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nursing care of patients with altered electrolyte balance
sodium
major cation of extracellular fluid
NaCl, cholride follows Na
water distribution
neuromuscular activity
muscle contraction
nerve impulses
most come from diet
normal range: 135-145mEq/L
kidneys
primary regulator of odium balance in the body
decreased blood volume leads to Na and water retention
increased blood volume leads to elimination of Na and water by the kidneys
hyponatremia: Na < 135mEq/L: Pathophysiology
GI loss: prolonged vomiting, NG suction, diarrhea, or laxative abuse
skin loss: burns or large amount of wound drainage, excessive diaphoresis
older adults have decrease locoorsse
adrenal insufficiency
prolonged use of diuretics
critical value <120mEq/L or >160mEq:L
hyponatremia: pathophysiology
excessive water intake - dilutes serum sodium
heart failure, renal failure, lover cirrhosis
decreae domdium)
SIADH (retaining water)
hyponatremia: manifestations
muscle cramps, weakness, and fatigue
anorexia, n/v, diarrhea
gi upeset n/v
headache, confusion, irritability, lethargy, tremors, seizures, coma
hyponatremia: treatment
fluid restriction
eat a “normal” diet, it will have enough sodium in it
sometimes oral rehydration with electrolyte solution
lactated ringers are given with hyponatremia
hyponatremia: nursing interventions
clients with neurologic symptoms are at risk for injury, safety is important
elevating the HOB may help control headaches
daily wts to monitor fluid status
monitor electrolytes
assess muscle strength and tone
hypernatremia: >Na 145mEq/L: pathophysiology
regulatory mechanism: the body is protected from hyponatremia when excess Na stimulates the release of ADH and the kidneys retain water
thirst mechanism is stimulated in response to high Na levels (increase water intake)
insensible water loss (high fever, burns, increase environmental temp) results in water loss
increased water intake could refer to 3% NS IV’s, saltwater near drowning, high sodium meds, while decrease water intake could occur if have limited access to water concentrated tube feedings
hypernatremia manifestations
thirst
neuro: lethargy, weakness, irritability
seizures, coma, death (if severe)
hypernatremia: treatment
diuretics (increase excretion of sodium)
low sodium
low sodium diet might include whole wheat bread, steamed carrots, chicken legs
would NOT include ham, cheese, cottage cheese
read the labels to determine sodium content
hypernatremia nursing interventions
client safety
daily weight
monitor neuro status
potassium
major cation of intracellular fluid
generated action potential and transmits nerve impulses
normal range-3.5-5mEw/L
control of potassium
enters the body by the food we eat
kidneys play a primary role in excretion, but some is lot via GI and skin
regulated by changes in plasma K+ and aldosterone levels
where Na+ goes, K+ doesnt
hypokalemia: K < 3.5mEq/L: pathophysiology
diuretics such as furosemide and HCTZ are a common cause of hypokalemia
increased aldosterone secretion and stress are other reasons a person may have excessive renal K+ excretion
diarrhea, vomiting, ileostomy drainage can result in K+ loss
hypokalemia: manifestations
client are prone to dysrhythmias and cardiac arrest
hypokalemia increases the sensitivity of excitable membranes to digoxin
toxic effects may occur with normal doses
hypokalemia: manifestations
muscle weakness usually doesnt start until K+ is less than 2.5mEq/L
muscle weakness can lead to respiratory failure
leg cramps, fatigue, paresthesias
confusion that develops may be the result of hypokalemia
hypokalemia: treatment
oral potassium
should not be given on an empty stomach
when given in a liquid form, K+ should be diluted in at least 4oz fluid
K+ is absorbed well from the GI tract
IV potassium replacement
IV potassium is NEVER given IVP
administer slowly
do not administer undiluted
not given faster than 10-20mEq/hr
monitor for manifestations of hyperkalemia
monitor I/O
A > 30 ml/hr urine output is recommended
people with normal K levels do not need cardiac monitoring
ex). keep in mind that all clients receiving KCl in their IV’s have cardiac monitoring
nursing diagnoses
risk for injury r/t muscle weakness
activity intolerance/impaired physical mobility
self care deficit
decreased cardiac output r/t dysrhythmias
clients are at risk for falls due to their muscle weakness
hypokalemia: nursing interventions
encourage intake of foods high in potassium such as milk, dried fruit, bananas, apricots, oranges, cantaloupes, baked potato with skin, meats, avocado, raw carrots, raw tomatoes
teach s/s of hypokalemia
if a diuretic is needed a potassium sparing diuretic such as spironolactone (Aldactone) should be given
hyperkalemia: K> 5mEq/L: pathophysiology
potassium irritates
more K+ means more irritability
increased excitability of nerves and muscles r/t increased resting potential
hyperkalemia: etiologies
watch for hyperkalemia in clients with renal failure (most common cause)
aldactone (spirnolactone) is a potassium-sparing diuretic
giving too much oral or IV potassium can result in hyperkalemia
be careful!
