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conducting zone
what structures doe is it include
function?
trachea, bronchi, bronchioles, & terminal bronchioles
Transports, warms, humidifies, and filters air
NO gas exchange
transitional ands respiratory zone
structures & function
alveolar ducts and sacs & respiratory bronchioles
Performs the actual exchange of oxygen and carbon dioxide between the air and the blood
steps of pulmonary circulation
1. Right ventricle → pumps deoxygenated blood to pulmonary artery.
2. Pulmonary arteries → carry deoxygenated blood to the lungs.
3. Lungs → blood releases CO₂ and picks up O₂.
4. Pulmonary veins → carry oxygenated blood back to the heart to left atrium.
5. Left atrium → receives the oxygenated blood, which then moves into the left ventricle for systemic circulation

Atelectasis
what is it?
most common after ?
how does anesthesia and pain affect ?
other factors
what V/Q imbalance?
Collapse or incomplete expansion of alveoli, reducing the surface area available for gas exchange
Most common postoperative pulmonary complication, especially after thoracic or upper abdominal surgery
anesthesia - slows breathing
pain after surgery - limits deep breathing and ambulation
pts also have mucus blocking airways and surfactant impairment
perfusion w/o ventilation
s/s of atelectasis
dyspnea + respiratory distress w/o lobar involvement
cough with or without sputum
tachypnea , tachycardia & anxiety
pleural pain
subtle AMS - due to lack of oxygen (early sign)
decreased chest expansion on affected side
cyanois (late sign)
increased WOB
fever r/t inflammation or infectious processes
when we listen to lungs on a pt w/ atelectatsis
when we percuss ?
we can hear crackles on inspiration when alveoli tries to open
diminished or absent breath sound
dullness to percussion
risks factorsof atelectasis
general anesthesia,
immobility,
pain limiting deep breathing/coughing,
obesity,
smoking history,
pre-existing lung disease
what diagnostic tests confirm atelectasis
Pulse ox Low
Chest xray
Ct scan
ABG (low PO2 and high CO2)
prevention & managing (ICOUGH) +3 more
Incentive spirometry
•Coughing (controlled with splinting) and deep breathing
•Oral care (brushing teeth and using mouthwash twice a day)
•Understanding (patient and staff education)
•Getting out of bed at least three times daily do AROM & q2hr turns
•Head-of-bed elevation + hydration (thins secretions)
manage pain
give bronchodilators & mucolytics as ordered
chest physiotherapy
how to use incentive spirometer and how often
10 breaths every hour they are awake
sit up
exhale, create seal around mouthpiece
Breathe in as slowly and deeply as you can through your mouth to raise the piston
When you reach your highest possible breath, hold it for 3 to 5 seconds while the piston slowly falls.
breathe out normally
for more severe atelectasis
2 procedures
3 devices
may require bronchoscopy to remove obstructive mucus plugs
thoracentesis for removing plueral fluid
CPAP
endotracheal intubation & mechanical vent
PEEP(positive end expiratory pressure ) - holds the alveoli open during exhalation, stopping them from collapsing
thoracentesis
what is it
purpose
N/C (needs what before? monitor ? document ? before any exercise?)
Removal of fluid in the pleural space via needle aspiration
can be therapeutic or diagnostic
needs consent/ educate and prep
monitor for any bleeding at the site and BP
document drainage & look at dressing
pre-medicate before exercises
Acute Tracheobronchitis
what is it
often after 2
Acute inflammation of the mucous membranes of the trachea and bronchial tree
often follows URI or inhalation of chemical irritants (smoke, pollen, asbestos, dust, fiber glass)
s/s of Acute Tracheobronchitis
how does cough change as disease progresses
other s/s
initially dry/ irritating / hacking cough
as disease progresses - increased sputum
chest pain form dry cough
dyspnea from inflammation (narrowing airway)
fever, chills, headache , night sweats, fatigue
medical management of Acute Tracheobronchitis
what Is not recommended
antibiotic if bacterial
analgesics + antipyretics
fluid to thin secretions
cool vapor or steam
DO not use antihistamines - dries secretions , harder to clear
Nursing care of tracheobronchitis
encourage ? 2
allow?
monitor for
bronchial hygiene - coughing + deep breathing
encourage fluids + proper nutrition
allow rest
monitor for complications in high risk patients
Respiratory Syncytial Virus (RSV)
what is it? cause ?
occurs when ?
most common cause of
transmitted ? precautions ?
reportable?
