1/290
Looks like no tags are added yet.
Why do we assess our clients?
- To gather baseline data like age, sex, and athletic norms
-To gather information to help with exercise prescription
- Evaluate profess in a training block
- Motivational
SMARRT Goals
Specific, measurable, aggressive but attainable, relevant, time-bound
When should we assess our clients?
- Prior to exercise training program
- Periodically during/ after training block
- Long-term
How do we interpret our assessments?
- External comparisons
- Internal Comparisons
External Comparisons
Compared to age, sex, athletic norms
Internal Comparisons
- Comparing bilaterally
Comparing longitudinally
Testing Organization
- Create testing protocol and organization of tests
- Have all data collection sheets prepared
- Check all equipment
- Room temperature (68-72)
Test Enviorment
- Test anxiety
- Client emotions
- Room temperature
- Room ventilation
General Indications for stopping and exercise test
- Onset of angia or angia like symptoms
- Drop in SBP less than or equal to 10mmHG with an increase in work rate
- Excessive rise in BP
- Shortness of breath, wheezing, leg cramping
- Signs of poor perfusion
- Noticeable change in heart rhythm by palpation or ausculation
Signs of poor perfusion
Light-headness, confusion, neasuea, pallor, cyanosis, cold and clammy skin
Test Administartion
- Selection and training of testers
- Recording forms
- Test format
- Testing batteries and multiple testing trials
- Preparing athletes for testing
How long should one rest between attempts that are close to the athletes maximum?
2 to 3 minutes
How long should one rest between tests are close to maximum
3 to 5
How long should be the rest between test batteries?
5
What is the kinetic chain?
Consists of the muscular system, skeletal system, and nervous system
Muscular system
Functional anatomy
Skeletal system
Bones and their articulation
Nervous system
As it relates to movement
Endomorph
Round and heavy body type
Mesomorph
Broad shoulder, narrow hips, muscular
Ectomorph
Narrow shoulders and hip, very little muscle or body fat
What are common deviations
- Upper crossed syndrome
-Lower crossed syndrome
- Pronation distortion syndrome
What muscles are tight or over active in upper crossed syndrome
Pec major/minor, Levator scapulae, upper trapezius, cervical extensors
What muscles are inhibited or weak in upper crossed syndrome
Rhomboids, middle/ lower trap, serratus anterior, cervical flexors
What is upper crossed syndrome?
forward head and rounded shoulders
What is lower crossed syndrome?
anterior tilt to the pelvis (arched lower back)
What muscles are tight or over active in lower crossed syndrome?
Hip flexors, gastrocnemius/ soleus, lumbar extensors, adductor complex
What muscles are inhibited or weak in lower crossed syndrome?
Abdominals, gluteus maximas, gluteus medius, and hamstrings
What is pronation distortion syndrome
Excessive foot pronation, knee flexion, internal rotation, and also prevents valgus stress during functional activity
What is the FMS?
A batteru of 7 fundamental movement patterns (tests) designed to evaluate movement dysfunction and asymmetries
What are movemnts of the FMS?
Deep squat, incline lunge, shoulder mobility, active straight leg raise, trunk stability pushup, rotary stability
What is a 3 in FMS Scoring?
Able to perform the test correctly
What is a 2 in FMS Scoring?
Able to perform the test correctly in a modified position or able to partially complete the test
What is a 1 in FMS Scoring?
Unable to successfully complete the test
What does a 0 mean in FMS Scoring?
Pain during the test or clearing test
What is ROM?
The total avilable amount of movement in a joint
How can we evaluate precise ROM?
Goniometer, inclinometer, and tape measure
What are the three types of ROM?
Active, passive, resisted
Active Range of Motion
The arc of motion attained by a subject during unassisted voluntary joint motion
Passive Range of Motion
The arc of motion attained by an examiner without assistance by the subject
End Feel
The limitation at the end of the ROM that prevents the movement from going any further
Normal end feel
Soft tissue approximation, firm muscular, capsular, or ligamentous stretch, hard bone contracting bone
Abnormal end feel
Soft tissue edema, synovitis, firm increase muscular tonus, capsular, ligamentous or muscular shortening, hard osteoarthritis, loose bodies in joint, myositis ossificans, fracture
Empty end feel
No real end feel, rom never reached due to pain, no resistance except patient gaurding, acute joint inflammation, brusitis, fracture, psychogenic origin
Goniometry
The use of instruments to measure the range of motion of body joints
Goniometer starting position
0 degrees
Exceptions for goniometer starting position
Shoulder and hip rotation and forarm pronation/supination
Strength assessment
-MMT/RROM
-Isokinetic
-Dynamometry
-Max strength tests???
