Exercise Physiology and Neuromuscular Control Review

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Vocabulary flashcards covering neuromuscular concepts, motor unit recruitment, autonomic nervous system pathways, and endocrine responses to exercise.

Last updated 3:11 PM on 10/9/26
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19 Terms

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Cerebellum

The brain center involved in correcting errors during fast movements.

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Basal ganglia

The brain center involved in initiating movement that has low dopamine in parkinston’s disease.

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Size principle

The principle that recruits motor units from small to large as force demand increases.

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Rate coding

The mechanism that allows muscle force to continue increasing through a higher firing frequency after motor unit recruitment is complete.

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Muscle spindle

The sensory receptor that detects rapid stretch and initiates the reflex causing the lower leg to kick forward when the patellar tendon is tapped.

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Preganglionic sympathetic neuron

A sympathetic neuron emerging from the spinal cord that releases acetylcholine (ACh\text{ACh}).

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Postganglionic sympathetic neuron to the heart

A sympathetic neuron that releases norepinephrine (NE\text{NE}) to increase heart rate.

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Sweat glands (sympathetic innervation)

A sympathetic nervous system exception that receives acetylcholine (ACh\text{ACh}) rather than norepinephrine.

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Plasma free fatty acid (FFA) reduction mechanisms during heavy exercise

Factors reducing free fatty acid availability at high intensity, including lactate promoting FFA re-esterification, H+H^+ inhibiting hormone-sensitive lipase (HSL), and reduced adipose blood flow.

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Intracellular control of glycogenolysis

The local, intracellular regulation within contracting muscle that primarily drives glycogen breakdown rather than systemic hormonal control alone.

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Hormonal response at 40% VO2 max40\%\,VO_2\text{ max}

An exercise intensity state in which cortisol will likely decrease and growth hormone (GH) shows a minimal or very modest increase.

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Hormonal response at 80% VO2 max80\%\,VO_2\text{ max}

An exercise intensity state in which both cortisol and growth hormone (GH) elevate significantly.

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Lactate inhibition of hormone-sensitive lipase (HSL)

The mechanism that contributes most to the decrease in free fatty acid (FFA) utilization during intense exercise despite elevated lipolytic hormones.

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Propranolol glycogenolysis test

The pharmacological blocking of β-adrenergic receptors that fails to completely prevent muscle glycogen breakdown, demonstrating that plasma epinephrine is not the sole initiator of glycogenolysis.

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Fast-acting hormones

The pair of hormones classified as rapidly acting in the context of exercise, consisting of epinephrine and norepinephrine.

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Blood glucose regulation at 70% VO2 max70\%\,VO_2\text{ max}

The combination of decreased insulin and increased glucagon that helps maintain blood glucose during a 3030-minute run.

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Post-training catecholamine response at identical absolute speed

The decrease in plasma epinephrine (E\text{E}) and norepinephrine (NE\text{NE}) levels during exercise following a 1212-week endurance program because the relative intensity (%VO2 max\%VO_2\text{ max}) is lower.

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Cortisol kinetics during low-intensity exercise

The physiological balance below threshold intensity where plasma cortisol concentration stays the same or decreases primarily because cortisol removal exceeds cortisol secretion.

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Dual control of muscle glycogen breakdown

The two primary regulatory factors responsible for glycogenolysis during exercise: epinephrine–cyclic AMP and Ca2+Ca^{2+}–calmodulin.