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purpose of classification
translate a lifelong, complex disease process into a short diagnostic statement
periodontitis is not identical between patients, between teeth, between sites, and at different times
disease can progress at different rates
host response and risk factors differ
treatment complexity varies
evolution of classification
the 1999 system included terms like chronic periodontitis and aggressive periodontitis
newer system does not classify aggressive periodontitis as a separate disease category
classic localized phenotype can be described using a molar/incisor pattern
older termed phenotypes can still exist clinically, but newer classification describes disease through stage, grade, and distribution/pattern
therapeutic goals around natural teeth
ultimate purpose of diagnosis is not the diagnosis itself, but rather the treatment to create a periodontal environment
minimal probing depths
no adequate furcation problems
adequate fiber barrier
harmonious occlusion
biological stability
effective plaque control
endpoint ā treatment must be maintainable by the patient
patient must be able to clean the area themselves
if a deep inaccessible pocket remains, plaque returns and the patient cannot clean effectively; disease risk remains
staging vs grading
staging ā evaluates severity and extent based on measurable extent of destroyed and damaged tissues attributable to periodontitis
assesses complexity through specific factors that may determine complexity of controlling current disease
assesses management of long-term function and esthetics
grading ā estimates future risk of periodontitis progression and responsiveness to standard therapeutic principles
guides intensity of therapy and monitoring
estimates potential health impact of periodontitis on systemic disease to guide monitoring and co-therapy with medical colleagues
clinical attachment level
measured from CEJ to base of pocket
different than probing depth, which measures from gingival margin to base of pocket
CEJ is fixed, whereas gingival margin can move due to enlargement or recession
CAL tells you attachment loss more accurately than probing depth
if gingival margin is at CEJ ā CAL = PD
if there is recession ā CAL = PD + recession
if gingiva is coronal to CEJ ā CAL = PD - coronal gingival excess
CAL vs radiographic bone loss
ideally, CAL = radiographic bone loss; but they may not match perfectly
bone architecture is three-dimensional, but radiographs compress anatomy into a 2D image
facial/lingual bone can obscure defects
vertical defects may be underestimated radiographically
recent changes may not yet be obvious radiographically
CAL can appear worse than radiographic bone loss
use clinical findings + CAL + radiographs to determine periodontal diagnosis
newer classification system describes radiographic bone loss by how far it extends along the root
divide the root into coronal, middle, and apical thirds
a 2mm of bone loss means something different on a short root vs a long root
proportional loss is more useful than millimeters alone
normal alveolar bone height on a radiograph is 0.5-2.0 mm
periodontitis staging
staging considers severity, complexity, and extent/distribution
stage I ā initial periodontitis
severity ā interdental CAL = 1-2 mm; RBL = coronal third (<15%); no tooth loss due to periodontitis
complexity ā maximum PD ⤠4mm; mostly horizontal bone loss
good prognosis with maintenance
stage II ā moderate periodontitis
severity ā interdental CAL = 3-4 mm; RBL = coronal third (15-33%); no tooth loss due to periodontitis
complexity ā maximum PD ⤠5mm; mostly horizontal bone loss
good prognosis with maintenance
stage III ā severe periodontitis with potential for tooth loss
severity ā CAL ā„ 5mm; RBL = middle third and beyond; ā¤4 tooth loss due to periodontitis
complexity ā PD ā„ 6mm; vertical bone loss ā„ 3mm; class II or III furcation involvement; moderate ridge defect
disease is severe and more difficult to maintain, at meaningful risk for additional tooth loss
fair prognosis with maintenance
stage IV ā severe periodontitis with potential for loss of dentition
severity ā CAL ā„ 5mm; RBL = middle third and beyond; ā„5 teeth lost due to periodontitis
complexity ā masticatory dysfunction, secondary occlusal trauma (tooth mobility degree ā„2), severe ridge defects, bite collapse, drifting, flaring, <20 teeth remaining (10 opposing pairs)
represents severe periodontal destruction + functional restorative collapse
questionable prognosis with maintenance
extent and distribution for each stage adds to stage as a descriptor
localized (<30% teeth involved), generalized, or molar/incisor pattern
tooth loss due to periodontitis is important
do not count a tooth lost due to caries, trauma, endodontic failure, or other untreated reasons
past periodontal tooth loss tells you disease has demonstrated severity, future management is more complex, and restoring the patient may be difficult
complexity can increase the stage
an apparent moderate case can become a higher stage due to deep pockets, furcations, vertical defects, tooth loss, mobility, functional problems, and complex rehabilitation needs
not classified only from CAL
goals of staging:
identify periodontitis as early as possible
separate stage I/II from stage III/IV
process to classify periodontitis
initial case overview
assess full-mouth probing depths, full-mouth radiographs, missing teeth, overall disease severity
initial staging determined by CAL and RBL, where tooth loss due to periodontitis modifies stage definition
establish stage
determine CAL, RBL, tooth loss due to periodontitis, complexity
establish grade
determine rate of previous progression, bone loss relative to age, phenotype, smoking, diabetes, other medical/systemic conditions
staging/grading applies only to periodontitis