hyperkalemia: manifestations
more depolarization
initially increases cells excitability (more irritable), but eventually produces weakness because of persistent neuromuscular depolarization
hyperkalemia: manifestations
the heart is very sensitive to potassium imbalances
dysrhythmias
muscle twitching and muscle cramps
seizures
nausea/ abdominal cramping/diarrhea
muscle weakness, leading to paralysis
weakness first noticed in the legs
hyperkalemia: treatment
monitor heart rhythm and rate (telemetry)
bradycardia
monitor pulse strength, BP (hypotension)
restrict potassium intake
avoid salt subsitutes
dialysis if indicated
hyperkalemia: medication
kayexalate (sodium polystyrene)
classification: K+ ion exchange resin
action: it exchanges sodium ions for potassium ions in the intestine
excess k+ is removed
side effects: hypokalemia, hypocalcemia, hypernatremia, constipation, fecal impaction, N&V, anorexia
calcium
contraction of smooth, cardiac, and skeletal muscles
blood coagulations
bones and teeth
nerve impulse transmission
normal range: 8.6-10.2 (8.9-10.1) mg/dL
control of calcium
about 30% comes from ingested food
absorbs in intestines
more than 99% of the calcium in the body is combined with phosphorus and is concentrated in the bone
this makes a readily available source
balance of calcium depends on Vitamin D, parathyroid hormone and calcitonin
hypocalcemia: Ca < 8.9mg/dL pathophysiology
calcium ions play an important role in cardiac contractility
hypocalcemia has a depressive effect on cardiac contractility
this leads to decreased cardiac output
if calcium is not available for clotting, bleeding problems occur
if calcium is not available for bone, the bones become weak
bone demineralization places a client at risk for injury
parathyroid glands can affect calcium and phosphorus
hypocalcemia: manifestations
tetany (leg/arm spasms, twitching)
numbness and tingling
carpal-pedal spasms
seizures
decreased BP
physical exam
how do you assess for Trousseau’s sign
inflate a BP cuff above systolic blood pressure level for 3 minutes
if positive, carpal spasms develop
how do you assess for chvostek’s sign
tap lightly over the facial nerve (in front of the ear)
this causes contraction of the facial muscles
hypocalcemia: treatment
can be an emergency if laryngeal spasms and respiratory arrest are imminent
monitor closely after thyroid and neck surgery
damage to the parathyroid gland may occur
IV calcium via a drip is preferred but can be given IVP (calcium chloride and calcium gluconate)
monitor calcium closely in client with parathyroid function
hypocalcemia: nursing interventions
1000-1500mg/day depending on age
foods high in C++ include milk and other dairy products, shellfish, and dark green leaft vegetables
ensure adequate vitamin D
hypocalcemia meds: calcium supplements
give supplements 1hr before or 1-2hrs after a meal
encourage fluids to avoid calcium-based urinary stones
foods high in Vitamin D include egg yolks, fortified milk, fish liver oils, whole grain cereals, wheat germ, liver, vegetable oils
hypercalcemia: Ca > 10.2mg/dL: pathophyiology
causes: hyperparathyroidism, malignancies (solid tumors and hematological malignancies), excess vitamin D, and aluminum intoxication
hypercalcemia: symptoms
things slow down
muscle weakness, fatigue, anorexia, nausea, vomiting, constipation, thirst
decreased strength, decreased deep tendon reflexes
confusion, lethargy
cardiac arrest (severe Ca > or = 17)
hypercalcemia: manifestations
renal calculi (kidney stone)
polyuria (increase urination)
impaired higher cerebral functioning, such as confusion, personality changes, emotional instability
lethargy, slurred speech
magnesium
present in intracellular fluid
needed for intracellular activites such as protein synthesis, carbohydrate metabolism, and muscle contractions
