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Eye Disorders and Treatments
Eye Disorders and Treatments
Dry Eye
Symptoms:
Rainy, sandy, gritty feeling, typically worse as the day goes on.
Diagnosis:
Schirmer's Test:
Filter paper placed in the lower lid; normal results are 10-15 mm of wet paper after 5 minutes.
Drops:
Dye disappears from the lacrimal cul-de-sac within one minute is considered normal.
Medical Management:
Artificial Tears:
Over-the-counter, doctor can recommend specific ones.
Cyclosporine:
Increases tear production, reduces inflammation (takes 3-6 months for improvement).
Environmental Factors:
Dust, sprays, aerosols; advise filters.
Surgical Repair:
If other methods fail.
Ectropion and Entropion
Non-infectious disorders of eyelid margins turning abnormally.
Ectropion:
Outward turning (EGG - Out).
Causes: Bell's palsy, birth defects, eye lacerations, injury.
Entropion:
Inward Turning (IN - In).
Causes: Atrophy of eyelid tissue and muscle, congenital conditions, trauma.
Medical Management:
Topical medications to reduce inflammation; surgery is preferred.
Interventions:
Monitor treatment, report progress; early diagnosis is better.
Cataracts
Lens becomes cloudy or opaque, decreasing visual acuity.
Causes:
Aging, injury, congenital problems, diabetes, UV rays, corticosteroids, toxins, hypertension, smoking.
Clinical Manifestations:
Painless, blurred vision, difficulty reading fine print, double vision, photosensitivity, glare (worse at night), abnormal color perception, hazy/fuzzy vision.
Diagnostic Tests:
Slit lamp exam to see opaqueness.
Ophthalmoscope to look at the lens.
Treatment:
Change eyewear prescription.
Surgical repair: Intracapsular (entire lens removal - not typical), extracapsular (preferred).
Post-op Education:
Outpatient procedure.
Antibiotics and corticosteroid drops to prevent infection and decrease inflammation.
Avoid bending, stooping, coughing, lifting, straining (increase IOP).
Stool softeners and high-fiber diet recommended to avoid straining.
Use eye shield at night.
Notify doctor if continued irritation or pain.
Follow-up: Typically the next day.
Visual field is hazy and fuzzy.
Diabetic Retinopathy
Disorder of retinal blood vessels characterized by microaneurysms, hemorrhage, exudates, and new vessel formation.
Common after 15 years of diabetes.
Nearly all patients with type 1 and 80% with type 2 diabetes have some degree of retinal disease.
Incidence increases with duration of diabetes and poor control.
Initial stage can last several years.
Clinical Manifestations:
Microaneurysms, floaters, visual loss.
Diagnosis:
Ophthalmoscopy to see dilated and narrowed arteries.
Surgical intervention:
Photocoagulation: Laser beam to destroy new blood vessels, seal leaking vessels, and prevent retinal edema.
Cryotherapy. Uses a topical anesthetic.
Early detection is key
Age-Related Macular Degeneration (ARMD)
Slow progressive loss of central and near vision.
Genetic component and risk factors like UV light exposure, hyperopia, smoking, female gender, obesity, race, light-colored eyes.
Dietary supplementation with vitamins C, E, A, copper, and zinc will slow development of advancing ARMD.
Two types: Wet and Dry (dry is 90% of cases).
Clinical Manifestations:
Drusen (yellow exudate beneath the retinal pigment).
Gradual bilateral loss of central vision.
Trouble distinguishing colors.
Distorted vision.
Dark empty area in the center of vision.
Diagnosis:
Fluorescein angiography.
Treatment:
Injections of Avastin and Maccogen, and antibiotics to prevent infections.
Visual aids: Telescopic and microscopic lenses, magnifying glasses, electronic magnifiers.
Retinal Detachment
Separation of the retina from the choroid, often due to a hole in the retina.
May be caused by severe trauma to the eye.
Clinical Manifestations:
Initially painless (only pain with initial trauma).
Floaters and flashes of light.
Feeling of a curtain or heaviness over the eye.
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Nervous System
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Studied by 28 people
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