Anatomy Exam Two: Chapter 18

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27 Terms

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<p>What are the <strong>4 heart chambers</strong> and their main <strong>structural differences</strong>?</p>

What are the 4 heart chambers and their main structural differences?

Atria receive blood; ventricles pump it. The left ventricle is thicker to pump at higher pressure to the body.

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<p>What is the pathway of the <strong>pulmonary circuit</strong>?</p>

What is the pathway of the pulmonary circuit?

right ventricle > pulmonary truck/arteries > lungs > pulmonary veins > left atrium

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<p>What is the pathway of the <strong>systemic circuit</strong>?</p>

What is the pathway of the systemic circuit?

left ventricle > aorta > body tissues > vena cava > right atrium

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<p>What is the role of <strong>intercalated discs</strong>?</p>

What is the role of intercalated discs?

Gap junctions allow rapid electrical spread, making the heart contract as a single unit.

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<p>What is the importance of <strong>Calcium Ca+ </strong>in contraction?</p>

What is the importance of Calcium Ca+ in contraction?

Extracellular entry triggers internal calcium release, unlocking muscle fiber sliding.

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<p>What is the metabolic <strong>energy requirement </strong>of cardiac muscle?</p>

What is the metabolic energy requirement of cardiac muscle?

strictly aerobic respiration (requires constant oxygen) and packed with mitochondria

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<p>What are the <strong>Atrioventricular valves (AV) </strong> names and functions?</p>

What are the Atrioventricular valves (AV) names and functions?

tricuspid valve (right) and bicuspid/mitral (left) prevent back-flow into atria during systole (contraction)

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<p>What are the <strong>Semilunar Valve (SL)</strong> names and functions?</p>

What are the Semilunar Valve (SL) names and functions?

pulmonary and aortic valves prevent back-flow into the ventricles during diastole (relaxation)

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<p>What <strong>anchors</strong> the AV valves?</p>

What anchors the AV valves?

chordae tendineae (heart strings) attached to papillary muscle

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<p>What happens during <strong>ventricle filling</strong>?</p>

What happens during ventricle filling?

heart relaxes (diastole), AV valves open and blood fills ventricles passively

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<p>What happens during <strong>ventricular systole</strong>?</p>

What happens during ventricular systole?

ventricles contract, AV valves slam shut (1st heart sound) and SL valves pop open and blood ejects

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<p>What happens during <strong>early diastole</strong>?</p>

What happens during early diastole?

ventricles relax and blood flows backwards and snaps SL valves shut (2nd heart sound)

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<p>Complete the <strong>sequence of excitation</strong>?</p>

Complete the sequence of excitation?

SA node > AV node > AV bundle > bundle branches > purkinji fibers

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<p>Why is the impulse delayed at the AV node? </p>

Why is the impulse delayed at the AV node?

allows atria to finish contracting and completing fill ventricles before they pump

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<p>What does the <strong>P wave</strong> represent? </p>

What does the P wave represent?

atrial depolarization ( triggers atrial contraction)

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<p>What does the <strong>QRS complex</strong> represent? </p>

What does the QRS complex represent?

ventricular depolarization (triggers ventricular contraction)

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<p>What does the <strong>T wave</strong> represent? </p>

What does the T wave represent?

ventricular repolarization (triggers ventricular relaxation)

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<p>List the formula for <strong>Cardiac output (CO)</strong>.</p>

List the formula for Cardiac output (CO).

Heart Rate X Stroke Volume

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<p>What is the <strong>preload</strong> and the <strong>Frank-Starling Law</strong>?</p>

What is the preload and the Frank-Starling Law?

degree of muscle stretch before contraction. More stretch (venous return) = stronger contraction = higher stroke volume

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<p>What is <strong>contractility</strong> and what increases it?</p>

What is contractility and what increases it?

muscle pumping strength independent of stretch and increased by sympathetic nerves, epinephrine and calcium.

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<p>What is <strong>afterload </strong>and how does it affect stroke volume?</p>

What is afterload and how does it affect stroke volume?

back pressure from atrial blood ventricles must overcome, a higher afterload (high blood pressure) = lower stroke volume

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<p>How does the autoimmune nervous system modify <strong>heart rate</strong>?</p>

How does the autoimmune nervous system modify heart rate?

sympathetic increases rate (norepinephrine), parasympathetic decreases rate (acetylchloline/vagus nerve).

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<p>Tachycardia vs Bradycardia</p>

Tachycardia vs Bradycardia

fast resting heart rate (over 100) vs slow resting heart rate (under 60).

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<p>What are <strong>atrial fibrillation</strong> EKG <strong>features</strong> and <strong>risks</strong>?</p>

What are atrial fibrillation EKG features and risks?

no p waves and irregular baseline, causes poor atrial pumping, risk of blood clots and stroke

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<p>What is <strong>ventricular fibrillation</strong>?</p>

What is ventricular fibrillation?

chaotic, electrical twitching, zero blood flow to the body, fatal

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<p>What is <strong>second-degree heart block</strong>?</p>

What is second-degree heart block?

impaired conduction where some SA node impulses fail to reach ventricles (dropped QRS complexes).

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<p>What are the common causes of <strong> congestive heart failure</strong> (CHF)?</p>

What are the common causes of congestive heart failure (CHF)?

weakened myocardium due to chronic high blood pressure, past heart attacks, coronary blockages or bad valves