Sports medicine exam 2 review

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291 Terms

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Why do we assess our clients?

- To gather baseline data like age, sex, and athletic norms

-To gather information to help with exercise prescription

- Evaluate profess in a training block

- Motivational

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SMARRT Goals

Specific, measurable, aggressive but attainable, relevant, time-bound

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When should we assess our clients?

- Prior to exercise training program

- Periodically during/ after training block

- Long-term

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How do we interpret our assessments?

- External comparisons

- Internal Comparisons

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External Comparisons

Compared to age, sex, athletic norms

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Internal Comparisons

- Comparing bilaterally

Comparing longitudinally

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Testing Organization

- Create testing protocol and organization of tests

- Have all data collection sheets prepared

- Check all equipment

- Room temperature (68-72)

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Test Enviorment

- Test anxiety

- Client emotions

- Room temperature

- Room ventilation

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General Indications for stopping and exercise test

- Onset of angia or angia like symptoms

- Drop in SBP less than or equal to 10mmHG with an increase in work rate

- Excessive rise in BP

- Shortness of breath, wheezing, leg cramping

- Signs of poor perfusion

- Noticeable change in heart rhythm by palpation or ausculation

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Signs of poor perfusion

Light-headness, confusion, neasuea, pallor, cyanosis, cold and clammy skin

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Test Administartion

- Selection and training of testers

- Recording forms

- Test format

- Testing batteries and multiple testing trials

- Preparing athletes for testing

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How long should one rest between attempts that are close to the athletes maximum?

2 to 3 minutes

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How long should one rest between tests are close to maximum

3 to 5

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How long should be the rest between test batteries?

5

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What is the kinetic chain?

Consists of the muscular system, skeletal system, and nervous system

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Muscular system

Functional anatomy

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Skeletal system

Bones and their articulation

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Nervous system

As it relates to movement

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Endomorph

Round and heavy body type

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Mesomorph

Broad shoulder, narrow hips, muscular

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Ectomorph

Narrow shoulders and hip, very little muscle or body fat

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What are common deviations

- Upper crossed syndrome

-Lower crossed syndrome

- Pronation distortion syndrome

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What muscles are tight or over active in upper crossed syndrome

Pec major/minor, Levator scapulae, upper trapezius, cervical extensors

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What muscles are inhibited or weak in upper crossed syndrome

Rhomboids, middle/ lower trap, serratus anterior, cervical flexors

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What is upper crossed syndrome?

forward head and rounded shoulders

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What is lower crossed syndrome?

anterior tilt to the pelvis (arched lower back)

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What muscles are tight or over active in lower crossed syndrome?

Hip flexors, gastrocnemius/ soleus, lumbar extensors, adductor complex

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What muscles are inhibited or weak in lower crossed syndrome?

Abdominals, gluteus maximas, gluteus medius, and hamstrings

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What is pronation distortion syndrome

Excessive foot pronation, knee flexion, internal rotation, and also prevents valgus stress during functional activity

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What is the FMS?

A batteru of 7 fundamental movement patterns (tests) designed to evaluate movement dysfunction and asymmetries

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What are movemnts of the FMS?

Deep squat, incline lunge, shoulder mobility, active straight leg raise, trunk stability pushup, rotary stability

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What is a 3 in FMS Scoring?

Able to perform the test correctly

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What is a 2 in FMS Scoring?

Able to perform the test correctly in a modified position or able to partially complete the test

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What is a 1 in FMS Scoring?

Unable to successfully complete the test

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What does a 0 mean in FMS Scoring?

Pain during the test or clearing test

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What is ROM?

The total avilable amount of movement in a joint

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How can we evaluate precise ROM?

Goniometer, inclinometer, and tape measure

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What are the three types of ROM?

Active, passive, resisted

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Active Range of Motion

The arc of motion attained by a subject during unassisted voluntary joint motion

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Passive Range of Motion

The arc of motion attained by an examiner without assistance by the subject

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End Feel

The limitation at the end of the ROM that prevents the movement from going any further

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Normal end feel

Soft tissue approximation, firm muscular, capsular, or ligamentous stretch, hard bone contracting bone

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Abnormal end feel

Soft tissue edema, synovitis, firm increase muscular tonus, capsular, ligamentous or muscular shortening, hard osteoarthritis, loose bodies in joint, myositis ossificans, fracture

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Empty end feel

No real end feel, rom never reached due to pain, no resistance except patient gaurding, acute joint inflammation, brusitis, fracture, psychogenic origin

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Goniometry

The use of instruments to measure the range of motion of body joints

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Goniometer starting position

0 degrees

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Exceptions for goniometer starting position

Shoulder and hip rotation and forarm pronation/supination

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Strength assessment

-MMT/RROM

-Isokinetic

-Dynamometry

-Max strength tests???

