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periodic compressions of air, water, or other media varying in amplitude and frequency
sound waves
intensity of a sound wave
amplitude
number of compressions or cycles per second measured in Hz cyclers per second
frequency
related to the aspect of perception, sounds higher in frequency are higher in this
pitch
represented by height of each wave
amplitude
measured by the number of waves per second
frequency
tone quality of complexity dependent on the harmonics of the tone
timbre
conveying emotional information by tone of voice
prosody
outer ear structure of flesh and cartilage that sticks out from each side of the head
pinna
alters the reflection of sound waves and helps us locate the source of a sound
pinna
sound waves passing through the auditory canal enter here, evolved ti amplify sound vibrations
middle ear
eardrum that vibrates in the middle ear and connects to three tiny bones (ossicles)
tympanic membrane
membrane of the inner ear that ossicles transmit vibrations to
oval window
structure of the inner ear containing auditory receptors set into motion the fluid by the oval window
cochlea
auditory receptors that lie along the basilar membrane in the cochlea abd stimulate the auditory nerve
hair cells
basilar membrane vibrates in synchrony with sound waves, causing auditory nerve axons to produce action potentials in the same frequency (50hz = 50 action potentions per second)
frequency theory
what is the caveat of frequency theory
refractory period limits the maximum firing rate of any neuronto about 1000hz, far short of the highest frequencies we hear
basilar membrane varies from stiff at its base from stiff at its base where the stirrup meets the cochlea, to floppy at the other end of the cochlea, the apex
place theory
pitch perception depends on which part of the inner ear has cells with the greatest activity level ; the point at which it peaks identifies the frequency of the sound
place theory
area in the superior temporal cortex in which cells respond best to tones of a particular frequency
primary auditory cortex (A1)
where do axons cross over to enable each hemisphere of the forebrain to get most of its input but not all from the opposite ear
the midbrain
this pathway in A1 combines input from the auditory system and the visual system, and it connects to systems for memory and decision making
what pathways
this pathway in A1 localizes sound sources and guides actions toward or away from them
where or how
processes the experience you have of a sound source moving from one place to another, connected to area MT for visual motion and combines visual and auditory information to locate something accurately
planum temporale
damage to this area causes motion deafness, one can hear the sound but can not detect that the sound source is moving
planum temporale
responds to imagined sound and becomes active when people view silent videos that suggest sound
A1
people with damage here have trouble with speech and music but identify and localize single sounds reasonably well
primary auditory cortex
cells from the primary auditory cortex that have a preferred tone and differ in detail from one to another
tonotopic map of sounds
surrounds the primary auditory cortex and responds mainly to meaningful sounds such as animal calls, birdsong,machinery noises, music, and speech
secondary auditory cortex
sound localization and comparing intensity of sound at the two ears, useful up to 2k-3k hz; moreso localize high frequencies
sound shadow
sound localization and sound originates to the side of the head and the sound wave strikes the two ears out of phase , useful up to 1500hz; moreso localize low frequencies
phase difference
cant tell if someone is singing off key and do not detect a wrong note in a melody, but have trouble gauging peoples mood from tone of voice; tone deafness
amusia
people with this have fewer than average connects to the fronal cortex and differences in memory and attention to pitch
amusia
hearing loss that occurs if the bones of the middle ear fail to transmit sound waves properly to the cochlea
conductive/middle ear deafness
can be corrected by surgery or hearing aids have normal cochlea and auditory nerve
conductive/middle ear deafness
damage to the cochlea, hair cells, or auditory nerve, confined to one part of the cochlea and impairs hearing only of certain frequencies
nerve/inner ear deafness
frequent or constant ringing in the ears and can resemble phantom limb in some cases
tinnitus
these senses respond the pressure, bending, or other distortions
mechanical senses
adjacent to the cochlea and monitors movements and directs compensatory movements of your eyes
vestibular organs
sensations from here detect the tilit of the head and the heads acceleration, used to guide eye movement and maintain balance, people with damage here wiggle and fall
vestibular organs
oriented in perpendicular planes filled with a fluid and line that move when the head is accelerated at any angle and sensitive to directional tilit, like on a roller coaster
semicircular canals
calcium carbonate particles that lie next to hair cells, push against hair cells and excite them when your head tilts, tell brain which direction you are moviing. like reading a book when ur moving ur head
