CARE: SKIN INTEGRITY (PRESSURE INJURIES AND CONTACT DERMATITIS)

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Last updated 4:34 PM on 9/28/26
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92 Terms

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pressure injury

Skin and/or underlying tissue damage caused by prolonged pressure or pressure combined with shear.

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commonsites for pressure injuries

Sacrum

Heels

Trochanters

Ischial tuberosities

Elbows

Occiput

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what may pressure wounds present as

intact skin, an open wound, deep discoloration, pain, or device-shaped injury.

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when are pressure injuries preventable

when risk is identified early and prevention is consistent

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pressure

Tissue compressed between bone and surface

  • EX: sacral injury from bedrest


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nursing prevention for pressure

Reposition, offload, support surfaces

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shear

Skin stays in place while deeper tissue slides

  • EX: sliding down in bed with HOB elevated


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nursing prevention for shear

Lift, don’t drag; limit HOB when appropriate

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friction

skin rubs against surface

  • elbows/heels rubbing on linens


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nursing preventions for friction

Protective dressings, smooth linens, careful transfers

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moisture

Skin becomes overhydrated and fragile

  • EX: incontinence- associated dermatitis


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nursing prevention for moisture

Cleanse, dry, barrier cream, toileting plan

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who is vulnerable for pressure injuries

Immobility: unable to shift weight

Poor perfusion : PVD, shock, diabetes

Altered sensation: spinal cord injury, neuropathy

Moisture: incontinence, diaphoresis

Poor nutrition: low protein/calories, dehydration

Age/frailty: thin skin, reduced reserve

Medical devices: oxygen tubing, masks, casts

Medications: sedatives, vasopressors, steroids

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what does the braden scale do

Identifies risk level and guides prevention bundles for pressure injuroes

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for the braden scale the lower the score

the higher the risk

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braden scale categories

Sensory perception

Moisture

Activity

Mobility

Nutrition

Friction/shear

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what does the braden score not replace

Focused skin assessment

Clinical judgment

Device checks

Patient-specific interventions

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when is a risk score useful

only if it triggers appropriate prevention

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wound assessment components

Location: Anatomic site; relationship to bony prominence or device

Size: Length × width × depth in cm

Wound bed: Granulation, epithelialization, slough, eschar

Drainage: Amount, color, consistency, odor

Edges/margins: Attached, rolled/epibole, undermining, maceration

Periwound skin: Erythema, warmth, induration, moisture, pain

Tunneling/undermining: Clock method and depth

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wound assessment what to look for

Persistent color change

Purple, blue, gray, or ashen tone

Localized shiny or taut skin

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wound assessment what should you feel

Temperature difference

Boggy or firm tissue

Swelling or induration

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what should you ask the patient during a wound assessment

Pain

Burning

Tingling

Change in sensation

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Stage 1 pressure ulcer key finding

Nonblanchable erythema of intact skin

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stage 1 pressure ulcer what you see

Skin intact; may show temperature, sensation, or firmness changes

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Stage 2 pressure ulcer key finding

Partial-thickness skin loss with exposed dermis

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Stage 2 pressure ulcer what you see

Pink/red moist wound bed or serum-filled blister; no adipose tissue

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Stage 3 pressure ulcer key finding

full thickness skin loss

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Stage 3 pressure ulcer what you see

Adipose may be visible; slough/eschar may appear; no exposed bone/tendon/muscle

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Stage 4 pressure ulcer key finding

Full-thickness skin and tissue loss

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Stage 4 pressure ulcer what you see

Exposed or palpable fascia, muscle, tendon, ligament, cartilage, or bone

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unstageable pressure ulcer key finding

depth is obscured

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unstageable pressure ulcer what you see

Slough or eschar prevents visualization of wound base

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deep tissue pressure injury key finding

Persistent deep red/maroon/purple discoloration

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deep tissue pressure injury what you see

Intact or non-intact skin; damage begins in deeper tissue

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pressure injury staging decision tree

knowt flashcard image
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Medical Device-Related Pressure injury

Caused by diagnostic or therapeutic devices.

Shape often matches device.

Stage using usual staging system

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mucosal membrane pressure injury

Occurs on mucous membranes from device use.

Examples: ET tube, NG tube, urinary catheter.

Cannot be staged

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STOP Stable Heel Eschar

Dry, adherent, intact eschar on ischemic heel/limb is usually left intact unless ordered otherwise.

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device related injuries are

preventable

  • inspect under and around devices


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Stage 1 Pressure Injury: Non- Blanchable Erythema of Intact Skin (NPIAP)

• Intact skin with a localized area of non-blanchable erythema.

• May be preceded by blanchable erythema or by changes in sensation, temperature, or firmness

• Does not include purple or maroon discoloration

• May appear differently in darkly pigmented skin.

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Stage 2 Pressure Injury: Partial-Thickness Skin Loss with Exposed Dermis (NPIAP)

• Partial thickness loss of skin with exposed dermis

• Wound bed is pink or red and moist

• May occur as an intact or ruptured serum filled blister

• Adipose or deeper tissues are not visible

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Stage 3 Pressure Injury: Full-Thickness Skin Loss (NPIAP)

• Full thickness loss of skin

• Granulation tissue and epibole often present

• Slough and or eschar may visible

• May be undermining or tunneling

• Depth varies by anatomic location and degree of adiposity

  • The injury can extend down to, but not through underlying fascia; there is no exposed muscle or bone.


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Stage 4 Pressure Injury: Full-Thickness Skin and Tissue Loss (NPIAP)

• Exposed or directly palpable fascia, muscle, tendon, ligament, cartilage, or bone in the wound.

