PEDS Exam 2

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Description and Tags

Neonatal conditions, respiratory, endocrine, neuro

31 Terms

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What is respiratory distress syndrome? (RDS) and what are the risk factors?

Results from surfactant deficiency in the lungs.

Risk factors: Pre-term babies, C-section, Nuchal cord, Meconium aspiration.

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What are the assessment findings of RDS? how do you know if its worsening?

  • Tachypnea (RR greater than 60 bpm)

  • Dyspnea

  • Crackles

  • Retractions

  • Expiratory grunt

  • Nasal flaring

  • Cyanosis or pallor

It’s worsening when the patient becomes unresponsive and or flaccid (floppy)

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What are the diagnostic tests for RDS?

ABG labs (will show respiratory acidosis b/c patient is holding onto Co2)

CXR (chest x-ray)

Patient is hypoxic

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How to manage RDS

  • Suction as needed

  • Ventilator support

  • Nitric oxide (helps with pulmonary dilation and relaxes smooth muscle)

  • Thermoregulation

  • Mouth and skin care

  • Decreased stimuli

  • Artificial surfactant (always check ABG before and after)


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What are the possible complications of RDS?

  • Pneumothorax

  • Retinopathy

  • Bronchopulmonary dysplasia

  • Infection

  • Intraventricular hemorrhage


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What is Bronchopulmonary dysplasia? (BPD)

  • Causes some form of alveoli damage

  • Need for supplemental oxygen for at least 28 days after birth

  • More common in infants less than 28 weeks gestation or less than 1000 g

  • Respiratory distress = BPD


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What are the assessment findings of BPD?

  • Inability to wean from O2 or ventilator

  • Signs of respiratory distress

  • Failure to thrive

  • Intermittent bronchospasms, mucus plugging, and chronic air trapping

  • Decreased pH Increased Co2, Respiratoy acidosis


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How to manage BPD?

Diagnosis

  • O2 or vent dependence

  • CXR

Treatment

  • Tracheostomy (long term fix)

  • Wean off vent/O2 source

  • Humidified O2

  • Fluid restriction

  • Increased calorie intake

  • CPT (chest physiotherapy)

  • Bronchodilators


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Abdominal wall defects: Omphalocele

Intestines on the outside of the body with a clear sac around them

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Abdominal wall defects: Gastroschisis

Intestines are on the outside of the body but no clear sac/ membrane around it

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How to manage abdominal wall defects post birth

  • Directly following birth, overexposed abdominal content and membranes with saline-soaked pads and a plastic drape

  • This prevents excess water loss, drying, and temp instability

  • Followed by surgical closure

  • If primary closure is not possible, a silo is created to help the intestines go back in.


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Pre-op Nursing considerations for abdominal wall defects

  • Infection is a constant threat before surgery

  • IV antibiotics

  • Nutrition

  • Careful w/ positioning

  • NO PRONE

  • Viscera should be protected with wet saline pads

  • Decompress GI tract

  • Fluid replacement

  • Heat and fluid loss watch out for!


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Post-op Nursing considerations for abdominal wall defects

  • Careful positioning to prevent disturbance of silastic material

  • Heat and fluid loss are major concerns for thermoregulation

  • Fluid and nutritional replacement is critical

  • Post op monitoring for complications and assessment of bowel function


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Possible complications for abdominal wall defects

  • Infection

  • Evisceration

  • Bowel perforation


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What is Necrotizing Enterocolitis? (NEC) and what are the risk factors?

There is no known exact cause; it is an inflammatory disease of the digestive tract.

Risk factors

  • Intrauterine growth restriction

  • Premature

  • Shock

  • Respiratory distress


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What are the assessment findings of NEC?

  • Abdominal distention

  • Bloody stool

  • Periods of apnea

  • Hypotension

  • Lethargy

  • Decreased urine output

  • Poor feeding


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What are possible reasons for NEC?

  • Ischemia

  • Hypoxia

  • Feeding

  • Bacteria

  • Immature GI tract


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How to diagnose NEC, Labs , and possible complications

Diagnosis

STOP FEEDING IMMEDIATELY

  • X-ray shows a sausage-shaped dilation of the intestine progressing to marked distention

  • Soapsuds appearance

Labs

  • Anemia

  • Leukopenia or leukocytosis

  • Metabolic acidosis

  • Electrolyte imbalance

  • Blood cultures = Bacteria

Complications

  • Short bowel

  • Failure to thrive

  • Sepsis


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Management of NEC

  • Main goal is to prevent it from happening or worsening

  • NG tube for decompression (NOT feeding)

  • Measure abdominal girth =Q4-Q8

  • Serial X-rays =Q4-Q6

  • Vitals

  • Gastric residual and bowel sounds

  • Avoid rectal temps

  • Position supine, no pressure on abdomen, no diaper.

  • Slow re-feeding; don’t start for 7-10 days after

  • Start very small


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What is Precocious puberty and it’s causes

Early puberty

  • Girls less than 8 and boys less than 9

Causes

  • Idiopathic

  • Congenital

  • Neoplasms

  • Trauma


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Complications of Precocious puberty and management

Complications

  • Early closing of growth plates

Management

  • Luteinizing hormone-releasing hormone → to prevent premature closing of growth plates

  • Monthly injections

  • Stop treatment when people reach normal puberty age

  • Psychosocial support → may feel down or upset b/c treatment so provide support


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Congenital Hypothyroidism causes and assessment findings

Causes

  • Trisomy 21

  • Mom used anti-thyroid

Assessment findings

  • Hypotonic

  • Cool extremities

  • Mottling

  • Difficulty feeding

  • Lethargy

  • Constipation

  • Hoarse cry

  • Enlarged tongue


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Congenital Hypothyroidism Diagnosis and Treatment

Diagnosis

  • Newborn screening

  • Labs

Treatment

  • Levothyroxine

  • Too much med = Hyperthyroidism, increased HR, irritability, decreased sleep, fever, diaphoresis.

  • Too little med = Hypothyroid, fatigue, sleepy, decreased appetite, constipated



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Hyperthyroidism assessment

  • Graves disease

  • Peaks at age 12-14

  • Can be mistaken for ADHD

Assessment findings

  • Goiter

  • Exophthalmos

  • Tachycardia

  • Irritable

  • Diaphoresis

  • Fatigue

  • Weight loss

  • Fatigue

  • Short attention span

  • Emotional lability


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