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(12) Major Symptoms of Acute Stress Disorder
Earlier onset, within 1 month of trauma
Involuntary, distressing, persistent memories of the event
Nightmares
Flashbacks
Intense distress
Negative mood
Dissociation (e.g., depersonalization)
Avoidance (e.g., avoiding external cues)
Arousal (e.g., sleep trouble, irritability, etc.)
(12) Major Symptoms of Adjustment Disorder
Psychological response to a common stressor
Can be in response to single/multiple events
Occurs within 3 months of stressor
Results in clinically significant behavioral/emotional symptoms
Symptoms disappear when stressor ends/person adapts
(12) How does the HPA axis become altered in response to trauma?
Elevated levels of stress hormones, cortisol levels decrease from chronic stress
(12) With the HPA axis altered, how does that affect the developing brain?
Lower cortisol levels can cause the stress response system to behave unpredictably, and impact structure and function; may account for later psychiatric problems
(12) What does the P stand for in PRACTICE?
Psychoeducation/parenting strategies
(12) What does the R stand for in PRACTICE?
Relaxation skills
(12) What does the A stand for in PRACTICE?
Affective expression/regulation
(12) What does the C stand for in PRACTICE?
Cognitive coping / Conjoint parent-child sessions
(12) What does the T stand for in PRACTICE?
Trauma narrative/processing
(12) What does the I stand for in PRACTICE?
In vivo exposure
(12) What does the E stand for in PRACTICE?
Enhancing personal safety and future growth
(12) What is the goal of the Trauma Narrative/Processing step?
To help the patient gradually approach, organize, and reframe memories so they lose the emotional intensity, and be integrated into the person’s life.
(5) Defining characteristics of Neurodevelopmental disorders
Early onset and persistent course
Lasting alterations in neural development
Accompanied by subtle delays in language, motor, and social development
Frequently co-occur
(5) Evolutionary degeneracy theory
Intellectual and social problems of children with intellectual disability were viewed as a regression to an “earlier period in human evolution”
Throwback to the “Mongolian race”
(5) The Eugenics Scare
“The science which deals with all influences that improve the inborn qualities of a race”
Individuals with intellectual disabilities were threats to society (“morons” or “moral imbeciles”)
(5) How were ID’s diagnosed past vs. present?
Mainly based on low IQ, but now adaptive functioning is more prominent
(5) Diagnostic criteria of intellectual disabilities
Deficits in intellectual functions, such as reasoning, problem-solving, planning, abstract thinking, judgment, academic learning, and learning from experience
Deficits in adaptive functioning
Results in failure to meet developmental/sociocultural standards for independence and social responsibility
Limits functioning in things like communication, social participation, and independent living
Onset of intellectual and adaptive deficits during the developmental period
(5) Major characteristics of Mild Severity
About 85% of people with ID
Not identified until early elementary years
Overrepresentation of minority group members
Develop social/communication skills
Live successfully in community as adults with appropriate support
(5) Major characteristics of Moderate Severity
About 10% of people with ID
Identified during preschool years
Applies to many people with down syndrome
Benefit from vocational training
Can perform supervised unskilled/semi-skilled work in adulthood
(5) Major characteristics of High Severity
About 3-4% of people with ID
Often associated with organic causes
Identified at a young age
Delays in developmental milestones/physical features
May have mobility or other health problems
Typically need assistance throughout life
Live in group homes/with family
(5) Major characteristics of Profound Severity
About 1-2%
Identified in infancy due to delays in development/biological anomalies
Learn only rudimentary communication skills
Require training for eating, grooming, toileting, and dressing
Require lifelong assistance and care
(5) Which cases do biological factors account for?
Severe and profound, but not mild
(5) Risk factor Biomedical - Prenatal
Chromosomal disorders
Single-gene disorders
Syndromes
Metabolic disorders
Maternal illness
Paternal age
(5) Risk factor Biomedical - Perinatal
Prematurity
Birth injury
Neonatal disorders
(5) Risk factor Biomedical - Postnatal
Traumatic brain injury
Malnutrition
Seizure disorders
Degenerative disorders
(5) Risk factor Social - Prenatal
Poverty
Maternal malnutrition
Domestic violence
Lack of access to prenatal care
(5) Risk factor Social - Perinatal
Lack of access to prenatal care
(5) Risk factor Social - Postnatal
Impaired child-caregiver interaction
Lack of adequate stimulation
Family poverty
Chronic illness in the family
Institutionalization
(5) Risk factor Behavioral - Prenatal
Parental drug use
Parental alcohol use
Parental smoking
Parental immaturity
(5) Risk factor Behavioral - Perinatal
Parental rejection of caretaking
Parental abandonment of child
(5) Risk factor Behavioral - Postnatal
Child abuse/neglect
Domestic violence
Inadequate safety measures
Social deprivation
Difficult child behaviors
(5) Risk factor Educational - Prenatal
Parental cognitive disability without supports
Lack of preparation for parenthood
(5) Risk factor Educational - Perinatal
Lack of medical referral for intervention services at discharge
(5) Risk factor Educational - Postnatal
Impaired parenting
Delayed diagnosis
Inadequate early intervention services
Inadequate special education services
Inadequate family support
(5) Factors that affect overall adjustment of a child with ID
Inheritance, Environment, Neurobiological, Social, Psychological
(6) Major symptoms of Autism Spectrum Disorder
Impairments in social interaction
Impairments in communication
Restricted, repetitive, and stereotypical patterns of behavior/interests/activities
(6) Factors that contribute to the spectrum nature of autism
May differ in level of intellectual ability
Vary in severity of language problems
Behavior changes with age
(6) Lack of Joint attention
