1/110
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai | Chat |
|---|
No analytics yet
Send a link to your students to track their progress
Cognition
The study of how the brain processes, stores, and uses information; topics include perception, attention, memory, social cognition, cognitive control, and consciousness. [Lecture 1]
Model (in cognitive science)
A simplified representation that helps researchers understand and predict cognitive processes. [Lecture 1]
Black box problem
The brain's internal processes cannot be observed directly, so researchers infer them from observable environmental inputs and behavior. [Lecture 1]
Behavioral paradigm
A controlled cognitive task used to infer mental processes by measuring behavior such as response time, accuracy, or eye movements. [Lectures 1-2]
Case study
An in-depth study of a person, often with brain damage, used to examine whether a brain area is necessary for a cognitive function. [Lectures 1-2]
Brain stimulation
Manipulating activity in a brain region to test whether it causally contributes to a function. [Lectures 1-2]
Brain imaging
Methods used to observe brain anatomy or activity; functional imaging usually identifies correlations rather than causation. [Lectures 1-2]
Mind-body problem
The question of how physical brain tissue can give rise to thoughts, feelings, emotions, and awareness. [Lecture 1]
Dualism
Descartes's view that the nonphysical, immortal mind and the physical, mortal brain are separate but interacting substances. [Lecture 1]
Dual-aspect theory
Spinoza's view that mind and brain are subjective and objective aspects of the same underlying thing. [Lecture 1]
Emergent property
A property that arises from interactions among simpler components but is not found in the components individually. [Lecture 1]
Reductionism
The view that the mind can be explained entirely in biological terms, such as neuronal firing patterns and neurotransmitters. [Lecture 1]
Thomas Willis
Seventeenth-century physician called the father of clinical neurology; linked brain damage to cognitive dysfunction through long-term observation and postmortem dissection. [Lecture 1]
Localism (localizationism)
The view that particular brain regions support particular functions. [Lecture 1]
Holism
The view that the whole brain participates in each behavior or function. [Lecture 1]
Phrenology
Early nineteenth-century practice claiming that use enlarged specific brain regions and created skull bumps that revealed mental traits; scientifically invalid but helped popularize localization. [Lecture 1]
Franz Gall
Leader of phrenology and an early advocate of the localizationist view. [Lecture 1]
Pierre Flourens
Supported holism after finding that birds with brain lesions could recover regardless of lesion location. [Lecture 1]
Paul Broca and patient Tan
Broca linked Tan's inability to produce speech to a lesion in the left anterior brain, supporting localization of speech production. [Lecture 1]
Broca's aphasia
Impaired speech production associated with damage to Broca's area. [Lectures 1-2]
Carl Wernicke
Linked loss of language comprehension to a lesion in a posterior brain region. [Lecture 1]
Wernicke's aphasia
Impaired language comprehension associated with damage to Wernicke's area. [Lectures 1-2]
Wilder Penfield
Neurosurgeon who stimulated brain tissue in epilepsy patients and mapped movements, sensations, and memories, helping develop the motor homunculus. [Lecture 1]
Motor homunculus
A topographic map of body movement represented along the motor cortex; cortical space reflects degree of use, not body-part size. [Lecture 1]
Somatosensory homunculus
A topographic map of body sensations represented along the somatosensory cortex. [Lecture 1]
Modern view of localization versus holism
Simple processes may be localized, while complex functions depend on networks of multiple brain regions working together. [Lecture 1]
Ganglia
Clusters of nerve cells. [Lecture 1]
Cerebrum
The brain other than the cerebellum; includes all four cerebral lobes. [Lecture 1]
Neuron
A nerve cell; a canonical neuron receives input through dendrites and sends output through an axon. [Lecture 1]
Dendrites
Branching neuron structures that primarily receive input. [Lecture 1]
Soma (cell body)