normal range is 1.3-2.3mEq/L
hypomagnesemia
causes: insufficient Mg intake, NG suction, vomiting, diarrhea, malabsorption, chronic alcoholism
assessment: tachycardia, HTN, seizures, tremors, change in mental statues (neuro)
interventions: discontinue diuretic therapy and administer IV magnesium sulfate, seizure precautions
eat Mg-rich foods, milk, green leafy veggies, pork, beefy, chicken
hypermagnesemia
overview- due to increased Mg intake or decreased excretion (renal failure)
assessment: bradycardia, hypotension, weakness, nausea, vomiting, lethargy
interventions: give diuretics to increase renal excretion of Mg
restrict dietary intake of Mg-rich food, avoid laxatives and antacids with magnesium
phosphate (PO4)
major anion in intracellular fluid
needed to activate B-complex vitamins
assists with cell division and carbohydrate, protein and fate metabolism
a decrease in serum phosphorus level is accompanied by an increase in serum calcium level
normal value is 2.5-4.5mg/dL
hypophosphatemia: <2.5mg/dL
overview- more likely in clients with chronic alcoholic pancreatitis due to insufficient intake; malnutrition; may also be due to impaired kidney excretion from renal failure, malignancy
assessment- irritability, fatigue, weakness, paresthesias, confusion, seizures, coma
interventions- administer oral vitamin D and phosphorus supplements
decrease intake of Ca foods and increase PO4 foods (fish, whole grains, meats, nuts)
CNS effects may include irritability, confusion, seizures
if client being treated suddenly has a positive Trousseaus sign, the phosphorus replacement is causing hypocalcemia
interventions also include assess systems noted under assessment
hyperphosphatemia: >4.5mg/dL
overview: problems that occur center on the hypocalcemia that results with increase in serum phosphorus
assessment: refer to assessment of hypocalcemia
interventions: instruct to avoid laxatives, decrease phosphorus intake
laxatives and enemas contain phosphates
nutrition
body mass index
determines whether a person wt is appropriate for height
may provide a useful estimate of malnutrition
ideal body weight (IBW) requires a balance between expenditure of energy and intake of nutrients
weight in pounds divided (ht in inches x ht inches) x 703
guide for BMI evalution
<18.5 underweight
18.5-24.9 normal
25-29.9 overweight
30-39.9 obesity, Class I and II
40 or >40 extreme obesity
factors affecting nutrition
age
older adults take in less calorie, and more sodium (generally)
gender (pregnancy, lactation)
increase calorie intake
health/medications
lifestyle/ alcohol abuse
religion and culture
cost of food/food fabs
personal preferences
factors affecting nutrition
medications
alter appetite
disturb taste perception
interfere with nutrients absorption or excretion
alter nutrient metabolism
factors affecting nutrition
dentition issues
dysphagia
GI issues —> anorexia, vomiting, diarrhea, and nausea
factors affecting nutrition
alcohol and tobacco use
advertising
other
socioeconomic factors; social isolation, support system
nutrition and the older adult
need fewer calories due to decrease in metabolism
loss of teeth, loss of taste
difficulties chewing and swallowing
decreased thirst
constipation
altered nutrition
malnutrition: includes undernutrition and overnutrition
anorexia nervosa and bulimia - medical diagnoses
assessment: shared responsibility between physician, dietitian and nursing
keeping track of intake of patient
nursing diagnoses
imbalanced nutrition: less than body requirements
obesity
overweight and risk for overweight
readiness for enhanced nutrition (wellness diagnosis)
special diets
clear liquid
apple, grape and cranberry juice
broth
popsicles
coffee/tea
full liquid
ice cream
milk
cream soups
yogurt
