Preschoolers: Dyspraxia and Enuresis

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Last updated 8:59 PM on 8/28/26
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23 Terms

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Dyspraxia

Impairment in the development of fine or/ gross motor coordination, oculomotor function, memory, and speech.

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may exist in isolation OR concurrent with_____

learning disabilities, autism, or attention deficit disorders

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What are the risk factors for dyspraxia

Preterm and SGA are at the highest risk

Effects boys more than girls

Strong familial tendency

Overall, affects 5-10% of children

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Signs of dyspraxia

- Delay in acquired early motor skills such as sitting, crawling and walking

- Difficulty getting dressed and using cutlery

- Poor understanding of spatial concepts, such as on/under/over/in front of

- Anxiety and low self-esteem

- Difficulty paying attention, reacts to all stimuli without discrimination

- Works better in small groups or 1:1

- Difficulty managing time

- Often loses things

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Is dyspraxia a cognitive impairment

No, as it is caused by reduced cortical thickness and grey matter volume in the cerebellum, amongst sensorimotor and planning locations

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Does dyspraxia affect academic achievement and/or activities of daily living

Yes, absolutely

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Enuresis

Repeated involuntary voiding by a child who has reached an age at which bladder control is expected.

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Primary enuresis

The child never achieved consistent nighttime dryness. Caused by

1) Delayed bladder maturation, or ADH maturation, Deep sleep patterns

2) Genetic factors (familial)

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Secondary enuresis

The child was previously dry for 6 months. Can be caused by:

1) Stressors

2) Medical conditions or illness, i.e., UTI, constipation, obstructive sleep apnea

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Is enuresis a disease

NO, enuresis is a sign typically of an underlying issue

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At what age do children develop the sensation of urinary fullness

years 2-4 years, this is when the muscular development occurs, as well as control contractions of detrusor and relaxation in bladder emptying

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When are the kidneys fully developed

By 2 years of age

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What is the enuresis criteria

Repeated (>2times/ week) involuntary voiding by a child who has reached the age of 5 years old, at which bladder control is expected.

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What is the average age to be toilet-trained

27 months

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Monosymptomatic enuresis (MSE)

Nighttime bedwetting without daytime urinary symptoms

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Nonmonosymptomatic enuresis (NMSE)

Day & night symptoms

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With nonmonosymptomatic enuresis, what questions do you want to ask

- When are they voiding, are they having frequent voids (>8 times per day) or (

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What are some symptoms of Primary monosymptomatic enuresis

- Difficulty waking to stimuli

- Familial, underdeveloped bladder

- Overactive detruser muscle

- Nocturnal polyuria, as kidneys do not concentrate urine appropriately, or there is a decrease in vasopressor release from the pituitary

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Symptoms of secondary monosymptomatic enuresis

Nocturnal enuresis, which can be supported with management of constipation, UTI, stressors, OSA and diabetic mellitus onset

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Causes of primary nonmonosymptomatic enuresis

1) constipation, diabetes mellitus, overactive bladder

2) Underlying neurological disorder

3) Urinary tract obstruction

4) Urinary tract malformation

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What assessment data is used for enuresis in regard to a bladder journal

1) Urinary frequency

2) Urinary volume

3) Dysfunction: interrupted, incomplete sensation, urgency, dysuria

4) Time of day

5) Intake: 1-3 hours prior to bedtime; caffeine

6) Abdominal assessment (constipation)

7) UTI: Fever, dysuria, urgency, irritability, FLACC

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How can you support children with primary monosymptomatic enuresis

1) Tell parents they will grow out of it

2) Give education, reassurance, and positive reinforcement

3) Void in AM & before bed. AND frequent voiding schedule (q2 to 3hr)

4) Take the child out of the diaper

5) Wear a vibrating watch

6) Optimal posture: sit upright, middle of the toilet, feet on a footstool

7) 80% of fluid intake before 4: 00 pm; no caffeine

8) High fibre diet (constipation)

9) Enuresis alarm

10) Desmopressin (special occasions, sleepover)

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Desmopressin

It is a medication that reduces urine production by acting like antidiuretic hormone (ADH), helping the kidneys reabsorb more water. Support normal circadian variation of plasma vasopressin