Clinical Presentation & Diagnostic Considerations of the Small Animal Cardiac Patient

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29 Terms

1
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Describe the normal cardiac auscultation.

  • Sounds

  • ECG


• S1: Mitral and tricuspid valves closure (AV valves)

  • Beginning of systole

  • Is longer, louder, lower pitched

• S2: Aortic and pulmonic valves closure.

→ Lub(S1) - dup (S2)

  • Is shorter, not as loud

  • Beginning of diastole


S4 & S3 - usually not heard in small animal patients

  • Atrial depolarization = S4

  • Then Isovolumetric contraction - pressure in left and right ventricles rises

  • Until aortic valve opens (Systole)

  • Pressure falls, aortic and pulmonic valve close or blood would reenter the ventricle

  • Isovolumetric relaxation


<p><strong>• S<sub>1</sub>: Mitral and tricuspid valves closure (AV valves)</strong></p><ul><li><p>Beginning of systole</p></li><li><p>Is longer, louder, lower pitched</p></li></ul><p>• S<sub>2</sub>: Aortic and pulmonic valves closure.</p><p>→ Lub(S<sub>1</sub>) - dup (S<sub>2</sub>)</p><ul><li><p>Is shorter, not as loud</p></li></ul><ul><li><p>Beginning of diastole</p></li></ul><p></p><p>S<sub>4 </sub>&amp; S<sub>3 </sub>- usually not heard in small animal patients</p><ul><li><p>Atrial depolarization = S<sub>4</sub></p></li><li><p>Then <strong>Isovolumetric contraction</strong> - pressure in left and right ventricles rises</p></li><li><p>Until aortic valve opens (Systole)</p></li><li><p>Pressure falls, aortic and pulmonic valve close or blood would reenter the ventricle</p></li><li><p><strong>Isovolumetric relaxation</strong></p></li></ul><p></p>
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What are the main qualities of a heart murmur?

Timing

Intensity

Location

Quality

Pitch


**First three to important to recognize

3
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Describe the features of the timing in regard to a heart murmur.

• Determined by the phase of the cardiac cycle, when did it occur?

• Systolic (If produced from S1 to S2)

  • S1 - beginning of systole

• Diastolic (S2 to S1)

  • S2 - beginning of diastole

• Continuous (machinery)

  • Hear it interrupted, throughout

• Early, Mid, Holo (through whole phase)

  • More nuanced, can be difficult to hear with high HR


4
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Describe the features of the intensity in regard to a heart murmur.

• How loud is the murmur?

• Classification of grade of intensity from I to VI

  • Degree of disease correlated to intensity of the murmur


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Describe the heart murmur scale - grades 1-6 and the definition for each.

  • Definition 1 - Heart Sounds

  • Definition 2 - Localization (How far are you from the source)


<ul><li><p>Definition 1 - Heart Sounds</p></li><li><p>Definition 2 - Localization (How far are you from the source)</p></li></ul><p></p>
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Describe the features of the location in regard to a heart murmur.

  • Where is the loudest murmur present?

  • Left: Apical (Mitral), Basal (Aortic and Pulmonic)-

  • Right: Apical (Tricuspid)


PAM → Side Change → T

<ul><li><p>Where is the loudest murmur present?</p></li><li><p> Left: Apical (Mitral), Basal (Aortic and Pulmonic)-</p></li></ul><ul><li><p> Right: Apical (Tricuspid)</p></li></ul><p></p><p>PAM → Side Change → T</p>
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Describe the features of the pitch and quality in regard to a heart murmur.

Describe how it behaves

• Pitch describes the auscultator's perception of the frequency of the murmur: high / medium / low

• Quality describes the "shape" of the murmur:

• Regurgitant (also called plateau shaped or rectangular, A)

• Ejection (crescendo-decrescendo or diamond shaped, B)

• Blowing (decrescendo, C)

<p>Describe how it behaves</p><p><strong>• Pitch</strong> describes the auscultator's perception of the frequency of the murmur: high / medium / low</p><p><strong>• Quality</strong> describes the "shape" of the murmur:</p><p><strong>• Regurgitant</strong> (also called plateau shaped or rectangular, A)</p><p><strong>• Ejection </strong>(crescendo-decrescendo or diamond shaped, B)</p><p><strong>• Blowing </strong>(decrescendo, C)</p>
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How can you diagnose an arrhythmia?

