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Trauma in pregnancy
Leading non-obstetric cause of maternal death
Common causes of trauma in pregnancy
Attempted suicide; motor vehicle accidents; falls; assault; blunt force; penetrating trauma; burns
Major complications of trauma in pregnancy
Preterm birth; placental abruption; uterine rupture; fetomaternal haemorrhage; fetal loss
Priority in maternal trauma
Maintain cardiopulmonary function and oxygenation to the woman and baby
Trauma management priority
Treat the woman's condition first
Airway assessment in trauma
Assess ability to talk/phonate; swelling; bleeding; crepitus; cervical spine injury; burns
Airway management in trauma
Intubation; spinal immobilisation; surgical airway if required
Breathing assessment in trauma
Assess bilateral breath sounds; respiratory effort; respiratory rate; oxygen saturation
Breathing management in trauma
Supplemental oxygen and emergency treatment based on injury
Circulation assessment in trauma
Assess colour; consciousness; bleeding; pulse; blood pressure
Circulation management in trauma
Direct pressure to bleeding; IV access; isotonic crystalloid; treat injury
Disability assessment in trauma
Assess consciousness; pupils; glucose; trauma signs; movement of all four extremities
GCS <8 management
Intubate; cranial imaging; optimise oxygenation
Exposure assessment in trauma
Remove clothing; avoid hypothermia; perform head-to-toe examination
Position after trauma >20 weeks
Left lateral position to reduce inferior vena cava compression
Alternative to left lateral positioning
Manual uterine displacement
Why left lateral positioning matters in maternal collapse
Reduces vena cava compression and improves effectiveness of CPR
Fetal wellbeing after trauma
CTG after 24 weeks gestation
Anaphylaxis
Anaphylaxis
Life-threatening emergency caused by an immune response to an allergen
Onset of anaphylaxis
Can occur within 10 minutes of allergen exposure
Routes of allergen exposure
Contact; inhalation; consumption
Effect of anaphylaxis on blood vessels
Vasodilation causing fluid accumulation and swelling
Severe complication of anaphylaxis
Pulmonary oedema causing respiratory collapse
Immunological anaphylaxis pathway
Allergen activates B cells → IgE antibodies → IgE binds mast cells → repeat exposure triggers cytokine release
Prevention of anaphylaxis
Avoid known triggers
Signs of anaphylaxis
Swelling; runny nose; hives; itching; pain; lightheadedness; diarrhoea; vomiting; shortness of breath; hoarseness
Initial management of anaphylaxis
Remove trigger and follow DRSABCD
Key treatments for anaphylaxis
Oxygen; adrenaline; IV fluids
Additional treatments for anaphylaxis
Bronchodilators; antihistamines; corticosteroids
Monitoring during anaphylaxis
Maternal vital signs and fetal condition
When expedited birth may be considered in anaphylaxis
If the woman continues to deteriorate
Maternal collapse and cardiac arrest
Maternal collapse
Sudden deterioration that may progress from shock and loss of consciousness to cardiac arrest or death
Causes of maternal collapse
Cardiac; respiratory; neurological; placental; uterine; haemorrhage; trauma; sepsis causes
A–H causes of maternal collapse
A anaesthetic/accident; B bleeding; C cardiac; D drugs; E embolic; F fever; G general/hypoxia/electrolytes; H hypertension
Amniotic fluid embolism
Amniotic fluid/fetal material enters maternal circulation causing an anaphylactic reaction and cardiorespiratory collapse
DIC
Disseminated intravascular coagulation causing widespread clotting followed by depletion of platelets/clotting factors and uncontrolled bleeding
Why cardiac arrest is more complex in pregnancy
Reduced venous return from uterine compression and physiological changes of pregnancy
Position during maternal CPR
Left tilt or manual uterine displacement to reduce vena cava compression
Chest compression considerations in pregnancy
Chest compressions may need to be positioned higher
Defibrillation in pregnancy
