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Benefits of randomization
ensuring that all else is equal between treatment and control except treatment itself
How can we check our randomization?
1) checking observable characteristics of groups
2) checking whether treatment effects are similar with and without controls
Heteroskedastic errors
standard errors whose variance is correlated with the regressors (no constant conditional variance across treatment)
Homoskedastic errors
constant conditional variance across the independent variable
When is an estimate also interpreted as elasticity
when its a log-log regression
Interpretation of an elasticity
the percentage change in Y when X changes by one percent
T-value calculation
treatment estimate - (our Null hypothesis = usually 0 because we assume no effect) divided by standard error
Two elements of omitted variable bias
1) relationship between treatment and omitted variable
2) effect of omitted variable on the outcome
Calculation of OVB if you have long and short coefficient
OVB = long minus short
Interpretation of OLS regression results (if not fully randomization is ensured)
never a causal effect always just an association
= rejecting the hypothesis that there is no effect (but not saying there is one)
Two major concerns of field experiments
1) treatment dilution
2) imperfect random assignment
Treatment dilution
individuals assigned to treatment group are not treated
ITT calculation (words)
Estimate of treatment minus estimate of control
True effect for IV (word)
LATE
true effect for IV (calculation)
Intent-to-treat divided by compliance rate
Compliance rate (phi in IV) formula
Percentage of people who were assigned treatment and did treatment MINUS percentage of people who were not assigned treatment and did treatment
Ihree IV assumptions
1) Independence
2) Relevance
3) Exclusion Restriction
How to estimate a bias on the true causal effect (when we have imperfect compliance)
without treatment, how would effect look like for each group
= e.g. those who do not take the treatment might not need/want it because XY
general characteristic influences the direction (higher costs + opting in = overestimating the effect)
simple direction of bias rule (when imperfect compliance)
Baseline Trait of noncompliers and how trait affects outcome (without treatment)
Thumb rule for over/underestimating effect
Does the treatment group consist of people who are naturally better or naturally worse at the final outcome?
= tendencies of overestimating/upwards bias vs underestimating/downwards bias effect
Standard deviation
measures the variability in data
used to make differences comparable between different studies
one standard deviation difference = big jump
Mathematical expectation of a variable
the population average of this variable
statistical significance
it is highly unlikely but not impossible that difference arose purely by chance
Dealing with heteroskedasticity
using robust standard errors
Central Limit theorem
with enough data, the distribution of the t-statistic is smooth and bell-shaped (also with dummy variable)
OLS estimator (formula words)
causal + selection
beta HAT OLS estimate
beta + Cov(e,D) / Var(D)
Selection bias in OLS estimator (formula)
COV(e,D) / Var (D)
OLS estimator if we have imperfect randomization
does not reflect the causal effect of taking the treatment but is biased by a selection term
Endogeneity (words)
the covariance of the treatment and the error term is not zero = OLS problematic
Exogenous (condition + OLS)
Covariance between the treatment and the error term is 0 = OLS no problem
IV method
uses three assumptions to characterise a chain reaction leading from the instrument to outcome
Three IV assumptions
1) Relevance of the instrument
2) independence assumption
3) exclusion restriction
IV Assumption 1 - Relevance
the instrument is correlated with the treatment
COV(Z, D) is unequal 0
IV assumption 2 - Independence
The instrument is not correlated with errors
IV assumption 3 - Exclusion restriction
The instrument only affects the outcome Y through the treatment D
Testing IV assumption
1) can be directly tested via regression from data
2) & 3) cannot be directly tested, the researcher needs to argue why this holds
Instrument is strong enough when
f-statistic of first regression > 10
OLS estimator (formula)
COV(D,Y) / Var(D)
IV estimator OLS (formula)
COV (Z,Y) /COV (Z,D)
IV estimator (words)
ratio of two OLS estimators (rho and phi)
IV - Phi (formula) as OLS estimator
COV(Z,D) /Var (Z)
IV - Phi (words) & OLS estimator
Covariance of Instrument and Treatment divided by variance of Instrument
Rho (formula) - (co)variances
COV (Z,Y) / Var (Z)
IV -Rho (words)
Covariance of the Instrument and Outcome divide by Variance of Instrument
Rho also called
reduced form
intent to treat
IV - Phi also called
first stage
to get causal effect via manual IV
estimate both regressions (reduced and first stage) and divide the reduced by the first one
RHO written as Expectations (formula)
E[Y | Z = 1] - E[Y | Z = 0]
Phi written as expectations (formula)
E [ D | Z = 1] - E[ D| Z = 0]
RHO in terms of expectations (words)
The expected outcome of those who have the instrument MINUS the expected outcome of those without the instrument
PHI in terms of expectations (words)
The likelihood of getting the treatment when having the instrument MINUS the likelihood of getting the treatment when NOT having the instrument
Why do we need a strong first stage?
so we do not divide by zero
How does IV get rid of the selection bias?
