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olfaction
smell
gustation
taste
odorants
dissolve in nasal mucous and bind to chemioreceptors to produce a smell
olfactory epithelium
smell sensory organ, contains olfactory receptor cells, supporting cells, and basal cells
olfactory receptor cells
bipolar neurons modified to detect odors, contain enlarged ends called olfactory bulbs
olfactory hairs
modified cilia with chemoreceptors
what happens when odorants bind to chemoreceptors
adenylate cyclase converts ATP to cAMP opening ion channels
t/f: olfactory nerves travel through the thalamus to the cerebral cortex
false
taste cells
receptors of taste, found in taste buds
taste buds
sensory organ of the mouth/throat, found in papillae
lingual papillae
bumps on your tongue, come in 4 shapes
shapes of papillae
filiform, vallate, foliate, fungiform
filiform papillae
only type of papillae to have no taste buds. most common. help manipulate food. found towards the middle of the tongue.
vallate papillae
form a v on the back of the tongue, only 8-12 in the mouth
foliate papillae
most sensitive taste buds, found on the sides of the tongue
fungiform papillae
scattered randomly along the tongue, appear as red dots
taste pore
apex of taste cell
taste hairs
microvilli extending through the taste pore
tastant
chemical that dissolves in saliva and enters taste pores to create a taste
types of tastes
sour, salty, sweet, bitter, umami
sour taste
occurs when hydrogen ions flood a cell. taste buds found on the inferior lateral parts of the tongue.
salty taste
when sodium diffuses through leak channels. taste buds found on the posterior lateral parts of the tongue.
bitter taste
when tastants bind to g-protein receptors. most sensitive taste. taste buds found on the back of the tongue
sweet taste
when tastants bind to g-protein receptors. taste buds found on the front of the tongue
umami taste
when amino acids bind to g-protein receptors
palpebrae
eyelids
lacrimal glands/ducts/canals/sac
the path tears take. through the gland to the duct, into your eyes, and drained out of the canals into the sac in your nose
fibrous tunic
outside layer of eye. contains cornea and sclera
vascular tunic
middle layer of the eye. contains iris, choroid, and ciliary body
nervous tunic
most deep layer of the eye, contains retina
how do we see (simply)
light hits the lens, which reflects it onto the retina, making it an action potential which travels along the optic nerve to the brain
visible spectrum
the part of the light spectrum we can see
refraction
bending of light when it hits an object denser than the air, causing its speed to slow
convex
thickest in the center, shape of the lens
focal point
where light rays converge
t/f: you see upside down and inverted, but your brain fixes it
true
accomodation
the ciliary muscles pull on the lens to change its shape and shift focus
what happens when you go from looking at something close up to something far away?
ciliary muscles relax, making suspensory ligaments tighten and pull on the lens
pupil constriction related to distance
small pupil means your looking ahead at a distance, big pupils mean you’re looking at something close
emmetropic
perfect vision
hyperopia
farsightedness: light focuses behind the retina instead of on it, causing blurry vision up close
myopia
nearsightedness: light focuses in front of the retina instead of on it, causing blurry vision at distances
presbyopia
age-related decline in vision, causing farsightedness
astigmatism
instead of forming one image due to the refraction, two images are created, resulting in blurred vision
rods
detect light levels
cones
detect color
dark current
photoreceptors continuously release glutamate. stops when light is perceived, which makes bipolar cell depolarize, generating an action potential
why are rods unique
they are depolarized at rest, light causes hyperpolarization
rhodopsin
chemical in membranous discs of rod, contains opsin + retinol
how do rods turn light into an action potential?
retinol changes shape from cis to trans when in contact with light, which makes the alpha proteins break off of the g protein, causing GPT to GTP and cGMP to GMP, which closes sodium leak channels, creating hyperpolarization
colors perceived by cones
red, blue, green
sounds with a bigger amplitude
louder
sounds with more frequency
higher pitched
timbre
how smooth a wave is
how do we hear?
soundwaves strike the tympanic membrane which vibrates causing the ossicles to also vibrate and hit the oval window. the sound travels through the scala vestibuli and produces a wave in its perilymph which distorts the endolymph and activates the hair cells which either depolarize or hyperpolarize depending on the input. while that information goes to the brain, the waves enter the scala tympani and exit through the round window.
scala vestibuli
runs from the oval window to the heliocotrema
scala tympani
runs from the heliocotrema to the round window
perilymph
fluid in scala vestibuli/tympani
endolymph
fluid in the basilar membrane/scala media
hair cells
sterocilia sensory receptors that detect wavelengths in the spiral organ/organ of Corti that convert wavelengths to action potentials
kinocilium
longest sensory hair, direction it points determines if the cell depolarizes or hyperpolarizes
movement towards kinocilium
potassium channels open, cell is depolarized, neurotransmitters are released
movement away from kinocilium
potassium channels close, cell is hyperpolarized
static labyrinth
evaluates the position of the head relative to acceleration, contains the utricle and saccule
dynamic labyrinth
evaluates the position of the head in a 3d plane, contains the semicircular canals
utricular macule
senses horizontal movement
saccular maccule
senses vertical movement
otoliths
tiny crystals embedded un the otolith membrane which weigh the membrane and the macular hairs down, making it more sensitive to movement
cupula
on top of the crista ampularis in the ampula, hair cells that act as a float and are displaced by movement