Shocks are appropriate
Cardiac arrest resuscitation
DRSABCDE and CPR
Amiodarone in pregnancy cardiac arrest
Notes state NO amiodarone
Emergency caesarean consideration during arrest
Notes indicate consideration after approximately 4 minutes of CPR without return of pulse
Multiple pregnancy
Multiple pregnancy
Pregnancy involving more than one fetus
Risk of multiple pregnancy
Increased significantly compared with singleton pregnancy
Risk factors for multiple pregnancy
Genetic predisposition; previous multiple pregnancy; high parity; advanced maternal age; assisted reproductive technology
Dizygotic twins
Twins arising from two or more fertilised eggs
Monozygotic twins
Twins arising when one fertilised egg splits into two or more identical fetuses
Chorionicity
Number of placentas present in a multiple pregnancy
Dizygotic chorionicity
Always diamniotic/dichorionic
Monozygotic DCDA timing
Division during the first 3 days after ovulation
Monozygotic MCDA timing
Division approximately 4–8 days after fertilisation
Monozygotic MCMA timing
Division approximately 8–12 days after fertilisation
Conjoined twins
Result from later division of the zygote
Early ultrasound in twins
Helps determine zygosity and chorionicity and assists care planning
Complications of multiple pregnancy
Low birth weight; preterm birth; placenta previa; placental abruption; IUGR; pre-eclampsia; GDM; anaemia; malpresentation; birth asphyxia; PROM; GBS; PPH
Twin-to-twin transfusion syndrome
Blood is transferred from one twin to the other through vessels in a shared placenta
Twin-to-twin transfusion risk
Occurs in monochorionic twins
Effect of twin-to-twin transfusion
Disproportionate growth between twins
Preterm birth management in twins
Tocolytics; antibiotics; corticosteroids; magnesium sulphate as indicated
Antenatal care for multiple pregnancy
Multidisciplinary care with increased surveillance and scans
Key early assessment in multiple pregnancy
Dating and determining chorionicity
Birth options for twins
Elective induction or caesarean birth may be offered
Vaginal birth for DCDA twins
Can be considered when the first twin is cephalic
Fetal monitoring during twin birth
CTG recommended
Postnatal consideration after twin birth
Support transition to parenting more than one baby and consider mode of birth and recovery needs
Prematurity
Preterm birth
Birth before 37 completed weeks
Extremely preterm
Less than 28 weeks
Very preterm
28 to less than 32 weeks
Moderate preterm
32 to 33+3 weeks
Late preterm
34 to 36+6 weeks
Major risks of prematurity
IVH; NEC; sepsis; thermoregulation difficulties; nutrition difficulties
Preterm infant skin
Immature epidermis causes thermal instability; fluid loss; skin breakdown; infection risk
Respiratory problems in preterm infants
Respiratory distress and apnoea due to immature respiratory systems
Other challenges of prematurity
Feeding difficulties and developmental challenges
Temperature complications in prematurity
Hypothermia due to limited physiological reserves and immature thermoregulation
Why hypothermia is dangerous in preterm infants
Energy is diverted from growth and healing toward heat production
Neutral thermal environment
Environment where normal temperature is maintained with minimal metabolic and oxygen expenditure
Common complications of preterm infants
Respiratory challenges; thermal instability; feeding difficulties; jaundice
Preterm infant nutrition
Immature gastrointestinal system and difficulties maintaining suck-swallow coordination
Neonatal thermoregulation
Why newborns lose heat easily
Immature thermoregulation; high surface-area-to-weight ratio; limited energy reserves
Effect of neonatal hypothermia
Increased oxygen consumption; metabolic acidosis; increased mortality risk
Non-shivering thermogenesis
Heat production through brown adipose tissue in response to cold stress
Brown adipose tissue
Tissue around the neck
Preterm brown fat
Reduced brown fat stores make preterm infants less effective at thermogenesis