BECAUSE of the assumption that the instrument is not correlated with the error (the thing causing selection bias)
How does the 2SLS get rid of selection bias?
using the fitted values for the treatment gets rid of the error (because fitted values are predicted values, not real ones = no error)
PHI when we have perfect compliance
is 1
IV - When Phi < 1
incomplete compliance
= the Intent to Treat is bigger than the local average treatment effect
LATE formula
gamma = rho / phi
Why are we estimating only the effect of compliers with IV?
because we only have variation in the treatment (e.g. we only observe treatment on those who otherwise would not get it)
never takers
those who would never take the treatment
always takers
those who would always take the treatment
Assumption needed to interpret IV beta as LATE
Monotonicity assumption
Monotonicity assumption
there are no defiers
Defiers
those who would only take treatment if they do not get the instrument (randomization)
Two-stage least squares
alternative way of computing ratio of rho and phi by means of a sequence of two regressions
Regression in the two-stage least squares
1) Regress the treatment on the instrument variable
2) save the predicted values from Regression 1 and regress the outcome variable on these predicted values
Advantages of 2SLS
1) allows as many control variables as you’d like
2) allows to efficiently use more than 1 instrument per exogenous covariate
Steps to 2SLS
1) test instrument
2) estimate the impact of instrument on treatment
3) Use fitted values of treatment for second-stage equation
2SLS - Regression 0
Yi = a0 + rho * Zi + y0 Ai + e0i
2SLS - Regression 1 (formula)
Di = a1 + Phi * Zi + y1Ai + e1i
2SLS - Regression 2 (formula)
Y_i = a_2 + beta_2SLS* D^HAT + y_2*Ai + e_2i
Parameters in 2SLS
rho = overall effect of instrument on outcome
phi = effect of instrument on treatment
beta = true causal effect of treatment on outcome
ei = confounder that affects relationship of treatment and outcome

Def. Sharp RDD (short)
one side is treated, the other is not
Def. Fuzzy RD (short)
difference in treatment intensity to left and the right side (can use IV)
Difference- in- Difference Designs
allow you to estimate causal effects using before and after comparison in time
Causal intuition of Diff In Diff
compare the outcomes of the treatment group before and after the start of the treatment to the outcomes of the control group before and after
Basic steps of Diff in Diff (very simple)
1) Take difference treatment and control after
2) subtract difference treatment and control before
3) Hopefully this makes ceteris paribus
Basic DD regression equation
Y_dt = a + b+Tread_d + gamma Post_t + delta(Treat_d * Post_t) + e_dt
Subscript d
indicates the group (needed for Y, Treatment and error)
Subscript t
indicates the time period (needed for Y, Post and error)
Advantage of Diff in Diff
comparing changes instead of levels = eliminate fixed differences between groups that might otherwise generate omitted variable bias
Manual calculation of DD estimate (formula)
(Y_1_t - Y_0_t) - (Y_1_t-1 - Y_0_t-1)
Manual calculation of DD estimate (words)
subtracting pre-treatment differences between two units from the post treatment differences
DD counterfactual
from strong assumption of common trends
Time effects
capture temporal changes in the outcome variable that are common to all units of observation
Group effects
difference between treatment and control group before the treatment
Interpretation alpha in Diff-in-Diff
constant
outcome for the control group in the pre-reform period
Interpretation beta (Group dummy)
difference between treatment and control before treatment
Interpretation gamma (Time dummy)
common difference in outcome for treatment and control group in post-reform period (seen in control group slope)
Interpretation delta ( Treatment effect/Interaction term dummy)
difference in outcome for treatment and control group in post-reform period MINUS the difference in outcome of treatment and control group in the pre-reform period
Extended DD Regression equation (only more time dummies)
Y_dt = alpha + beta Tread_d + SUM_s(gamma_s POST_s) + delta(Treat_d + Post_t) + e_dt
Extended DD Regression (year and treatment dummies)
Y_dt = alpha +beta Treat_d + SUM_s(gamma_s Post_s) + SUM_u(delta_u (Treat_d * Post_u) + e_dt
Why include multiple dummies in Diff-in-Diff
to capture individual year effects
and to capture build up of treatment
How to account for different trends of treatment and control group in Diff in Diff
adding differential trends (e.g. demographic characteristics that change over time)
Notation differential trends
n_treat( Treat_d * t)
n_control(Control_d * t)
Adding control variable notation
pi * Xi (vector of control variables)
Key assumptions of DD
1) common time effects
2) exogeneity of the reform
3) No anticipation of the reform
Common time effects assumption
In absence of the reform both groups would have evolved parallel
Exogeneity of the reform
reform not related to different development of treatment and control group
No anticipation of the reform
Treatment group did not respond before the reform