• Slough and or eschar may be visible

• Epibole, undermining, and or tunneling may occur

• Depth varies by anatomic location

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Pressure Injury Prevention Bundle

Risk assess: Braden + clinical judgment

Inspect skin: head-to-toe + under devices

Reposition: turn schedule; offload heels

Manage moisture: cleanse, dry, barrier product

Reduce shear: lift, don't drag; HOB mindful

Support surfaces: mattress/chair cushions

Nutrition: protein, calories, hydration

Mobilize: ambulate/ROM as able

  • These preventions work best as a bundle not individual interventions


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What not to do to a pressure injury

✕ Do not massage bony prominences: May damage fragile tissue.

✕ Do not drag patients across linens: Increases friction and shear.

✕ Do not use donut cushions: Can concentrate pressure.

✕ Do not leave heels resting on bed: Offload heels completely.

✕ Do not ignore device pressure: Assess under tubing, masks, braces, collars.

✕ Do not remove stable heel eschar: Unless ordered/clinically indicated

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why is protein and calories needed for for pressure injuries

Needed for tissue repair, immune function, and collagen synthesis.

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what is hydration needed for pressure injuries

Supports perfusion and skin turgor

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what to monitor during hydration

Monitor intake, output, and contraindications

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what micronutrients are needed for pressure injuries

Vitamin C, zinc, copper, vitamin A may be considered when deficient or ordered.

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when is wound healing impaired

when perfusion, nutrition, or infection control is inadequate

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wound care principles: think TIME

TIME

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TIME stand for

Tissue

infection/inflammation

mositure balance

edge

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tissue clincial question

Is nonviable tissue present?

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tissue nursing action

Debridement when appropriate; protect stable heel eschar

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infection/inflammation clinical question

Are there local or systemic infection cues?

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infection/inflammation nursing action

Culture/antibiotics if ordered; report fever, odor, purulent drainage

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moisture balance clinical question

Is the wound too dry or too wet?

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moisture balance nursing action

Select dressing to maintain moist healing and protect periwound

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Edge clincial question

are edges advancing

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edge nursing action

Monitor size/depth, epibole, undermining; consult WOC nurse

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dry wound bed dressing goal

Add/maintain moisture as ordered

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heavy exudate wound dressing goal

Absorb drainage and protect periwound skin

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Fragile surrounding skin dressing

Use gentle adhesive or non-adhesive options

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tunneling/dead space wound goal

Fill dead space without overpacking

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infection concern dressing

Assess/report; antimicrobial dressing may be ordered

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high risk bony area dressing

Protective foam may reduce friction/shear

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When to consult WOC nursing

Unstageable or deep tissue pressure injury

Stage 3 or Stage 4 pressure injury

Rapidly worsening wound

Complex dressing needs

Heavy exudate, odor, tunneling, or undermining

Concern for infection or delayed healing

Medical device-related injury

Need for patient/family education on wound care

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team members for WOC nursing

WOC nurse, provider, dietitian, PT/OT, case management, patient/family

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WOC nursing prioirty

Escalate early. Waiting often increases wound severity and cost

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Complications of Pressure Injury

Local infection: increased pain, erythema, drainage, odor

Cellulitis: spreading warmth/redness/swelling

Osteomyelitis: bone involvement, delayed healing

Sepsis: fever, tachycardia, hypotension, altered LOC

Pain/QOL: sleep disruption, mobility limits, distress

Cost/LOS: longer hospitalization and more intensive care

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systemic infection cues from pressure injuires require

prompt escalation

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Irritant Contact Dermatitis: mechanism

Direct skin damage from irritant

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Irritant Contact Dermatitis: common triggers

Soaps, detergents, chemicals, frequent hand hygiene

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Irritant Contact Dermatitis: timing

Can occur quickly after exposure

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Irritant Contact Dermatitis: symptoms

Burning, pain, redness, dryness/fissures

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Irritant Contact Dermatitis: mangament

Remove irritant, protect barrier, moisturize

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Allergic Contact Dermatitis: mechanism

Delayed immune hypersensitivity reaction

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Allergic Contact Dermatitis: commn triggers

Latex, nickel, poison ivy, adhesives, fragrances

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Allergic Contact Dermatitis: timing

Often delayed 24–72 hr after exposure

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Allergic Contact Dermatitis: symtoms

Intense itching, erythema, vesicles, oozing/crusting

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Allergic Contact Dermatitis: management

Avoid allergen, topical steroids if ordered, antihistamines for itching

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what are you doing when doing an assessment of contact dermitisis

find the exposure pattern

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contact dermatitis history

New soaps, detergents, gloves, plants, metals, adhesives, workplace chemicals, skin products.

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contact dermatitis distribution

Where is the rash?

Does it match a device, jewelry, glove, plant exposure, or topical product

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contact dermatitis complications

Scratching can lead to secondary infection.

Assess for drainage, warmth, fever,

spreading erythema.

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Contact Dermatitis Management

remove

relieve

treat

protect

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Contact Dermatitis Management: remove

Stop exposure to irritant/allergen

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Contact Dermatitis Management: relieve

Cool wet compresses, reduce itching

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Contact Dermatitis Management: treat

Topical corticosteroid or antihistamine if ordered

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Contact Dermatitis Management: protect

Barrier creams, gloves, trigger avoidance

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contact dermatitis patient teaching

Avoid scratching. Keep nails short. Report infection signs.

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Contact Dermatitis: safety→ allergy

Latex allergy requires clear documentation and latex-free supplies.