otoliths
what does the vestibular organ consist of
saccule, utricle, and three semiciruclar canals
sensory network that monitors the surface of the body and its movements including touch, deep pressure, pain, and temperature
somatosensory system
these detect vibrations or sudden displacements on the skin
pacinian corpuscle
chemical found in hot peppers that produces a painful burning sensation
capsaicin
responds to discrimitive touch and vibration
meissners corpuscles
responds to light touch
merkels disks
responds to skin stretch and roughness
ruffini endings
quickly adapting receptors
pacinian and meissner corpuscles
small receptive fields
meissner corpuscles and merkels disks
large receptive field and slowly adapting receptor
ruffini endings
where does information from touch receptors in the head enter
the central nervous system through cranial nerves
area of the body connected to a particular spinal nerve
dermatome
essential for touch experiences
primary somatosensory cortex, S1
where does information from skin sensations go to , responds to pleasantness or unpleasantness of the sensation
anterior cingulate cortex and insular cortex
what does damage to S1 (somatosensory cortex) do
impairs body perception
in S1, what do unmyelinated axons do
produce emotional aspect of touch, but not conscious sensation of it
guessing the location of a touch while insistinf that they did not feel it, inactivity in S1
numbsense
where do pain and temperature pathway cross
immediately from the receptors to a tract on the contralateral side of the spinal cord
where does touch information cross
travels up the ipsilateral side of the spinal cord to the medulla, then crosses to the contralateral side
what conveys sharp pain
thicker and faster axons
what conveys dull pain
thinner axons
what does mild pain release
glutamate
what does strong pain release
glutamate, neuropeptides, substance P, and CGRP
what part of the brain is active and responsive to emotional aspects of pain
the cingulate cortex and sensory areas responsive to physical pain
the brain puts the brakes on prolonged pain by__, systems that respond to opiate drugs and similar chemicals
opioid mechanisms
area of the brainstem that is rich in endorphin synapses; opiates bind to receptors here and in the spinal cord
periaqueductal gray area
what do opiates act on
the nervous system
nervous systems own opiate type chemicals , transmitters that attach to the same receptors as morphine
endorphins
spinal cord neurons receive messages from pain receptors also receive input from other inputs that can close the pathway for the pain messages
gate theory
relieve certain kinds of pain and act on the periphery of the body as well as the CNS
cannabinoids
condition of chronic pain that lasts long after original cause of pain has ended, can be treated with cannabinoids
neuropathic pain
what causes neuropathic pain
damaged tissues release, histamine, NGF, but also magnify response of nearby heat and pain receptors
what is Nav1.7
sodium voltage gate 7 important for axons conveying pain and olfactory sensations
receptors on the tongue that detect certain chemicals
taste buds
structures on a tongues surface that contain taste buds
papillae
contains the protein miraculin and modifies sweet receptors, enabling acids to stimulate them
miracle berry
decreased response to a stimulus as a result of recent exposure to it
adaptation
reduced response to one taste after exposure to another
cross adaptation
what are the 5 taste receptors
sweet, salty, sour, bitter, umami
this taste receptor permits sodium ions on the tongue to cross its membrane, thereby detects the presense of sodium
salty
this receptor detects the presence of acids
sour
these receptors resemble metabotropic synpases , binding to them activates a G protein that releases a second messenger within the cell
sweetness, bitterness, and umami
how do we detect taste partly by
timing pattern of neuron responses
anything that excites a pain axon reaches the brain as a pain sensation
labelned line
what coding does color vision require
across fiber pattern or combinatorial coding
taste information reaches cells here and can respond also to odors hunger and emotions
insula in the cortex
people with a higher than average number of tastebuds
supertasters
response to chemicals that contact the membranes inside the nose
olfaction
neurons responsible for smell, located on the olfactory epithelium in the rear of the nasal air passages
olfactory cells
receptors on the olfactory cells cilia that extend into the mucous surface of the nasal passage and detect the chemicals of the air you inhale
orthonasal olfaction
receportors on the olfactory cells cilia that extend into the mucous surface of the nasal passage and detect chemicals given off by food in your mouth
retronasal olfaction
in olfactory coding, receptors sensitive to a particular chemical or combination of chemicals connect to a single cluster in the olfactory bulb
glomerulus
the experience some people have in which stimulation of one sense evokes a perception of that sense and another one also , some have increased gray matter and alterned connections
synethesia
where do illusions, or misperceived stimulus occur
in the cerebral cortex