other categories include health, biofilm-induced gingivitis, non-biofilm-induced gingival disease, peri-implant conditions and are not staged/graded the same as natural-tooth periodontiis
periodontitis grading
start every patient at grade B, then use evidence to determine whether the patient should move grades
grade A ā slow progression
no bone loss/CAL over 5 years
<0.25% bone loss / age
heavy biofilm with little destruction
despite significant plaque challenge, tissues have broken down relatively slowly
nonsmoker, normoglycemic with no diagnosis of diabetes
grade B ā moderate progression
< 2mm bone loss/CAL over 5 years
0.25-1.0% bone loss / age
destruction is commensurate with biofilm
amount of disease approximately makes sense for amount of local challenge
<10 cigarettes/day, diabetes with HbA1c <7.0%
grade C ā rapid progression
ā„ 2mm bone loss/CAL over 5 years
>1.0% bone loss / age
destruction exceeds what is expected from biofilm
little plaque/calculus, young patient, major attachment loss
ā„10 cigarettes/day, diabetes with HbA1c ā„7.0%
grading aims to indicate rate of periodontitis progression, responsiveness to therapy, and potential impact on systemic health
primary criteria ā direct evidence (CAL or RBL) and indirect evidence (phenotype, % bone loss/age)
case phenotype compares amount of biofilm with amount of periodontal destruction
grade modifiers ā risk factors (smoking, diabetes)
push toward a worse grade
health on a reduced periodontium
a patient can have significant previous attachment loss but currently have healthy gingiva with stable attachment and no active periodontitis
health a reduced periodontium in a non-periodontitis patient vs health on a reduced periodontium in a stable patient
no need to restore lost bone to normal to maintain periodontal health
reduced periodontium can remain stable for many years with good plaque control and appropriate peridontal maintenance
often approximately 3-4 month recall intervals
periodontitis case definition
2mm attachment loss on at least 2 non-adjacent teeth
periodontitis requires a pattern of attachment loss rather than one isolated abnormal measurement
clinical gingival health ā <10% bleeding sites with no pockets >3mm
bleeding on probing is central when distinguishing health from gingival inflammation
gingival recession classification
newer classification system also describes recession based on interproximal attachment loss
recession type 1 ā gingival recession without interproximal attachment loss
recession type 2 ā recession + interproximal attachment loss
interproximal attachment loss ⤠buccal attachment loss
recession type 3 ā recession + interproximal attachment loss
interproximal attachment loss > buccal attachment loss
more inteproximal loss results in worse predictability for root-coverage grafting
mucogingival conditions
newer classification system expands how mucogingival deformities are described based on whole soft-tissue phenotype and associated clinical problems
periodontal phenotype ā thin/scalloped, thick/scalloped, thick/flat
gingival recession
gingival thickness
gingival width
cervical caries
esthetic concerns
hypersensitivity
lack of keratinized gingiva
decreased vestibular depth
abnormal frenum / muscle position
gingival excess
endo-perio lesions
classification distinguishes lesions with root damage vs lesions without root damage
with root damage:
root fracture or crackling
root canal or pulp chamber perforation
external root resorption
without root damage:
endo-periodontal lesions in periodontitis patients
endo-periodontal lesions in non-periodontitis patients
narrow vs wide vs deep pockets
one vs multiple tooth surfaces
isolated periodontal defect may not be purely periodontal
always consider endodontic origin + root damage + combined lesions
peri-implant disease categories
peri-implant disease is not given the same stage I-IV / grade A-C system
peri-implant health
peri-implant mucositis
bleeding, erythema, swelling, and/or supporation with or without increased probing depth compared to previous examinations
no bone loss
reversible
peri-implantitis
bleeding and/or suppuration on gentle probing
increased probing depth compared to previous examinations
bone loss beyond changes from initial remodeling
irreversible
in absence of previous data:
bleeding and/or suppuration on gentle probing
probing depth ā„ 6mm
ā„ 3mm of bone loss
hard and soft-tissue deficiencies
probing around implant is different
natural teeth have PDL and fiber insertion into cementum/tooth
implants do not have same fiber attachment to implant surface
probe penetration can naturally be deeper around an implant
increased probing depth around an implant does not mean the same thing as the identical probing depth around a natural tooth
use clinical findings + radiographs when diagnosing peri-implant disease
initial implant bone remodeling vs peri-implantitis
bone loss around an implant does not automatically equal peri-implantitis, and radiographic bone loss should be used in conjunction with CAL
some bone changes may have existed at implant placement, from initial remodeling, or because of initial hard-tissue deficiencies
compare with baseline / previous records whenever possible
disease means progressive bone loss beyond expected initial change
peri-implant health ā absence of bone loss beyond crestal bone level changes resulting from initial bone remodeling
no inflammation and no bleeding
peri-implant mucositis ā absence of bone loss beyond crestal bone level changes resulting from initial bone remodeling
signs of inflammation and bleeding and/or suppuration on gentle probing
peri-implantitis ā presence of bone loss beyond crestal bone level changes resulting from initial bone remodeling
probing depths ā„ 6mm
bleeding and/or suppuration on gentle probing
bone levels ā„ 3mm apical of most coronal portion of intraosseous part of implant