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Neonatal conditions, respiratory, endocrine, neuro
What is respiratory distress syndrome? (RDS) and what are the risk factors?
Results from surfactant deficiency in the lungs.
Risk factors: Pre-term babies, C-section, Nuchal cord, Meconium aspiration.
What are the assessment findings of RDS? how do you know if its worsening?
Tachypnea (RR greater than 60 bpm)
Dyspnea
Crackles
Retractions
Expiratory grunt
Nasal flaring
Cyanosis or pallor
It’s worsening when the patient becomes unresponsive and or flaccid (floppy)
What are the diagnostic tests for RDS?
ABG labs (will show respiratory acidosis b/c patient is holding onto Co2)
CXR (chest x-ray)
Patient is hypoxic
How to manage RDS
Suction as needed
Ventilator support
Nitric oxide (helps with pulmonary dilation and relaxes smooth muscle)
Thermoregulation
Mouth and skin care
Decreased stimuli
Artificial surfactant (always check ABG before and after)
What are the possible complications of RDS?
Pneumothorax
Retinopathy
Bronchopulmonary dysplasia
Infection
Intraventricular hemorrhage
What is Bronchopulmonary dysplasia? (BPD)
Causes some form of alveoli damage
Need for supplemental oxygen for at least 28 days after birth
More common in infants less than 28 weeks gestation or less than 1000 g
Respiratory distress = BPD
What are the assessment findings of BPD?
Inability to wean from O2 or ventilator
Signs of respiratory distress
Failure to thrive
Intermittent bronchospasms, mucus plugging, and chronic air trapping
Decreased pH Increased Co2, Respiratoy acidosis
How to manage BPD?
Diagnosis
O2 or vent dependence
CXR
Treatment
Tracheostomy (long term fix)
Wean off vent/O2 source
Humidified O2
Fluid restriction
Increased calorie intake
CPT (chest physiotherapy)
Bronchodilators
Abdominal wall defects: Omphalocele
Intestines on the outside of the body with a clear sac around them
Abdominal wall defects: Gastroschisis
Intestines are on the outside of the body but no clear sac/ membrane around it
How to manage abdominal wall defects post birth
Directly following birth, overexposed abdominal content and membranes with saline-soaked pads and a plastic drape
This prevents excess water loss, drying, and temp instability
Followed by surgical closure
If primary closure is not possible, a silo is created to help the intestines go back in.
Pre-op Nursing considerations for abdominal wall defects
Infection is a constant threat before surgery
IV antibiotics
Nutrition
Careful w/ positioning
NO PRONE
Viscera should be protected with wet saline pads
Decompress GI tract
Fluid replacement
Heat and fluid loss watch out for!
Post-op Nursing considerations for abdominal wall defects
Careful positioning to prevent disturbance of silastic material
Heat and fluid loss are major concerns for thermoregulation
Fluid and nutritional replacement is critical
Post op monitoring for complications and assessment of bowel function
Possible complications for abdominal wall defects
Infection
Evisceration
Bowel perforation
What is Necrotizing Enterocolitis? (NEC) and what are the risk factors?
There is no known exact cause; it is an inflammatory disease of the digestive tract.
Risk factors
Intrauterine growth restriction
Premature
Shock
Respiratory distress
What are the assessment findings of NEC?
Abdominal distention
Bloody stool
Periods of apnea
Hypotension
Lethargy
Decreased urine output
Poor feeding
What are possible reasons for NEC?
Ischemia
Hypoxia
Feeding
Bacteria
Immature GI tract
How to diagnose NEC, Labs , and possible complications
Diagnosis
STOP FEEDING IMMEDIATELY
X-ray shows a sausage-shaped dilation of the intestine progressing to marked distention
Soapsuds appearance
Labs
Anemia
Leukopenia or leukocytosis
Metabolic acidosis
Electrolyte imbalance
Blood cultures = Bacteria
Complications
Short bowel
Failure to thrive
Sepsis
Management of NEC
Main goal is to prevent it from happening or worsening
NG tube for decompression (NOT feeding)
Measure abdominal girth =Q4-Q8
Serial X-rays =Q4-Q6
Vitals
Gastric residual and bowel sounds
Avoid rectal temps
Position supine, no pressure on abdomen, no diaper.
Slow re-feeding; don’t start for 7-10 days after
Start very small
What is Precocious puberty and it’s causes
Early puberty
Girls less than 8 and boys less than 9
Causes
Idiopathic
Congenital
Neoplasms
Trauma
Complications of Precocious puberty and management
Complications
Early closing of growth plates
Management
Luteinizing hormone-releasing hormone → to prevent premature closing of growth plates
Monthly injections
Stop treatment when people reach normal puberty age
Psychosocial support → may feel down or upset b/c treatment so provide support
Congenital Hypothyroidism causes and assessment findings
Causes
Trisomy 21
Mom used anti-thyroid
Assessment findings
Hypotonic
Cool extremities
Mottling
Difficulty feeding
Lethargy
Constipation
Hoarse cry
Enlarged tongue
Congenital Hypothyroidism Diagnosis and Treatment
Diagnosis
Newborn screening
Labs
Treatment
Levothyroxine
Too much med = Hyperthyroidism, increased HR, irritability, decreased sleep, fever, diaphoresis.
Too little med = Hypothyroid, fatigue, sleepy, decreased appetite, constipated
Hyperthyroidism assessment
Graves disease
Peaks at age 12-14
Can be mistaken for ADHD
Assessment findings
Goiter
Exophthalmos
Tachycardia
Irritable
Diaphoresis
Fatigue
Weight loss
Fatigue
Short attention span
Emotional lability