The main part of a neuron containing the nucleus and much of the cell's machinery. [Lecture 1]
Axon
The neuron structure that carries output away from the cell body. [Lecture 1]
Golgi stain
A cell-staining method that reveals neuron cell bodies and processes, helping researchers study cytoarchitecture. [Lecture 1]
Cytoarchitecture
The organization of tissue based on cell types and their arrangement. [Lecture 1]
Brodmann's areas
Cortical subdivisions identified from differences in cell types and cytoarchitecture in postmortem brains. [Lecture 1]
Gray matter
Brain tissue made primarily of cell bodies and dendrites. [Lecture 1]
White matter
Brain tissue made primarily of axons. [Lecture 1]
Gyrus (plural: gyri)
A ridge or protrusion on the cerebral cortex. [Lecture 1]
Sulcus (plural: sulci)
A groove or invagination on the cerebral cortex. [Lecture 1]
Corpus callosum
Large bundle of axons connecting the brain's left and right hemispheres. [Lecture 1]
Frontal lobe
Anterior cerebral lobe involved broadly in planning, decision-making, cognitive control, and motor functions. [Lecture 1]
Parietal lobe
Superior-posterior cerebral lobe involved broadly in somatosensation and spatial processing. [Lecture 1]
Temporal lobe
Lateral-inferior cerebral lobe involved broadly in audition, memory, object recognition, and language. [Lecture 1]
Occipital lobe
Posterior cerebral lobe involved primarily in vision. [Lecture 1]
Precentral gyrus
Cortical ridge anterior to the central sulcus that contains primary motor cortex. [Lecture 1]
Postcentral gyrus
Cortical ridge posterior to the central sulcus that contains primary somatosensory cortex. [Lecture 1]
Dorsal / superior
Toward the top of the brain. [Lecture 1]
Ventral / inferior
Toward the bottom of the brain. [Lecture 1]
Anterior / rostral
Toward the front of the brain. [Lecture 1]
Posterior / caudal
Toward the back of the brain. [Lecture 1]
Lateral
Toward the outside or away from the brain's midline. [Lecture 1]
Medial
Toward the inside or the brain's midline. [Lecture 1]
Sagittal plane
A vertical plane dividing the brain into left and right portions. [Lecture 1]
Coronal plane
A vertical plane dividing the brain into anterior and posterior portions. [Lecture 1]
Axial plane
A horizontal plane dividing the brain into superior and inferior portions. [Lecture 1]
Limbic system
A group including the hippocampus, hypothalamus, parts of the thalamus, amygdala, and parts of the basal ganglia; involved in emotion, behavior, learning, and memory. [Lecture 1]
Sternberg short-term memory task
Participants decide whether a target item appeared in a memorized set; response time across set sizes tests serial versus parallel memory search. [Lecture 2]
Serial processing
Items are processed one at a time; in Sternberg's task, response time rises linearly as memory-set size increases. [Lecture 2]
Parallel processing
Multiple items are processed simultaneously; predicts a relatively flat response-time function as set size increases. [Lecture 2]
Sternberg task slope
The increase in response time for each additional memory item; the slides illustrate about 50 ms per item as evidence of serial scanning. [Lecture 2]
Self-terminating search
A search that stops as soon as the target is found; target-present trials should vary with target position. [Lecture 2]
Exhaustive search
A search that scans every item before responding, even after the target is found. [Lecture 2]
Sternberg experiment conclusion
Short-term memory items were scanned serially and exhaustively during memory search. [Lecture 2]
Lesion
Any damage to the brain. [Lecture 2]
Necessary brain region
A region is necessary for a function if damage to it impairs that function. [Lecture 2]
Sufficient brain region
A region is sufficient for a function if activating that region alone can produce the function; lesion evidence by itself does not establish sufficiency. [Lecture 2]
Common causes of human brain lesions
Stroke, cerebral hemorrhage, aneurysm, anoxia, head injury, tumors or tumor removal, and neurodegenerative disease such as Alzheimer's. [Lecture 2]
Focal lesion
Damage concentrated in a particular brain area, used to relate that area to impaired and spared cognitive processes. [Lecture 2]
Spatial memory
Memory for where an object was located. [Lecture 2]