pudding
sherbet
mechanically altered
chopped, ground, mashed soft, thickened liquids for dysphagia
mashed potatoes
scrambled eggs
applesauce
modifications
consistent carb (DM)
carb count
fat and calorie restricted (weight loss)
low salt (cardiac diet)
renal diet (low potassium)
fats and label reading
more unsaturatered fats
increased fiber
assisting patients with meals
weakened individuals: feeding vs assisting
visually impaired, alternations in cognition (dementia)
use of adaptive feeding aids
vitamins
water soluble
vitamin C
B- complex vitamins
B1 (thiamin)
B2 (riboflavin)
B3 (niacin)
B6 (pyridoxine)
B12 (cyanocobalamin), pantothenic acid and biotin
fat soluble
Vitamins A, D, E, K
major minerals
calcium
phosphorus
magnesium
potassium
sodium and chloride
trace minerals
iron
red meat
oral supplements
zinc
manganese
chromium, copper
molybdenum and selenium
fluoride, iodine
nursing care of the adult with needs r/t fluid balance
homeostasis
the state of equilibrium in the internal environment of the body
body fluids transport nutrients, electrolytes and oxygen to cells
fluids carry wastes products away from the cells
water regulated body temperature and lubricates joints and membranes
water is the most common substance in the body
hydration
state of fluid balance
normal assessment
alert
moist eyes and mucous membranes
intake = output
adequate skin turgor
fluid and electrolytes imbalances
may be caused by illness or injury or disease
therapeutic measures
IV fluids (if less)
diuretics may cause or contribute to F/E imbalance (if too much)
kidneys
regulate volume and composition of ECF
acute renal failure can be cause by dehydration and hypovolemia
potassium
eliminate BUN and creatinine
hypovolemia stimulates renin release
activates the renin-angiotensin-aldosterone system
heart and blood vessels
natriuretic peptides
hormones secreted by special cells that line the atria and ventricles of the heart
secreted in response to increased blood volume and BP
ANF (atrial natriuretic factor)
primary actions: vasodilation and increased urinary excretion of Na and H20 which decreases blood volume
lungs
regulates body temperature
fluid excess
pulmonary congestion, pulmonary edema
fluid deficit
increased resp rate due to decreased tissue perfusion and hypoxia
endocrine system
aldosterone
antidiuretic hormone (ADH)
natriuretic peptide (NP)
the endocrine system is one of the two primary communicating and coordinating systems in the body
it communicates through chemical substances or hormones
the endocrine system helps control F/E balance
2 hormones that help control these critical balances are;
fluid loss is adjusted by;
adrenal glands
secrete aldosterone
essential for the maintenance of F/E balance
maintain ECF volume
promote reabsorption of Na and excretion of K ions (kidney)
aldosterone
hormone secreted by the adrenal cortex
protects sodium balance by preventing sodium loss
excretes K+
helps regulate water balance
in exchange for sodium —> potassium ions are excreted
pituitary
a body fluid deficit or increased plasma osmolality releases ADH
ADH regulates water retention and decreases osmolality
small gland - about the size of a pea
has been referred to as the master gland
located in base of the brain
works closely with the hypothalamus
two lobes; anterior and posterior
plasma osmolality = a measure of solute concentration of circulating blood
syndrome of inappropriate antidiuretic hormone (SIADH)
causes water retention
CNS disorder
brain tumors
brain injury
cancer
malignancies
antidiuretic hormone
ADH or vasopressin
synthesized in the hypothalamus and stored in the posterior pituitary
causes water reabsorption
POTENT VASOCONSTRICTOR
ADH acts in the renal distal and collecting tubules causing water reabsorption
thirst center —> hypothalamus
