Understand the pathophysiology of left- and right-sided heart failure.
Define acute heart failure.
List causes of acute and chronic heart failure.
Identify signs and symptoms of acute and chronic heart failure.
Assist in planning nursing care for patients undergoing diagnostic tests for heart failure.
Explain medical treatments used for acute and chronic heart failure.
Plan nursing care and education for patients and their families with heart failure.
Definition: Heart failure (HF) is a syndrome resulting from the progressive inability of the heart to pump enough blood to meet the body’s oxygen and nutrient needs.
Older Term: Previously referred to as Congestive Heart Failure (CHF), as congestion is not always present.
Most Common Cause: Coronary artery disease.
Other Causes:
Cardiomyopathy
Hypertension (HTN)
Heart valve disorders
Myocardial infarction
Ventricular Function: Both ventricles pump equal amounts of blood. Impairment in one leads to an overall decrease in pump effectiveness.
Progression: Typically, the left ventricle weakens first which can lead to the failure of the right ventricle.
Afterload: The force generated by the left ventricle to eject blood into the aorta.
HTN increases afterload, causing additional strain on the left ventricle.
Consequences: Blood backs up into the lungs, causing alveolar edema and impairing gas exchange.
Paroxysmal nocturnal dyspnea
Elevated pulmonary capillary wedge pressure
Restlessness and confusion
Orthopnea (difficulty breathing when lying flat)
Pulmonary congestion indicated by:
Cough (sometimes blood-tinged)
Crackles and wheezes
Tachypnea (rapid breathing)
Tachycardia
Fatigue and cyanosis
Mainly results from left-sided heart failure.
Cor Pulmonale: Right ventricle hypertrophies and fails due to increased pressures in the pulmonary artery.
Backward Build-up: Causes peripheral edema due to increased venous pressure.
Fatigue
Increased peripheral venous pressure
Ascites and hepatosplenomegaly (enlarged liver and spleen)
Distended jugular veins
Anorexia and GI distress
Dependent edema (swelling in hands and fingers)
Cardiac Output: Low cardiac output triggers compensatory mechanisms:
Sympathetic nervous system activation increases heart rate.
Renin-angiotensin-aldosterone system causes fluid retention.
Heart chambers enlarge and muscle mass increases (hypertrophy).
However, these mechanisms ultimately worsen heart failure by increasing oxygen demands.
Definition: A severe condition with life-threatening fluid congestion in the alveoli.
Signs and Symptoms:
Classic presentation includes pink, frothy sputum.
Severe dyspnea (difficulty breathing) and rapid respirations using accessory muscles.
Crackles, wheezes, anxiety, and clammy skin.
Diagnosis:
Important diagnostic tools include arterial blood gases, pulmonary pressures, and chest X-ray.
Therapeutic Interventions:
Positioning (semi-Fowler or Fowler), oxygen therapy, diuretics, inotropic agents, and vasodilators.
Impaired gas exchange
Decreased cardiac output
Excess fluid volume
Acute pain
Definition: A progressive condition where signs and symptoms may worsen over time.
Signs and Symptoms:
Fatigue, exertional dyspnea, orthopnea, paroxysmal nocturnal dyspnea, cough.
Auscultation may reveal crackles/wheezes and tachycardia.
Patients may experience chest pain, Cheyne-Stokes respiration, edema, anemia, nocturia, cyanosis, altered mental status, and malnutrition.
Enlarged organs (hepatomegaly and splenomegaly)
Pleural effusion (fluid surrounding lungs)
Risk of thrombosis and emboli
Cardiogenic shock due to inadequate blood flow.
Screening Tests: Serum BNP, BUN, creatinine, liver function tests.
Diagnostic imaging: Chest X-ray, echocardiography, ECG, cardiac stress testing, catheterization, sleep studies.
Optimize heart function and reduce oxygen demand:
Identify and correct underlying causes,
Improve contraction strength,
Maintain fluid and sodium balance,
Decrease heart workload.
Oxygen therapy,
ACE inhibitors, ARBs, or angiotensin receptor neprilysin inhibitors,
Beta-blockers,
Diuretics,
Inotropic agents,
Vasodilators,
Ivabradine (to slow heart rate),
Anticoagulants.
Hypokalemia, hypomagnesemia, and hypercalcemia:
Symptoms include abdominal pain, nausea, vomiting, anorexia, visual changes, bradycardia.
Activity management, sodium and weight control, and device implantation (e.g., pacemakers, implantable cardioverter-defibrillators).
Coronary artery bypass grafting,
Valve replacement,
Ventricular reconstruction.
Impaired gas exchange,
Decreased cardiac output,
Acute pain,
Anxiety,
Excess fluid volume.
Administration of oxygen,
Guiding patient rest and activity levels,
Fluid management and education on lifestyle changes (low-sodium diet, weight control).
Patient: Mrs. Garcia, 72, diagnosed with HF 3 years ago, presents with dyspnea and bilateral ankle swelling related to missed diuretic doses.
Vital signs indicate hypoxic conditions affecting care plan and management strategies.
Indicated for end-stage heart failure, involving strict selection criteria, immunosuppressive therapy, and lifelong anti-rejection medications.
Risks include rejection, infection, malignancies, and side effects of anti-rejection medicines.