Cardiac Rhythms

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Last updated 1:52 AM on 10/10/26
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20 Terms

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Normal Sinus Rhythm (NSR)

60-100 bpm

Normal P wave, QRS, PR interval

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Sinus Bradycardia

< 60 bpm (normal in atheltes)

Signs:  Hypotension, pale/cool skin, weakness, angina, SOB, confusion, dizziness or syncope (from reduced cerebral perfusion).

Administer IV Atropine. Pacemaker, dopamine, or epinephrine infusion if needed.

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Sinus Tachycardia

101 to 180 bpm

Signs: Dizziness, dyspnea, hypotension, angina in patients with CAD.

β-blockers, adenosine, calcium channel blockers

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Premature Atrial Contraction (PAC)

Contraction starting from an ectopic focus in the atrium (location other than SA node)

Distorted P wave.

Signs: Palpitations (awareness of weird heartbeat), heart "skips a beat".

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Paroxysmal Supraventricular Tachycardia (PSVT)

151 to 220 bpm

a PAC triggers a run of repeated premature beats. "Paroxysmal" indicates abrupt onset and termination.

Signs: Palpitations, dyspnea, angina, hypotension.

IV Adenosine, IV β-blockers, Calcium channel blockers

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Atrial Flutter

Atrial Rate: 200 to 350 bpm

Symptoms result from high ventricular rate and loss of atrial "kick"

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Atrial Fibrillation (AFib)

Atrial Rate: 350-600 bpm with chaotic, fibrillatory waves replacing P waves

amiodarone. ECG, anticoag., ablation, Left atrial Appendage Occlussion (prevents clots)

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Junctional Dysrhythmias

AV node becomes the pacemaker when the SA node fails to fire or the impulse is blocked

Serves as a safety escape mechanism—do not suppress escape rhythms. Treat if symptomatic (Atropine for escape rhythm)

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First-Degree AV Block

Prolonged PR interval; associated with aging, disease states, and certain drugs.

Usually not serious and patients are asymptomatic. No immediate treatment required.


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Second-Degree AV Block, Type 1 (Mobitz I / Wenckebach)

Progressive lengthening of PR interval until a QRS complex is dropped

Treat if symptomatic with Atropine or temporary pacemaker. If asymptomatic, observe closely.

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Second-Degree AV Block, Type 2 (Mobitz II)

Constant PR interval with non-conducted P waves (dropped QRS complexes).

Requires transcutaneous pacing or temporary/permanent pacemaker.

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Third-Degree AV Block (Complete Heart Block)

No impulses conducted from atria to ventricles. Results in severe bradycardia, decreased CO, ischemia, HF, shock, and syncope.

Pacemaker therapy is required. Drugs (epinephrine/dopamine) to increase HR while awaiting pacing.

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Premature Ventricular Contractions (PVCs)

Premature, wide, and distorted QRS complex

Administer β-blockers, lidocaine, or amiodarone if needed.

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Accelerated Idioventricular Rhythm (AIVR)

Develops when intrinsic pacemaker rate (SA/AV node) falls below rate of ventricular ectopic pacer. Rate is 40 to 100 beats/min.

Do NOT suppress the rhythm. Administer Atropine if patient is symptomatic, or use temporary pacing.

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Ventricular Tachycardia (VT)

Ectopic focus takes over as pacer. Life-threatening due to severe decrease in CO and risk of deterioration to VF.

Causes: Heart disease, long QT syndrome, electrolyte imbalances, drug toxicity, CNS disorders, hypoxia.

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VT with Pulse

(Stable): Antidysrhythmics (amiodarone) or synchronized cardioversion.

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Polymorphic VT with prolonged QT

(Torsades de Pointes): IV Magnesium, isoproterenol, or antitachycardia pacing.

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Pulseless VT

Emergency CPR and rapid defibrillation.

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Ventricular Fibrillation (VF)

Severe, chaotic electrical activity with no effective ventricular contraction or CO. Pt is unresponsive, pulseless, and apneic. Fatal within minutes if untreated.

Immediate high-quality CPR, ACLS measures, rapid defibrillation, and epinephrine/amiodarone.

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Asystole & Pulseless Electrical Activity (PEA)

Asystole: Total absence of ventricular electrical activity

PEA: Electrical activity is observed on ECG, but no pulse.

High-quality CPR, ACLS measures, intubation, IV Epinephrine. Defibrillation is NOT indicated for Asystole/PEA.