Object memory
Memory for what an object was. [Lecture 2]
Dissociation
Evidence that one cognitive function is impaired while another is spared. [Lecture 2]
Single dissociation
Damage to one brain area impairs Function 1 but spares Function 2; may be explained by task difficulty or a general deficit. [Lecture 2]
Double dissociation
Area A damage impairs Function 1 but not Function 2, while Area B damage shows the opposite pattern, supporting independent processes. [Lecture 2]
Cross-over interaction
Opposite impairment patterns across two lesions and two tasks; evidence that process independence is not merely due to uncontrolled task factors. [Lecture 2]
Temporal-versus-parietal memory double dissociation
Temporal-lobe damage selectively disrupts object memory, while parietal-lobe damage selectively disrupts spatial memory. [Lecture 2]
Broca-Wernicke double dissociation
Broca's-area damage selectively impairs language production, while Wernicke's-area damage selectively impairs language comprehension. [Lecture 2]
Two single dissociations without a double dissociation
If Area A damage impairs both functions and Area B damage spares both, the results do not show opposite selective deficits and therefore are not a double dissociation. [Lecture 2]
Limitations of neuropsychology
Damage may be widespread, neural plasticity may mask deficits, and impairment may result from broken connections rather than the damaged area's own function. [Lecture 2]
Neural plasticity
The brain's ability to reorganize or adapt, which can compensate for damage and mask a deficit. [Lecture 2]
Brain stimulation and causal inference
If manipulating a brain region changes a function, researchers can infer a causal relationship between the region and that function. [Lecture 2]
Invasive brain stimulation
Stimulation delivered directly to exposed brain tissue through an open skull. [Lecture 2]
Noninvasive brain stimulation (NIBS)
External methods such as TMS, tDCS, and tACS that alter brain activity without surgery. [Lecture 2]
Transcranial magnetic stimulation (TMS)
A noninvasive, highly localized magnetic pulse that reaches only a few centimeters into the brain. [Lecture 2]
Transcranial direct current stimulation (tDCS)
Noninvasive electrical stimulation that can reach deeper structures but affects many brain areas and is therefore less localized. [Lecture 2]
Transcranial alternating current stimulation (tACS)
Deep, nonlocalized alternating-current stimulation used to manipulate brain oscillations. [Lecture 2]
Temporal resolution
Precision in determining when an event or neural process occurs. [Lecture 2]
Spatial resolution
Precision in determining where an event or neural process occurs. [Lecture 2]
Single-unit recording
An electrode records the activity of an individual neuron under different conditions; provides high spatial and high temporal resolution. [Lecture 2]
Electroencephalography (EEG)
Scalp electrodes measure changes in electrical field potentials produced by neural activity; high temporal but low spatial resolution. [Lecture 2]
Brain oscillation
A rhythmic pattern of neural electrical activity described by its frequency. [Lecture 2]
Delta band
EEG activity around 1-3 Hz, associated in the slides with slow-wave sleep. [Lecture 2]
Theta band
EEG activity around 4-7 Hz, associated in the slides with non-REM sleep. [Lecture 2]
Alpha band
EEG activity around 8-12 Hz, associated with being awake and relaxed or zoning out. [Lecture 2]
Beta band
EEG activity around 12-25 Hz, associated with being mentally active. [Lecture 2]
Gamma band
EEG activity above about 25 Hz, associated in the slides with local synchrony. [Lecture 2]
Event-related potential (ERP)
EEG activity averaged and time-locked in relation to a specific event. [Lecture 2]
ERP component
A positive or negative waveform deflection associated with an underlying cognitive process; names describe polarity and approximate timing. [Lecture 2]
P1 component
A positive ERP wave occurring at approximately 100 ms. [Lecture 2]
N2 component
A negative ERP wave occurring at approximately 200 ms. [Lecture 2]
Feature-based attention ERP finding
Attended colors produce larger P1 and N1 responses than unattended colors, suggesting attention boosts color perception. [Lecture 2]