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Handheld Dynamometry

Easy and objective measurement of strength

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Fusiform

Fibers arranged parallel to line form origin insertion

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Pennate

Fibers inserted obliquely into tendon that extends length of muscle on one side or through the belly

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Does a long fusiform increase or decrease the vulnerability to stretch

Increase

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Does pennate increase or decrease the vulnerability to stretch

Decrease

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Fan Shaped fasciculi arrangement

Looks like a group of muscles formed side by side in a fan shape

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Subluxation

Excessive translation of the humeral head without complete seperation

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Anterior dislocation

The result of an anterior force on the shoulder, forced abduction, and external rotation

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Posterior dislocation

Occurs due to forced adduction and internal rotation or fallon on an extended and internally rotated shoulder

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Characteristics of an anterior dislocation

Flattened deltoid, prominent humeral head in axilla, arm carried in slight abduction and external rotation, and moderate pain and disability

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Characteristics of posterior dislocation

Severe pain and disability, arm carried in adduction and internal rotation, prominent acromion and coracoid process, limited external rotation and elevation

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Bankart Lesion

Permanent anterior defect of labrum

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Hill Sachs Lesion

Caused by compression of cancellous bone against anterior glenoid rim creating a diviot in the humeral head

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SLAP lesion

Defect in superior labrum that begins posteriorly and extends anteriorly impacting attachment of long head of biceps on labrum

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Possible complications of shoulder dislocations

Bracial nerves and vessels may be compromised, rotator cuff injuries, fractures, bicipital tendon subluxation and transverse ligament rupture

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Type 1 SLAP

Fraying of the superior labrum with no detatchment of the biceps insertion

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Type II SLAP

Detachment of the superior labrum and biceps tendon

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Type III SLAP

Bucket handle tear of the superior labrum with the biceps tendon

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Type SLAP IV

Bucket handle tear of superior labrum with biceps tendon tearing

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Anterior chronic instability

May have clicking or pain; complain of dead arm during cocking phase; pain posteriorly possibly impingment, positive apprehension test

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Posterior Chronic Instability

Possible impingement, loss of internal rotation; crepitation; increased laxity; pain anteriorly and posteriorly

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Multidirectional Chronic Instability

Inferior laxity; positive sulcus sign; pain and clicking with arm at side, possible signs and sympotoms associated with anterior and posterior instability

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What is an acromioclavicular sprain commonly called?

Seperated shoulder

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How does an acromioclavicular strain happen?

Direct blow that is upward force from humerus

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What is an acromioclavicular sprain?

Force driving clavicle away from scapula

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What is ruptured during an acromioclavicular sprain?

Ac and or coracoclavicular ligament

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Grade 1 AC joint sprain

Point tenderness and pain w/ movement; no disruption of AC joint

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Grade 2 AC joint sprain

tear or rupture of AC ligament, partial displacement of lateral end of clavicle; pain, point tenderness and decreased ROM (abduction/adduction)

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Grade 3 AC Joint Sprain

Rupture of AC and CC ligaments

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Grade 4 AC Joint Sprain

Posterior dislocation of clavicle

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Grade 5 AC Joint Sprain

Loss of AC and CC ligament; tearing of deltoid and trapezius

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Grade 6 AC Joint Sprain

Displacement of clavicle behind the coracobrachials

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AC Joint Sprain Management

Ice, stabilization, referral to physcian

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What AC Joint sprains are non-opprative?

1-3

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What AC Joint sprains require surgey?

4-6

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How do fractures of the elbow occur?

FOOSH and a direct blow

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What bones are still aligned during a dislocation of the elbow?

Condyles and humerus

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What may condyle fractures result in?

Gunstock deformity

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Postolateral dislocation stage 1

Disruptions of the ulnar portion of the laterally collateral ligament complex with postolateral subluxation and spontaneous reduction

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postolateral dislocation stage II

Continues disruption anteriorly and posteriorly with accompanying partial dislocation

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postolateral dislocation stage 3A

A disruption of all soft tissues except for the anterior bundle of the MCL. Complete posterior dislocation

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Postolateral dislocation stage 3B

Complete rupture of the MCL complex with gross valgus and vargus instability

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Scaphoid fracture MOI

FOOSH- compressing scaphoid between radius and second row of carpal bones

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Signs and symptoms of schaphoid fracture

- Pain and swelling in anatomical snuff box

- Pain with radial deviation

- May be confused with a sprain

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Scaphoid fracture management

- Immobilization = 6wks

- Remobilize --> restore strength

Often has complications

May require surgery

Protect against impact = 3months

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Impingment Syndrome

Strong relationship between rotator cuff weakness, altered glenohumeral and scapulothoracic kinemetics, and impingement

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Impingement Syndrome Signs and Symptoms

• pain with palpation of subacromial space or tendons

• Overhead athletes = may see increased GH external rotation and decreased internal rotation (GIRD)

• Positive impingement and empty can tests

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Neer's progressive stages of shoulder impingement: Stage 1

- Supraspinatus or biceps tendon injury

- Point tenderness

- Pain with abduction and resisted supination with external rotation; edema, thickening of rotator cuff and bursa

- Occurs in patients less than 25 years old

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Neer's progressive stages of shoulder impingement: Stage 2

- Permanent thickening

- Fibrosis of supraspinatus and biceps tendon

- Aching during activity that worsens at night

- May experience restricted arm motion

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Neer's progressive stages of shoulder impingement: Stage 3

- History of shoulder problems and pain

- Tendon defect (3/8") or possible muscle tear and permenant scar tissue and thickening of rotator cuff

- Patients 25-40 years old

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Neer's progressive stages of shoulder impingement: Stage 4

- Infraspinatus and supraspinatus wasting

- Pain during abduction

- Tendon defect greater than 3/8"

- Limited active ROM

- Weak resistive ROM and clavicle degeneration

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What is lateral epicondylitis known as

Tennis elbow