1/22
Looks like no tags are added yet.
cardiomyopathy
disease of heart muscle which impairs ability of fibers to contract/relax → heart failure
acquired or hereditary!
can be seen in kids and adults
symptoms:
dyspnea
orthopnea
palpitations
peripheral edema/JVD
types:
dilated
hypertrophic
restrictive
dilated cardiomyopathy
enlargement of heart chambers, impaired pumping → decreased SV & CO
Causes:
CAD/MI
ETOH abuse
systemic HTN
DM
viral infections
toxins
pregnancy
drugs
or idiopathic!
Symptoms: fatigue, weakness, chest pain, BP is normal/low
Treatment (depends on cause)
lifestyle mod
meds
surgery
cardiac device implantation
hypertrophic cardiomyopathy
LV hypertrophy & rigid ventricular wall, reduction in chamber size
decreased SV & CO
usually genetic
most common CM
often asymptomatic & undiagnosed!!!
heart failure symptoms (SOB, lower leg edema, fatigue)
can cause sudden death in young athletes!!!
types: obstructive or non-obstructive
Treatment:
meds - cardiac myosin inhibitor
surgical: septal myectomy, septal ablation, implantation of cardiac device, transplant
restrictive cardiomyopathy
marked endocardial scarring (fibrosis) → impairs diastolic filling → decreased CO
risk factors: sarcoidosis, connective tissue disorder, some cancer treatments
Symptoms: heart failure symptoms
Treatment: manage symptoms and treat underlying cause
takotsubo cardiomyopathy
stress cardiomyopathy or broken heart syndrome!!
transient form of CM that causes LV to temporarily weaken/balloon out
cause: elevated catecholamines that are released due to stress
symptoms: SOB, chest pain, syncope
heart failure
chronic progressive condition where heart muscle is unable to pump enough blood to meet body’s needs → decreased CO
symptoms: fatigue, SOB, lower leg edema, weight gain
risk factors/causes:
CAD/MI
HTN
DM/metabolic syndrome
family history
obesity
chronic pulmonary disease
inflammation/infection
some cardiotoxic agents - chemo
BNP > 400
NT-ProBNP > 1800
imaging/tests: chest x-ray, echo, EKG, stress test
Treatment:
diet change/less water intake
ACE inhib/ARBs
diuretics
beta blockers
inotropes
exercise!!! → reduces failty
PT Assessment!!!: 6MWT → <300 m = poor prognosis, MCID = 32 m
HF → low aerobic capacity
right heart failure
venous congestion & systemic symptoms!
congestion of peripheral tissues
liver congestion → impaired liver function
GI congestion → anorexia, GI distress, weight loss
edema & ascites
main causes:
CAD of right ventricle MI
COPD
pulmonary HTN
pulmonary stenosis
PE
tricuspid regurgitation
s3 heart sound!!!
JVD: increased volume in venous system or R side heart failure!
left heart failure
reduced cardiac output & pulmonary symptoms
low CO → activity intolerance & decreased tissue perfusion
pulmonary congestion
impaired gas exchange → cyanosis
pulmonary edema → orthopnea, paroxysmal nocturnal dyspnea, cough with frothy sputum
Common causes:
CAD of left ventricle MI
HTN
valvular heart disease
cardiomyopathies
myocarditis
systolic heart failure
HFrEF (reduced ejection fraction)
impaired contraction/pumping
enlarged dilated heart within myocardium
EF <40%
diastolic heart failure
HFpEF (preserved ejection fraction)
impaired filling/relaxation
ventricular wall hypertrophy
often have EF >50%
acute decompensated heart failure
sudden or gradual onset of signs/sympts of HF that requires unplanned medical visit
hospitalizations are due to:
noncomplicance to meds
dietary restrictions
uncontrolled HTN
ischemia
arrhythmias
COPD
treatment: diuretics, vasodilators, inotropic meds
NYHA stages class I
no symptoms
can perform ordinary activities without any limitations
NYHA stages class II
mild symptoms
occasional swelling
somewhat limited ability to exercise
no symptoms at rest
NYHA stages class III
noticeable limitations in ability to exercise
comfortable only at rest
NYHA stages class IV
unable to do any physical activity without discomfort
symptoms at rest
cardiac resynchronization therapy
special type of pacemaker that treats HF
paces both ventricles & R atrium → coordinate myocardial contractions → stronger contraction = increased CO
can improve symptoms and decrease mortality
NOT all patients achieve expected results
intra aortic balloon pump
used for patients with cardiogenic shock!
catheter is inserted into large artery (femoral or axillary)
inflates during diastole
deflates during systole
decreases afterload!
increases coronary blood flow!
impella device
mini ventricular assist device that provides short term support of left ventricle; continuously draws blood from LV then expels it to ascending aorta
decreases LV work load & myocardial demand
increases MAP, CO, systemic perfusion, and coronary flow
epicardial pacemaker
temporary PM
placed after CT surgery
sewn onto outside of heart and wires exit the skin
able to be mobilized!!!
transvenous pacemaker
temporary PM
placed in emergent situations
pacing wire inserted into large vein → RV
usually on BED REST!
permanent pacemaker
implantable pulse generator and lead wires that connect to the myocardium
endocardial leads attached to RA/ventricles with generator placed below clavical
precautions:
NO shoulder flex/abd >90
no extension
no lifting/pushing/pulling for 4-6 wks
groin precautions → 1 week if used for placing PM
leadless pacemaker
all components of PM are inside one device, placed in RV, doesn’t need leads or generator
can only pace one ventricle
cannot defibrillate
NO post op precautions!!
AICD
detects and corrects life threatening arrhythmias by delivering electrical shock
vtach or vfib!!
same post op precautions as PM
KNOW what rate the device fires at & keep HR 10-15 beats below the rate !!!!
PT considerations in acute care setting:
may have blunted or delayed HR response to activity
observe EKG & ensure PM is working
know firing rate
monitor for signs of exercise intolerance
teach modifications for ADLs
ROM/lifting precautions