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Cortical Lesion: Hemiplegic Posture
Retraction & depression of scapula; internal rotation of the shoulder; flexion of elbow, wrist, fingers; pronation of the forearm; lateral flexion of trunk toward involved side; elevation & retraction of pelvis; internal rotation of hip; extension of hip / knee; supination of foot; plantar flexion of ankle/toes.
Cortical Lesion: Hypotonia
Deceased muscle tone - less-than-normal resistance of a muscle to passive elongation - affected limb feels limp and heavy
Cortical Lesion: Hypertonia
Increased muscle tone - more-than-normal resistance of a muscle to passive elongation - neural (spasticity) & mechanical (soft-tissue stiffness) factors contribute
Cortical Lesion: Spasticity
Neural component of hypertonus - velocity-dependent increase in tonic stretch reflexes and exaggerated tendon reflexes (often with clonus & clasp-knife reflex).
Cortical Lesion: Clonus
Oscillating contraction & relaxation of limb segment - alternating patterns of stretch reflex & inverse stretch reflex of a specific muscle
Cortical Lesion: Clasp Knife
Resistance to passive stretch of a spastic muscles that suddenly gives way (like the blade of a jack knife).
Cortical Lesion: Loss of Fractionation
Inability to move a single joint without producing unnecessary movements in other joints - predictable, repetitive combinations of movements that occur together (rather than selective, flexible movement patterns).
Cortical Lesion: Apraxia
Inability to perform goal-directed motor activity in the absence of paresis, ataxia, sensory loss, or abnormal muscle tone - omissions, disturbed order of submovements within a sequence, clumsiness, preservation
Cortical Lesion: Lead Pipe Rigidity
Hypertonus in agonist and antagonist muscles, resistance to movement that is not velocity dependent, felt throughout the range of motion
Cerebellar Lesion: Intention Tremor
Rhythmic oscillating movement, develops during intentional movements, involuntary alternating contractions of opposing muscles.
Cerebellar Lesion: Dysmetria
Inability to judge distance accurately, overshooting or undershooting a specific target.
Cerebellar Lesion: Dysynergia
Movements broken up into multiple simple movements (rather than one smooth movement)
Cerebellar Lesion: Dysdiadochokinesia
Impairment in ability to perform repeated alternating movements, such as pronation & supination, rapidly & smoothly.
Cerebellar Lesion: Adiadochokinesia
Loss of ability to perform rapid alternating movements
Cerebellar Lesion: Ataxia
Unsteadiness, incoordination, clumsiness.
Cerebellar Lesion: Ataxic Gait
Wide-based, unsteady, staggering gait, tendency to veer side to side.
Basal Ganglia Lesions: Tremors at Rest/Nonintentional Tremors
Stop at the initiation of voluntary movement - resume during the holding phase of motor tasks when attention wanes or is diverted to another task - fatiguing
Basal Ganglia Lesions: Hypokinesia
Slowness or reduced amount or range of spontaneous movement - symptoms are reflected in lack of facial expression, monotone speech, reduced eye movements, diminished arm swing during walking,
Basal Ganglia Lesions: Hypokinesia - Akinesia
Difficulty initiating voluntary movements
Basal Ganglia Lesions: Hypokinesia - Bradykinesia
Slowness in carrying out movements
Basal Ganglia Lesions: Festinating Gait
Small, fast, shuffling steps propel the body forward at an increasing rate - difficulty stopping or changing directions
Basal Ganglia Lesions: Athetosis
Slow, writhing involuntary movements - neck, face, and extremities - tone may be increased or decreased
Basal Ganglia Lesions: Dystonia
Powerful, sustained contractions of muscles - cause twisting & writhing of limb or whole body - distorted postures trunk and proximal extremities
Basal Ganglia Lesions: Chorea
Sudden involuntary purposeless, rapid, jerky movements and/or grimacing - primarily in the distal extremities and face (e.g., Huntington chorea)
Basal Ganglia Lesions: Hemibllismus
Unilateral chorea - violent, forceful, flinging movements of extremities on one side of the body