gastrointestinal (GI)
think GI tract accounts for most water intake
a small amount of water is eliminated by GI tract
Diarrhea and vomiting can lead to significant F/E loss
water intake includes fluids, and water pressent in solid foods
lean meats
fruits
vegetables
most of the bodys water is excreted by the kidneys
NG
parathyroid gland
secrete PTH (parathyroid hormone)
regulates blood level of calcium and phosphate
calcium and phosphate help to balance each other out (high CA, low phosphate and vice versa)
increases calcium reabsorption and phosphate excretion in the kidneys
skin
regulates body temperature
excessive sweating caused by fever or high environmental temperatures may lead to large losses of water and electrolytes
fluid volume deficit - FVD - (hypovolemia)
assessment abnormalities
poor skin turgor (dehydrated)
rapid, weak and thready pulse
heart is working harder to perfuse bc not enough fluid
hypotension
not enough fluid volume
increased respirations
lungs are working harder to perfuse bc not enough fluid
behavior, mental state
restlessness, indifference
third space fluid shift (third spacing)
occurs when fluid accumulates in a portion of the body from which it is not easily exchanged with ECF (technically fluid loss)
ascites - the accumulation of serous fluid in the peritoneal or abdominal cavity
edema associated with burns
fluid in the abdominal cavity with peritonitis
the fluid is trapped and unavailable for use
not being exchanged properly
3rd spaced fluid is trapped and essentially unavailable for functional use
risk factors for dehydration
vomiting/ diarrhea
burns
large, draining wounds
diabetes
hemorrhage
other; heat, exercise, decreased thirst sensation (poor oral intake)
insensible fluid loss from skin or lungs
invisible vaporization of water
amount increases by accelerating body metabolism
occurs in increased body temp and exercise
clinical manifestations of dehydration
cardiovascular
increased HR
decreased BP
respiratory
increased respirations
skin —> assess
skin color
moisture
skin turgor
edema
neurologic
confusion (esp. older people)
renal
U.O less than 500ml/day
other
wt loss
the minimum amount of urine per day needed to dissolve and excrete toxic waste products is 400-600ml
if less wastes are retained and cause lethal electrolytes imbalances, acidosis and toxic build up of nitrogen
treatment
IV therapy
diet therapy, oral intake
drug therapy
antiemetics
antipyretics - reduce temp
nursing interventions
monitor lab values
electrolytes
increased hematocrit (percentage of red blood cells)
blood is “thick”, no fluid to “thin” it out
increased urine specific gravity (weight of urine)
would be higher because of “thickness”, not enough fluid to “thin” out
normal is 1.005-1.03
VS, I/O, daily weight
adequate oral intake and routine mouth care
fluid volume excess - FVE - (hypervolemia)
assessment abnormalities
pitting edema
lower extremities
bounding pulse
strong (because of extra fluid)
hypertension
more fluid volume
lungs
SOB, moist crackles
confusion, irritability
weight gain
fluid volume excess - FVE
common causes
renal failure
CHF
treatment
drug therapy
diet therapy
fluid restriction
low sodium
treatment/ nursing interventions
diruetics
fluid and sodium restrictions
monitor I/O, daily weights
oral care
elevate edematous extremities - promotes venous return and fluid reabsorption
labs
decreased hemacrit
urine specific gravity
COP - prevents skin breakdown
macronutrients
macronutrients
carbs
fats
proteins - vital component of every living cell
milk
eggs
meats
nuts
anthoropometric mesurements
height
weight
BMI
waist/ wrist circumference
numbers used to look over general health and nutrition
2 fat soluble vitamins
vitamin D - helps calcium to work better
vitamin K - role in blood clotting