Handheld Dynamometry
Easy and objective measurement of strength
Fusiform
Fibers arranged parallel to line form origin insertion
Pennate
Fibers inserted obliquely into tendon that extends length of muscle on one side or through the belly
Does a long fusiform increase or decrease the vulnerability to stretch
Increase
Does pennate increase or decrease the vulnerability to stretch
Decrease
Fan Shaped fasciculi arrangement
Looks like a group of muscles formed side by side in a fan shape
Subluxation
Excessive translation of the humeral head without complete seperation
Anterior dislocation
The result of an anterior force on the shoulder, forced abduction, and external rotation
Posterior dislocation
Occurs due to forced adduction and internal rotation or fallon on an extended and internally rotated shoulder
Characteristics of an anterior dislocation
Flattened deltoid, prominent humeral head in axilla, arm carried in slight abduction and external rotation, and moderate pain and disability
Characteristics of posterior dislocation
Severe pain and disability, arm carried in adduction and internal rotation, prominent acromion and coracoid process, limited external rotation and elevation
Bankart Lesion
Permanent anterior defect of labrum
Hill Sachs Lesion
Caused by compression of cancellous bone against anterior glenoid rim creating a diviot in the humeral head
SLAP lesion
Defect in superior labrum that begins posteriorly and extends anteriorly impacting attachment of long head of biceps on labrum
Possible complications of shoulder dislocations
Bracial nerves and vessels may be compromised, rotator cuff injuries, fractures, bicipital tendon subluxation and transverse ligament rupture
Type 1 SLAP
Fraying of the superior labrum with no detatchment of the biceps insertion
Type II SLAP
Detachment of the superior labrum and biceps tendon
Type III SLAP
Bucket handle tear of the superior labrum with the biceps tendon
Type SLAP IV
Bucket handle tear of superior labrum with biceps tendon tearing
Anterior chronic instability
May have clicking or pain; complain of dead arm during cocking phase; pain posteriorly possibly impingment, positive apprehension test
Posterior Chronic Instability
Possible impingement, loss of internal rotation; crepitation; increased laxity; pain anteriorly and posteriorly
Multidirectional Chronic Instability
Inferior laxity; positive sulcus sign; pain and clicking with arm at side, possible signs and sympotoms associated with anterior and posterior instability
What is an acromioclavicular sprain commonly called?
Seperated shoulder
How does an acromioclavicular strain happen?
Direct blow that is upward force from humerus
What is an acromioclavicular sprain?
Force driving clavicle away from scapula
What is ruptured during an acromioclavicular sprain?
Ac and or coracoclavicular ligament
Grade 1 AC joint sprain
Point tenderness and pain w/ movement; no disruption of AC joint
Grade 2 AC joint sprain
tear or rupture of AC ligament, partial displacement of lateral end of clavicle; pain, point tenderness and decreased ROM (abduction/adduction)
Grade 3 AC Joint Sprain
Rupture of AC and CC ligaments
Grade 4 AC Joint Sprain
Posterior dislocation of clavicle
Grade 5 AC Joint Sprain
Loss of AC and CC ligament; tearing of deltoid and trapezius
Grade 6 AC Joint Sprain
Displacement of clavicle behind the coracobrachials
AC Joint Sprain Management
Ice, stabilization, referral to physcian
What AC Joint sprains are non-opprative?
1-3
What AC Joint sprains require surgey?
4-6
How do fractures of the elbow occur?
FOOSH and a direct blow
What bones are still aligned during a dislocation of the elbow?
Condyles and humerus
What may condyle fractures result in?
Gunstock deformity
Postolateral dislocation stage 1
Disruptions of the ulnar portion of the laterally collateral ligament complex with postolateral subluxation and spontaneous reduction
postolateral dislocation stage II
Continues disruption anteriorly and posteriorly with accompanying partial dislocation
postolateral dislocation stage 3A
A disruption of all soft tissues except for the anterior bundle of the MCL. Complete posterior dislocation
Postolateral dislocation stage 3B
Complete rupture of the MCL complex with gross valgus and vargus instability
Scaphoid fracture MOI
FOOSH- compressing scaphoid between radius and second row of carpal bones
Signs and symptoms of schaphoid fracture
- Pain and swelling in anatomical snuff box
- Pain with radial deviation
- May be confused with a sprain
Scaphoid fracture management
- Immobilization = 6wks
- Remobilize --> restore strength
Often has complications
May require surgery
Protect against impact = 3months
Impingment Syndrome
Strong relationship between rotator cuff weakness, altered glenohumeral and scapulothoracic kinemetics, and impingement
Impingement Syndrome Signs and Symptoms
• pain with palpation of subacromial space or tendons
• Overhead athletes = may see increased GH external rotation and decreased internal rotation (GIRD)
• Positive impingement and empty can tests
Neer's progressive stages of shoulder impingement: Stage 1
- Supraspinatus or biceps tendon injury
- Point tenderness
- Pain with abduction and resisted supination with external rotation; edema, thickening of rotator cuff and bursa
- Occurs in patients less than 25 years old
Neer's progressive stages of shoulder impingement: Stage 2
- Permanent thickening
- Fibrosis of supraspinatus and biceps tendon
- Aching during activity that worsens at night
- May experience restricted arm motion
Neer's progressive stages of shoulder impingement: Stage 3
- History of shoulder problems and pain
- Tendon defect (3/8") or possible muscle tear and permenant scar tissue and thickening of rotator cuff
- Patients 25-40 years old
Neer's progressive stages of shoulder impingement: Stage 4
- Infraspinatus and supraspinatus wasting
- Pain during abduction
- Tendon defect greater than 3/8"
- Limited active ROM
- Weak resistive ROM and clavicle degeneration
What is lateral epicondylitis known as
Tennis elbow