Acute lower respiratory tract infection, usually caused by a virus
Occurs in annual epidemics: October–March
most common cause of bronchiolitis.
transmitted through direct contact with respiratory secretions and contaminated surfaces
Contact + Droplet Precaution (mask, gown, gloves & goggles)
Outbreaks or unusual clusters – reportable to DOH
what increases the risk of severe RSV/bronchiolitis, hospitalization, and respiratory complications
immunosuppression,
low birth weight,
lung disease,
severe neuromuscular disease
congenital heart defects
s/s of RSV/ bronchitis mild
rhinitis
coughing
low grade fever
wheezing
tachycardia
vomiting & diarrhea
s/s of RSV/ bronchitis severe
tachypnea >70 (peds)
grunting - severe distress
wheezing
retractions
cyanosis
nasal flaring
lethargy - poor feeding + exhaustion (late sign)
diagnostic tests of RSV/ bronchitis
nasal/nasopharyngeal swab - confirmed with Enzyme linked immunosorbent assay (ELISA) or immunofluorescent assay (IFA)
managing RSV/ bronchitis
5 resp
symptom management ?
never use
Oxygen – humidified with or without heat
Nebulized saline
May need mechanical ventilation
Suctioning
ABG monitoring
Fluids, antipyretics, Rest - symptom management
never use aspirin in children ! - reyes syndrome
RSV vaccine
only given to
what kind of immunity
palivizumab –Synagis
only given to high risk infants
provides passive immunity
Ribavirin
what is needed with it
aerosolized/inhaled via nebulizer - rarely used, reserved for severe RSV
Small-Particle Aerosol Generator (SPAG) needed if administering Ribavirin. Keeps it out of the air!
what is pneumonia
lobar vs bronchopneumonia
which is more common
aspiration pneumonia
Inflammation/infection of the lung parenchyma/alveoli caused by bacteria, viruses or fungi
reportable to DOH
Lobar pneumonia refers to the involvement of part or entire lobe
Bronchopneumonia refers to spots of alveoli affected throughout the bronchial tree (more common)
aspiration pneumonia -
Inhalation of oropharyngeal or gastric contents into the lower airway
pathophysiology of pneumonia
what kind of V/Q
Pathogens infiltrate the normally sterile lower respiratory tract via inhalation, aspiration, or bloodstream
Spreads and triggers an inflammatory response
Alveoli fill with fluid and exudate (consolidation), impairing gas exchange
Results in perfusion-without-adequate-ventilation problem
LOOK at Table 19-2 for risk factors and preventative measures
Community-acquired pneumonia (CAP)
Develops outside the healthcare setting or within 48 hours of admission
Hospital-acquired pneumonia (HAP)
Develops 48+ hours after admission, in a patient not on mechanical ventilation
Ventilator-associated pneumonia (VAP)
could be from
Develops 48+ hours after endotracheal intubation
from lack of oral care or suctioning, not being turned and positioned frequently enough
not in semi fowlers, could be lying flat - not promoting lung expansion
Health care–associated pneumonia (HCAP)
Occurs in non-hospitalized patients with extensive health care contact
LTC / nursing home pts
Dialysis pts
Chemo pts
Home infusion therapy pts
antibiotic/ wound care pts
Hospitalization for ≥2 days in an acute care facility within 90 days of infection
N/C of Immunompromised pts (HIV, transplant, cancer pts) with pneumonia
Higher risk for opportunistic infections
May have a more severe or atypical presentation
Requires a high index of suspicion & closer monitoring
Early diagnosis and treatment are important
• Treatment depends on the suspected organism and severity
common risk factors of pneumonia (look at table 19-2) SALAMI
smoking
advanced age + altered level of consciousness
lung disease
alcohol use
malnutrition + mechanical vent
Immunocompromised + immobility
prevention of pnuemonia
smoking cessation,
hand hygiene,
early mobilization,
aspiration precautions (1:1 supervised feeding, no fluids with food, soft foods & thickened liquid)
oral care
who is required to take the pneumococcal vaccine
adults 65 years of age or older
19 years old & older with weak immune system, smoke cigarettes or have chronic heart, lung or liver disease or alcoholism
assessment findings when pt has pneumonia
auscultation ?
palpation?
percussion ?