Difficulty to coordinate visual focus on the same object, event, or activity with another person for a social purpose
(6) Atypical processing of faces
Differences in how the brain perceives, recognizes, and extracts social/emotional information from faces
(6) Lack of Protodeclarative and Expressive Gestures
Significant disruptive in early nonverbal communication and join attention; serve as foundational blocks for spoken language, social bonding, and cognitive growth
(6) Preservative speech
The repetition of words, phrases, sounds, or specific tone of conversation
(6) Impairments in Pragmatics
Difficulties using verbal and non-verbal communication appropriately in social contexts
(6) Repetitive sensory and Motor behaviors
Repeated movements, sounds, or physical actions; “stimming”
(6) Insistence on Sameness
Strong preference for routine, a deep dislike of unexpected change, and a need for predictability
(6) Intellectual Strengths and Deficits
About 70% have co-occuring intellectual impairment
Low verbal scores and high nonverbal scores
About 25% have splinter skills or islets of ability
5% display isolated and remarkable talents
(6) Theory of Mind deficit in ASD
Difficulty understanding or being aware of others’ and their own mental states
(6) Youth processing information
Tend to process information in bits and pieces rather than look at the big picture
(6) Reliable detection age and recommended screening age
12-18 months detected; 18-24 months screening
(6) ASD heritability
Highly heritable
(6) Cause of ASD
Biologically based, neurodevelopment disorder related to problems in early development, genetic influences, and brain abnormalities
(6) Siblings of Individuals with ASD
15-20% have broader phenotype
(6) Key features of Childhood-Onset Schizophrenia
Delusions
Hallucinations
Thought disturbances
Disorganized
Catatonic behavior
Inappropriate affect
Significant impairment in functioning
(6) Difference between positive and negative symptoms of COS
Positive: not normally present
Negative: normally present
(6) Positive symptoms of COS
Delusions
Hallucinations
(6) Negative symptoms of COS
Slowed thinking
Speech
Movement
Emotional apathy
Lack of drive
(6) Hallucinations vs. Delusions
Sensory perception vs. fixed, false belief
(6) COS prevalence & history
Extremely rare in children under 12; if present, children have history of behavioral, social, and psychiatric disturbances
(6) COS Biological model
Strong genetic contribution and disturbance in the development of the brain
(8) Core Characteristics of ADHD
Inattention
Hyperactivity-impulsivity
Highly correlated
Predict different behavior/cognitive impairments
(8) Facet of attention that is most affected
Sustained attention
(8) DSM-5 Criteria for ADHD
Inattention
Difficulty sustaining attention
Easily sidetracked
Avoids/dislikes tasks that require sustained mental effort
Often forgetful in daily activities
Fidgety
Often cannot remained seated
Runs/climbs in inappropriate situations
“Driven by a motor”
Talks excessively
Difficulty waiting their turn
Interrupts others
Predominantly Inattentive Type (ADHD-PI)
Inattentive, drowsy, daydreamy, spacey, in a fog, easily confused
Specific learning disorders, process information slowly, trouble remembering things, low academic achievement
Anxious, apprehensive, socially withdrawn, mood disorders
Predominantly Hyperactive-Impulsive Type (ADHD-HI)
Primarily symptoms of hyperactivity-impulsivity
Fidgety, cannot remained seated, running, “on the go”, talking excessively, difficulty waiting, interrupting
Includes preschoolers, limited validity for older children
Rarest of the 3 subtypes
Combined type (ADHD-C)
Children who have symptoms of both inattention and hyperactivity-impulsivity
Most often referred for treatment
(8) Children with ADHD Intelligence
Normal intelligence, but impaired academic functioning; often diagnosed with learning disabilities
(8) Family problems
Negativity
Child noncompliance
Excessive parental control
Sibling conflict
Maternal depression
Paternal anti-social behavior
Marital conflict
(8) Peer problems
Often bothersome, stubborn, socially awkward, socially insensitive
Often disliked and rejected by peers
Few friends
Unable to apply social understanding in social situations
Positive friendships can buffer negative outcomes
(8) Accompanying Psychological Disorders
Oppositional Defiant Disorder
Conduct disorder
Anxiety disorders (25%)
Mood disorders
(8) Why is ADHD more commonly diagnosed in boys?
ADHD in girls often unrecognized/unreported, DSM criteria may be more appropriate to boys
(8) Unique experience for ADHD in Girls
Have severe problems in adolescence
Peer rejection, conduct problems, deficits in academics, attentional skills, language abilities
More likely to develop eating disorder symptoms
(8) ADHD in Infancy
May be present at birth but no reliable/valid methods to identify
(8) ADHD in Preschool
Hyperactive-impulsivity symptoms more visible and significant ages 3-4
Symptoms that last at least 1 year are likely to have difficulties later in life
(8) ADHD in Elementary
Evident when child starts school
Oppositional defiant behaviors may increase/develop
Ages 8-12, defiance/hostility may take form of serious problem
Self-care, personal responsibility, chores, trustworthiness, independence, social relationships, academic performance
(8) ADHD in Adolescence & Adulthood
Many do not outgrow problems; some can get worse
At least 50% of clinic-referred elementary continue to suffer
Either outgrow/learn to cope
Boredom, work difficulties, impaired social relation, depression, low self-concept, substance abuse
(8) Genetic risk factors
If parent has ADHD, child has 60% chance of having ADHD
75% heritability estimates in twins
Genes may contribute to expression of ADHD
Focus specifically in dopamine regulation, psychomotor activity, sensation seeking
(8) Prenatal risk factors in ASD
Mother’s use of cigarettes, alcohol, or other drugs during pregnancy
(8) Association with frontostriatal circuitry implication
Affects attention, executive function, delayed responding, and response organization
(8) Association with Dopamine and Norepinephrine
Deficiency in the availability of dopamine and norepinephrine