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Boston Classification System
aphasia classification system that is developed by Harold Goodglass, Edith Kaplan, and Norman Geschwind called the Boston Diagnostic Aphasia Examination (BDAE)
most widely used in the US, favored by researchers, neurologists, and other professionals
takes a syndrome view
diagnostic criteria are fluency, auditory comprehension, and repetition
syndrome
group of symptoms which consistently occur together, condition characterized by a set of associated symptoms
syndrome view
different types of aphasia that vary by symptoms, Boston Classification System uses this
anomic aphasia
intact fluency, auditory comprehension, and repetition
conduction aphasia
intact fluency, auditory comprehension, and poor repetition
transcortical sensory aphasia
intact fluency, poor auditory comprehension, and intact repetition
Wernicke’s aphasia
intact fluency, poor auditory comprehension, and repetition
transcortical motor aphasia
non fluent, intact auditory comprehension, and repetition
Broca’s aphasia
non fluent, intact auditory comprehension, and poor repetition
transcortical mixed aphasia
non fluent, poor auditory comprehension, and intact repetition
global aphasia
non fluent, poor auditory comprehension, and repetition
unitary view of aphasia/Schuell’s classification system
there aren’t different types of aphasia and everyone that has aphasia just has it
the variation is on severity, it is classified by the severity, sensory/motor deficits, and prognosis for recovery
irreversible aphasia and simple aphasia
irreversible aphasia
very impaired, poor prognosis for recovery
simple aphasia
mild aphasia, good prognosis for recovery
Weisenberg and McBride classification system
receptive and expressive aphasia
do not use this, tells you very little about the aphasia because there are expressive and receptive difficulties in all types of aphasia
Gardner classification system
fluent and nonfluent aphasias
can be used as a broad characterization as a starting point
nonfluent aphasias in the Boston classification system
transcortical motor, Broca’s, transcortical mixed, and global aphasia
fluent aphasias
anomic, conduction, transcortical sensory, and Wernicke’s aphasia
words of caution
Boston assumes the aphasia types result from disruptions to certain areas, not everyone agrees and that aphasia types are from damage to specific areas
functional specialization and localization, and they do not always hold true although the lesion site is often involved
functional specialization
the idea that structures within the hemispheres have specific abilities
localization
if damage to the area causes impairment, then that area is responsible for the impaitment (lesion type)
characteristics that Boston does not use to classify
reading comprehension and written expression
reading comprehension
mirrors auditory comprehension and usually more severe
written expression
mirrors oral expression and usually more severe
using written and spoken modalities together
can improve overall comprehension and written expression can improve oral expression
helpful in severe cases as well
evolution of aphasia
aphasia can evolve over time, symptoms can change, most people live with it for the rest of their lives
when symptoms change, the diagnosis changes
symptoms should not get worse
aphasia and deafness
they can gesture but cannot produce and understand the sign
left language hemisphere whether the language output is verbal or gestural
crossed aphasia
damaged hemisphere and the dominant hand on the same side, very rare
does not fall typically on the Boston and there are extralinguistic issues
ex. damage to the right hemisphere and right-handed
extralinguistic issues
difficulty understanding prosody, tone, pitch
difficulties with nonliteral language and abstract concepts
subcortical aphasia
damages to the subcortical areas causing aphasia
symptoms do not fit the Boston, classified with the lesion site
impairments tend to be milder and make better recovery
subcortex
within the white matter is the basal ganglia and the thalamus
thalamus
largest subcortical structure, communicates information between the brain stem and the cerebrum
basal ganglia
crucial for motor movements
thalamic lesion
good auditory comprehension, repetition, fluent speech, a lot of semantic paraphasias and neologisms
nonthalamic lesion
not in the thalamus, could be basal ganglia, have mild impaired auditory comprehension, repetition, nonfluent, reminded of Broca’s without agrammatism, also present with dysarthria
primary progressive aphasia (PPA)
dementia syndrome, progressive neurodegenerations in the language networks of the brain
language is first affected and over time it gets worse and cognitive abilities are lost
with present with global dementia ~5 years and language has to be the only thing affected for about 2 years
three variants of primary progressive aphasia (PPA)
nonfluent/agrammatic, semantic, and logopenic
alexia
reading problems due to recent neurological impairment, loss of reading ability, impaired comprehension
agraphia
writing problems associated with recent neurological impairment, loss of ability to write, using language to communicate in written language
pure aphasia
it is possible have an impairment in only one modality while the others are intact
rare, identify and treat using classifications
pure alexia
reading comprehension difficulties with no other deficits, pure word blindness
can write and spell but unable to read it back seconds later
often caused by PCA damage
pure agraphia
isolated impairment of writing, verbal and reading remain intact, decreased ability to retrieve and manipulate the letters
stroke in the angular gyrus, supramarginal gyrus, and posterior regions of the temporal lobe
agnosia
problem in recognition
neurological condition where one does not recognize sensory information even though the senses are operating normally
auditory agnosias
verbal auditory agnosia, nonverbal auditory agnosia, amusia
verbal auditory agnosia
pure word deafness, hears speech, but unable to understand it