  • What is a normal rhythm?


• Auscultation provides the first clue to the presence of an arrhythmia and in some cases the rhythm diagnosis.

• When an arrhythmia is auscultated or suspected, however, an ECG (Electrocardiogram) should always be obtained to confirm the diagnosis.

• Normal finding: Sinus rhythm

  • Regular rhythm, characterized by a monotonous S1-S2 cadence.

  • Approximately 60 - 160 bpm in dogs, 140 - 220 in cats.


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What are normal arrhythmias?

• Normal finding: Sinus arrhythmia

• Regularly irregular pattern, associated with breathing.

• Normal or slow heart rate.


• Vagal Tone (VTo) induced:

• Inspiratory → Vto decreases, HR faster.

  • In healthy, relaxed dogs, means heart is saving energy

• Cats = May be a sign of disease with increased vagal tone

  • Since are usually stressed at the vet


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What are some abnormal arrhythmias?

• Atrial fibrillation:

• Generally rapid, irregularly irregular rhythm.

• Variable loudness of heart sounds.

  • ECG - random appearance of peaks


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What are the features of normal respiratory sounds?

• Normal lung sounds are created by the turbulent movement of air in the tracheobronchial tree in large (> 2 mm diameter) airways → Transmission to the chest.

• Ā«BronchovesicularĀ» and Ā«vesicular breath soundsĀ» implies Ā«made in the alveoliĀ» but are transmitted from the large airways.

• Inspiratory: Lobar and large subsegmental bronchi.

• Exhalation: Trachea and the first one to two divisions of bronchi.

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Why do abnormal lung sounds occur?

• Disease state → Altered sound transmission →

  • Efficient high-frequency sound transmission.

• Pleural disease/Pneumothorax → increased reflection of sound

  • Reduced intensity of lung sounds.

  • Not allowing sound waves to reverberate as they should.


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What produces adventitious lung sounds?

• Abnormal lung sounds that are superimposed on normal (or increased/decreased) lung sounds.

• Three categories:

  • Crackles

    • Fine

    • Coarse

  • Wheezes

  • Ronchi


<p>• Abnormal lung sounds that are superimposed on normal (or increased/decreased) lung sounds.</p><p>• Three categories:</p><ul><li><p>Crackles</p><ul><li><p>Fine</p></li><li><p>Coarse</p></li></ul></li><li><p>Wheezes</p></li><li><p>Ronchi</p></li></ul><p></p>
14
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What do ā€œfine cracklesā€ sound like upon auscultation?

  • Common causes?


• High pitch (>800 Hz)

• Quickly muffled, unmusical lung sounds.

• Usually at the beginning/end of breathing.


Findings, not specific for disease, high overlap:

  • Bronchitis

  • Lung oedema

  • Pneumonia

  • Lung fibrosis


Fine Crackles

  • Brief, popping and high pitched, similar to wood burning in a fireplace

  • Heard in both phases, sometimes heard more in one phase than another


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What do ā€œcourse cracklesā€ sound like upon auscultation?

• Usually lower pitch (≄700-800 Hz) and longer duration (<50 msec) than fine crackles.

• You can hear them over the trachea/mouth with/without stethoscopes.

These indicate the presence of fluid in the large airways!

  • Bubbling sound quality, popping, low-pitched, longer and louder, heard in both phases


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What do ā€œwheezesā€ sound like upon auscultation?


• Longer duration (>80-100 msec), relatively high frequency (oft >1000 Hz) compared to crackles.

• Sinusoidal (musical), narrowing of airway, caused by vibration of small bronchial walls.

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What do ā€œronchus/ronchiā€ sound like upon auscultation?


• Longer duration(>100 ms), but in contrast to Wheezes they have deeper frequency (<2-300 Hz).

• Vibration of large airways or air- fluid interface disruption.

• Snoring sound.

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Describe the sounds characteristic of stridor.

• Harsh high-pitched sound produced by obstruction of upper airways:

  • Nasal stridor

  • Pharyngeal stridor

  • Laryngeal stridor

  • Tracheal stridor


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Describe the sounds characteristic of stertor?

• Low pitch nasopharyngeal obstruction.

