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Primary intention
separation is small
bridge of cells binds ends of wounds together
minor wounds, sutures
secondary intentions
separation is large
tissue must fill space
more scarring, longer healing time
what is considered a normal, physiological response to trauma
inflammation
what is the role of phagocytosis
body ridding itself of debris, foreign material, dying tissue so it can heal itself
what is the goal of phase 1 (inflammation)
to control active inflammation and limit scope of original injury
what can too much inflammation lead to
secondary cell death
for phase 1 (inflammation phase) , what is responsible for clearing debris and bacteria
macrophages
is the lymphatic system active during phase 1 inflammation
it is passive, why elevation is encouraged
what is secondary damage/cell death
damage due to inflammation
what is happening during phase 2 of healing
tissue formation, re-epithelialization, capillarization
how long can phase 2 of healing usually last for?
2-4 weeks
what is the goal of phase 2 of healing
very active phase (controlled movement) and encourage repair
what is considered the longest phase of the healing process
maturation phase (phase 3)
Is chronic inflammation (possible phase 4) beneficial?
no, maturation phase (phase 3) can't normally progress to full recovery
chronic inflammation (possible phase 4)
simultaneous progression of active inflammation, tissue destruction, and healing
what are some systemic factors influencing healing
age, infection or disease, metabolic status, nutrition, hormones, medication, fever, oxygen
what are some local factors influencing healing
type, size, and location of injury
infection
vascular supply
movement/excessive pressure
what is a chronic lesion
repetitive/micro trauma that leads to segment alignment problems and developmental deficiencies
what are acute lesion injuries
high energy trauma and mechanic orgin
what kind of origin is chronic injury, micro-trauma considered?
mechanic origin
avascular necrosis is what type of origin
vascular origin
is a tendon supposed to have a lot of vasculature
no, if so we will see a decrease in strength
how may a tendon become increasingly vascularized
chronic injury
what is the main issue with an open fracture
the high risk of infection
greenstick fracture
bending and incomplete break of a bone; most often seen in children
comminuted fracture
fracture in which the bone is splintered or crushed
what are the stages of fracture healing
inflammation (0-2 wks)
callus formation (2-3 wks)
union (4-6 wks)
consolidation (6-8 wks)
remodeling (1-2 years)
stage 1 treatment of osteoarthritis
conservative approach ~ PT focused
stage 2 treatment of osteoarthritis
pharmacologic non-invasive approach + PT
stage 3 treatment of osteoarthritis
pharmacologic invasive approach (steroid injections)
stage 4 treatment of osteoarthritis
surgery
tendonitis is considered
repetitive trauma to tendons
tendinosis is considered
chronic microtrauma to tendons ~ degenerative disease of tendon's collagen
what is the best kind of exercise for tendinosis and tendonitis
eccentric exercises
chronic disuse disease of muscle
atrophy
metabolic disease of muscles include
cachexia and duchene
cachexia
a condition of physical wasting away due to the loss of weight and muscle mass that occurs in patients with diseases such as advanced cancer or AIDS
spastic/flaccid muscles could be due to...
neurologic disease
contracture
tightening of muscles, tendon, skin, etc
compartment syndrome
increased pressure within a muscle compartment