can understand other auditory stimuli but not spoken
nonverbal auditory agnosia
cannot make sense of nonverbal sounds, all other modalities intact
amusia
inability to distinguish music
visual agnosia
impairment in recognizing visual input but the function of the eyes is intact
lesions are in the parietal and occipital lobe and often in the right hemisphere
acute care hospital
the first place that people go, don’t go for very long and on average people stay for 11 days
there are different kinds and could be community or a larger one with hundreds of beds, federally funded and some hospitals are specialized
rehabilitation hospital
must tolerate a minimum of 3 hours of therapy a day 5 days a week
stay for about 2 weeks, some are a separate facility than the hospital
assessment and treatment happen here
skilled nursing facility/subacute
24-hour nursing care and therapy services, trying to get the patient ready to go home
less intense than a rehab hospital, ranges in sizes
most insurance companies say you have to be in the acute care hospital for 3 days
therapists are on staff through an agency, less often for clients here
long term care
nursing home, there for the rest of their lives
may see clients here, may have complex medical needs and typically here not because of aphasia alone
shared room with 2 beds, therapists on staff through an agency
assisted living
in own apartment with different levels of care
very expensive, gives all meals, helps with meds
therapists are hired by an agency and very rare full-time work for SLPs
homecare/visiting nurse services
people are seen at home, may be with family or at assisted living
not seen for very long (10 sessions allowed total) and very expensive
fills the gap to outpatient
outpatient
live at home and travel for services
rehabilitation/hospital setting, private office, university, telehealth
community
places like Adler Aphasia Center
discharge
typically, due to insurance, there is a time where clients cannot get therapy anymore
it is very scary for most, so it is important to give them a home program and resources
they can also return when insurance will allow, this should never be a surprise, and you have to tell clients this
interdisciplinary team
doctors, nurses, PT, OT, recreational therapy, rehab techs, psychologists, neuropsychologists, dietitians, social workers
assessment
rooted in medicine and uses the clinical methods
process varies on level of care, where the client is, and how long they have lived with aphasia
first step of assessment
screening, happens at acute care when they get to the hospital
tells you if they have signs of aphasia, symptoms, cannot tell you the type
results of screening
determine whether testing is necessary, pass/fail dependent, need a doctor’s order
screening probably done by nurse or physician
common screening tools
FAST, ALPS, AASP, BEST-2, MAST, LAST
most screenings look at all modalities, but some do skip
time of screening
should be very short, 15-20 minutes, should be done without a lot of fuss
assessment
necessary to make a diagnosis of aphasia and specify the type
determines the severity and nature, important for prognosis
can be used to determine outcome or efficacy of treatment as well
formal assessments
standardized, strict protocol and directions, quantitative
assesses linguistic skills and good at telling you what an individual cannot do
informal assessment
non-standardized, based on the client’s needs, and very flexible
good at telling goals, can tell you what helps them
can answer the question when do you have difficulties
assessment steps
1 case history
2 conduct an interview
3 formal assessments
4 informal assessments
5 analyze
6 make a differential diagnosis
7 counsel
aphasia batteries
a large scale comprehensive standardized assessment for aphasia
western aphasia battery-revised (WAB-R)
3 sections and you don’t have to give all 3
there is a bedside version that takes 15 minutes
aphasia quotient (AQ) of the WAB-R
45-minute section that is most often given
language quotient (LQ) of the WAB-R
includes the AQ and reading and writing
takes 1 hour
cortical quotient (CQ) of the WAB-R
includes the AQ, LQ, block design, drawing, calculation, and nonverbal reasoning
takes an extra 16 minutes
spontaneous speech score task WAB AQ
spontaneous speech information content, spontaneous speech fluency, picture description and personal questions
auditory verbal score task WAB AQ
yes/no questions, auditory word recognition, sequential commands
repetition score task WAB AQ
repeat words, sentences, and phrases
naming score task WAB AQ
object naming, word fluency (divergent naming), sentence computation, responsive speech
understanding scores WAB AQ
tasks within scores make up specific scores, overall score is out of 100 which determines severity
indicates patterns of strengths, weaknesses, and discrepancies
0-25: very severe
26-50: severe
51-75: moderate
76+: mild
93.8- cutoff of score, considered latent, if scored within this category do other testing to help get services
WAB-R bedside
spontaneous speech information content
spontaneous speech fluency
yes and no questions
sequential commands
repetition
object naming
reading
writing
BDAE-3 Boston Diagnostic
2 options, long form (1-5 hours) and short form (40-60 minutes)
same components but long form has more items
subtests of the BDAE-3
conversational and expository speech (discourse)
auditory comprehension
oral expression
reading
writing
comprehensive aphasia test (CAT)
3 sections, takes 90-120 minutes and usually done over 2 sessions
cognitive screen, impact questionnaire, and language portion
language portion of the CAT
comprehension of receptive language skills, spoken and written, words, sentences and paragraphs
expression section of repetition, naming (objects and actions), reading, writing
Informal assessment
test all 4 modalities of language
informal auditory comprehension
respond to commands that vary in complexity and get more difficult
picture/object identification
yes/no questionnaire responses
end with story retell, listen to a story