LOC?
crackles or diminished/ absent lung sounds over affected area or wheezing
Increased tactile fremitus
Dullness to percussion
Older adults may present with confusion/delerium/falls or functional decline instead of classic fever and cough
other s/s of pnuemonia
varies with the type of pneumonia causal agent and presence of underlying disease, but generally:
Cough - (dry/productive, sputum: purulent, rust colored)
Headache, fever, chills, fatigue
pleuritic chest pain
tachypnea
Use of accessory muscles,
↓ appetite
diagnostic tests for pneumonia
4 tests
what are invasive ways can we get sputum
who would we use bronchoscopy on
CXR,
blood culture
sputum examination - (first thing in morning )
• Invasive procedures may be used for specimen collection
• Nasotracheal/orotracheal suctioning, fiberoptic bronchoscopy
Bronchoscopy: for severe chronic, reoccurring infections, compromised immune systems, and mechanically ventilated patient
risk facts for pneumonia for pediatric pts (0-12)
RSV/viruses & Strep pneumoniae (younger)
Mycoplasma ("walking pneumonia" - child still functioning) more common school-age
Prematurity/low birth weight,
incomplete vaccinations
aspiration,
recent viral URI,
smoke exposure,
daycare/crowded settings,
malnutrition
underlying heart, lung, or immune disorders
risk facts for pneumonia for adolescent/ adult pts
Strep pneumoniae (most common CAP)
Mycoplasma, Chlamydophila
Smoking, vaping
alcohol use - suppresses immune system and interferes with gag reflex
close-contact living
COPD
immunosuppression
risk facts for pneumonia for older adult pts
Strep pneumoniae, H. influenzae, gram-negatives,
aspiration pneumonia
dysphagia,
immobility,
↓ cough reflex,
comorbidities,
institutional living
how can we prevent VAP
position
routine ?
based on assessment
spontaneously ?
monitor ?
semi fowlers
routine oral care with chlorhexidine at least 1x a day or whenever you see secretions
suctioning when you assess adventitious lung sounds or see secretions
wake your pt spontaneously daily
spontaneous breathing trial - see if pt can breathe on their own and be extubated
monitor cuff presssure on ETT
complications of pneumonia
atelectasis
plural effusion - excess fluid in plural space
empyema - pus in plural space
lung abcess
sepsis
shock
respiratory failure
covid
precautions?
similar to ? unique symptoms
tx? meds? position?
can lead to
airborne, droplet and contact
similar to pneumonia with symptoms like ageusia (loss of taste) & anosmia (loss of smell)
tx with antivirals, steroids, o2, vent, prone position
may lead to shock and resp. failure
SARS - severe acute respiratory syndrome
type of ?
spread via?
symptoms?
clinical manifestations?
protocol ?
type of corona virus
spread via droplet and contact
contagious when symptomatic
high fever, headache , myalgia, diarrhea, cough
progressive hypoxia → pneumonia
same protocol as pneumonia
transmission of TB
what PPE
what kind of room
if pat has to leave room
what kind of care
airborne and droplet
need N95 mask
pt needs negative pressure room with 6 air exchanges per hour
if pt has to leave room they need surgical mask
clustered care is important - limit exposure to the disease
risk factors of TB - TB RISK +2
tight living quarters / institutional living (LTC, prison, psych facility)
below or at poverty line (homeless, lack of healthcare)
Refugee/ immigrant
immune system suppression (HIV, transplant, cancer, corticosteroids use)
Substance abuse (IV drug use, alcohol abuse)
Kids under 5 - immune system under developed
comorbidities - diabetes, chronic kidney disease, malnourishment
health care workers
s/s pf active TB
early vs late ?
may have ?
insidious - slow and subtle but deadly
early - low grade fever, cough, night sweats, fatigue and weight loss
may have hemoptysis - coughing up blood
late - high fever, chest pain , diminished bronchial sounds from consolidation, crackles
Mantoux skin test
what is it
read after ?
what are we looking for ?
positive result ? for immunocompromised?
considerations?
