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26 Terms
1
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what is short bowel syndrome?
occurs when bowel is surgically shortened to extent that the remaining bowel is unable to absorb levels of nutrients to meet individuals needs
most common reasons of resection: Crohn’s traumatic abdominal injuries, malignant tumors, mesenteric infarction
2
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what affects nutrition of people with short bowel syndrome?
the amount and location of resected remaining bowel
pts who have 150cm or more w/o colon or 60-90 cm of small bowel with colon may need PN and progress to oral diet over 1-2 year period
factors influencing adaptation: length present, pt’s age, whether ileocecal valve remains, health of remaining bowel, health of stomach, liver, and pancreas
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describe nutrition therapy for short bowel syndrome
in early months, PN is major source of nutrition and hydration
consuming intact nutrients promotes bowel adaptation b/c they stem bowel to flow to intestine and secretion of pancreatic enzymes and bile acids
six to eight small meals per day
if pt’s colon intact, fat intake is restricted to avoid steatorrhea and increased fluid loss
4
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what is IBS?
most frequently diagnosed digestive disorder in US
many factors in etiology
symptoms that can not be explained: lower abd pain, constipation, diarrhea, alternating periods of constipation and diarrhea, bloating, mucus in stools
can significantly impair QOL
rule out other diagnosis
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what is nutrition therapy for IBS?
inconclusive evidence of current treatment used
pharm treatment options
complementary therapies
elimination diet
probiotics
grade a level evidence for 5g of guar gum daily (soluble, nongelling fiber)
low FODMAP diet: restriction of fermentable oligosaccharides, disaccharides, monosaccharides, and pyols
avoid large meals, reduce lactose, reduce fat, reduce sugar alcohols, reduce fructose, reduce gas producing foods
6
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describe the low roadmap diet
total elimination of high roadmap for 2-6 weeks
6 week re-introduction phase: each week introduce one of the types of sugars back into diet and see how tolerated
7
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what is diverticular disease?
caused by increase pressure within the intestinal lumen
diverticulosis is usually asymptomatic
diverticulitis occurs when diverticula become inflamed
symptoms of diverticulitis: cramping, alternating diarrhea and constipation, flatus, abd distention, low grad fever
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what are complications of diverticular disease?
occult blood loss and acute rectal bleeding leading to iron deficiency anemia
accesses and bowel perforation lead to peritonitis
fistula formation causing bowel obstruction
bacterial overgrowth that leads to malabsorption of fat and vitamin B12
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what is nutrition therapy like for diverticular disease?
high-fiber intake may prevent and improve symptoms and prevent diverticulosis
avoid nuts, seeds, and popcorn
during acute phase of diverticulitis: NPO until bleeding and diarrhea subside, oral intake resumes w/ clear liquids and progresses to low-fiber diet until inflammation and bleeding no longer risk
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what are ileostomies/colostomies?
performed after part or all of colon, anus, and rectum are removed
potential nutritional problems, smaller length of colon = greater potential for complications
ileostomies cause a decrease in fat, bile acids dn vitamin B12 absorption
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describe nutrition therapy for ostomies
goal: minimize symptoms and replenish losses
initially clear liquids low in simple sugars, advance slowly based on tolerance, low residue 6-8 week post op
avoid swallowing air and gas formation
take small bites and chew throroughly
add foods that may decrease odor: buttermilk, parsley, yogurt, cranberry juice
add foods that may thicken stool: pectin, past, potatoes, cheese, banana, applesauce
regular diet resumes abt 6-8 weeks after surgery
obtains adequate fluids and lytes is main concern
12
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define liver disease
after absorption, almost all nutrients are transported to liver
vital for detoxifying drugs, alcohol, ammonias and other poisonous substances
liver damage can have profound and devastating effects on metabolism
failure can occur from chronic liver disease or secondary to critical illnesses
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what is fatty liver disease?
abnormal fat deposition in liver, occurs in clients w/ alcoholic liver disease
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what is non alcoholic fatty liver disease (NAFLD)?
simple hepatic steatosis, often aysmptomatic
benign to nonalcoholic steatohepatitis characterized by inflammation and liver cell damage
complications such as advanced fibrosis, cirrhosis, and heptocellular carcinoma may result
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what is cirrhosis?
irreversible liver disease that occurs when damage liver cells are replace by functionless scar tissue, impairs function and disrupt circulation
strongly associated with obesity and metabolic syndrome
twice as likely to die of cardiovascular disease than liver disease
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describe nutrition therapy for NAFLD
reduce calories to promote gradual weight loss: target is 7-10%
mediterranean diet is most recommended
avoid fructose, limit alcohol
coffee actually has protective effect, so no liver related restrictions
aerobic and resistance training reduce liver fat
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describe hepatitis
inflammation of liver caused by virus, bacteria, toxins, obstruction, parasites, or chemicals
early symptoms: anorexia, nausea, vomiting, fever, fatigue, weight loss, headache
later: dark colored urine, jaundice, liver tenderness, and possible liver enlargement
cell damage is reversible
nutrition therapy: adequate energy, protein, and micronutrients
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what is alcoholic hepatitis?
toxic liver injury associated with chronic ethanol consumption
presents with fever, jaundiced and hepatomegaly
advanced signs of ascites, portal hypertensive bleeding, and hepatic encephalopathy
increase susceptibility to infections
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what are the goals of nutrition therapy for liver disease?
up to 1.6 g/kg/day protein depending on degree of malnutrition and other complications
ascites: 2000 mg Na and fluid restriction
20
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describe nutrition therapy for liver transplant
moderate to severe malnutrition increase risk of complications and death after transplant
small, frequent meals and commercial supplements help maximize intake
recommendations based on nutritional status, weight, tolerance, and lab values
food safety due to immunosuppressants
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what is pancreatitis?
inflammation of pancreas
may develop hyperglycemia related to insufficient insulin secretion
alcohol abuse and gallstones account for more than 70% of cases of acute pancreatitis
characterized by intermittent pain made worse by eating
malabsorption does not occur until enzyme secretion is less than 10% of normal
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describe acute pancreatitis
symptoms: upper abd pain radiating to back, worsening w/ food ingestion, n/v, abd distentions, steatorrhea
diagnosis based on symptoms and abnormal serum amylase and/or lipase
mild: IV hydration and pain meds
severe: many complications may develop within hours or days such as shock, pulmonary failure, renal failure, GIB, MSOF. treat with fluid and electrolytes, antibiotics, neutron support, and possible surgery
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what is chronic pancreatitis?
chronic irreversible inflammation that leads to fibrosis w/ tissue calcification
manifestations: chronic abd pain, normal or mildly elevated pancreatic enzymes
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what is nutrition therapy for acute pancreatitis?
clear liquids and advance as tolerated.
if severe then j tube feedings of hydrolyzed/elemental formula, small peptide formulas w/ 70% fat, TPN if unable to tolerate j tube
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describe nutrition therapy for chronic pancreatitis