• Brachycephalic syndrome!

20
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Describe how the pulse is evaluated.

  • Pulse Pressure: Difference between systolic and diastolic pressure

    • Wide difference: Strong, hyperkinetic.

    • Small difference: Weak, hypokinetic (can be high or low)

• Anacrotic Pulse (parvus et tardus): weak and delayed.

• Pulsus alternans: Strong/Weak alternation.

• Pulsus paradoxus: Weak arterial pulse during inspiration, strong during expiration.

  • Sometimes with pericardial effusion or tamponade

• Pulsus bigeminus: Regular alteration of ventricular contractionised by premature

  • Additional beat or pulse wave


<ul><li><p><strong>Pulse Pressure: </strong>Difference between systolic and diastolic pressure</p><ul><li><p>Wide difference: Strong, hyperkinetic.</p></li><li><p>Small difference: Weak, hypokinetic (can be high or low)</p></li></ul></li></ul><p>• Anacrotic Pulse (parvus et tardus): weak and delayed.</p><p>• Pulsus alternans: Strong/Weak alternation.</p><p>• Pulsus paradoxus: Weak arterial pulse during inspiration, strong during expiration.</p><ul><li><p>Sometimes with pericardial effusion or tamponade</p></li></ul><p>• Pulsus bigeminus: Regular alteration of ventricular contractionised by premature</p><ul><li><p>Additional beat or pulse wave</p></li></ul><p></p>
21
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What does the hepatojugular reflex tell you?

Hepatojugular Reflex: cranial part of abdomen squeezed, enters right side, jugular vein becomes enlarged

• Right heart failure: Increase RA → Hydrostatic Pressure Caval Veins

→ Backwards Failure

• Pressure on the liver → rise in venous pressure.

  • May also see ascites - intravascular hydrostatic pressure increases, fluid leaks out


22
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What does CRT evaluation allow you to assess?

• Capillary Refill Time (CRT) parameter to evaluate peripheral perfusion. Normal is below two seconds.

• Cyanosis: bluish discoloration of the mucous membranes

  • Central: systemic deoxygenation of arterial blood, not from lungs

  • Peripheral: localized reduction in oxygenated haemoglobin.

  • Differential: deoxygenation of caudal part of body, with normal oxygenation of head and forelimbs, certain region of body


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What is a cough and how relevant is it in cardiac disease?

• Sudden expiratory effort in which a loud expulsion of air occurs.

• Defensive Mechanism (Stimulus: Irritation).

• Larynx, Trachea, Bronchi, Mediastinum.

• Cardiac disease? NOT in cats!

  • Can produce a cough in dogs, receptors in upper areas or larynx and, trachea and large bronchi

  • Pulomary oedema in alveoli should not trigger coughing reflex, unless it reaches bronchi

• Haemoptysis: Bloody Cough.

<p>• Sudden expiratory effort in which a loud expulsion of air occurs.</p><p>• Defensive Mechanism (Stimulus: Irritation).</p><p>• Larynx, Trachea, Bronchi, Mediastinum.</p><p>• Cardiac disease? <strong>NOT in cats!</strong></p><ul><li><p>Can produce a cough in dogs, receptors in upper areas or larynx and, trachea and large bronchi</p></li><li><p>Pulomary oedema in alveoli should not trigger coughing reflex, unless it reaches bronchi</p></li></ul><p>• Haemoptysis: Bloody Cough.</p>
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What are some potential differentials for coughs?

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Describe the main features of respiratory distress?

• Eupnoea: normal, unlaboured breathing.

• Hyperpnea (more volume) / Tachypnoea (higher frequency)

• Dyspnoea: respiratory difficult

• Paradoxical breathing: Breathing pattern in which the chest moves in on inhalation and out on exhalation.

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Lung depend on _____ and _____.

Ventilation, Circulation

V & Q

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What are differential diagnosis for dyspnoea?

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What is syncope?

• Defined as transient loss of consciousness (TLOC) due to cerebral hypoperfusion, characterized by a rapid onset, short duration, and spontaneous complete recovery.

• Presyncope (near-syncope): used to indicate symptoms and signs that occur before unconsciousness in syncope.

  • Prodrome of syncope

  • Not followed by loss of consciousness


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How can you distinguish a seizure form a syncope event?

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