Neuro 670 test 2 pt 1

autoimmune neuro syndromes

Lupus, MS, myasthenia gravis

idiopathic and neurotransmitter disorders

epilepsy, delirium, mood disorders

infectious and deficiency neuro disorders

meningitis, lymes, prion

these deficiencies are associated with neuro disorders

thiamine, vitamin C

defining aspect of idiopathic & neurotransmitter disorders

initially there’s no IDed structural damage

dysphagia

difficulty swallowing

paralytic dysphagia

LMN lesion, decreased motor control and sensation

pseudobulbar dysphagia

UMN lesion, change in tone, poor coordination

mechanical dysphagia

structural change

CN V damage

weak chewing/bolus manipulation, low facial sensation

CN VII damage

loss of taste, poor mouth closure

CN IX damage

loss of taste, poor swallow reflex, low sensation in back of mouth

CN X damage

larynx, pharynx, poor closure of airway

CN XI damage

poor protection of airway

CN XII damage

poor tongue control

demyelinating disorder, slow progression, no cure

MS

relapsing remitting MS

relapses w/ recovery and stability periods

secondary progressive MS

follows relapse-remit

primary progressive MS

continuous decline

progressive relapsing MS

least common, progressive decline w/ large relapses

MS symptoms, besides pain, weakness, and fatigue

heat sensitivity, spasticity, ataxia, intention tremor, dysphagia

ALS

UMN/LMN destroyed, post-diagnosis lifespan 2-5 years, 6 stages

ALS symptoms

UMN: asthenia, spasticity, hyperreflexia

LMN: fasticulation, cramps, atrophy, speech, swallowing, breathing

GBS

demyelinating, rapid, LMN, very recoverable

phases of GBS

1. acute inflammatory 2. plateau 3. progressive recovery

MG

autoimmune, disruption to AcH receptor sites, fluctuating weakness, trunk strength preserved, visual impairments

seizure disorders are classed as

idiopathic

OT intervention for seizure disorders

med management, safety plans, IEPs, community mobility

transverse myelitis

inflammation of SC, interventions mimic SC injury

acoustic neuroma

benign tumor on vestibular N

Traditional Neuro development theory

reflex theory: sensory driven, primitive reflexes

Rood

sensory input drives motor response, develop in predetermined sequence

tap, stretch, light touch, temperature create motor development

Rood interventions

Bobath

inhibit abnormal movement patterns, postural control=foundation

physical handling, we learn thru sensation not doing

Bobath interventions

Brunnstrom

sequential movement, synergies, more adaptive techniques

reinforce synergy, controlled movement, adaptive techniques

Brunnstrom interventions

proprioceptive NM facilitation

diagonal patterns, integrate both sides

CNS is viewed as

heterarchical (dynamic)

UMN

CNS, come from brain

LMN

peripheral