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Vocabulary flashcards covering neuromuscular concepts, motor unit recruitment, autonomic nervous system pathways, and endocrine responses to exercise.
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Cerebellum
The brain center involved in correcting errors during fast movements.
Basal ganglia
The brain center involved in initiating movement that has low dopamine in parkinston’s disease.
Size principle
The principle that recruits motor units from small to large as force demand increases.
Rate coding
The mechanism that allows muscle force to continue increasing through a higher firing frequency after motor unit recruitment is complete.
Muscle spindle
The sensory receptor that detects rapid stretch and initiates the reflex causing the lower leg to kick forward when the patellar tendon is tapped.
Preganglionic sympathetic neuron
A sympathetic neuron emerging from the spinal cord that releases acetylcholine (ACh).
Postganglionic sympathetic neuron to the heart
A sympathetic neuron that releases norepinephrine (NE) to increase heart rate.
Sweat glands (sympathetic innervation)
A sympathetic nervous system exception that receives acetylcholine (ACh) rather than norepinephrine.
Plasma free fatty acid (FFA) reduction mechanisms during heavy exercise
Factors reducing free fatty acid availability at high intensity, including lactate promoting FFA re-esterification, H+ inhibiting hormone-sensitive lipase (HSL), and reduced adipose blood flow.
Intracellular control of glycogenolysis
The local, intracellular regulation within contracting muscle that primarily drives glycogen breakdown rather than systemic hormonal control alone.
Hormonal response at 40%VO2 max
An exercise intensity state in which cortisol will likely decrease and growth hormone (GH) shows a minimal or very modest increase.
Hormonal response at 80%VO2 max
An exercise intensity state in which both cortisol and growth hormone (GH) elevate significantly.
Lactate inhibition of hormone-sensitive lipase (HSL)
The mechanism that contributes most to the decrease in free fatty acid (FFA) utilization during intense exercise despite elevated lipolytic hormones.
Propranolol glycogenolysis test
The pharmacological blocking of β-adrenergic receptors that fails to completely prevent muscle glycogen breakdown, demonstrating that plasma epinephrine is not the sole initiator of glycogenolysis.
Fast-acting hormones
The pair of hormones classified as rapidly acting in the context of exercise, consisting of epinephrine and norepinephrine.
Blood glucose regulation at 70%VO2 max
The combination of decreased insulin and increased glucagon that helps maintain blood glucose during a 30-minute run.
Post-training catecholamine response at identical absolute speed
The decrease in plasma epinephrine (E) and norepinephrine (NE) levels during exercise following a 12-week endurance program because the relative intensity (%VO2 max) is lower.
Cortisol kinetics during low-intensity exercise
The physiological balance below threshold intensity where plasma cortisol concentration stays the same or decreases primarily because cortisol removal exceeds cortisol secretion.
Dual control of muscle glycogen breakdown
The two primary regulatory factors responsible for glycogenolysis during exercise: epinephrine–cyclic AMP and Ca2+–calmodulin.