble the target activity?

Training specificity improves transfer to the intended task.

72
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Give three aerobic training modalities.

Walking/running, cycling and swimming (choose for ability and goals).

73
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What are the major components of an aerobic prescription?

Frequency, intensity, time/duration, type, progression and monitoring.

74
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Define continuous aerobic training.

Sustained exercise at a relatively steady submaximal intensity.

75
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What aerobic intensity is cited for continuous training in these notes?

Often about 60–80% VO₂max; adapt to the person and context.

76
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Define high-intensity interval training (HIIT).

Repeated hard exercise bouts separated by lower-intensity recovery intervals.

77
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Define sprint interval training (SIT).

Repeated near-maximal or all-out sprint efforts separated by recovery.

78
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List the nine key HIIT programming decisions.

Mode; work intensity/duration; rest intensity/duration; reps; sets; between-set time/intensity.

79
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Name four ways to prescribe aerobic exercise intensity.

%VO₂max, %HRmax, RPE and %MAS (or work rate/power).

80
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What is a MET?

A multiple of resting metabolic rate; conventionally 1 MET ≈ 3.5 mL O₂/kg/min.

81
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What is MAS (or vVO₂max)?

Minimum running speed associated with attainment of VO₂max, measured or estimated.

82
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What does RPE quantify?

An individual’s subjective rating of exercise effort.

83
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What is the usual range on the Borg 6–20 RPE scale?

6 (no exertion) to 20 (maximal exertion).

84
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What is a limitation of predicting HRmax as 220 − age?

It is a rough population estimate with wide individual error.

85
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For age 70, what is HRmax from the 220 − age estimate?

150 bpm (approximate, not measured).

86
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For estimated HRmax 150, what is 64–76% HRmax?

96–114 bpm (moderate band used in the notes).

87
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For estimated HRmax 150, what is 77–95% HRmax?

About 116–143 bpm (vigorous band used in the notes).

88
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Why must HIIT volume be controlled as intensity increases?

Higher-intensity sessions add fatigue and can elevate injury risk if poorly progressed.

89
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What is anaerobic speed reserve (ASR)?

Maximum sprint speed minus maximal aerobic speed (MAS).

90
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Why monitor high-speed efforts near maximal sprint speed?

They produce substantial fatigue and musculoskeletal loading.

91
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How should rest usually change as work-interval intensity rises?

Increase recovery duration and/or lower recovery intensity.

92
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What is a common starting HIIT work:rest ratio at 100–120% MAS?

About 1:1, modified to the session goal and individual.

93
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What is a possible work:rest range for very fast sprint efforts?

About 1:2 to 1:6 or more; adequate recovery depends on goals.

94
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How is session-RPE training load often calculated?

Session RPE × session duration (minutes) = arbitrary units (AU).

95
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Calculate training load for RPE 14 lasting 30 minutes.

14 × 30 = 420 AU, using the stated RPE scale.

96
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Calculate weekly load for three 10-minute sessions at RPE 12.

3 × (10 × 12) = 360 AU.

97
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Which variables can be increased as a person adapts?

Frequency, duration and intensity; progress according to tolerance.

98
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Is the 10%-per-week progression rule universal?

No. It is a heuristic, not a validated injury-proof ceiling.

99
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Why include recovery or active-rest days?

To allow adaptation and help manage cumulative fatigue.

100
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What should guide rate of progression?

Age, health status, functional capacity, response and current training load.