0.1 mL intradermal injection under skin
read after 48-72 hours
interpret induration & erythema (both are needed for a positive result)
for immunocompromised pt - 5mm or more = positive ACTIVE TB
10mm or more = positive ACTIVE TB result
BCG (Bacillus Calmette Guerin) vaccine - Will always have a positive PPD - people born in foreign countries
what other dx tests are used to determine TB infection
Sputum analysis - first thing in morning, before brushing teeth and mouthwash or breakfast
Chest X-Ray - will show white lung tissue if positive
Blood test- Interferon Gamma Release Assay - QuantiFERON TB Gold & T-
SPOT
how do we treat TB
how long is the tx?
meds ?
vitamin ?
initial vs continuation phase ?
when is the pt non-infectious
instruct?
ensuring med compliance ?
anti-TB agents for 6-12 months
4 or more of the following meds
• R = Rifampin and Rifamate (rifampin & isoniazid)
• I = Isoniazid
• P = Pyrazinamide
• E = Ethambutol
vitamin B6 for neuropathy
initial phase - 1x a day for 8 weeks
continuation phase - lasts for 4-7 months depending on sputum tests
non-infectous after 2-3 weeks of consistent treatment (must be complaint to meds)
instruct pt to take meds on empty stomach
DOT - direct observed therapy to ensure med compliance
multidrug resistant TB
extensive drug resistant TB
what adds to it
multi-resistant : resistant to at least isoniazid and rifampin
extensive- resistant: resistant to isoniazid, rifampin, plus a fluoroquinolone and at least one injectable second-line drug
non compliance adds to drug resistance
Lung Abcess
what is it?
most are ?
what is usually the cause
Lesion of the lung parenchyma containing pus that collapses and forms a cavity of about 2 cm.
Most are a complication of bacterial pneumonia
Aspiration of bacterium is usually the cause
who is at risk for lung abscess
Immobilized patients - feed pts sitting up, to decrease r/o aspiration
CNS disorders - ensure oral care
Elderly - dental issues, inadequate clearing of bacteria
Tube fed patients -
Post-op
s/s of lung abscess
Productive cough with foul sputum
• Pleurisy with dull chest pain
Anorexia and weight loss
• Empyema
• Leukocytosis (increased WBC)/ fever/ fatigue
• Bronchopleural fistula
Lung Abscess- Diagnostics & Assessment Findings
lung sounds ?
what scans? what test?
what procedure
culture ?
• Decreased or absent breath sounds
• Pleural Friction Rub – grating sound, crackles
• Chest X-Ray shows a cavity with an air-fluid level/ ct scan
• Sputum culture identifies source
• May require bronchoscopy to dx or to treat
Management of Lung Abscess'
positioning?
meds
what do we encourage
support?
• Postural drainage and positioning – chest physiotherapy
• IV antimicrobial therapy, followed by PO
• Deep breathing and coughing exercises
• Nutritional support - weight loss common
Prevention of Lung Abscess
Turning and positioning-chest PT
• Deep breathing and coughing exercises
• Pre-dental antibiotic therapy for those with infected teeth/gums
• Appropriate antibiotic therapy for resp illnesses (based on culture)
Sarcoidosis
• Interstitial Lung disease of unknown cause
• Can involve any tissue or organ but mostly seen in the lung (pumlomary sarcoidosis) - also effects lymph nodes
pathophysiology of sarcodosis
Hypersensitive response to bacteria, fungus, viruses or chemicals
Hypersensitivity leads to Inflammation
inflammation leads to granuloma (collection of macrophages) formation on the walls of alveoli
over time leads to to fibrosis(scar tissue ), which decreases lung compliance
Granulomatous Disease
what it it
leads to ?
Granulomas can appear on the walls of the alveoli or on the walls of the bronchioles
Results in: Low lung compliance, impaired diffusion and decrease lung volume
s/s of sarcoidosis
Dyspnea, Cough, Hemoptysis, Congestion
Diagnostics - Sarcoidosis
• Chest X-Ray and CT scans show nodules in the lungs
• Transbronchial biopsy confirms diagnosis
• Pulmonary function test
• Arterial blood gases
meds for sarcoidosis
• Oral corticosteroids: tapered – can be up to 12 months
• Immune modulator: methotrexate